inositol has been researched along with Parkinson Disease, Secondary in 2 studies
Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.
Parkinson Disease, Secondary: Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Rocha, EM | 1 |
| Smith, GA | 1 |
| Park, E | 1 |
| Cao, H | 1 |
| Graham, AR | 1 |
| Brown, E | 1 |
| McLean, JR | 1 |
| Hayes, MA | 1 |
| Beagan, J | 1 |
| Izen, SC | 1 |
| Perez-Torres, E | 1 |
| Hallett, PJ | 1 |
| Isacson, O | 1 |
| Yamasue, H | 1 |
| Fukui, T | 1 |
| Fukuda, R | 1 |
| Kasai, K | 1 |
| Iwanami, A | 1 |
| Kato, N | 1 |
| Kato, T | 1 |
2 other studies available for inositol and Parkinson Disease, Secondary
| Article | Year |
|---|---|
Sustained Systemic Glucocerebrosidase Inhibition Induces Brain α-Synuclein Aggregation, Microglia and Complement C1q Activation in Mice.
Topics: alpha-Synuclein; Animals; Autophagy; Axonal Transport; Cerebral Cortex; Complement Activation; Compl | 2015 |
Drug-induced parkinsonism in relation to choline-containing compounds measured by 1H-MR spectroscopy in putamen of chronically medicated patients with schizophrenia.
Topics: Adult; Antipsychotic Agents; Aspartic Acid; Basal Ganglia Diseases; Choline; Chronic Disease; Creati | 2003 |