Page last updated: 2024-09-03

imatinib mesylate and Cerebrovascular Disorders

imatinib mesylate has been researched along with Cerebrovascular Disorders in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Carreon, D; Doan, C; Durrant, LM; Kim, D; Manaenko, A; McCoy, L; Obenaus, A; Pearce, WJ; Tang, J; Zhang, JH1
Gastl, G; Geissler, K; Greil, R; Hantschel, O; Lang, A; Linkesch, W; Lion, T; Petzer, AL; Pittermann, E; Pleyer, L; Thaler, J; Valent, P; Wolf, D1
Bessler, WK; Clapp, DW; Conway, SJ; Horn, WE; Ingram, DA; Lasater, EA; Li, F; Mead, LE1

Reviews

1 review(s) available for imatinib mesylate and Cerebrovascular Disorders

ArticleYear
Nilotinib as frontline and second-line therapy in chronic myeloid leukemia: open questions.
    Critical reviews in oncology/hematology, 2012, Volume: 82, Issue:3

    Topics: Antineoplastic Agents; Benzamides; Cerebrovascular Disorders; Clinical Trials as Topic; Comorbidity; Contraindications; Diabetes Mellitus; Drug Administration Schedule; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Patient Selection; Piperazines; Practice Guidelines as Topic; Protein Kinase Inhibitors; Protein-Tyrosine Kinases; Pyrimidines; Treatment Outcome

2012

Other Studies

2 other study(ies) available for imatinib mesylate and Cerebrovascular Disorders

ArticleYear
Imatinib attenuates cerebrovascular injury and phenotypic transformation after intracerebral hemorrhage in rats.
    American journal of physiology. Regulatory, integrative and comparative physiology, 2016, 12-01, Volume: 311, Issue:6

    Topics: Animals; Cerebral Arteries; Cerebral Hemorrhage; Cerebrovascular Circulation; Cerebrovascular Disorders; Dose-Response Relationship, Drug; Imatinib Mesylate; Male; Phenotype; Rats; Rats, Sprague-Dawley; Receptors, Platelet-Derived Growth Factor; Treatment Outcome

2016
Nf1+/- mice have increased neointima formation via hyperactivation of a Gleevec sensitive molecular pathway.
    Human molecular genetics, 2008, Aug-01, Volume: 17, Issue:15

    Topics: Animals; Antineoplastic Agents; Arterial Occlusive Diseases; Benzamides; Cardiovascular Diseases; Carotid Arteries; Cell Proliferation; Cerebrovascular Disorders; Disease Models, Animal; Drug Resistance, Neoplasm; Extracellular Signal-Regulated MAP Kinases; Genes, Neurofibromatosis 1; Imatinib Mesylate; Mice; Mice, Mutant Strains; Neurofibromatosis 1; Piperazines; Pyrimidines; Tunica Intima

2008