inositol has been researched along with Mitochondrial Diseases in 3 studies
Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.
Mitochondrial Diseases: Diseases caused by abnormal function of the MITOCHONDRIA. They may be caused by mutations, acquired or inherited, in mitochondrial DNA or in nuclear genes that code for mitochondrial components. They may also be the result of acquired mitochondria dysfunction due to adverse effects of drugs, infections, or other environmental causes.
Excerpt | Relevance | Reference |
---|---|---|
"Gaucher disease is an autosomal recessive disease, caused by a lack or functional deficiency of the lysosomal enzyme, glucocerebrosidase (GCase)." | 1.42 | Glucocerebrosidase deficiency and mitochondrial impairment in experimental Parkinson disease. ( Alvarez-Fischer, D; Andreas, H; Hirsch, EC; Höglinger, GU; Höllerhage, M; Lu, L; Noelker, C; Oertel, WH; Roscher, R; Sturn, A; Vulinovic, F, 2015) |
"Patients with mitochondrial disorders offer a unique window through which we can begin to understand the association between psychiatric symptoms and mitochondrial dysfunction in vivo." | 1.38 | Psychiatric symptoms correlate with metabolic indices in the hippocampus and cingulate in patients with mitochondrial disorders. ( Anglin, RE; Mazurek, MF; Noseworthy, MD; Rosebush, PI; Tarnopolsky, M, 2012) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (33.33) | 29.6817 |
2010's | 2 (66.67) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Noelker, C | 1 |
Lu, L | 1 |
Höllerhage, M | 1 |
Vulinovic, F | 1 |
Sturn, A | 1 |
Roscher, R | 1 |
Höglinger, GU | 1 |
Hirsch, EC | 1 |
Oertel, WH | 1 |
Alvarez-Fischer, D | 1 |
Andreas, H | 1 |
Anglin, RE | 1 |
Rosebush, PI | 1 |
Noseworthy, MD | 1 |
Tarnopolsky, M | 1 |
Mazurek, MF | 1 |
Prestel, J | 1 |
Gempel, K | 1 |
Hauser, TK | 1 |
Schweitzer, K | 1 |
Prokisch, H | 1 |
Ahting, U | 1 |
Freudenstein, D | 1 |
Bueltmann, E | 1 |
Naegele, T | 1 |
Berg, D | 1 |
Klopstock, T | 1 |
Gasser, T | 1 |
3 other studies available for inositol and Mitochondrial Diseases
Article | Year |
---|---|
Glucocerebrosidase deficiency and mitochondrial impairment in experimental Parkinson disease.
Topics: 1-Deoxynojirimycin; 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Animals; Brain; Cell Count; Cells, | 2015 |
Psychiatric symptoms correlate with metabolic indices in the hippocampus and cingulate in patients with mitochondrial disorders.
Topics: Adult; Aged; Anxiety; Aspartic Acid; Case-Control Studies; Caudate Nucleus; Creatine; Female; Glutam | 2012 |
Clinical and molecular characterisation of a Parkinson family with a novel PINK1 mutation.
Topics: Adult; Aged; Basal Ganglia; Biomarkers; Cell Line; DNA Mutational Analysis; Energy Metabolism; Femal | 2008 |