Page last updated: 2024-08-18

isomethyleugenol and Congenital Immunodeficiency Disease

isomethyleugenol has been researched along with Congenital Immunodeficiency Disease in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Cheng, C; Gao, WQ; Hou, J; Ji, Z; Li, L; Li, X; Liu, K; Miao, J; Shen, L; Sheng, Y; Sun, J; Wang, J; Wang, X; Xu, L; Yao, J; Zhang, K; Zhao, H; Zhou, W; Zhu, HH	1

Other Studies

1 other study(ies) available for isomethyleugenol and Congenital Immunodeficiency Disease

Article	Year
The histone methyltransferase Setd2 is indispensable for V(D)J recombination. Nature communications, 2019, 07-26, Volume: 10, Issue:1 Topics: Amino Acid Motifs; Animals; B-Lymphocytes; Cell Differentiation; Child, Preschool; Epigenesis, Genetic; Histone-Lysine N-Methyltransferase; Histones; Humans; Infant; Lysine; Male; Methylation; Mice; Mice, Inbred C57BL; Mice, Knockout; Mutation; Primary Immunodeficiency Diseases; Receptors, Antigen, T-Cell, alpha-beta; T-Lymphocytes; V(D)J Recombination	2019

Article

Year

The histone methyltransferase Setd2 is indispensable for V(D)J recombination.

Nature communications, 2019, 07-26, Volume: 10, Issue:1

Topics: Amino Acid Motifs; Animals; B-Lymphocytes; Cell Differentiation; Child, Preschool; Epigenesis, Genetic; Histone-Lysine N-Methyltransferase; Histones; Humans; Infant; Lysine; Male; Methylation; Mice; Mice, Inbred C57BL; Mice, Knockout; Mutation; Primary Immunodeficiency Diseases; Receptors, Antigen, T-Cell, alpha-beta; T-Lymphocytes; V(D)J Recombination

2019