Page last updated: 2024-10-28

ifenprodil and Neurodegenerative Diseases

ifenprodil has been researched along with Neurodegenerative Diseases in 3 studies

ifenprodil: NMDA receptor antagonist

Neurodegenerative Diseases: Hereditary and sporadic conditions which are characterized by progressive nervous system dysfunction. These disorders are often associated with atrophy of the affected central or peripheral nervous system structures.

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Trippier, PC1
Jansen Labby, K1
Hawker, DD1
Mataka, JJ1
Silverman, RB1
Rui, M1
Rossino, G1
Coniglio, S1
Monteleone, S1
Scuteri, A1
Malacrida, A1
Rossi, D1
Catenacci, L1
Sorrenti, M1
Paolillo, M1
Curti, D1
Venturini, L1
Schepmann, D1
Wünsch, B1
Liedl, KR1
Cavaletti, G1
Pace, V1
Urban, E1
Collina, S1
Mazumder, MK1
Borah, A1

Reviews

1 review available for ifenprodil and Neurodegenerative Diseases

ArticleYear
Target- and mechanism-based therapeutics for neurodegenerative diseases: strength in numbers.
    Journal of medicinal chemistry, 2013, Apr-25, Volume: 56, Issue:8

    Topics: Alzheimer Disease; Amyotrophic Lateral Sclerosis; Antioxidants; Calcium Channels; Drug Combinations;

2013

Other Studies

2 other studies available for ifenprodil and Neurodegenerative Diseases

ArticleYear
Identification of dual Sigma1 receptor modulators/acetylcholinesterase inhibitors with antioxidant and neurotrophic properties, as neuroprotective agents.
    European journal of medicinal chemistry, 2018, Oct-05, Volume: 158

    Topics: Acetylcholinesterase; Animals; Antioxidants; Blood-Brain Barrier; Cell Survival; Cholinesterase Inhi

2018
Piroxicam inhibits NMDA receptor-mediated excitotoxicity through allosteric inhibition of the GluN2B subunit: an in silico study elucidating a novel mechanism of action of the drug.
    Medical hypotheses, 2014, Volume: 83, Issue:6

    Topics: Allosteric Site; Anti-Inflammatory Agents, Non-Steroidal; Apoptosis; Calcium; Cells, Cultured; Human

2014