inositol has been researched along with Status Epilepticus in 7 studies
Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.
Status Epilepticus: A prolonged seizure or seizures repeated frequently enough to prevent recovery between episodes occurring over a period of 20-30 minutes. The most common subtype is generalized tonic-clonic status epilepticus, a potentially fatal condition associated with neuronal injury and respiratory and metabolic dysfunction. Nonconvulsive forms include petit mal status and complex partial status, which may manifest as behavioral disturbances. Simple partial status epilepticus consists of persistent motor, sensory, or autonomic seizures that do not impair cognition (see also EPILEPSIA PARTIALIS CONTINUA). Subclinical status epilepticus generally refers to seizures occurring in an unresponsive or comatose individual in the absence of overt signs of seizure activity. (From N Engl J Med 1998 Apr 2;338(14):970-6; Neurologia 1997 Dec;12 Suppl 6:25-30)
| Excerpt | Relevance | Reference |
|---|---|---|
| " We have previously shown that myoinositol (MI) daily treatment for 28 days prevents certain biochemical changes that are triggered by kainic acid (KA) induced status epilepticus (SE)." | 7.83 | Myoinositol Attenuates the Cell Loss and Biochemical Changes Induced by Kainic Acid Status Epilepticus. ( Kikvidze, M; Lepsveridze, E; Lordkipanidze, T; Nozadze, M; Solomonia, R; Tsverava, L, 2016) |
| "It is known that myo-inositol pretreatment attenuates the seizure severity and several biochemical changes provoked by experimentally induced status epilepticus." | 7.79 | The protective effect of myo-inositol on hippocamal cell loss and structural alterations in neurons and synapses triggered by kainic acid-induced status epilepticus. ( Bikashvili, T; Bolkvadze, T; Japaridze, NJ; Kiladze, M; Kotaria, N; Solomonia, RO; Zhvania, MG, 2013) |
| " We have previously shown that myo-Inositol (MI) daily treatment prevents certain biochemical changes that are triggered by kainic acid (KA)-induced status epilepticus (SE)." | 7.79 | Myo-inositol treatment and GABA-A receptor subunit changes after kainate-induced status epilepticus. ( Dzneladze, D; Gogichaishvili, N; Kiguradze, T; Lepsveridze, E; Nozadze, M; Solomonia, R, 2013) |
| " In this study, we investigated the metabolism in the thalamus to identify the changes that occur following Coriaria lactone (CL)-induced status epilepticus (SE) and to observe whether the epileptiform discharges could present a difference between the left and right thalami." | 7.78 | Longitudinal ¹H MRS assessment of the thalamus in a Coriaria lactone-induced rhesus monkey status epilepticus model. ( Gong, QY; Li, HX; Li, JM; Wang, L; Yang, ZY; Zhang, XY; Zhou, D, 2012) |
| " We have previously shown that myoinositol (MI) daily treatment for 28 days prevents certain biochemical changes that are triggered by kainic acid (KA) induced status epilepticus (SE)." | 3.83 | Myoinositol Attenuates the Cell Loss and Biochemical Changes Induced by Kainic Acid Status Epilepticus. ( Kikvidze, M; Lepsveridze, E; Lordkipanidze, T; Nozadze, M; Solomonia, R; Tsverava, L, 2016) |
| "It is known that myo-inositol pretreatment attenuates the seizure severity and several biochemical changes provoked by experimentally induced status epilepticus." | 3.79 | The protective effect of myo-inositol on hippocamal cell loss and structural alterations in neurons and synapses triggered by kainic acid-induced status epilepticus. ( Bikashvili, T; Bolkvadze, T; Japaridze, NJ; Kiladze, M; Kotaria, N; Solomonia, RO; Zhvania, MG, 2013) |
| " We have previously shown that myo-Inositol (MI) daily treatment prevents certain biochemical changes that are triggered by kainic acid (KA)-induced status epilepticus (SE)." | 3.79 | Myo-inositol treatment and GABA-A receptor subunit changes after kainate-induced status epilepticus. ( Dzneladze, D; Gogichaishvili, N; Kiguradze, T; Lepsveridze, E; Nozadze, M; Solomonia, R, 2013) |
| " In this study, we investigated the metabolism in the thalamus to identify the changes that occur following Coriaria lactone (CL)-induced status epilepticus (SE) and to observe whether the epileptiform discharges could present a difference between the left and right thalami." | 3.78 | Longitudinal ¹H MRS assessment of the thalamus in a Coriaria lactone-induced rhesus monkey status epilepticus model. ( Gong, QY; Li, HX; Li, JM; Wang, L; Yang, ZY; Zhang, XY; Zhou, D, 2012) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (14.29) | 29.6817 |
| 2010's | 6 (85.71) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kotaria, N | 1 |
| Kiladze, M | 1 |
| Zhvania, MG | 1 |
| Japaridze, NJ | 1 |
| Bikashvili, T | 1 |
| Solomonia, RO | 1 |
| Bolkvadze, T | 1 |
| Wu, Y | 1 |
| Pearce, PS | 1 |
| Rapuano, A | 1 |
| Hitchens, TK | 1 |
| de Lanerolle, NC | 1 |
| Pan, JW | 1 |
| Tsverava, L | 1 |
| Lordkipanidze, T | 1 |
| Lepsveridze, E | 3 |
| Nozadze, M | 3 |
| Kikvidze, M | 1 |
| Solomonia, R | 3 |
| Rabeson, H | 1 |
| Fauvelle, F | 1 |
| Testylier, G | 1 |
| Foquin, A | 1 |
| Carpentier, P | 1 |
| Dorandeu, F | 1 |
| van Ormondt, D | 1 |
| Graveron-Demilly, D | 1 |
| Mikautadze, E | 1 |
| Kuchiashvili, N | 1 |
| Kiguradze, T | 2 |
| Zhang, XY | 1 |
| Yang, ZY | 1 |
| Li, JM | 1 |
| Li, HX | 1 |
| Wang, L | 1 |
| Gong, QY | 1 |
| Zhou, D | 1 |
| Gogichaishvili, N | 1 |
| Dzneladze, D | 1 |
7 other studies available for inositol and Status Epilepticus
| Article | Year |
|---|---|
The protective effect of myo-inositol on hippocamal cell loss and structural alterations in neurons and synapses triggered by kainic acid-induced status epilepticus.
Topics: Analysis of Variance; Animals; Cell Count; Cerebrum; Dendrites; Hippocampus; Inositol; Kainic Acid; | 2013 |
Metabolic changes in early poststatus epilepticus measured by MR spectroscopy in rats.
Topics: Animals; Antigens, Nuclear; Aspartic Acid; Brain Chemistry; Creatine; Epilepsy, Temporal Lobe; Gluta | 2015 |
Myoinositol Attenuates the Cell Loss and Biochemical Changes Induced by Kainic Acid Status Epilepticus.
Topics: Animals; Blotting, Western; Calibration; Caspase 3; Cell Count; Hippocampus; Inositol; Kainic Acid; | 2016 |
Quantitation with QUEST of brain HRMAS-NMR signals: application to metabolic disorders in experimental epileptic seizures.
Topics: Acetates; Alanine; Algorithms; Animals; Aspartic Acid; Brain; Choline; gamma-Aminobutyric Acid; Glut | 2008 |
Myo-inositol treatment prevents biochemical changes triggered by kainate-induced status epilepticus.
Topics: Animals; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Convulsants; Hippocampus; HSP90 Heat-Sh | 2010 |
Longitudinal ¹H MRS assessment of the thalamus in a Coriaria lactone-induced rhesus monkey status epilepticus model.
Topics: Animals; Creatinine; Disease Models, Animal; Glutamic Acid; Inositol; Lactones; Macaca mulatta; Magn | 2012 |
Myo-inositol treatment and GABA-A receptor subunit changes after kainate-induced status epilepticus.
Topics: Animals; Inositol; Kainic Acid; Male; Protein Subunits; Rats; Rats, Wistar; Receptors, GABA-A; Statu | 2013 |