Page last updated: 2024-10-19

inositol and Blastocyst Disintegration

inositol has been researched along with Blastocyst Disintegration in 2 studies

Inositol: An isomer of glucose that has traditionally been considered to be a B vitamin although it has an uncertain status as a vitamin and a deficiency syndrome has not been identified in man. (From Martindale, The Extra Pharmacopoeia, 30th ed, p1379) Inositol phospholipids are important in signal transduction.
inositol : Any cyclohexane-1,2,3,4,5,6-hexol.
1D-chiro-inositol : Belonging to the inositol family of compounds, D-chiro-inositol (DCI) is an isomer of glucose. It is an important secondary messenger in insulin signal transduction.
muco-inositol : An inositol that is cyclohexane-1,2,3,4,5,6-hexol having a (1R,2R,3r,4R,5S,6r)-configuration.

Research Excerpts

ExcerptRelevanceReference
"Valproic acid (VPA) is an anticonvulsant drug that is widely used therapeutically for a variety of neurological conditions."1.33Myo-inositol enhances teratogenicity of valproic acid in the mouse. ( Finnell, RH; Giavini, E; Massa, V; Wlodarczyk, B, 2006)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Franke, B1
Klootwijk, R1
Lemmers, B1
de Kovel, CG1
Steegers-Theunissen, RP1
Mariman, EC1
Massa, V1
Wlodarczyk, B1
Giavini, E1
Finnell, RH1

Other Studies

2 other studies available for inositol and Blastocyst Disintegration

ArticleYear
Phenotype of the neural tube defect mouse model bent tail is not sensitive to maternal folinic acid, myo-inositol, or zinc supplementation.
    Birth defects research. Part A, Clinical and molecular teratology, 2003, Volume: 67, Issue:12

    Topics: Animals; Diet; Disease Models, Animal; DNA; Embryo Loss; Female; Genotype; Inositol; Leucovorin; Lit

2003
Myo-inositol enhances teratogenicity of valproic acid in the mouse.
    Birth defects research. Part A, Clinical and molecular teratology, 2006, Volume: 76, Issue:3

    Topics: Abnormalities, Drug-Induced; Administration, Oral; Animals; Anticonvulsants; Drug Synergism; Embryo

2006