imiquimod has been researched along with Dermatitis in 141 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (0.71) | 18.2507 |
| 2000's | 1 (0.71) | 29.6817 |
| 2010's | 60 (42.55) | 24.3611 |
| 2020's | 79 (56.03) | 2.80 |
| Authors | Studies |
|---|---|
| Amento, E; Kang, GJ; Kaur, M; Mechanic, J; Ravikumar, P; Welihinda, A; Yadav, S | 1 |
| Asahina, R; Chow, Z; Dainichi, T; Egawa, G; Ishida, Y; Kabashima, K; Kitoh, A; Miyake, T; Nomura, T; Okuno, T; Otsuka, M; Yokomizo, T | 1 |
| Vinardell, MP | 1 |
| Agócs, R; Dobosy, P; Kemény, L; Németh, I; Pajtók, C; Pap, D; Szabó, AJ; Szebeni, B; Tulassay, T; Vannay, Á; Veréb, Z; Veres-Székely, A | 1 |
| Iida, K; Ishikawa, J; Iwamoto, T; Iwata, A; Kageyama, T; Makita, S; Nakajima, H; Suto, A; Suzuki, K; Tanaka, S; Yabe, Y; Yokota, M | 1 |
| Blauvelt, A; Boki, H; Kimura, T; Miyagaki, T; Okochi, H; Sato, S; Suga, H; Sugaya, M | 1 |
| Deng, M; Li, S; Li, X; Luo, S; Su, Y; Tan, S; Tang, G; Wu, R; Zhang, P; Zhang, Q; Zhang, S; Zhang, X; Zhao, M; Zhu, Y | 1 |
| Chen, J; Dang, E; Fang, H; Li, Q; Ma, J; Pang, B; Qiao, H; Qiao, P; Shao, S; Sun, Z; Wang, G; Wang, L; Xue, K; Yu, C | 1 |
| Guo, H; Li, M; Liu, H | 1 |
| Chen, Y; Chen, Z; Guo, C; Hu, Y; Shi, Y; Wang, Y; Xu, P; Yu, Q; Yu, Z; Zhang, X; Zhao, Z; Zhou, X | 1 |
| Rasool, M; Srikanth, M | 1 |
| Fang, X; Li, W; Ling, S; Liu, X; Luo, Y; Wang, A; Xu, B; Yao, X; Zhang, S; Zhou, Y; Zong, W | 1 |
| Bonifaz, LC; Cancino-Diaz, JC; Cancino-Diaz, ME; Castro-Escamilla, O; Martínez-Torres, I; Perez-Tapia, SM; Rodríguez-Martínez, S; Tepale-Segura, A | 1 |
| Du, Z; Gu, H; Huang, J; Li, C; Liu, G; Shi, Y; Xu, Y; Zhang, L; Zhou, W | 1 |
| Fu, FH; Li, XX; Lin, YW; Liu, B; Ma, L; Qi, R; Xing, X | 1 |
| Correa Atella, G; Donato-Trancoso, A; Romana-Souza, B | 1 |
| Chen, LC; Cheng, YP; Guo, JW; Liu, CY | 1 |
| Chen, Z; Hu, L; Lei, X; Li, S; Liu, J; Liu, Y; Song, Z; Wei, Z; Xiang, R; Yang, Y | 1 |
| Cai, Y; Chen, L; Ding, C; Hu, X; Li, X; Liu, N; Qin, H; Rouchka, EC; Tieri, D; Wang, L; Xu, Q; Xue, F; Yan, J; Zheng, J | 1 |
| Guo, H; Lai, Y; Li, L; Li, X; Liu, Y; Luo, X; Mo, C; Puno, P; Wang, Y; Wu, X; Yan, B; Yang, S | 1 |
| Abe, T; Hida, T; Ichimiya, S; Ikegami, I; Inoue, KI; Kamada, Y; Kamekura, R; Kamiya, S; Kamiya, T; Kobayashi, K; Sato, T; Takaki, H; Uhara, H; Yanagi, M | 1 |
| Wu, X; Xiang, S; Xiang, Y | 1 |
| Cai, H; Chen, X; Chen, Y; Hu, W; Hua, L; Liang, S; Lin, G; Liu, Z; Ou, Y; Sun, P; Wu, X; Yang, Z; Zhou, Y | 1 |
| AAl-Haidari, R; Ah Mostafa, M; Alhaddad, A; N Al Busaidi, H; Samman, W; Samuel Kelechi, M; Sarkar, D; Sm, F | 1 |
| He, L; Li, C; Li, M; Qiao, P; Shao, S; Wang, G; Wang, Z; Zhang, J; Zhang, K; Zhang, Y | 1 |
| Li, M; Liu, C; Liu, S; Lu, J; Tang, J; Zhou, X; Zhu, L | 1 |
| Chen, Y; Chikazawa, S; Ichimasu, N; Katagiri, K; Kobayashi, K; Suzuki, S | 1 |
| Duan, J; Duan, R; Gao, S; Li, J; Li, X; Ma, X; Qin, Y; Shi, R; Zhang, L; Zhao, R | 1 |
| Asaji, M; Chang, J; Hattori, H; Higo, T; Hikoso, S; Ichi, I; Inui, H; Ito, K; Kamada, Y; Kanno, K; Koseki, M; Nishida, M; Nishihara, S; Ohama, T; Okada, T; Okuzaki, D; Omatsu, T; Ono, M; Saga, A; Saibara, T; Sakata, Y; Tanaka, K; Yamashita, S; Zhu, Y | 1 |
| Chenyang, W; Fushida, N; Hamaguchi, Y; Horii, M; Ikawa, Y; Kitano, T; Komuro, A; Matsushita, T; Nakabori, I; Sawada, K; Xibei, J | 1 |
| Bai, M; Deng, H; Fan, X; Guo, M; Li, Z; Liu, W; Meng, Q; Shi, Y; Sun, R; Yang, X; Yuan, D | 1 |
| Chen, H; Huang, Y; Jiang, J; Jin, Z; Kong, Y; Li, L; Liu, X; Tong, Q; Wei, F; Yin, J; Zhang, Y | 1 |
| Lian, LH; Nan, JX; Wu, YL; Yang, HX; Zhan, ZY; Zhang, ZH | 1 |
| Christensen, PKF; Hansen, AK; Høyer-Hansen, MH; Koch, J; Larsen, J; Martel, BC; Skov, S | 1 |
| Clausen, BE; Hsiao, YP; Kelel, M; Lee, CH; Lee, YL; Liang, PH; Liu, FT; Lu, CH; Su, SH; Tan, YF; Tsai, CH; Tsai, TF; Weng, HJ; Yeh, CY | 1 |
| Chen, H; Chen, K; Hu, Y; Huang, D; Ju, M; Liu, L; Luan, C; Zhang, J; Zhou, X | 1 |
| Chen, X; Lei, Y; Liu, Y; Lyu, C; Yang, Y; Ye, Y; Zhang, T; Zhou, X | 1 |
| Burnet, M; Cloos, N; Cruces, S; Fischer, T; Geiger, J; Geiger, S; Guezguez, J; Guse, JH; Keppler, M; Laufer, S; Martorelli, M; Riexinger, L; Sandri, TL; Schwamborn, A; Späth, N; Straß, S | 1 |
| Chang, YC; Chen, CH; Chen, PJ; Chen, SH; Fang, SY; Hwang, TL; Tsai, SC; Tsai, YF; Tseng, HH; Wang, YH | 1 |
| Benoist, C; Hanna, BS; Hsu, YC; Mathis, D; Ortiz-Lopez, A; Sassone-Corsi, M; Shwartz, Y; Zarin, P | 1 |
| Hillman, PF; Jung, YR; Kang, JH; Kang, TB; Kim, JH; Lee, SB; Nam, SJ; Sim, EJ | 1 |
| Dummer, R; Roshardt Prieto, NM; Wyss, M | 1 |
| Cai, X; Du, J; Gao, W; Gong, H; Lu, H; Xiao, H; Xu, J; Yang, Y | 1 |
| Amalia, SN; Baral, H; Fujiwara, C; Hosoi, M; Inoue, Y; Ishikawa, M; Kosaka, K; Motegi, SI; Ogino, S; Sekiguchi, A; Shibasaki, K; Torii, R; Uchiyama, A; Yamazaki, S; Yokoyama, Y | 1 |
| Dai, SM; Deng, H; Guo, M; Liu, N; Liu, W; Meng, Q; Su, Y; Wei, M; Zhuang, H | 1 |
| Endo, K; Makabe, H; Nakashima, A; Sawa, T; Tanaka, S; Tanaka, Y | 1 |
| Lin, J; Lin, P; Lu, YY; Shi, HY | 1 |
| Han, X; Li, P; Lin, Y; Liu, X; Xie, X; Zhang, L | 1 |
| Fu, D; Fu, X; Hu, H; Li, J; Li, M; Song, X; Tian, Z; Wang, Q; Yan, D; Yang, Z; Zheng, S | 1 |
| Luo, F; Luo, M; Pan, Y; Peng, Y; Ran, C; Wang, B; Wang, H; Wang, L; Yan, YN; Yi, T; Zhang, Y; Zhou, R | 1 |
| Antal, D; Bai, P; Demény, MÁ; Dull, K; Kiss, B; Kovács, I; Póliska, S; Pór, Á; Szántó, M; Szegedi, A; Szöllősi, AG; Ujlaki, G | 1 |
| Pu, S; Sun, L; Zhao, K; Zhou, D | 1 |
| Li, P; Lin, Y; Liu, X; Wang, N; Xie, X; Zhang, L | 1 |
| Li, Y; Liu, Q; Ma, X; Wang, A; Wei, F; Zhang, H | 1 |
| Becker, M; Bezdek, S; Bieber, K; Gross, N; Gullberg, D; Gupta, Y; Hdnah, A; Ibrahim, SM; Künzel, S; Ludwig, RJ; Mousavi, S; Prüssmann, J; Sadik, CD; Sayegh, JP; Sezin, T; Vorobyev, A | 1 |
| Chan, CI; Chen, SJ; Hseu, JH; Hseu, YC; Vadivalagan, C; Wu, PY; Yang, HL; Yen, HR | 1 |
| Cai, Z; Wang, W; Zeng, Y; Zhang, Y | 1 |
| Chen, H; Chen, Q; Chen, S; Chen, X; Chen, Y; Gu, H; Li, M; Lian, N; Yang, Y; Zhang, Y | 1 |
| Arnold-Schild, D; Bartneck, J; Grabbe, S; Hartmann, AK; Klein, M; Langguth, P; Meiser, SL; Muth, S; Pielenhofer, J; Probst, HC; Radsak, MP; Schild, H; Stassen, M | 1 |
| Chen, X; Fang, Z; Huang, B; Jiang, R; Wang, Y; Yin, M | 1 |
| Funasaka, Y; Kanda, N; Saeki, H; Yamamoto, M; Yoshida, M | 1 |
| Bai, J; Fu, D; Guo, Y; Hu, H; Li, J; Li, M; Qu, Z; Si, W; Song, G; Song, X; Tian, Z; Wang, K; Wu, M; Wu, X; Xu, M; Yan, D; Yang, Z; Zhou, X | 1 |
| Guo, Q; Guo, ZX; Huang, ZZ; Luo, YJ; Mai, SZ; Shi, ZR; Tang, ZQ; Xiong, H; Xu, M; Xu, Y | 1 |
| Chen, W; He, Q; Long, J; Shi, Q; Zhang, B | 1 |
| Feng, F; Feng, Z; Lyu, R; Ma, T | 1 |
| Du, Z; Li, N; Lu, C; Wu, D; Yuan, X; Zhang, M; Zhu, W | 1 |
| Cao, J; Che, D; Gao, J; Geng, S; Han, H; Hao, Y; He, L; Kong, S; Liu, R; Peng, B; Shi, J; Wang, J; Zhang, Y; Zheng, Y | 1 |
| Fallon, PG; Hernández-Santana, YE; Leon, G; St Leger, D; Walsh, PT | 1 |
| Di, T; Fu, J; Guo, J; Li, N; Li, P; Meng, Y; Wang, Y; Zhang, L; Zhao, J | 1 |
| Fujii, K; Mizutani, Y; Saito, K; Seishima, M; Yamamoto, Y | 1 |
| Gyulai, R; Horváth, S; Kemény, Á; Pintér, E | 1 |
| Fu, J; Li, P; Wang, Y; Zeng, Z; Zhang, L | 1 |
| Dang, EL; Fu, M; Qiao, HJ; Wang, G; Xiao, CY; Zhang, C; Zhu, ZL | 1 |
| Chen, W; Hu, X; Jiang, J; Liu, J; Liu, W; Qie, C; Xie, X | 1 |
| Hayashi, M; Murayama, T; Otake, S; Otsubaki, T; Ueda, Y; Uesato, N | 1 |
| Aira, LE; Debes, GF | 1 |
| Cui, Y; Fei, WM; Guo, XD; Hao, YY; Liang, L; Zhang, C; Zhao, ZQ | 1 |
| Chung, BY; Kang, SY; Kim, HO; Kim, HR; Kim, JC; Park, CW | 1 |
| Chang, L; Fujita, Y; Hashimoto, K; Hashimoto, Y; Ishima, T; Matsue, H; Shinno-Hashimoto, H; Wei, Y | 1 |
| Alavala, S; Jerald, MK; Nalban, N; Sangaraju, R; Sistla, R | 1 |
| Anwaar, S; Ashraf, A; Cai, P; Ganguly, D; Huang, J; Liu, B; Liu, X; Lou, J; Ni, H; Rahaman, O; Wang, L; Wen, J; Yang, CY; Zhao, J | 1 |
| Chan, TC; Chang, WY; Huang, WC; Krueger, JG; Lee, MS; Tsai, TF | 1 |
| Asano, Y; Miyagaki, T; Oka, T; Sato, S; Shibata, S; Sugaya, M; Takahashi, N; Takahashi, T | 1 |
| Balázs, B; Bodnár, VÁ; Bolla, SB; Csányi, E; Erős, G; Kemény, L; Manczinger, M; Papp, BT; Szabó, K; Szél, E | 1 |
| Cho, S; Jin, SP; Kim, MW; Koh, SJ; Lee, DH; Park, HS; Yoon, HS; Yu, DA; Yun, HT | 1 |
| Chuang, TH; Hsu, LC; Lai, CY; Lin, KI; Su, YW | 1 |
| Adachi, A; Dainichi, T; Egawa, G; Ginhoux, F; Honda, T; Ikuta, K; Kabashima, K; Kitoh, A; Kunisawa, J; Nagatake, T; Nakamizo, S; Nomura, T; Otsuka, A | 1 |
| Chen, H; Duan, X; Gan, L; He, M; Hu, F; Liu, X; Ming, Z; Poorun, D; Wang, X; Xu, J; Zhang, S; Zhu, K | 1 |
| Kim, DS; Kim, HJ; Kim, SH; Kim, TG; Lee, M; Lee, MG; Park, JY | 1 |
| Fukushige, T; Hashiguchi, T; Higashi, Y; Ibusuki, A; Kanekura, T; Yamakuchi, M | 1 |
| Kainoh, M; Koga, Y; Takahashi, T | 1 |
| Palmer, G; Palomo, J; Rodriguez, E; Talabot-Ayer, D; Troccaz, S | 1 |
| Adachi, A; Arita, M; Dainichi, T; Egawa, G; Hanakawa, S; Honda, T; Kabashima, K; Kitoh, A; Kusuba, N; Miyachi, Y; Murata, T; Nakajima, S; Nakashima, C; Narumiya, S; Ono, S; Otsuka, A; Sawada, Y; Ueharaguchi, Y | 1 |
| Chao, YH; Chu, CL; Lee, YR; Lin, CC; Lin, FC; Pan, YG; Wu, JJ | 1 |
| Elizei, SS; Ghahary, A; Ghoreishi, M; Kilani, R; Mahmoudi, S; Pakyari, M | 1 |
| Carey, CD; Diaz-Perez, JA; Falo, LD; Killeen, ME; Mathers, AR; Yang, Y | 1 |
| Huang, J; Lin, X; Songyang, Z; Wang, M; Zhang, S; Zhao, X; Zheng, G | 1 |
| Blanco, L; Calvo, A; Espuelas, S; Fernández-Rubio, C; Irache, JM; Larrea, E; Moreno, E; Nguewa, P; Sanmartín, C; Schwartz, J | 1 |
| Adame, S; Adhami, VM; Babatunde, AS; Bharali, DJ; Chamcheu, JC; Esnault, S; Longley, BJ; Massey, RJ; Mousa, SA; Mukhtar, H; Siddiqui, IA; Wood, GS | 1 |
| Kang, NW; Kim, DD; Kim, KT; Kim, MH; Lee, JY; Lee, SY; Park, JH; Sohn, SY | 1 |
| Davis, R; Griffith, AD; Hadiono, M; Pietro, A; Popkin, DL; Yang, JS; Zaidi, AK | 1 |
| Bezdek, S; Bucala, R; Busch, H; Dahlke, M; Leng, L; Mousavi, S; Rades, D; Sadik, CD; Zillikens, D | 1 |
| Harada, K; Iwakura, Y; Nakae, S; Numata, T; Shimura, E; Sudo, K; Tsuboi, R; Yamaguchi, S; Yoshizaki, T | 1 |
| Aoki, R; Arai, MM; Harada, Y; Hisamatsu, T; Kanai, T; Kiyohara, H; Koda, Y; Mikami, Y; Miyamoto, K; Mizuno, S; Naganuma, M; Nomura, E; Sujino, T; Teratani, T | 1 |
| Cai, QW; Li, N; Li, X; Liu, LP; Lu, CJ; Wu, DH; Yu, WL; Zhang, MM; Zhu, W | 1 |
| Jeon, SH; Kim, CH; Koo, DB; Lee, JH; Lim, CY; Park, MY; Yoo, JK | 1 |
| Chen, M; Li, Y; Tang, F; Tong, M; Wen, H; Xiao, R; Zhang, G; Zhao, M | 1 |
| Chen, H; Duan, J; Duan, X; Gan, L; Liu, D; Liu, X; Lu, X; Poorun, D; Zhang, S | 1 |
| Boehncke, WH; Borowczyk-Michalowska, J; Brembilla, NC; Conrad, C; Fallon, PG; Kavanagh, RD; Kaya, G; Mylonas, A; Senra, L; Wrobel, LJ; Yawalkar, N | 1 |
| Gyulai, R; Horváth, S; Kemény, Á; Komlódi, R; Perkecz, A; Pintér, E | 1 |
| Galiano, RD; Hong, SJ; Mao, R; Mustoe, TA; Qi, S; Xie, P; Zhao, J | 1 |
| Chen, J; Chen, Y; Chen, Z; Fan, H; Gu, J; Shi, Y; Wang, Y; Yu, Z; Zhang, X; Zhou, Q | 1 |
| Kuang, H; Li, T; Sun, Y; Wang, Q; Wei, Z | 1 |
| Choi, EJ; Hong, MP; Kie, JH; Kim, HR; Lee, A; Lee, HK; Lim, W; Moon, BI; Seoh, JY | 1 |
| Detmar, M; Huggenberger, R; Kerl, K; Zgraggen, S | 1 |
| Byamba, D; Jee, H; Kim, DS; Kim, DY; Kim, SH; Kim, TG; Lee, MG; Lee, SK; Park, TY; Yang, SH | 1 |
| Anderson, RR; Chen, X; Shah, D; Wang, J; Wu, MX | 1 |
| Binks, MH; Dickson, MC; Garthside, SJ; Ludbrook, VJ; Perry, HC; Wilson, PA | 1 |
| Choi, DK; Im, M; Kim, CD; Lee, JH; Lee, Y; Lee, YH; Li, ZJ; Lim, SK; Seo, YJ; Shin, JM; Sohn, KC; Yoon, TJ | 1 |
| Kim, CH; Kim, JY; Lee, AY | 1 |
| Chen, F; Chen, Y; Cheng, L; Hao, F; Niu, J; Qian, T; Shi, X; Song, Z; Wang, H; Yan, H; Zhai, Z; Zhang, D; Zhang, N; Zhong, B | 1 |
| Ayithan, N; Hwang, ST; Imai, Y; Wang, L; Wu, X; Yuan, Y | 1 |
| Fan, H; Harris, J; Jones, SA; Morand, EF; Perera, DN; Russ, BE | 1 |
| Asano, Y; Hau, CS; Kadono, T; Kadowaki, T; Kamata, M; Kubota, N; Kurokawa, M; Masamoto, Y; Mitsui, A; Sato, S; Shibata, S; Sugaya, M; Tada, Y; Yamauchi, T | 1 |
| Ando, N; Aoki, R; Ishimaru, K; Nakamura, Y; Nakao, A; Ogawa, H; Okumura, K; Shibata, S; Shimada, S | 1 |
| Blackshear, PJ; Curtis, AD; Flake, GP; Hill, GD; Lai, WS; Mannie, MD; Patial, S; Stumpo, DJ | 1 |
| Morimura, S; Oka, T; Sato, S; Sugaya, M | 1 |
| Cavanagh, J; Graham, GJ; McColl, A; Nerurkar, L; Thomson, CA | 1 |
| Bezdek, S; Hdnah, A; Ibrahim, S; Ludwig, RJ; Mousavi, S; Sadik, CD; Sezin, T; Zillikens, D | 1 |
| Chong, HT; Cowin, AJ; Ibbetson, J; Kopecki, Z; Sidhu, S; Yang, GN | 1 |
| Liu, J; Luo, H; OuYang, Q; Pan, Y; Xuan, C | 1 |
| Bando, Y; Honma, M; Igawa, S; Iinuma, S; Ishida-Yamamoto, A; Kishibe, M; Saito, N; Yoshida, S | 1 |
| Botto, M; Buang, N; Cook, HT; Crawford, G; Dazzi, F; Giacomassi, C; Ling, GS; Scott, D; Strid, J | 1 |
| Cho, KA; Kim, JY; Lee, KH; Park, M; Woo, SY | 1 |
| Gudjonsson, JE; Hawkes, JE; Ward, NL | 1 |
| Cooper, KD; Fu, PF; Griffith, AD; McCormick, TS; Popkin, DL; Soler, DC; Young, AE | 1 |
| Chan, MP; Zimarowski, MJ | 1 |
| de Heusch, M; Dumoutier, L; Dunussi-Joannopoulos, K; Fouser, LA; Hendrickx, E; Lemaire, MM; Renauld, JC; Van Belle, AB; Warnier, G | 1 |
| Dapavo, P; Hirsch, E; Ji, H; Okkenhaug, K; Perino, A; Roller, A; Soro, E | 1 |
| Baker, D; Edwards, L; Eron, L; Ferenczy, A; Fox, TL; Hougham, AJ; Owens, ML; Schmitt, KA | 1 |
| Arndt, KA; Dover, JS; Lipper, GM | 1 |
2 review(s) available for imiquimod and Dermatitis
| Article | Year |
|---|---|
Methodological shortcomings in the reports of the imiquimod psoriatic model.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Mice; Psoriasis; Skin | 2022 |
Natural Modulators of Endosomal Toll-Like Receptor-Mediated Psoriatic Skin Inflammation.
Topics: Aminoquinolines; Animals; Anti-Inflammatory Agents, Non-Steroidal; Curcumin; Cytokines; Dermatitis; Endosomes; Humans; Imiquimod; Indazoles; Isonicotinic Acids; Mice; Psoriasis; Resveratrol; Signal Transduction; Skin; Stilbenes; Toll-Like Receptor 7; Toll-Like Receptor 8; Toll-Like Receptor 9; Toll-Like Receptors | 2017 |
1 trial(s) available for imiquimod and Dermatitis
| Article | Year |
|---|---|
Self-administered topical 5% imiquimod cream for external anogenital warts. HPV Study Group. Human PapillomaVirus.
Topics: Adjuvants, Immunologic; Administration, Cutaneous; Adult; Aminoquinolines; Anus Diseases; Condylomata Acuminata; Dermatitis; Double-Blind Method; Drug Administration Schedule; Erythema; Female; Follow-Up Studies; Genital Diseases, Female; Genital Diseases, Male; Humans; Imiquimod; Incidence; Interferon Inducers; Male; Ointments; Pharmaceutical Vehicles; Placebos; Recurrence; Remission Induction; Safety; Self Administration; Treatment Outcome | 1998 |
138 other study(ies) available for imiquimod and Dermatitis
| Article | Year |
|---|---|
Positive Allosteric Modulation of A
Topics: Adenosine; Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Immunity; Interleukin-17; Interleukin-23; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2022 |
Cutaneous Liver X Receptor Activation Prevents the Formation of Imiquimod-Induced Psoriatic Dermatitis.
Topics: Administration, Cutaneous; Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Liver X Receptors; Psoriasis; Skin | 2022 |
High salt diet impairs dermal tissue remodeling in a mouse model of IMQ induced dermatitis.
Topics: Animals; Biomarkers; Body Weight; Cell Movement; Cells, Cultured; Cytokines; Dermatitis; Dermis; Disease Models, Animal; Extracellular Matrix; Fibroblasts; Humans; Imiquimod; Inflammation; Inflammation Mediators; Leukocytes, Mononuclear; Male; Mice, Inbred C57BL; Probiotics; Sodium Chloride, Dietary | 2021 |
NF-κB1 Contributes to Imiquimod-Induced Psoriasis-Like Skin Inflammation by Inducing Vγ4
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Interleukin-17; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2022 |
Lymphatic Dysfunction Exacerbates Cutaneous Tumorigenesis and Psoriasis-Like Skin Inflammation through Accumulation of Inflammatory Cytokines.
Topics: Animals; Carcinogenesis; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Lymphatic System; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2022 |
Decreased microRNA-126 expression in psoriatic CD4
Topics: Animals; Cell Differentiation; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Mice; Mice, Inbred BALB C; MicroRNAs; Psoriasis; Skin; Th17 Cells | 2022 |
LCN2 Mediates Skin Inflammation in Psoriasis through the SREBP2‒NLRC4 Axis.
Topics: Animals; Apoptosis Regulatory Proteins; Calcium-Binding Proteins; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Keratinocytes; Lipocalin-2; Mice; Psoriasis; Skin; Sterol Regulatory Element Binding Protein 2 | 2022 |
Selenium-Rich Yeast Peptide Fraction Ameliorates Imiquimod-Induced Psoriasis-like Dermatitis in Mice by Inhibiting Inflammation via MAPK and NF-κB Signaling Pathways.
Topics: Animals; Cell Line; Dermatitis; Disease Models, Animal; Female; Humans; Hydrogen Peroxide; Imiquimod; Inflammation; Interleukin-17; Keratinocytes; Mice; Mice, Inbred BALB C; NF-kappa B; Peptides; Psoriasis; RAW 264.7 Cells; Selenium; Signal Transduction; Skin; Yeasts | 2022 |
Human umbilical cord-derived mesenchymal stem cells ameliorate psoriasis-like dermatitis by suppressing IL-17-producing γδ T cells.
Topics: Animals; Dermatitis; Humans; Imiquimod; Inflammation; Interleukin-17; Mesenchymal Stem Cell Transplantation; Mesenchymal Stem Cells; Mice; Psoriasis; T-Lymphocytes; Umbilical Cord | 2022 |
3, 3'- diindolylmethane hinders IL-17A/IL-17RA interaction and mitigates imiquimod-induced psoriasiform in mice.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Indoles; Interleukin-17; Janus Kinases; Keratinocytes; Mice; Psoriasis; Signal Transduction; Skin; STAT Transcription Factors; Vascular Endothelial Growth Factor A | 2022 |
Transglutaminase 3 Attenuates Skin Inflammation in Psoriasis by Inhibiting NF-κB Activation through Phosphorylated STAT3-TET3 Signaling.
Topics: Animals; Cysteamine; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Keratinocytes; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; RNA, Small Interfering; Skin; STAT3 Transcription Factor; Transglutaminases | 2022 |
The Protective Role of pVHL in Imiquimod-Induced Psoriasis-like Skin Inflammation.
Topics: Animals; Dermatitis; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; Imiquimod; Inflammation; Interleukin-17; Mice; Psoriasis; Tumor Necrosis Factor-alpha; Von Hippel-Lindau Tumor Suppressor Protein | 2022 |
The Essential Oil Derived from
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Interleukin-1; Interleukin-17; Interleukin-23; Interleukin-6; Keratinocytes; Mice; Mice, Inbred BALB C; Oils, Volatile; Perilla frutescens; Psoriasis; Skin | 2022 |
Notch1/Hes1‑PTEN/AKT/IL‑17A feedback loop regulates Th17 cell differentiation in mouse psoriasis‑like skin inflammation.
Topics: Animals; Cell Differentiation; Dermatitis; Feedback; Imiquimod; Inflammation; Interleukin-17; Lymphadenopathy; Mechanistic Target of Rapamycin Complex 1; Mice; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Psoriasis; Skin; Splenomegaly; Th17 Cells; Transcription Factor HES-1 | 2022 |
Dietary olive oil intake aggravates psoriatic skin inflammation in mice via Nrf2 activation and polyunsaturated fatty acid imbalance.
Topics: Animals; Dermatitis; Diet; Disease Models, Animal; Fatty Acids, Unsaturated; Humans; Imiquimod; Inflammation; Keratinocytes; Mice; Mice, Inbred BALB C; NF-E2-Related Factor 2; Olive Oil; Psoriasis; Skin; Skin Diseases | 2022 |
Lithosepermic Acid Restored the Skin Barrier Functions in the Imiquimod-Induced Psoriasis-like Animal Model.
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2022 |
Tiamulin inhibits TNF-α and alleviates psoriasis-like dermatitis.
Topics: Animals; Dermatitis; Disease Models, Animal; Diterpenes; Imiquimod; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; Tumor Necrosis Factor-alpha | 2022 |
Dynamic trafficking patterns of IL-17-producing γδ T cells are linked to the recurrence of skin inflammation in psoriasis-like dermatitis.
Topics: Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammation; Interleukin-17; Mice; Psoriasis; Skin; T-Lymphocytes | 2022 |
Sis-25, a meroditerpenoid derivative with a cyclobutane scaffold, inhibits activated T cell proliferation by targeting GSK3β in vitro and in vivo.
Topics: Cell Proliferation; Cyclobutanes; Dermatitis; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Humans; Imiquimod; Immunosuppressive Agents; Phosphatidylinositol 3-Kinases | 2022 |
Functional Interplay between IL-9 and Peptide YY Contributes to Chronic Skin Inflammation.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Interleukin-9; Keratinocytes; Mice; Peptide YY; Psoriasis; Skin | 2022 |
Sinomenine Suppressed Keratinocyte Proliferation and Imiquimod-Induced Psoriasis-Like Dermatitis by Regulating lncRNA XIST.
Topics: Animals; Anti-Inflammatory Agents; Cell Proliferation; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; RNA, Long Noncoding; Skin Diseases | 2022 |
Chlorquinaldol inhibits the activation of nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing protein 3 inflammasome and ameliorates imiquimod-induced psoriasis-like dermatitis in mice.
Topics: Animals; Carrier Proteins; Caspase 1; Chlorquinaldol; Cytokines; Dermatitis; Female; Imiquimod; Inflammasomes; Interleukin-1beta; Male; Mice; Mice, Inbred C57BL; NLR Family, Pyrin Domain-Containing 3 Protein; Nucleotides; Psoriasis; Pyrin Domain | 2022 |
Effect on ethanolic extract of sechiumedule fruitson imquimod-induced psoriasis like dermatitis in wistar rats.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Male; Mice; Psoriasis; Rats; Rats, Wistar; Skin | 2022 |
Decreasing GDF15 Promotes Inflammatory Signals and Neutrophil Infiltration in Psoriasis Models.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; Neutrophil Infiltration; Psoriasis; Skin | 2023 |
Inhibition of JAK1/STAT3 pathway by 2-methoxyestradiol ameliorates psoriatic features in vitro and in an imiquimod-induced psoriasis-like mouse model.
Topics: 2-Methoxyestradiol; Animals; Cell Proliferation; Dermatitis; Disease Models, Animal; Endothelial Cells; Humans; Imiquimod; Interleukin-17; Janus Kinase 1; Keratinocytes; Mercaptoethanol; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Ointments; Psoriasis; Skin; STAT3 Transcription Factor; Vascular Endothelial Growth Factor A | 2022 |
Oestrogen inhibits psoriasis-like dermatitis induced by imiquimod in mice in relation to increased IL-10 producing cells despite elevated expression of IL-22, IL-23, IL-17 mRNA.
Topics: Animals; Dermatitis; Disease Models, Animal; Estrogens; Female; Imiquimod; Inflammation; Interleukin-10; Interleukin-17; Interleukin-22; Interleukin-23; Interleukins; Mice; Mice, Inbred BALB C; Psoriasis; RNA, Messenger | 2023 |
Allicin ameliorates imiquimod-induced psoriasis-like skin inflammation via disturbing the interaction of keratinocytes with IL-17A.
Topics: Animals; Dermatitis; Disease Models, Animal; Guinea Pigs; Imiquimod; Inflammation; Interleukin-17; Keratinocytes; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; Rabbits; Skin | 2023 |
A Dietary Oxysterol, 7-Ketocholesterol, Exacerbates Imiquimod-Induced Psoriasis-like Dermatitis in Steatohepatitic Mice.
Topics: Animals; Dermatitis; Diet, High-Fat; Disease Models, Animal; Imiquimod; Ketocholesterols; Mice; Mice, Inbred C57BL; Non-alcoholic Fatty Liver Disease; Oxysterols; Psoriasis | 2022 |
FcγRIIB inhibits inflammation in a murine model of psoriasis.
Topics: Animals; Dermatitis; Disease Models, Animal; Forkhead Transcription Factors; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2022 |
Inhibition of Serum- and Glucocorticoid-Regulated Protein Kinase-1 Aggravates Imiquimod-Induced Psoriatic Dermatitis and Enhances Proinflammatory Cytokine Expression through the NF-kB Pathway.
Topics: Animals; Cytokines; Dermatitis; Glucocorticoids; Imiquimod; Mice; NF-kappa B; Protein Kinases; Psoriasis; Toll-Like Receptor 7 | 2023 |
Narciclasine inhibits phospholipase A2 and regulates phospholipid metabolism to ameliorate psoriasis-like dermatitis.
Topics: Animals; Anti-Inflammatory Agents; Dermatitis; Group IV Phospholipases A2; Imiquimod; Lipid Metabolism; Mice; Phospholipids; Psoriasis | 2022 |
Potential skin health promoting benefits of costunolide: a therapeutic strategy to improve skin inflammation in imiquimod-induced psoriasis.
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Health Promotion; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; Psoriasis; Sesquiterpenes; Skin | 2023 |
Imiquimod induces skin inflammation in humanized BRGSF mice with limited human immune cell activity.
Topics: Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammation; Mice; Psoriasis; Skin | 2023 |
PD-L1 Enhanced by cis-Urocanic Acid on Langerhans Cells Inhibits Vγ4
Topics: Animals; B7-H1 Antigen; Dermatitis; Humans; Imiquimod; Inflammation; Interleukin-23; Langerhans Cells; Mice; Psoriasis; Ultraviolet Rays; Urocanic Acid | 2023 |
IL-33-mediated activation of mast cells is involved in the progression of imiquimod-induced psoriasis-like dermatitis.
Topics: Animals; Dermatitis; Imiquimod; Interleukin-33; Mast Cells; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2023 |
Fufang Shengdi mixture alleviates psoriasis-like skin inflammation via promoting Annexin-A proteins expression.
Topics: Animals; Annexins; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Leukocytes, Mononuclear; Mice; Mice, Inbred BALB C; Psoriasis; Skin; Skin Diseases; Tumor Necrosis Factor-alpha | 2023 |
Isostearic acid is an active component of imiquimod formulations used to induce psoriaform disease models.
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; Skin; Toll-Like Receptor 7 | 2023 |
Palbociclib blocks neutrophilic phosphatidylinositol 3-kinase activity to alleviate psoriasiform dermatitis.
Topics: Animals; Anti-Inflammatory Agents; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammation; Mice; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Psoriasis | 2023 |
Treg cells require Izumo1R to regulate γδT cell-driven inflammation in the skin.
Topics: Animals; Dermatitis; Imiquimod; Inflammation; Mice; Psoriasis; Receptors, Cell Surface; Skin; Skin Diseases; T-Lymphocytes, Regulatory | 2023 |
Topics: Caspase 1; Caspases; Cornus; Dermatitis; DNA-Binding Proteins; HEK293 Cells; Humans; Imiquimod; Inflammasomes; Inflammation; Interleukin-1beta; NLR Family, Pyrin Domain-Containing 3 Protein; Plant Extracts; Psoriasis; Seeds | 2023 |
mRNA COVID 19 vaccine-induced recall dermatitis after topical imiquimod.
Topics: Administration, Topical; Aminoquinolines; COVID-19; COVID-19 Vaccines; Dermatitis; Humans; Imiquimod; RNA, Messenger | 2023 |
Piperine ameliorates psoriatic skin inflammation by inhibiting the phosphorylation of STAT3.
Topics: Alkaloids; Animals; Cytokines; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; Phosphorylation; Psoriasis; Skin; STAT3 Transcription Factor | 2023 |
TRPV4 Regulates the Development of Psoriasis by Controlling Adenosine Triphosphate Expression in Keratinocytes and the Neuroimmune System.
Topics: Adenosine Triphosphate; Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; Mice, Knockout; Neuropeptides; Psoriasis; Skin; TRPV Cation Channels | 2023 |
SIRT3 alleviates imiquimod-induced psoriatic dermatitis through deacetylation of XBP1s and modulation of TLR7/8 inducing IL-23 production in macrophages.
Topics: Animals; Dermatitis; Imiquimod; Inflammation; Interleukin-23; Macrophages; Mice; Psoriasis; Sirtuin 3; Toll-Like Receptor 7; X-Box Binding Protein 1 | 2023 |
Procyanidin B2 3,3''-di-O-gallate ameliorates imiquimod-induced skin inflammation by suppressing TLR7 signaling through the inhibition of endosomal acidification in dendritic cells.
Topics: Animals; Cytokines; Dendritic Cells; Dermatitis; Endosomes; Hydrogen-Ion Concentration; Imiquimod; Inflammation; Mice; Toll-Like Receptor 7 | 2023 |
Centella asiatica alleviates psoriasis through JAK/STAT3-mediated inflammation: An in vitro and in vivo study.
Topics: Animals; Antioxidants; Centella; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; Skin | 2023 |
Liangxue Jiedu formula improves imiquimod-induced psoriasiform dermatitis with circadian desynchrony by regulating Th17 cell differentiation based on network pharmacological analysis.
Topics: Animals; Cell Differentiation; Dermatitis; Disease Models, Animal; Imiquimod; Interleukin-17; Mice; Mice, Inbred BALB C; Psoriasis; Skin; Th17 Cells | 2023 |
Anti-interleukin 33 treatment alleviates psoriatic dermatitis in mice induced imiquimod.
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Interleukin-33; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2023 |
NEK2 overexpression aggravates IL-22-induced keratinocyte proliferation and cytokine level increases and IMQ-induced psoriasis-like dermatitis.
Topics: Animals; Cell Proliferation; Cytokines; Dermatitis; DNA; Imiquimod; Inflammation; Interleukin-22; Keratinocytes; Mice; Protein Kinases; Psoriasis; Skin | 2023 |
PARP2 promotes inflammation in psoriasis by modulating estradiol biosynthesis in keratinocytes.
Topics: Animals; Aromatase; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammation; Keratinocytes; Mice; Mice, Inbred BALB C; NF-kappa B; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases; Psoriasis; RNA, Messenger; Skin | 2023 |
Gentiopicroside-Loaded Chitosan Nanoparticles Inhibit TNF-α-Induced Proliferation and Inflammatory Response in HaCaT Keratinocytes and Ameliorate Imiquimod-Induced Dermatitis Lesions in Mice.
Topics: Animals; Cell Proliferation; Chitosan; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Interleukin-17; Interleukin-6; Keratinocytes; Mice; Mice, Inbred BALB C; Nanoparticles; Psoriasis; Tumor Necrosis Factor-alpha; Vascular Endothelial Growth Factor A | 2023 |
IL-17A exacerbates psoriasis in a STAT3 overexpressing mouse model.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Interleukin-17; Mice; Psoriasis; Skin | 2023 |
Evidence on the therapeutic role of thiolutin in imiquimod-induced psoriasis-like skin inflammation in mice.
Topics: Animals; Dermatitis; Forkhead Transcription Factors; Imiquimod; Inflammasomes; Inflammation; Mice; NLR Family, Pyrin Domain-Containing 3 Protein; Psoriasis | 2023 |
Forward genetics and functional analysis highlight Itga11 as a modulator of murine psoriasiform dermatitis.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Integrin alpha Chains; Mice; Psoriasis; Skin | 2023 |
Tranexamic acid improves psoriasis-like skin inflammation: Evidence from in vivo and in vitro studies.
Topics: Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Inflammasomes; Inflammation; Interleukin-17; Keratin-17; Keratinocytes; Mice; Mice, Inbred BALB C; NF-E2-Related Factor 2; NF-kappa B; NLR Family, Pyrin Domain-Containing 3 Protein; Psoriasis; Skin; Tranexamic Acid | 2023 |
Curcumin alleviates imiquimod-induced psoriasis-like inflammation and regulates gut microbiota of mice.
Topics: Animals; Curcumin; Dermatitis; Gastrointestinal Microbiome; Imiquimod; Inflammation; Interleukin-23; Mice; Tumor Necrosis Factor-alpha | 2023 |
Gasdermin D-mediated keratinocyte pyroptosis as a key step in psoriasis pathogenesis.
Topics: Animals; Dermatitis; Disulfiram; Gasdermins; Imiquimod; Inflammation; Keratinocytes; Ki-67 Antigen; Mice; Psoriasis; Pyroptosis | 2023 |
Optimized dithranol-imiquimod-based transcutaneous immunization enables tumor rejection.
Topics: Adjuvants, Immunologic; Animals; Anthralin; CD8-Positive T-Lymphocytes; Dermatitis; Humans; Imiquimod; Immunization; Mice; Mice, Inbred C57BL; Neoplasms; Vaccination | 2023 |
Depletion of G9A attenuates imiquimod-induced psoriatic dermatitis via targeting EDAR-NF-κB signaling in keratinocyte.
Topics: Animals; Dermatitis; Edar Receptor; Humans; Imiquimod; Keratinocytes; Mice; NF-kappa B; Psoriasis; Receptors, Ectodysplasin; Receptors, Tumor Necrosis Factor | 2023 |
Dietary Fiber Inulin Improves Murine Imiquimod-Induced Psoriasis-like Dermatitis.
Topics: Animals; Dermatitis; Imiquimod; Interleukin-17; Inulin; Ki-67 Antigen; Mice; Propionates; Psoriasis; RNA, Ribosomal, 16S | 2023 |
Staphylococcus warneri strain XSB102 exacerbates psoriasis and promotes keratinocyte proliferation in imiquimod-induced psoriasis-like dermatitis mice.
Topics: Animals; Cell Proliferation; Dermatitis; Disease Models, Animal; Homeodomain Proteins; Humans; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; Psoriasis; Repressor Proteins; Skin; Staphylococcus | 2023 |
Artesunate alleviates imiquimod-induced psoriasis-like dermatitis in BALB/c mice.
Topics: Animals; Anti-Inflammatory Agents; Artesunate; Dermatitis; Imiquimod; Intraepithelial Lymphocytes; Lymph Nodes; Male; Mice, Inbred BALB C; Psoriasis; Skin; Th17 Cells | 2019 |
Ginsenoside Rg1 abolish imiquimod-induced psoriasis-like dermatitis in BALB/c mice via downregulating NF-κB signaling pathway.
Topics: Animals; Dermatitis; Disease Models, Animal; Down-Regulation; Ginsenosides; Humans; Imiquimod; Interleukin-22; Interleukin-23; Interleukins; Male; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; Signal Transduction; Transcription Factor RelA; Tumor Necrosis Factor-alpha | 2019 |
[Esculentoside reduces expression of Th17 cell-related cytokines in the imiquimod-induced psoriasis-like dermatitis mouse model].
Topics: Animals; Cytokines; Dermatitis; Female; Imiquimod; Mice; Mice, Inbred BALB C; Oleanolic Acid; Psoriasis; Random Allocation; Saponins; Skin; Th17 Cells | 2019 |
Taxifolin attenuates IMQ-induced murine psoriasis-like dermatitis by regulating T helper cell responses via Notch1 and JAK2/STAT3 signal pathways.
Topics: Animals; Cell Line; Cell Proliferation; Cyclosporine; Cytokines; Dermatitis; Humans; Imiquimod; Inflammation; Janus Kinase 2; Keratinocytes; Lipopolysaccharides; Male; Mice; Mice, Inbred BALB C; Models, Animal; Psoriasis; Quercetin; Receptor, Notch1; Signal Transduction; Skin; STAT3 Transcription Factor; T-Lymphocytes; Transcription Factors | 2020 |
Imiquimod-related dermatitis is mainly mediated by mast cell degranulation via Mas-related G-protein coupled receptor B2.
Topics: Animals; Calcium Signaling; Cell Degranulation; Dermatitis; Disease Models, Animal; Drug-Related Side Effects and Adverse Reactions; HEK293 Cells; Humans; Imiquimod; Male; Mast Cells; Mice; Mice, Inbred C57BL; Mice, Knockout; Receptors, G-Protein-Coupled | 2020 |
Keratinocyte interleukin-36 receptor expression orchestrates psoriasiform inflammation in mice.
Topics: Animals; Cytokines; Dermatitis; Female; Humans; Imiquimod; Inflammation; Intraepithelial Lymphocytes; Keratinocytes; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Psoriasis; Receptors, Interleukin-1; Skin | 2020 |
Stress aggravates and prolongs imiquimod-induced psoriasis-like epidermal hyperplasis and IL-1β/IL-23p40 production.
Topics: Animals; Anxiety; Dendritic Cells; Dermatitis; Emotions; Epidermis; Hyperplasia; Imiquimod; Inflammation; Interleukin-12 Subunit p40; Interleukin-1beta; Male; Mice, Inbred BALB C; Mice, Inbred C57BL; Neurokinin-1 Receptor Antagonists; Neurotransmitter Agents; Nociceptors; Psoriasis; Receptors, Neurokinin-1; Stress, Psychological; Substance P; Up-Regulation | 2020 |
Topics: Animals; Cytokines; Dermatitis; Female; Gene Expression Regulation; Imiquimod; Indoleamine-Pyrrole 2,3,-Dioxygenase; Inflammation; Interleukin-17; Interleukin-23 Subunit p19; Mice, Inbred C57BL; Mice, Knockout; Psoriasis; Skin; Tumor Necrosis Factor-alpha | 2020 |
A Localized Aldara (5% Imiquimod)-Induced Psoriasiform Dermatitis Model in Mice Using Finn Chambers.
Topics: Animals; Dermatitis; Disease Models, Animal; Female; Imiquimod; Mice; Mice, Inbred C57BL; Psoriasis; Skin | 2020 |
4'-O-β-D-glucosyl-5-O-methylvisamminol ameliorates imiquimod-induced psoriasis-like dermatitis and inhibits inflammatory cytokines production by suppressing the NF-κB and MAPK signaling pathways.
Topics: Animals; Chromones; Cytokines; Dermatitis; Glucosides; Imiquimod; Inflammation; Lipopolysaccharides; MAP Kinase Signaling System; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis | 2020 |
Small interfering RNA targeting of keratin 17 reduces inflammation in imiquimod-induced psoriasis-like dermatitis.
Topics: Animals; Dermatitis; Disease Models, Animal; Female; Humans; Imiquimod; Inflammation; Keratin-17; Mice; Mice, Inbred BALB C; Psoriasis; RNA, Small Interfering; Skin | 2020 |
M351-0056 is a novel low MW compound modulating the actions of the immune-checkpoint protein VISTA.
Topics: Animals; Cytokines; Dermatitis; Imiquimod; Immune Checkpoint Proteins; Inflammation; Ligands; Lymphocyte Activation; Membrane Proteins; Mice | 2021 |
Topical Application of BMS-509744, a Selective Inhibitor of Interleukin-2-Inducible T Cell Kinase, Ameliorates Imiquimod-Induced Skin Inflammation in Mice.
Topics: Animals; CD3 Complex; Dermatitis; Imiquimod; Interleukin-17; Interleukin-23; Mice; Mice, Inbred BALB C; Protein-Tyrosine Kinases; Psoriasis; Skin; Th17 Cells; Tumor Necrosis Factor-alpha | 2021 |
Skin-Homing Regulatory B Cells Required for Suppression of Cutaneous Inflammation.
Topics: Animals; B-Lymphocytes, Regulatory; Cell Movement; Dermatitis; Disease Models, Animal; Female; Humans; Imiquimod; Interleukin-10; Male; Mice; Psoriasis; Skin | 2021 |
Improved imiquimod-induced psoriasis like dermatitis using microneedles in mice.
Topics: Administration, Cutaneous; Animals; Cytokines; Dermatitis; Disease Models, Animal; Drug Delivery Systems; Female; Imiquimod; Inflammation; Mice; Mice, Inbred BALB C; Needles; Pharmaceutical Preparations; Polyesters; Polymers; Psoriasis; Skin; Swine | 2021 |
Rapamycin Alleviates 2,3,7,8-Tetrachlorodibenzo-p-dioxin-Induced Aggravated Dermatitis in Mice with Imiquimod-Induced Psoriasis-Like Dermatitis by Inducing Autophagy.
Topics: Animals; Autophagy; Cell Differentiation; Cells, Cultured; Cytochrome P-450 CYP1A1; Dermatitis; Gene Expression Regulation; Humans; Imiquimod; Inflammation; Keratinocytes; Mice; NADPH Oxidase 4; NF-E2-Related Factor 2; Polychlorinated Dibenzodioxins; Psoriasis; Receptors, Aryl Hydrocarbon; Sirolimus | 2021 |
Abnormal composition of microbiota in the gut and skin of imiquimod-treated mice.
Topics: Animals; Dermatitis; Disease Models, Animal; Female; Gastrointestinal Microbiome; Imiquimod; Lactic Acid; Mice; Mice, Inbred C57BL; Microbiota; Psoriasis; Skin; Staphylococcus; Succinic Acid | 2021 |
Galangin ameliorates Imiquimod-Induced psoriasis-like skin inflammation in BALB/c mice via down regulating NF-κB and activation of Nrf2 signaling pathways.
Topics: Animals; Anti-Inflammatory Agents; Antioxidants; Body Weight; Cytokines; Dermatitis; Disease Models, Animal; Down-Regulation; Flavonoids; Heme Oxygenase-1; Imiquimod; Male; Membrane Proteins; Mice, Inbred BALB C; NF-E2-Related Factor 2; NF-kappa B; Oxidative Stress; Peroxidase; Psoriasis; Signal Transduction; Spleen | 2021 |
The RNase MCPIP3 promotes skin inflammation by orchestrating myeloid cytokine response.
Topics: Adaptor Proteins, Signal Transducing; Animals; Cell Cycle Proteins; Chemokine CCL2; Cytokines; Dendritic Cells; Dermatitis; Endoribonucleases; Epigenomics; Humans; Imiquimod; Inflammation; Interleukin-6; Macrophages; Mice; Mice, Knockout; Myeloid Cells; Psoriasis; Ribonucleases; Skin; Tumor Necrosis Factor-alpha | 2021 |
Capsaicin attenuates imiquimod-induced epidermal hyperplasia and cutaneous inflammation in a murine model of psoriasis.
Topics: Administration, Topical; Animals; Antineoplastic Agents; Antipruritics; Capsaicin; Dermatitis; Disease Models, Animal; Epidermis; Female; Hyperplasia; Imiquimod; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2021 |
CXCL17 Attenuates Imiquimod-Induced Psoriasis-like Skin Inflammation by Recruiting Myeloid-Derived Suppressor Cells and Regulatory T Cells.
Topics: Aminoquinolines; Animals; Chemokine CCL4; Chemokine CCL5; Chemokines; Chemokines, CXC; Chemotaxis; Cytokines; Dermatitis; Epidermis; Humans; Imiquimod; Keratinocytes; Mice; Myeloid-Derived Suppressor Cells; Psoriasis; Skin; T-Lymphocytes, Regulatory | 2017 |
Drug Repurposing by Simulating Flow Through Protein-Protein Interaction Networks.
Topics: Algorithms; Animals; Cell Line; Computer Simulation; Dermatitis; Drug Evaluation, Preclinical; Drug Repositioning; Ear, External; Gene Regulatory Networks; Humans; Imiquimod; Machine Learning; Mice; Mice, Inbred BALB C; NF-kappa B; Protein Interaction Domains and Motifs; Protein Interaction Maps; Psoriasis; RNA; Support Vector Machine; Tumor Necrosis Factor-alpha | 2018 |
Imiquimod-applied Interleukin-10 deficient mice better reflects severe and persistent psoriasis with systemic inflammatory state.
Topics: Adjuvants, Immunologic; Animals; Body Weight; Cytokines; Dermatitis; Disease Models, Animal; Epidermis; Imiquimod; Inflammation; Interleukin-10; Interleukin-17; Keratinocytes; Leukocyte Common Antigens; Male; Mice; Mice, Inbred C57BL; Organ Size; Psoriasis; RNA, Messenger; Skin; Spleen; Time Factors | 2018 |
High fat diet exacerbates murine psoriatic dermatitis by increasing the number of IL-17-producing γδ T cells.
Topics: Animals; Chemokine CCL20; Dermatitis; Diet, High-Fat; Disease Models, Animal; Endothelial Cells; Imiquimod; Interleukin-17; Keratinocytes; Male; Mice, Inbred C57BL; Mice, Obese; Obesity; Psoriasis; Skin; T-Lymphocytes | 2017 |
Resolvin D1 attenuates imiquimod-induced mice psoriasiform dermatitis through MAPKs and NF-κB pathways.
Topics: Administration, Cutaneous; Aminoquinolines; Animals; Anti-Inflammatory Agents; Dermatitis; Disease Models, Animal; Docosahexaenoic Acids; Drug Evaluation, Preclinical; Humans; Imiquimod; Male; Mice; Mice, Inbred BALB C; Mitogen-Activated Protein Kinases; NF-kappa B; Psoriasis; Severity of Illness Index; Signal Transduction; Skin; Skin Cream | 2018 |
Interleukin-21 receptor signalling is not critically required for imiquimod-induced psoriasiform dermatitis in mice.
Topics: Animals; CD4-Positive T-Lymphocytes; Dermatitis; Imiquimod; Inflammation; Interferon Inducers; Interleukin-21 Receptor alpha Subunit; Interleukin-23 Subunit p19; Intraepithelial Lymphocytes; Mice; Mice, Knockout; Mice, Transgenic; Psoriasis; Receptors, Interleukin-21; Signal Transduction | 2018 |
High-fat diet exacerbates imiquimod-induced psoriasis-like dermatitis in mice.
Topics: Animals; Body Weight; Cholesterol; Cytokines; Dermatitis; Diet, High-Fat; Disease Models, Animal; Disease Progression; Hyperlipidemias; Imiquimod; Inflammasomes; Inflammation; Male; Mice; Mice, Inbred C57BL; Neutrophils; Psoriasis; Skin | 2018 |
Anti-IL-12/IL-23p40 antibody ameliorates dermatitis and skin barrier dysfunction in mice with imiquimod-induced psoriasis-like dermatitis.
Topics: Aminoquinolines; Animals; Antibodies, Monoclonal; Cell Proliferation; Dermatitis; Imiquimod; Interleukin-12; Interleukin-23; Keratinocytes; Male; Mice; Mice, Inbred BALB C; Psoriasis; Skin | 2018 |
The severity of imiquimod-induced mouse skin inflammation is independent of endogenous IL-38 expression.
Topics: Aminoquinolines; Animals; Cells, Cultured; Dermatitis; Disease Models, Animal; Drug Eruptions; Female; Imiquimod; Inflammation; Interleukin-1; Interleukins; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Severity of Illness Index; Skin | 2018 |
Thromboxane A
Topics: Animals; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Interleukin-17; Mice; Mice, Inbred C57BL; Mice, Knockout; Psoriasis; Receptors, Antigen, T-Cell, gamma-delta; Receptors, G-Protein-Coupled; Receptors, Thromboxane A2, Prostaglandin H2; T-Lymphocytes; Thromboxane A2; Thromboxane-A Synthase | 2018 |
Gold lotion from citrus peel extract ameliorates imiquimod-induced psoriasis-like dermatitis in murine.
Topics: Aminoquinolines; Animals; Citrus; Cytokines; Dermatitis; Fruit; Humans; Imiquimod; Keratinocytes; Male; Mice; Mice, Inbred BALB C; Plant Extracts; Psoriasis | 2018 |
IDO-expressing Fibroblasts Suppress the Development of Imiquimod-induced Psoriasis-like Dermatitis.
Topics: Animals; Dermatitis; Female; Fibroblasts; Flow Cytometry; Imiquimod; Immunohistochemistry; Indoleamine-Pyrrole 2,3,-Dioxygenase; Interleukin-17; Interleukin-23; Mice; Mice, Inbred BALB C; Psoriasis | 2018 |
Extracellular ATP and IL-23 Form a Local Inflammatory Circuit Leading to the Development of a Neutrophil-Dependent Psoriasiform Dermatitis.
Topics: Adenosine Triphosphate; Animals; Dermatitis; Disease Models, Animal; Extracellular Space; Female; Humans; Imiquimod; Inflammation Mediators; Interleukin-17; Interleukin-1beta; Interleukin-23; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Neutrophils; NLR Family, Pyrin Domain-Containing 3 Protein; Psoriasis; Receptors, Purinergic P2X7; Signal Transduction; Skin | 2018 |
Gain-of-Function Mutation of Card14 Leads to Spontaneous Psoriasis-like Skin Inflammation through Enhanced Keratinocyte Response to IL-17A.
Topics: Adaptor Proteins, Signal Transducing; Animals; CARD Signaling Adaptor Proteins; Cell Line; Cytokines; Dermatitis; Gain of Function Mutation; Gene Expression Regulation; Guanylate Kinases; HEK293 Cells; Humans; Imiquimod; Interleukin-17; Keratinocytes; Mice, Inbred C57BL; Mice, Knockout; NF-kappa B; Psoriasis; Signal Transduction; T-Lymphocyte Subsets; TNF Receptor-Associated Factor 6 | 2018 |
Combination of paromomycin plus human anti-TNF-α antibodies to control the local inflammatory response in BALB/ mice with cutaneous leishmaniasis lesions.
Topics: Animals; Antibodies; Antiprotozoal Agents; Cells, Cultured; Dermatitis; Disease Models, Animal; Drug Therapy, Combination; Female; Host-Pathogen Interactions; Imiquimod; Inflammation Mediators; Leishmania major; Leishmaniasis, Cutaneous; Macrophages; Mice, Inbred BALB C; Neutrophil Infiltration; Paromomycin; Skin; Tumor Necrosis Factor-alpha | 2018 |
Chitosan-based nanoformulated (-)-epigallocatechin-3-gallate (EGCG) modulates human keratinocyte-induced responses and alleviates imiquimod-induced murine psoriasiform dermatitis.
Topics: Administration, Topical; Aminoquinolines; Animals; Antineoplastic Agents; Antioxidants; Catechin; Cell Differentiation; Cells, Cultured; Chitosan; Dermatitis; Filaggrin Proteins; Humans; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; Nanoparticles; Psoriasis | 2018 |
Curcumin-loaded lipid-hybridized cellulose nanofiber film ameliorates imiquimod-induced psoriasis-like dermatitis in mice.
Topics: Administration, Topical; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cellulose; Curcumin; Dermatitis; Drug Carriers; Imiquimod; Lipids; Male; Mice; Nanofibers; Psoriasis | 2018 |
A requirement for slc15a4 in imiquimod-induced systemic inflammation and psoriasiform inflammation in mice.
Topics: Animals; Cytokines; Dermatitis; Disease Models, Animal; Imiquimod; Inflammation; Membrane Glycoproteins; Membrane Transport Proteins; Mice; Mice, Transgenic; Psoriasis; Skin; Toll-Like Receptor 7 | 2018 |
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Interleukin-23; Intramolecular Oxidoreductases; Macrophage Migration-Inhibitory Factors; Mice; Mice, Knockout; Psoriasis; Skin | 2018 |
IL-36α is involved in hapten-specific T-cell induction, but not local inflammation, during contact hypersensitivity.
Topics: Animals; Cell Movement; Dendritic Cells; Dermatitis; Dermatitis, Contact; Haptens; Imiquimod; Inflammation; Interleukin-1; Keratinocytes; Lymph Nodes; Mice, Inbred C57BL; Psoriasis; Skin; T-Lymphocytes | 2018 |
Toll-Like Receptor 7 Agonist-Induced Dermatitis Causes Severe Dextran Sulfate Sodium Colitis by Altering the Gut Microbiome and Immune Cells.
Topics: Animals; B-Lymphocytes; Cell Movement; Colitis; Dermatitis; Dextran Sulfate; Disease Progression; Fecal Microbiota Transplantation; Female; Gastrointestinal Microbiome; Hematopoietic Stem Cells; Imiquimod; Immunoglobulin D; Immunoglobulin M; Intestines; Lactobacillus; Lymph Nodes; Lymphocyte Depletion; Mice, Inbred C57BL; Permeability; Psoriasis; Toll-Like Receptor 7 | 2019 |
PSORI-CM02 alleviates IMQ-induced mouse dermatitis via differentially regulating pro- and anti-inflammatory cytokines targeting of Th2 specific transcript factor GATA3.
Topics: Animals; Anti-Inflammatory Agents; Antineoplastic Agents; Cytokines; Dermatitis; Drugs, Chinese Herbal; GATA3 Transcription Factor; Imiquimod; Inflammation Mediators; Male; Mice; Mice, Inbred BALB C; Th2 Cells | 2019 |
Anti-psoriatic effect of myeloid-derived suppressor cells on imiquimod-induced skin inflammation in mice.
Topics: Animals; Cytokines; Dermatitis; Female; Imiquimod; Mice; Mice, Inbred C57BL; Myeloid-Derived Suppressor Cells; Psoriasis; T-Lymphocytes, Regulatory; Th1 Cells; Th17 Cells | 2019 |
Rutaecarpine inhibited imiquimod-induced psoriasis-like dermatitis via inhibiting the NF-κB and TLR7 pathways in mice.
Topics: Animals; Dermatitis; Disease Models, Animal; Imiquimod; Indole Alkaloids; Male; Membrane Glycoproteins; Mice; Mice, Inbred BALB C; NF-kappa B; Psoriasis; Quinazolines; Signal Transduction; Skin; Skin Diseases; Th17 Cells; Toll-Like Receptor 7 | 2019 |
Cold atmospheric plasma ameliorates imiquimod-induced psoriasiform dermatitis in mice by mediating antiproliferative effects.
Topics: Animals; Antineoplastic Agents; Apoptosis; Dermatitis; Female; Humans; Imiquimod; Mice; Mice, Inbred BALB C; Plasma Gases; Psoriasis | 2019 |
IL-17E (IL-25) Enhances Innate Immune Responses during Skin Inflammation.
Topics: Acute Generalized Exanthematous Pustulosis; Adjuvants, Immunologic; Animals; Dermatitis; Disease Models, Animal; Female; Humans; Imiquimod; Immunity, Innate; Interleukin-17; Interleukins; Macrophages; Mice; Mice, Knockout; Neutrophils; p38 Mitogen-Activated Protein Kinases; Pyoderma Gangrenosum; Skin; Up-Regulation | 2019 |
Methodological refinement of Aldara-induced psoriasiform dermatitis model in mice.
Topics: Animals; CD11b Antigen; Cytokines; Dermatitis; Disease Models, Animal; Female; Imiquimod; Mice, Inbred C57BL; Petrolatum; Psoriasis | 2019 |
Imiquimod-induced skin inflammation is relieved by knockdown of sodium channel Na
Topics: Animals; Dermatitis; Epidermis; Female; Gene Knockdown Techniques; Imiquimod; Inflammation; Psoriasis; Rabbits; RNA Interference; Skin; Voltage-Gated Sodium Channels | 2019 |
Ginsenoside compound K ameliorates imiquimod-induced psoriasis-like dermatitis through inhibiting REG3A/RegIIIγ expression in keratinocytes.
Topics: Animals; Apoptosis; Cell Line, Tumor; Cell Proliferation; Cell Survival; Dermatitis; Female; Ginsenosides; Humans; Imiquimod; Interleukin-1; Keratinocytes; Mice; Mice, Inbred C57BL; Pancreatitis-Associated Proteins; Psoriasis; Skin | 2019 |
Withanolides, Extracted from Datura Metel L. Inhibit Keratinocyte Proliferation and Imiquimod-Induced Psoriasis-Like Dermatitis via the STAT3/P38/ERK1/2 Pathway.
Topics: Animals; Cell Proliferation; Datura metel; Dermatitis; Humans; Imiquimod; Keratinocytes; MAP Kinase Signaling System; Mice; Psoriasis; STAT3 Transcription Factor; Withanolides | 2019 |
Reactive oxygen species prevent imiquimod-induced psoriatic dermatitis through enhancing regulatory T cell function.
Topics: Acetylcysteine; Aminoquinolines; Animals; Dermatitis; Disease Progression; Glutathione Peroxidase; Glutathione Peroxidase GPX1; Hyperbaric Oxygenation; Imiquimod; Indoleamine-Pyrrole 2,3,-Dioxygenase; Mice, Inbred C57BL; NADPH Oxidases; Naphthoquinones; Psoriasis; Reactive Oxygen Species; T-Lymphocytes, Regulatory | 2014 |
An important role of the SDF-1/CXCR4 axis in chronic skin inflammation.
Topics: Aminoquinolines; Animals; Chemokine CXCL12; Chronic Disease; Dermatitis; Imiquimod; Mice; Mice, Inbred C57BL; Neovascularization, Pathologic; Receptors, CXCR4; Up-Regulation | 2014 |
Skin-penetrating methotrexate alleviates imiquimod-induced psoriasiform dermatitis via decreasing IL-17-producing gamma delta T cells.
Topics: Aminoquinolines; Animals; CD11c Antigen; CD4-Positive T-Lymphocytes; Cytokines; Dermatitis; DNA, Complementary; Female; Imiquimod; Inflammation; Interleukin-17; Interleukin-23; Methotrexate; Mice; Mice, Inbred BALB C; Peptides; Permeability; Psoriasis; Skin | 2014 |
A micro-sterile inflammation array as an adjuvant for influenza vaccines.
Topics: Adjuvants, Immunologic; Age Factors; Aminoquinolines; Animals; Dendritic Cells; Dermatitis; Imiquimod; Influenza Vaccines; Interferon Inducers; Interferon Regulatory Factor-7; Laser Therapy; Lasers; Male; Mice, Inbred BALB C; Mice, Mutant Strains; Skin; Squalene; Sus scrofa | 2014 |
A model of skin inflammation in humans leads to a rapid and reproducible increase in the interferon response signature: a potential translational model for drug development.
Topics: 2',5'-Oligoadenylate Synthetase; Adolescent; Adult; Aminoquinolines; Biopsy; Chemokine CXCL10; Dermatitis; Drug Discovery; Humans; Imiquimod; Interferon Type I; Male; MicroRNAs; Middle Aged; Models, Genetic; Protein Processing, Post-Translational; Signal Transduction; Skin; Skin Cream; Surgical Tape; Young Adult | 2015 |
Inhibitory effect of cucurbitacin B on imiquimod-induced skin inflammation.
Topics: Aminoquinolines; Base Sequence; Dermatitis; DNA Primers; Humans; Imiquimod; In Vitro Techniques; Real-Time Polymerase Chain Reaction; Triterpenes | 2015 |
Therapeutic and immunomodulatory effects of glucosamine in combination with low-dose cyclosporine a in a murine model of imiquimod-induced psoriasis.
Topics: Aminoquinolines; Animals; Cyclosporine; Dermatitis; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Therapy, Combination; Female; Glucosamine; Imiquimod; Immunomodulation; Mice; Mice, Inbred C57BL; Organ Size; Psoriasis; Spleen; T-Lymphocytes, Regulatory | 2015 |
Downregulation of TNIP1 Expression Leads to Increased Proliferation of Human Keratinocytes and Severer Psoriasis-Like Conditions in an Imiquimod-Induced Mouse Model of Dermatitis.
Topics: Aminoquinolines; Animals; CCAAT-Enhancer-Binding Protein-beta; Cell Proliferation; Cells, Cultured; Dermatitis; Disease Models, Animal; DNA-Binding Proteins; Down-Regulation; Humans; Imiquimod; Keratinocytes; Mice; Mice, Inbred BALB C; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Psoriasis; RNA Interference; Severity of Illness Index; Signal Transduction; Skin | 2015 |
Cutting Edge: PD-1 Regulates Imiquimod-Induced Psoriasiform Dermatitis through Inhibition of IL-17A Expression by Innate γδ-Low T Cells.
Topics: Aminoquinolines; Animals; Antibodies; Dermatitis; Disease Models, Animal; Gene Expression Regulation; Humans; Imiquimod; Injections, Intraperitoneal; Interleukin-17; Keratinocytes; Mice; Mice, Inbred C57BL; Mice, Knockout; Neutrophil Infiltration; Neutrophils; Programmed Cell Death 1 Receptor; Receptors, Antigen, T-Cell, alpha-beta; Receptors, Antigen, T-Cell, gamma-delta; Signal Transduction; Th17 Cells | 2015 |
GILZ regulates Th17 responses and restrains IL-17-mediated skin inflammation.
Topics: Aminoquinolines; Animals; Cell Differentiation; Cell Proliferation; Cells, Cultured; Cytokines; Dendritic Cells; Dermatitis; Enzyme-Linked Immunosorbent Assay; Flow Cytometry; Gene Expression; Humans; Imiquimod; Interleukin-17; Interleukin-6; Lipopolysaccharides; Mice, Inbred C57BL; Mice, Knockout; Psoriasis; Reverse Transcriptase Polymerase Chain Reaction; Th17 Cells; Transcription Factors | 2015 |
Adiponectin regulates psoriasiform skin inflammation by suppressing IL-17 production from γδ-T cells.
Topics: Adiponectin; Adjuvants, Immunologic; Adult; Aminoquinolines; Animals; CD4-Positive T-Lymphocytes; CD8-Positive T-Lymphocytes; Dermatitis; Female; Humans; Imiquimod; Immunoblotting; Interleukin-17; Interleukin-23; Male; Mice; Middle Aged; Psoriasis; Real-Time Polymerase Chain Reaction; Receptors, Adiponectin; Receptors, Antigen, T-Cell, gamma-delta; Reverse Transcriptase Polymerase Chain Reaction; Skin; Subcutaneous Fat | 2015 |
Circadian Gene Clock Regulates Psoriasis-Like Skin Inflammation in Mice.
Topics: Aminoquinolines; Animals; Circadian Rhythm; CLOCK Proteins; Dermatitis; Female; Imiquimod; Interleukin-23; Mice; Mice, Inbred C57BL; Mutation; Period Circadian Proteins; Psoriasis; Receptors, Antigen, T-Cell, gamma-delta; T-Lymphocytes | 2015 |
Enhanced stability of tristetraprolin mRNA protects mice against immune-mediated inflammatory pathologies.
Topics: Aminoquinolines; Animals; Arthritis, Experimental; Cells, Cultured; Collagen; Dermatitis; Encephalomyelitis, Autoimmune, Experimental; Imiquimod; Inflammation; Mice; Mice, Transgenic; Mutation; RNA, Messenger; Tristetraprolin | 2016 |
CX3CR1 deficiency attenuates imiquimod-induced psoriasis-like skin inflammation with decreased M1 macrophages.
Topics: Aminoquinolines; Animals; Cell Count; Chemokine CX3CL1; CX3C Chemokine Receptor 1; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Interleukins; Macrophages; Mice; Mice, Inbred C57BL; Mice, Knockout; Psoriasis; Receptors, Chemokine; Skin; Tumor Necrosis Factor-alpha | 2016 |
TLR7-mediated skin inflammation remotely triggers chemokine expression and leukocyte accumulation in the brain.
Topics: Aminoquinolines; Animals; Brain; CD3 Complex; Chemokines; Chemotaxis, Leukocyte; Dermatitis; Disease Models, Animal; Doublecortin Domain Proteins; Female; Flow Cytometry; Gene Expression Profiling; Imiquimod; Interferon Inducers; Leukocytes; Membrane Glycoproteins; Mice; Microtubule-Associated Proteins; Neuropeptides; Oligonucleotide Array Sequence Analysis; Phorbol Esters; RNA, Messenger; Toll-Like Receptor 7 | 2016 |
The genetic difference between C57Bl/6J and C57Bl/6N mice significantly impacts Aldara™-induced psoriasiform dermatitis.
Topics: Aminoquinolines; Animals; Dermatitis; Disease Models, Animal; Imiquimod; Mice, Inbred C57BL; Psoriasis; Skin; Species Specificity | 2017 |
Reducing Flightless I expression decreases severity of psoriasis in an imiquimod-induced murine model of psoriasiform dermatitis.
Topics: Administration, Cutaneous; Aminoquinolines; Animals; Antibodies, Monoclonal; Antibodies, Neutralizing; Carrier Proteins; Cytoskeletal Proteins; Dermatitis; Disease Models, Animal; Drug Eruptions; Female; Humans; Imiquimod; Irritants; Male; Mice, Inbred BALB C; Microfilament Proteins; Middle Aged; Psoriasis; RNA, Messenger; Toll-Like Receptor 4; Trans-Activators | 2017 |
MAD ointment ameliorates Imiquimod-induced psoriasiform dermatitis by inhibiting the IL-23/IL-17 axis in mice.
Topics: Aminoquinolines; Animals; Cell Proliferation; Centella; Dermatitis; Disease Models, Animal; Female; Humans; Imiquimod; Interleukin-17; Interleukin-23; Mice; Mice, Inbred BALB C; Ointments; Psoriasis; Signal Transduction; Skin; Th17 Cells; Triterpenes | 2016 |
Kallikrein-related peptidase 6 promotes psoriasiform skin inflammation through a protease-activated receptor 2-independent mechanism.
Topics: Aminoquinolines; Animals; Dermatitis; Epidermis; Imiquimod; Kallikreins; Mice, Inbred C57BL; Mice, Knockout; Psoriasis; Receptor, PAR-2 | 2017 |
Complement C3 Exacerbates Imiquimod-Induced Skin Inflammation and Psoriasiform Dermatitis.
Topics: Adjuvants, Immunologic; Aminoquinolines; Complement Activation; Complement C3; Dermatitis; Humans; Imiquimod; Immunity, Cellular; Skin | 2017 |
Polyinosinic-polycytidylic acid (poly(I:C)) attenuates imiquimod-induced skin inflammation in mice by increasing cutaneous PD-L1 expression.
Topics: Aminoquinolines; Animals; B7-H1 Antigen; Cell Line, Transformed; Dermatitis; Disease Models, Animal; Humans; Imiquimod; Keratinocytes; Mice; Poly I-C; Psoriasis; Toll-Like Receptor 3 | 2017 |
The Snowballing Literature on Imiquimod-Induced Skin Inflammation in Mice: A Critical Appraisal.
Topics: Aminoquinolines; Animals; Dermatitis; Dermatology; Disease Models, Animal; Humans; Imiquimod; Inflammation; Mice; Psoriasis; Research Design | 2017 |
Overexpression of AQP3 and AQP10 in the skin exacerbates psoriasiform acanthosis.
Topics: Aminoquinolines; Animals; Aquaporin 3; Aquaporins; Cell Differentiation; Dermatitis; Detergents; Epidermis; Filaggrin Proteins; Imiquimod; Intermediate Filament Proteins; Keratin-10; Keratinocytes; Membrane Proteins; Mice; Up-Regulation; Weight Loss | 2017 |
Lupus erythematosus-like reaction in imiquimod-treated skin: a report of 2 cases.
Topics: Administration, Cutaneous; Aged; Aged, 80 and over; Aminoquinolines; Antineoplastic Agents; Dermatitis; Diagnosis, Differential; Female; Humans; Hutchinson's Melanotic Freckle; Imiquimod; Keratosis, Actinic; Lupus Erythematosus, Systemic; Male; Skin Neoplasms | 2011 |
IL-22 is required for imiquimod-induced psoriasiform skin inflammation in mice.
Topics: Adjuvants, Immunologic; Aminoquinolines; Animals; Antigens, Differentiation; Chemokine CCL3; Dermatitis; Disease Models, Animal; Gene Expression Regulation; Imiquimod; Immunity, Innate; Interleukin-22; Interleukins; Mice; Mice, Knockout; Neutrophil Infiltration; Neutrophils; Psoriasis; Receptors, Antigen, T-Cell, gamma-delta; Skin; T-Lymphocytes | 2012 |
Blockade of phosphatidylinositol 3-kinase PI3Kδ or PI3Kγ reduces IL-17 and ameliorates imiquimod-induced psoriasis-like dermatitis.
Topics: Aminoquinolines; Animals; Class I Phosphatidylinositol 3-Kinases; Class Ib Phosphatidylinositol 3-Kinase; Dermatitis; Disease Models, Animal; Female; Gene Knock-In Techniques; Humans; Imiquimod; Interleukin-17; Lymph Nodes; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Middle Aged; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Psoriasis; T-Lymphocyte Subsets | 2012 |
Recent therapeutic advances in dermatology.
Topics: Adjuvants, Immunologic; Aminoquinolines; Condylomata Acuminata; Dermatitis; Dermatology; Humans; Imiquimod; Interferon Inducers; Keratolytic Agents; Stevens-Johnson Syndrome; Tacrolimus | 2000 |