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ibuprofen and Autosomal Dominant Striatonigral Degeneration

ibuprofen has been researched along with Autosomal Dominant Striatonigral Degeneration in 1 studies

Midol: combination of cinnamedrine, phenacetin, aspirin & caffeine

Research Excerpts

ExcerptRelevanceReference
"Machado-Joseph disease or spinocerebellar ataxia type 3 is an inherited neurodegenerative disease associated with an abnormal glutamine over-repetition within the ataxin-3 protein."5.51Ibuprofen enhances synaptic function and neural progenitors proliferation markers and improves neuropathology and motor coordination in Machado-Joseph disease models. ( Brinkhaus, M; Henriques, D; Kaspar, BK; Matos, C; Mendonça, LS; Moreira, R; Nóbrega, C; Pereira de Almeida, L; Tavino, S; Tomé, S, 2019)
"Machado-Joseph disease or spinocerebellar ataxia type 3 is an inherited neurodegenerative disease associated with an abnormal glutamine over-repetition within the ataxin-3 protein."1.51Ibuprofen enhances synaptic function and neural progenitors proliferation markers and improves neuropathology and motor coordination in Machado-Joseph disease models. ( Brinkhaus, M; Henriques, D; Kaspar, BK; Matos, C; Mendonça, LS; Moreira, R; Nóbrega, C; Pereira de Almeida, L; Tavino, S; Tomé, S, 2019)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Mendonça, LS1
Nóbrega, C1
Tavino, S1
Brinkhaus, M1
Matos, C1
Tomé, S1
Moreira, R1
Henriques, D1
Kaspar, BK1
Pereira de Almeida, L1

Other Studies

1 other study available for ibuprofen and Autosomal Dominant Striatonigral Degeneration

ArticleYear
Ibuprofen enhances synaptic function and neural progenitors proliferation markers and improves neuropathology and motor coordination in Machado-Joseph disease models.
    Human molecular genetics, 2019, 11-15, Volume: 28, Issue:22

    Topics: Animals; Ataxin-3; Cell Line, Tumor; Cell Proliferation; Cerebellum; Disease Models, Animal; Fibrobl

2019