hypericin has been researched along with Pituitary Neoplasms in 3 studies
Pituitary Neoplasms: Neoplasms which arise from or metastasize to the PITUITARY GLAND. The majority of pituitary neoplasms are adenomas, which are divided into non-secreting and secreting forms. Hormone producing forms are further classified by the type of hormone they secrete. Pituitary adenomas may also be characterized by their staining properties (see ADENOMA, BASOPHIL; ADENOMA, ACIDOPHIL; and ADENOMA, CHROMOPHOBE). Pituitary tumors may compress adjacent structures, including the HYPOTHALAMUS, several CRANIAL NERVES, and the OPTIC CHIASM. Chiasmal compression may result in bitemporal HEMIANOPSIA.
| Excerpt | Relevance | Reference |
|---|---|---|
| "When hypericin treated cells were not exposed to light growth inhibition was insignificant." | 5.31 | Light-induced photoactivation of hypericin inhibits cellular growth in pituitary adenoma cell line AtT20/D16v-F2 (hypericin inhibits cellular growth of AtT20/D16v-F2). ( Cerman, J; Mareková, M; Vávrová, J; Vokurková, D, 2001) |
| "Two established pituitary adenoma cell lines, AtT-20 and GH4C1, were treated with hypericin in tissue culture for defined periods following passage." | 5.29 | Inhibition of cellular growth and induction of apoptosis in pituitary adenoma cell lines by the protein kinase C inhibitor hypericin: potential therapeutic application. ( Chen, ZH; Couldwell, WT; Gopalakrishna, R; Hamilton, HB; Hinton, DR; Law, RE; Su, YZ; Weiss, MH, 1996) |
| "Hypericin was administered in four doses (1 mg/kg) at 28-h intervals prior to light exposure, wherein those rats treated with light were exposed to a light source four hours after the last hypericin dose." | 1.35 | Hypericin-mediated photodynamic therapy of pituitary tumors: preclinical study in a GH4C1 rat tumor model. ( Chin, SS; Cole, CD; Couldwell, WT; Liu, JK; Schmidt, MH; Sheng, X; Weiss, MH, 2008) |
| "When hypericin treated cells were not exposed to light growth inhibition was insignificant." | 1.31 | Light-induced photoactivation of hypericin inhibits cellular growth in pituitary adenoma cell line AtT20/D16v-F2 (hypericin inhibits cellular growth of AtT20/D16v-F2). ( Cerman, J; Mareková, M; Vávrová, J; Vokurková, D, 2001) |
| "Two established pituitary adenoma cell lines, AtT-20 and GH4C1, were treated with hypericin in tissue culture for defined periods following passage." | 1.29 | Inhibition of cellular growth and induction of apoptosis in pituitary adenoma cell lines by the protein kinase C inhibitor hypericin: potential therapeutic application. ( Chen, ZH; Couldwell, WT; Gopalakrishna, R; Hamilton, HB; Hinton, DR; Law, RE; Su, YZ; Weiss, MH, 1996) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cole, CD | 1 |
| Liu, JK | 1 |
| Sheng, X | 1 |
| Chin, SS | 1 |
| Schmidt, MH | 1 |
| Weiss, MH | 2 |
| Couldwell, WT | 2 |
| Hamilton, HB | 1 |
| Hinton, DR | 1 |
| Law, RE | 1 |
| Gopalakrishna, R | 1 |
| Su, YZ | 1 |
| Chen, ZH | 1 |
| Mareková, M | 1 |
| Vávrová, J | 1 |
| Vokurková, D | 1 |
| Cerman, J | 1 |
3 other studies available for hypericin and Pituitary Neoplasms
| Article | Year |
|---|---|
Hypericin-mediated photodynamic therapy of pituitary tumors: preclinical study in a GH4C1 rat tumor model.
Topics: Animals; Anthracenes; Apoptosis; In Situ Nick-End Labeling; Perylene; Photochemotherapy; Pituitary N | 2008 |
Inhibition of cellular growth and induction of apoptosis in pituitary adenoma cell lines by the protein kinase C inhibitor hypericin: potential therapeutic application.
Topics: Adenoma; Animals; Anthracenes; Apoptosis; Cell Division; Perylene; Pituitary Neoplasms; Protein Kina | 1996 |
Light-induced photoactivation of hypericin inhibits cellular growth in pituitary adenoma cell line AtT20/D16v-F2 (hypericin inhibits cellular growth of AtT20/D16v-F2).
Topics: Adenoma; Animals; Anthracenes; Apoptosis; Cell Cycle; Cell Division; DNA, Neoplasm; Dose-Response Re | 2001 |