Page last updated: 2024-08-18

isomethyleugenol and Chromosomal Triplication

isomethyleugenol has been researched along with Chromosomal Triplication in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19901 (25.00)18.7374
1990's2 (50.00)18.2507
2000's0 (0.00)29.6817
2010's1 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Benedetti, D; Bomben, R; Caldana, C; D'Arena, G; Dal Bo, M; Del Poeta, G; Di Raimondo, F; Gaidano, G; Gattei, V; Hartmann, TN; Hutterer, E; Pozzato, G; Pozzo, F; Rossi, D; Rossi, FM; Tissino, E; Zaja, F; Zucchetto, A1
Anisowicz, A; Gadi, IK; Harrison, JJ; Raffeld, M; Sager, R1
Bliek, J; Boavida, M; Coad, N; Feinberg, AP; Hoovers, JM; Little, P; Mannens, M; Redeker, E; Steenman, M; Verjaal, M1
Artifoni, L; Aviv, H; Baccichetti, C; Bernasconi, F; Franzoni, E; Robinson, WP; Schinzel, AA; Shih, LY; Suslak, L; Wagstaff, J1

Other Studies

4 other study(ies) available for isomethyleugenol and Chromosomal Triplication

ArticleYear
CD49d is overexpressed by trisomy 12 chronic lymphocytic leukemia cells: evidence for a methylation-dependent regulation mechanism.
    Blood, 2013, Nov-07, Volume: 122, Issue:19

    Topics: Azacitidine; B-Lymphocytes; Cell Proliferation; Cells, Cultured; Chromosomes, Human, Pair 12; Decitabine; Disease Progression; Gene Expression Regulation, Leukemic; Humans; Integrin alpha4; Leukemia, Lymphocytic, Chronic, B-Cell; Methylation; Sequence Analysis, DNA; Signal Transduction; Survival Analysis; Trisomy

2013
Azacytidine-induced tumorigenesis of CHEF/18 cells: correlated DNA methylation and chromosome changes.
    Proceedings of the National Academy of Sciences of the United States of America, 1983, Volume: 80, Issue:21

    Topics: Adipose Tissue; Animals; Azacitidine; Cell Line; Chromosome Aberrations; Chromosome Disorders; Cricetinae; DNA, Neoplasm; Karyotyping; Methylation; Mice; Mice, Nude; Neoplasms, Experimental; Trisomy

1983
Parental imprinting of human chromosome region 11p15.3-pter involved in the Beckwith-Wiedemann syndrome and various human neoplasia.
    European journal of human genetics : EJHG, 1994, Volume: 2, Issue:1

    Topics: Beckwith-Wiedemann Syndrome; Child; Chromosome Aberrations; Chromosome Mapping; Chromosomes, Human, Pair 11; DNA; Female; Gene Expression; Genes, Tumor Suppressor; Humans; In Situ Hybridization, Fluorescence; Insulin; Insulin-Like Growth Factor II; Karyotyping; Male; Methylation; Neoplastic Syndromes, Hereditary; Parents; Pedigree; Polymorphism, Restriction Fragment Length; Sex Factors; Translocation, Genetic; Trisomy; Wilms Tumor

1994
Uniparental disomy explains the occurrence of the Angelman or Prader-Willi syndrome in patients with an additional small inv dup(15) chromosome.
    Journal of medical genetics, 1993, Volume: 30, Issue:9

    Topics: Angelman Syndrome; Child; Child, Preschool; Chromosome Deletion; Chromosome Inversion; Chromosomes, Human, Pair 15; Fathers; Humans; Karyotyping; Male; Methylation; Mothers; Prader-Willi Syndrome; Trisomy

1993