isoflurophate has been researched along with Seizures in 43 studies
Isoflurophate: A di-isopropyl-fluorophosphate which is an irreversible cholinesterase inhibitor used to investigate the NERVOUS SYSTEM.
Seizures: Clinical or subclinical disturbances of cortical function due to a sudden, abnormal, excessive, and disorganized discharge of brain cells. Clinical manifestations include abnormal motor, sensory and psychic phenomena. Recurrent seizures are usually referred to as EPILEPSY or seizure disorder.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Seizures were elicited within ∼8 minutes after DFP exposure that progressively developed into persistent SE lasting for hours." | 5.48 | Midazolam-Resistant Seizures and Brain Injury after Acute Intoxication of Diisopropylfluorophosphate, an Organophosphate Pesticide and Surrogate for Nerve Agents. ( Kuruba, R; Reddy, DS; Wu, X, 2018) |
| "Donepezil pre-treatment by itself induced only a comparatively weaker inhibition of acetylcholinesterase staining and infrequent yawning." | 5.36 | Donepezil inhibits diisopropylfluorophosphate-induced seizures and up-regulation of synaptotagmin 4 mRNA. ( Pregelj, P; Saghafi, MM; Zivin, M, 2010) |
| "A timely and effective control of seizures with pharmacological agents can minimize the secondary and long-term neuropathology that may result from this damage." | 5.35 | Imidazenil, a non-sedating anticonvulsant benzodiazepine, is more potent than diazepam in protecting against DFP-induced seizures and neuronal damage. ( Auta, J; Costa, E; Guidotti, A; Kadriu, B, 2009) |
| " To avoid these treatment complications but safely protect against DFP-induced seizures and other CNS toxicity, we adopted the strategy of administering mice with (i) small doses of huperzine A (HUP), a reversible and long-lasting (half-life approximately 5 h) inhibitor of AChE, and (ii) imidazenil (IMI), a potent positive allosteric modulator of GABA action selective for alpha(5)-containing GABA(A) receptors." | 3.74 | The combination of huperzine A and imidazenil is an effective strategy to prevent diisopropyl fluorophosphate toxicity in mice. ( Auta, J; Costa, E; Guidotti, A; Kadriu, B; Kozikowski, AP; Pibiri, F; Pinna, G, 2008) |
| " We have compared the acute effects of PBN on the development of seizures induced by the irreversible acetylcholinesterase (AChE) inhibitor diisopropylphosphorofluoridate (DFP), the reversible AChE inhibitor physostigmine (PHY), the muscarinic cholinergic receptor agonist pilocarpine (PIL) and the glutamatergic receptor agonist kainic acid (KA)." | 3.70 | Nitrone spin trapping compound N-tert-butyl-alpha-phenylnitrone prevents seizures induced by anticholinesterases. ( Dettbarn, WD; Milatovic, D; Zivin, M, 1999) |
| "Exposures to seizure-inducing chemical threat agents are a major public health concern." | 2.53 | Models to identify treatments for the acute and persistent effects of seizure-inducing chemical threat agents. ( Bruun, DA; Hammock, BD; Lein, PJ; Pessah, IN; Rogawski, MA; Tancredi, DJ; Wulff, H; Zolkowska, D, 2016) |
| "In addition to these epileptiform seizure-like events, DFP-exposed larvae showed increased neuronal apoptosis, which were both partially alleviated by diazepam treatment, suggesting a causal link between neuronal hyperexcitation and cell death." | 1.56 | Organophosphorus diisopropylfluorophosphate (DFP) intoxication in zebrafish larvae causes behavioral defects, neuronal hyperexcitation and neuronal death. ( Bar, O; Brenet, A; Dal-Bo, G; Dupuis, N; Hassan-Abdi, R; Igert, A; Nachon, F; Romain, C; Saurat, D; Somkhit, J; Soussi-Yanicostas, N; Taudon, N; Yanicostas, C, 2020) |
| "DFP triggered rapid and sustained seizure behavior irrespective of PB pretreatment, and there was no significant difference in average seizure behavior score during the first 4 h following injection between DFP animals pretreated with PB or not." | 1.51 | Pretreatment with pyridostigmine bromide has no effect on seizure behavior or 24 hour survival in the rat model of acute diisopropylfluorophosphate intoxication. ( Bruun, DA; Guignet, M; Harvey, DJ; Lein, PJ, 2019) |
| "Rapid control of seizure activity is important to minimize neuronal injury and the resulting neurological and behavioral disorders; however, early treatment will not be possible after mass release of OPs or NAs." | 1.51 | Antiseizure and neuroprotective effects of delayed treatment with midazolam in a rodent model of organophosphate exposure. ( Bealer, SL; Dudek, FE; Pouliot, W; Roach, B; Spampanato, J, 2019) |
| "No (sarin and VX) or brief (DFP) EEG seizure activity was evoked in PND14 rats, while SE progressively increased in severity as a function of age in PND21, 28 and 70 animals." | 1.48 | Age-dependent behaviors, seizure severity and neuronal damage in response to nerve agents or the organophosphate DFP in immature and adult rats. ( Bealer, SL; Dudek, FE; Ekstrand, JJ; Lehmkuhle, MJ; McDonough, JH; Miller-Smith, SM; Scholl, EA, 2018) |
| "Seizures were elicited within ∼8 minutes after DFP exposure that progressively developed into persistent SE lasting for hours." | 1.48 | Midazolam-Resistant Seizures and Brain Injury after Acute Intoxication of Diisopropylfluorophosphate, an Organophosphate Pesticide and Surrogate for Nerve Agents. ( Kuruba, R; Reddy, DS; Wu, X, 2018) |
| "Diazepam (10mg/kg) was administered 10min and 30min after the onset of continuous EEG seizures induced by paraoxon and it terminated SE in a majority of animals at both time points." | 1.38 | Characterization of status epilepticus induced by two organophosphates in rats. ( Balint, CA; Cowan, ML; Kapur, J; Sun, C; Todorovic, MS, 2012) |
| "The severity of DFP-induced ECoG seizures was assessed by continuous radio telemetry recordings in unrestrained and freely moving rats." | 1.37 | Acute imidazenil treatment after the onset of DFP-induced seizure is more effective and longer lasting than midazolam at preventing seizure activity and brain neuropathology. ( Auta, J; Costa, E; Davis, JM; Guidotti, A; Kadriu, B, 2011) |
| "Imidazenil is an imidazo-benzodiazepine derivative with high intrinsic efficacy and selectivity for α2-, α3-, and α5- but low intrinsic efficacy for α1-containing GABA(A) receptors." | 1.37 | Absence of tolerance to the anticonvulsant and neuroprotective effects of imidazenil against DFP-induced seizure and neuronal damage. ( Auta, J; Davis, JM; Gocel, J; Guidotti, A; Kadriu, B; Larson, J; Nambiar, MP, 2011) |
| "Donepezil pre-treatment by itself induced only a comparatively weaker inhibition of acetylcholinesterase staining and infrequent yawning." | 1.36 | Donepezil inhibits diisopropylfluorophosphate-induced seizures and up-regulation of synaptotagmin 4 mRNA. ( Pregelj, P; Saghafi, MM; Zivin, M, 2010) |
| "A timely and effective control of seizures with pharmacological agents can minimize the secondary and long-term neuropathology that may result from this damage." | 1.35 | Imidazenil, a non-sedating anticonvulsant benzodiazepine, is more potent than diazepam in protecting against DFP-induced seizures and neuronal damage. ( Auta, J; Costa, E; Guidotti, A; Kadriu, B, 2009) |
| "Convulsions are major and life-threatening signs of organophosphate (OP) nerve agents induced neurotoxicity." | 1.32 | Imidazenil: a potent and safe protective agent against diisopropyl fluorophosphate toxicity. ( Auta, J; Costa, E; Davis, J; Guidotti, A, 2004) |
| "Acute effects of seizure-inducing doses of the organophosphate compound diisopropylphosphorofluoridate (DFP, 1." | 1.31 | Nitric oxide modulates high-energy phosphates in brain regions of rats intoxicated with diisopropylphosphorofluoridate or carbofuran: prevention by N-tert-butyl-alpha-phenylnitrone or vitamin E. ( Dettbarn, WD; Gupta, RC; Milatovic, D, 2001) |
| "Rats were observed for seizure activity for 2 h, survivors were killed 24 h later and edema was measured in samples from parietal and piriform cortices, dorsal thalmus, and hippocampus." | 1.28 | Effect of LiCl pretreatment on cholinomimetic-induced seizures and seizure-induced brain edema in rats. ( Nelson, SR; Padzernik, TL; Terry, JB, 1990) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 14 (32.56) | 18.7374 |
| 1990's | 4 (9.30) | 18.2507 |
| 2000's | 6 (13.95) | 29.6817 |
| 2010's | 14 (32.56) | 24.3611 |
| 2020's | 5 (11.63) | 2.80 |
| Authors | Studies |
|---|---|
| Rojas, A | 1 |
| Wang, J | 1 |
| Glover, A | 1 |
| Dingledine, R | 1 |
| González, EA | 2 |
| Rindy, AC | 1 |
| Guignet, MA | 2 |
| Calsbeek, JJ | 1 |
| Bruun, DA | 7 |
| Dhir, A | 1 |
| Andrew, P | 1 |
| Saito, N | 2 |
| Rowland, DJ | 3 |
| Harvey, DJ | 4 |
| Rogawski, MA | 2 |
| Lein, PJ | 7 |
| Enderlin, J | 1 |
| Igert, A | 2 |
| Auvin, S | 1 |
| Nachon, F | 2 |
| Dal Bo, G | 1 |
| Dupuis, N | 2 |
| Supasai, S | 1 |
| Hobson, B | 1 |
| Soares, S | 1 |
| Singh, V | 1 |
| Wulff, H | 2 |
| Brenet, A | 1 |
| Somkhit, J | 1 |
| Hassan-Abdi, R | 1 |
| Yanicostas, C | 1 |
| Romain, C | 1 |
| Bar, O | 1 |
| Saurat, D | 1 |
| Taudon, N | 1 |
| Dal-Bo, G | 1 |
| Soussi-Yanicostas, N | 1 |
| Hobson, BA | 1 |
| Sisó, S | 1 |
| Garbow, JR | 1 |
| Scholl, EA | 1 |
| Miller-Smith, SM | 1 |
| Bealer, SL | 2 |
| Lehmkuhle, MJ | 1 |
| Ekstrand, JJ | 1 |
| Dudek, FE | 2 |
| McDonough, JH | 1 |
| Vengerovich, NG | 1 |
| Drachkova, IM | 1 |
| Yudin, MA | 1 |
| Bykov, VN | 1 |
| Sarana, AM | 1 |
| Wu, X | 1 |
| Kuruba, R | 1 |
| Reddy, DS | 1 |
| Guignet, M | 1 |
| Spampanato, J | 1 |
| Pouliot, W | 1 |
| Roach, B | 1 |
| Ferchmin, PA | 1 |
| Andino, M | 1 |
| Reyes Salaman, R | 1 |
| Alves, J | 1 |
| Velez-Roman, J | 1 |
| Cuadrado, B | 1 |
| Carrasco, M | 1 |
| Torres-Rivera, W | 1 |
| Segarra, A | 1 |
| Martins, AH | 1 |
| Lee, JE | 1 |
| Eterovic, VA | 1 |
| Pessah, IN | 1 |
| Tancredi, DJ | 1 |
| Zolkowska, D | 1 |
| Hammock, BD | 1 |
| Pibiri, F | 1 |
| Kozikowski, AP | 1 |
| Pinna, G | 1 |
| Auta, J | 5 |
| Kadriu, B | 4 |
| Costa, E | 4 |
| Guidotti, A | 5 |
| Zaja-Milatovic, S | 1 |
| Gupta, RC | 2 |
| Aschner, M | 1 |
| Milatovic, D | 3 |
| Davis, JM | 2 |
| Saghafi, MM | 1 |
| Pregelj, P | 1 |
| Zivin, M | 2 |
| Li, Y | 2 |
| Liu, C | 2 |
| Tewolde, T | 2 |
| Ford, G | 1 |
| Ford, BD | 2 |
| Gocel, J | 1 |
| Larson, J | 1 |
| Nambiar, MP | 1 |
| Todorovic, MS | 1 |
| Cowan, ML | 1 |
| Balint, CA | 1 |
| Sun, C | 1 |
| Kapur, J | 1 |
| Giulivi, C | 1 |
| Ford, GD | 1 |
| Ross-Inta, C | 1 |
| ESSIG, CF | 2 |
| HAMPSON, JL | 2 |
| HIMWICH, HE | 5 |
| HEATH, DF | 1 |
| GREEN, VA | 1 |
| SULLIVAN, WT | 1 |
| STRONG, LM | 1 |
| JOHNS, RJ | 2 |
| BALES, PD | 2 |
| BOUZARTH, WF | 1 |
| Davis, J | 1 |
| Tuovinen, K | 1 |
| FREEDMAN, AM | 1 |
| Wada, JA | 1 |
| Dettbarn, WD | 2 |
| Hoover, DB | 1 |
| Craig, CR | 1 |
| Colasanti, BK | 1 |
| Golovko, AI | 1 |
| Sofronov, GA | 1 |
| Nikolaeva, TV | 1 |
| Kliuntina, TV | 1 |
| Savolainen, KM | 1 |
| Muona, O | 1 |
| Nelson, SR | 2 |
| Samson, FE | 1 |
| Pazdernik, TL | 1 |
| Terry, JB | 1 |
| Padzernik, TL | 1 |
| Domino, EF | 1 |
| Chusid, JG | 1 |
| Kopeloff, LM | 1 |
| Rump, S | 2 |
| Grudzińska, E | 2 |
| Edelwejn, Z | 1 |
2 reviews available for isoflurophate and Seizures
| Article | Year |
|---|---|
Models to identify treatments for the acute and persistent effects of seizure-inducing chemical threat agents.
Topics: Animals; Anticonvulsants; Brain; Bridged-Ring Compounds; Cholinesterase Inhibitors; Disease Models, | 2016 |
SOME CENTRAL EFFECTS OF ORGANOPHOSPHORUS ANTICHOLINESTERASES.
Topics: Atropine; Cats; Cell Respiration; Cholinesterase Inhibitors; Diphosphates; Dogs; Isoflurophate; Orga | 1963 |
41 other studies available for isoflurophate and Seizures
| Article | Year |
|---|---|
Urethane attenuates early neuropathology of diisopropylfluorophosphate-induced status epilepticus in rats.
Topics: Acetylcholinesterase; Animals; Brain Injuries; Diazepam; Disease Models, Animal; Electroencephalogra | 2020 |
The chemical convulsant diisopropylfluorophosphate (DFP) causes persistent neuropathology in adult male rats independent of seizure activity.
Topics: Acetylcholinesterase; Animals; Behavior, Animal; Brain; Brain Waves; Cholinesterase Inhibitors; Conv | 2020 |
Characterization of organophosphate-induced brain injuries in a convulsive mouse model of diisopropylfluorophosphate exposure.
Topics: Animals; Brain; Brain Injuries; Cholinesterase Inhibitors; Disease Models, Animal; Electrocorticogra | 2020 |
Acute administration of diazepam or midazolam minimally alters long-term neuropathological effects in the rat brain following acute intoxication with diisopropylfluorophosphate.
Topics: Animals; Brain Diseases; Cholinesterase Inhibitors; Diazepam; GABA Modulators; Gliosis; Isoflurophat | 2020 |
Organophosphorus diisopropylfluorophosphate (DFP) intoxication in zebrafish larvae causes behavioral defects, neuronal hyperexcitation and neuronal death.
Topics: Acetylcholinesterase; Animals; Apoptosis; Behavior, Animal; Brain; Calcium; Cell Death; Isoflurophat | 2020 |
From the Cover: MagneticResonance Imaging Reveals Progressive Brain Injury in Rats Acutely Intoxicated With Diisopropylfluorophosphate.
Topics: Animals; Behavior, Animal; Brain; Cholinesterase Inhibitors; Diffusion Magnetic Resonance Imaging; I | 2017 |
Age-dependent behaviors, seizure severity and neuronal damage in response to nerve agents or the organophosphate DFP in immature and adult rats.
Topics: Animals; Behavior, Animal; Brain; Female; Isoflurophate; Male; Nerve Agents; Organophosphorus Compou | 2018 |
Experimental Substantiation of Inhalation Administration of Pathogenic Therapy of Toxic Convulsive Disorder for Correction of External Respiration Disorders.
Topics: Administration, Inhalation; Albuterol; Animals; Atropine; Bronchodilator Agents; Convulsants; Diazep | 2018 |
Midazolam-Resistant Seizures and Brain Injury after Acute Intoxication of Diisopropylfluorophosphate, an Organophosphate Pesticide and Surrogate for Nerve Agents.
Topics: Animals; Anticonvulsants; Benzodiazepines; Brain; Brain Injuries; Cholinesterase Inhibitors; Drug Re | 2018 |
Pretreatment with pyridostigmine bromide has no effect on seizure behavior or 24 hour survival in the rat model of acute diisopropylfluorophosphate intoxication.
Topics: Acetylcholinesterase; Animals; Brain; Cholinesterase Inhibitors; Disease Models, Animal; GPI-Linked | 2019 |
Antiseizure and neuroprotective effects of delayed treatment with midazolam in a rodent model of organophosphate exposure.
Topics: Animals; Anticonvulsants; Disease Models, Animal; Isoflurophate; Male; Midazolam; Neuroprotective Ag | 2019 |
4R-cembranoid protects against diisopropylfluorophosphate-mediated neurodegeneration.
Topics: Acetylcholinesterase; Animals; Brain Injuries; CA1 Region, Hippocampal; Cell Death; Cholinesterase I | 2014 |
The combination of huperzine A and imidazenil is an effective strategy to prevent diisopropyl fluorophosphate toxicity in mice.
Topics: Alkaloids; Animals; Benzodiazepines; Cognition Disorders; Drug Therapy, Combination; Imidazoles; Iso | 2008 |
Imidazenil, a non-sedating anticonvulsant benzodiazepine, is more potent than diazepam in protecting against DFP-induced seizures and neuronal damage.
Topics: Animals; Anticonvulsants; Behavior, Animal; Benzodiazepines; Brain; Cholinesterase Inhibitors; Diaze | 2009 |
Protection of DFP-induced oxidative damage and neurodegeneration by antioxidants and NMDA receptor antagonist.
Topics: Animals; Antioxidants; Biomarkers; Brain; Cholinesterase Inhibitors; Cyclic N-Oxides; Disease Models | 2009 |
Acute imidazenil treatment after the onset of DFP-induced seizure is more effective and longer lasting than midazolam at preventing seizure activity and brain neuropathology.
Topics: Animals; Anticonvulsants; Antigens, Nuclear; Benzodiazepines; Brain; Dose-Response Relationship, Dru | 2011 |
Donepezil inhibits diisopropylfluorophosphate-induced seizures and up-regulation of synaptotagmin 4 mRNA.
Topics: Acetylcholine; Acetylcholinesterase; Animals; Anticonvulsants; Brain; Cholinesterase Inhibitors; Don | 2010 |
Spatiotemporal pattern of neuronal injury induced by DFP in rats: a model for delayed neuronal cell death following acute OP intoxication.
Topics: Animals; Brain; Cholinesterase Inhibitors; Isoflurophate; Male; Models, Animal; Neurons; Rats; Rats, | 2011 |
Absence of tolerance to the anticonvulsant and neuroprotective effects of imidazenil against DFP-induced seizure and neuronal damage.
Topics: Animals; Anticonvulsants; Benzodiazepines; Cholinesterase Inhibitors; Diazepam; Disease Models, Anim | 2011 |
Characterization of status epilepticus induced by two organophosphates in rats.
Topics: Animals; Anticonvulsants; Atropine; Brain; Diazepam; Electroencephalography; Isoflurophate; Male; Ne | 2012 |
Neuregulin-1 is neuroprotective in a rat model of organophosphate-induced delayed neuronal injury.
Topics: Animals; Atropine; Brain; Cholinesterase Inhibitors; Immunohistochemistry; Isoflurophate; Male; Neur | 2012 |
Biochemically induced circling behavior.
Topics: Animals; Behavior; Isoflurophate; Seizures | 1953 |
ALTERATIONS IN THE ACTIVITY OF PENTOTHAL, PHENOBARBITAL, PENTYLENETETRAZOL, AND STRYCHNINE BY CHOLINESTERASE INHIBITORS.
Topics: Acetylcholine; Anesthesia; Atropine; Cholinesterase Inhibitors; Isoflurophate; Mice; Neostigmine; Pe | 1964 |
Microbiological assay of glutamic acid in normal rabbit brain, after the injection of DFP and after convulsions.
Topics: Animals; Biological Assay; Brain; Glutamates; Glutamic Acid; Injections; Isoflurophate; Rabbits; Sei | 1955 |
A central action of some antihistamines. Correction of forced circling movements and of seizure brain waves produced by the intracarotid injection of diisopropyl fluorophosphate (DFP).
Topics: Brain; Brain Waves; Fluorides; Histamine Antagonists; Isoflurophate; Phosphates; Seizures | 1950 |
The effects of DFP on the convulsant dose of theophylline, theophylline-ethylenediamine and 8-chlorotheophylline.
Topics: Aminophylline; Convulsants; Humans; Isoflurophate; Organophosphonates; Phosphorous Acids; Seizures; | 1951 |
Mechanism of seizures induced by di-isopropyl fluorophosphate.
Topics: Fluorides; Isoflurophate; Phosphates; Seizures | 1952 |
Imidazenil: a potent and safe protective agent against diisopropyl fluorophosphate toxicity.
Topics: Animals; Benzodiazepines; Diazepam; Imidazoles; Isoflurophate; Male; Neuroprotective Agents; Rats; R | 2004 |
Organophosphate-induced convulsions and prevention of neuropathological damages.
Topics: Acetylcholinesterase; Adenosine; Animals; Antidotes; Atropine; Brain; Cholinesterase Inhibitors; Dia | 2004 |
Effect of trimethadione (tridione) and other drugs on convulsions caused by di-isopropyl fluorophosphate (DFP).
Topics: Fluorides; Isoflurophate; Phosphates; Phosphorus; Phosphorus Compounds; Seizures; Trimethadione | 1950 |
Mechanism of amygdaloid convulsive seizure development.
Topics: Amygdala; Animals; Behavior, Animal; Electroencephalography; Isoflurophate; Kindling, Neurologic; Ma | 1982 |
Nitrone spin trapping compound N-tert-butyl-alpha-phenylnitrone prevents seizures induced by anticholinesterases.
Topics: Acetylcholinesterase; Animals; Anticonvulsants; Behavior, Animal; Brain; Cholinesterase Inhibitors; | 1999 |
Nitric oxide modulates high-energy phosphates in brain regions of rats intoxicated with diisopropylphosphorofluoridate or carbofuran: prevention by N-tert-butyl-alpha-phenylnitrone or vitamin E.
Topics: Adenosine Triphosphate; Amygdala; Animals; Antioxidants; Brain; Carbofuran; Cerebral Cortex; Choline | 2001 |
Cholinergic involvement in cobalt-induced epilepsy in the rat.
Topics: Acetylcholine; Animals; Brain Chemistry; Cerebral Cortex; Cholinesterases; Cobalt; Hemicholinium 3; | 1977 |
[Effects of diisopropylfluorophosphate, paraoxon and dichlophos on [3H] quinuclidinyl benzylate binding to the rat striatum synaptic membranes].
Topics: Animals; Corpus Striatum; Dichlorvos; In Vitro Techniques; Isoflurophate; Male; Paraoxon; Quinuclidi | 1992 |
Lithium modifies convulsions and brain phosphoinositide turnover induced by organophosphates.
Topics: Animals; Brain; Chlorides; Chromatography, Gas; Drug Synergism; Inositol Phosphates; Insecticides; I | 1991 |
Effect of LiCl pretreatment on cholinomimetic-induced seizures and seizure-induced brain edema in rats.
Topics: Animals; Brain; Brain Edema; Chlorides; Isoflurophate; Lithium; Lithium Chloride; Male; Organ Specif | 1990 |
Comparative seizure inducing properties of various cholinesterase inhibitors: antagonism by diazepam and midazolam.
Topics: Animals; Atropine; Behavior, Animal; Cholinesterase Inhibitors; Diazepam; Isoflurophate; Male; Midaz | 1987 |
Use of chronic irritative foci in laboratory evaluation of antiepileptic drugs.
Topics: Aluminum; Aminobutyrates; Animals; Bemegride; Central Nervous System Stimulants; Chlordiazepoxide; C | 1969 |
Investigations on the effects of diazepam in acute experimental intoxication with fluostigmine.
Topics: Action Potentials; Animals; Anticonvulsants; Atropine; Diazepam; Drug Combinations; Female; Isofluro | 1974 |
Effects of diazepam on abnormalities of bioelectrical activity of the rabbit's brain due to fluostigmine.
Topics: Animals; Brain; Diazepam; Electrodes, Implanted; Evoked Potentials; Isoflurophate; Organophosphorus | 1972 |