Page last updated: 2024-08-18

isomethyleugenol and Celiac Disease

isomethyleugenol has been researched along with Celiac Disease in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19901 (33.33)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Bhagat, G; Bilbao, JR; Castellanos-Rubio, A; Garcia-Santisteban, I; Godbout, JK; Green, PH; He, C; Herrero, L; Huerta, A; Irastorza, I; Mendoza, LM; Mera, P; Olazagoitia-Garmendia, A; Rodriguez, JA; Sebastian-DelaCruz, M; Serra, D; Verdu, EF; Zhang, L1
Kaikkonen, MU; Kurppa, K; Lindfors, K; Mäki, M; Oittinen, M; Popp, A; Viiri, K1
Kim, YS; Spritz, N1

Other Studies

3 other study(ies) available for isomethyleugenol and Celiac Disease

ArticleYear
Gluten-induced RNA methylation changes regulate intestinal inflammation via allele-specific
    Gut, 2022, Volume: 71, Issue:1

    Topics: Adenosine; Animals; Celiac Disease; Cell Line; Disease Models, Animal; Epithelial Cells; Exportin 1 Protein; HLA-DQ Antigens; Humans; Intestinal Mucosa; Karyopherins; Methylation; Mice, Knockout; NF-kappa B; Polymorphism, Single Nucleotide; Receptors, Cytoplasmic and Nuclear; RNA

2022
Polycomb Repressive Complex 2 Enacts Wnt Signaling in Intestinal Homeostasis and Contributes to the Instigation of Stemness in Diseases Entailing Epithelial Hyperplasia or Neoplasia.
    Stem cells (Dayton, Ohio), 2017, Volume: 35, Issue:2

    Topics: Animals; Celiac Disease; Cell Differentiation; Cell Proliferation; Enterocytes; Gene Expression Regulation, Neoplastic; Glutens; Histones; Homeostasis; Humans; Hyperplasia; Intestinal Mucosa; Intestinal Neoplasms; Lysine; Methylation; Mice, Inbred C57BL; Models, Biological; Polycomb Repressive Complex 2; Stem Cell Niche; Stem Cells; Wnt Signaling Pathway

2017
Hydroxy acid excretion in steatorrhea of pancreatic and nonpancreatic origin.
    The New England journal of medicine, 1968, 12-26, Volume: 279, Issue:26

    Topics: Celiac Disease; Chromatography; Fatty Acids; Fatty Acids, Nonesterified; Feces; Humans; Lipids; Methods; Methylation; Pancreatic Diseases; Stearic Acids

1968