Page last updated: 2024-08-18

isomethyleugenol and Cleft Spine

isomethyleugenol has been researched along with Cleft Spine in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (16.67)18.2507
2000's3 (50.00)29.6817
2010's2 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dai, XL; Jin, MH; Liu, GC; Mi, NN; Ren, XY; Wang, J; Wang, SQ; Zhang, J1
Chen, XR; Fan, Y; Li, L; Liu, QL; Qin, P; Wang, JX; Yang, HY; Zhang, D1
Barbaux, S; Doolin, MT; Hoess, K; McDonnell, M; Mitchell, LE; Whitehead, AS1
Burren, KA; Chitty, LS; Copp, AJ; Doudney, K; Dunlevy, LP; Greene, ND; Scott, R; Stanier, P; Stojilkovic-Mikic, T1
Eskes, TK1
Daskalakis, I; Hinkins, M; Lucock, M; Yates, Z1

Reviews

1 review(s) available for isomethyleugenol and Cleft Spine

ArticleYear
Neural tube defects, vitamins and homocysteine.
    European journal of pediatrics, 1998, Volume: 157 Suppl 2

    Topics: Animals; Female; Folic Acid; Homocysteine; Humans; Methionine; Methylation; Methylenetetrahydrofolate Reductase (NADPH2); Mutation; Neural Tube Defects; Oxidoreductases Acting on CH-NH Group Donors; Rats; Spinal Dysraphism

1998

Other Studies

5 other study(ies) available for isomethyleugenol and Cleft Spine

ArticleYear
An Inframe Trinucleotide Deletion in MTRR Exon 1 is Associated with the Risk of Spina Bifida.
    Neuromolecular medicine, 2017, Volume: 19, Issue:2-3

    Topics: 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase; Adult; Amino Acid Sequence; Cells, Cultured; Exons; Female; Ferredoxin-NADP Reductase; Folic Acid; Genetic Predisposition to Disease; Genotype; Homocysteine; Humans; Hyperhomocysteinemia; Infant, Newborn; Methylation; Models, Molecular; Pregnancy; Pregnancy Complications; Protein Conformation; Recombinant Proteins; Sequence Alignment; Sequence Deletion; Spinal Dysraphism; Young Adult

2017
Preliminary investigation of methylation status of microRNA-124a in spinal cords of rat fetuses with congenital spina bifida.
    The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians, 2017, Volume: 30, Issue:1

    Topics: Animals; Biomarkers; Blotting, Western; Case-Control Studies; Down-Regulation; Female; Gene Expression Regulation, Developmental; Immunohistochemistry; Male; Methylation; MicroRNAs; Rats; Rats, Sprague-Dawley; Real-Time Polymerase Chain Reaction; Spinal Cord; Spinal Dysraphism

2017
Maternal genetic effects, exerted by genes involved in homocysteine remethylation, influence the risk of spina bifida.
    American journal of human genetics, 2002, Volume: 71, Issue:5

    Topics: 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase; Algorithms; Computational Biology; Ferredoxin-NADP Reductase; Homocysteine; Linear Models; Methylation; Polymorphism, Single Nucleotide; Risk Factors; Spinal Dysraphism

2002
Abnormal folate metabolism in foetuses affected by neural tube defects.
    Brain : a journal of neurology, 2007, Volume: 130, Issue:Pt 4

    Topics: Anencephaly; Animals; Antimetabolites; Deoxyuridine; Female; Ferredoxin-NADP Reductase; Fetal Diseases; Fetus; Fibroblasts; Folic Acid; Genotype; Humans; Methylation; Mice; Neural Tube Defects; NIH 3T3 Cells; Polymorphism, Genetic; Pregnancy; S-Adenosylhomocysteine; S-Adenosylmethionine; Spinal Dysraphism

2007
An examination of polymorphic genes and folate metabolism in mothers affected by a spina bifida pregnancy.
    Molecular genetics and metabolism, 2001, Volume: 73, Issue:4

    Topics: Adult; Alleles; England; Erythrocytes; Female; Folic Acid; Gene Frequency; Humans; Methionine; Methylation; Mutation; Polyglutamic Acid; Polymorphism, Genetic; Polymorphism, Single Nucleotide; Pregnancy; Pteroylpolyglutamic Acids; Spinal Dysraphism; Vitamin B 12

2001