inositol has been researched along with Ciliary Motility Disorders in 2 studies
Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.
Ciliary Motility Disorders: Conditions caused by abnormal CILIA movement in the body, usually causing KARTAGENER SYNDROME, chronic respiratory disorders, chronic SINUSITIS, and chronic OTITIS. Abnormal ciliary beating is likely due to defects in any of the 200 plus ciliary proteins, such as missing motor enzyme DYNEIN arms.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Activation of the inositol cycle by a factor capable of by-passing the normal controls on exocrine secretion by an interaction with a coupling protein could produce effects similar to a calcium ionophore or the ciliary dyskinesia factor." | 7.67 | The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation? ( van Woerkom, AE, 1987) |
| "Activation of the inositol cycle by a factor capable of by-passing the normal controls on exocrine secretion by an interaction with a coupling protein could produce effects similar to a calcium ionophore or the ciliary dyskinesia factor." | 3.67 | The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation? ( van Woerkom, AE, 1987) |
| "Mutants that have only exencephaly are fourfold more frequent than those that have spina bifida aperta with or without exencephaly." | 2.46 | An update to the list of mouse mutants with neural tube closure defects and advances toward a complete genetic perspective of neural tube closure. ( Harris, MJ; Juriloff, DM, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (50.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Harris, MJ | 1 |
| Juriloff, DM | 1 |
| van Woerkom, AE | 1 |
1 review available for inositol and Ciliary Motility Disorders
| Article | Year |
|---|---|
An update to the list of mouse mutants with neural tube closure defects and advances toward a complete genetic perspective of neural tube closure.
Topics: Animals; Ciliary Motility Disorders; Disease Models, Animal; Epigenomics; Female; Folic Acid; Humans | 2010 |
1 other study available for inositol and Ciliary Motility Disorders
| Article | Year |
|---|---|
The end organ defect in cystic fibrosis; a hypothesis: disinhibited inositol cycle activation?
Topics: Blood Proteins; Calcium; Cell Membrane Permeability; Chlorides; Ciliary Motility Disorders; Cystic F | 1987 |