hypoxanthine and Amylo-1,6-Glucosidase Deficiency

hypoxanthine has been researched along with Amylo-1,6-Glucosidase Deficiency in 2 studies

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19901 (50.00)18.7374
1990's1 (50.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Azumi, T; Hisatome, I; Kinugawa, T; Kitamura, H; Kotake, H; Mashiba, H; Miyakoda, H; Ohno, K; Saito, M; Takeda, A1
Hara, N; Kawachi, M; Kiyokawa, H; Kono, N; Mineo, I; Shimizu, T; Tarui, S; Wang, YL; Yamada, Y1

Other Studies

2 other study(ies) available for hypoxanthine and Amylo-1,6-Glucosidase Deficiency

ArticleYear
Excess release of hypoxanthine from exercising muscle in two gout patients with partial HGPRTase deficiency: lack of ammonium release.
    The American journal of medicine, 1991, Volume: 90, Issue:4

    Topics: Adult; Ammonia; Child; Exercise Test; Glycogen Storage Disease Type III; Gout; Humans; Hypoxanthine; Hypoxanthine Phosphoribosyltransferase; Hypoxanthines; Male; Muscles

1991
Myogenic hyperuricemia. A common pathophysiologic feature of glycogenosis types III, V, and VII.
    The New England journal of medicine, 1987, Jul-09, Volume: 317, Issue:2

    Topics: Adult; Ammonia; Glycogen Storage Disease; Glycogen Storage Disease Type III; Glycogen Storage Disease Type V; Glycogen Storage Disease Type VII; Humans; Hypoxanthine; Hypoxanthines; Inosine; Male; Middle Aged; Muscles; Physical Exertion; Rest; Uric Acid

1987