inositol has been researched along with Dementias, Transmissible in 4 studies
Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Prion diseases are fatal chronic neurodegenerative diseases." | 1.34 | MRI and MRS alterations in the preclinical phase of murine prion disease: association with neuropathological and behavioural changes. ( Anthony, DC; Blamire, AM; Broom, KA; Griffin, JL; Lowe, JP; Perry, VH; Scott, H; Sibson, NR; Styles, P, 2007) |
| "Inherited prion diseases are caused by mutations in the gene which codes for prion protein (PrP), leading to proliferation of abnormal PrP isomers in the brain and neurodegeneration; they include Gerstmann-Sträussler-Scheinker disease (GSS), fatal familial insomnia (FFI) and familial Creutzfeldt-Jakob disease (fCJD)." | 1.33 | Regional brain metabolite abnormalities in inherited prion disease and asymptomatic gene carriers demonstrated in vivo by quantitative proton magnetic resonance spectroscopy. ( Collinge, J; Cordery, RJ; Godbolt, A; MacManus, DG; Rossor, MN; Waldman, AD, 2006) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cudalbu, C | 1 |
| Craveiro, M | 1 |
| Mlynárik, V | 1 |
| Bremer, J | 1 |
| Aguzzi, A | 1 |
| Gruetter, R | 1 |
| Maeda, Y | 1 |
| Kinoshita, T | 1 |
| Waldman, AD | 1 |
| Cordery, RJ | 1 |
| MacManus, DG | 1 |
| Godbolt, A | 1 |
| Collinge, J | 1 |
| Rossor, MN | 1 |
| Broom, KA | 1 |
| Anthony, DC | 1 |
| Lowe, JP | 1 |
| Griffin, JL | 1 |
| Scott, H | 1 |
| Blamire, AM | 1 |
| Styles, P | 1 |
| Perry, VH | 1 |
| Sibson, NR | 1 |
1 review available for inositol and Dementias, Transmissible
| Article | Year |
|---|---|
Structural remodeling, trafficking and functions of glycosylphosphatidylinositol-anchored proteins.
Topics: Animals; Biological Transport; Endocytosis; Endoplasmic Reticulum; Fatty Acids; Glycosylphosphatidyl | 2011 |
3 other studies available for inositol and Dementias, Transmissible
| Article | Year |
|---|---|
In Vivo Longitudinal (1)H MRS Study of Transgenic Mouse Models of Prion Disease in the Hippocampus and Cerebellum at 14.1 T.
Topics: Animals; Aspartic Acid; Brain Chemistry; Cerebellum; Glutamic Acid; Hippocampus; Inositol; Lactic Ac | 2015 |
Regional brain metabolite abnormalities in inherited prion disease and asymptomatic gene carriers demonstrated in vivo by quantitative proton magnetic resonance spectroscopy.
Topics: Adult; Aspartic Acid; Brain; Case-Control Studies; Creatine; Female; Heterozygote; Humans; Inositol; | 2006 |
MRI and MRS alterations in the preclinical phase of murine prion disease: association with neuropathological and behavioural changes.
Topics: Animals; Aspartic Acid; Astrocytes; Body Water; Brain; Choline; Creatine; Diffusion; Disease Models, | 2007 |