hydroxyproline has been researched along with Heart Failure in 22 studies
Hydroxyproline: A hydroxylated form of the imino acid proline. A deficiency in ASCORBIC ACID can result in impaired hydroxyproline formation.
hydroxyproline : A proline derivative that is proline substituted by at least one hydroxy group.
Heart Failure: A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.
| Excerpt | Relevance | Reference |
|---|---|---|
| " We hypothesized that simvastatin will improve these dynamics in experimental hypercholesterolemia (HC)." | 7.77 | Simvastatin preserves diastolic function in experimental hypercholesterolemia independently of its lipid lowering effect. ( Bonetti, PO; Herrmann, J; Lavi, R; Lerman, A; Lerman, LO; Mannheim, D, 2011) |
| "Canrenone attenuates LV interstitial remodeling and reduces filling pressures in rats with postinfarction heart failure." | 7.71 | Effects of canrenone on myocardial reactive fibrosis in a rat model of postinfarction heart failure. ( Casaburi, C; Cittadini, A; Di Gianni, A; Monti, MG; Saccà, L; Saldamarco, L; Scherillo, G; Serpico, R; Vanasia, M, 2002) |
| " We hypothesized that simvastatin will improve these dynamics in experimental hypercholesterolemia (HC)." | 3.77 | Simvastatin preserves diastolic function in experimental hypercholesterolemia independently of its lipid lowering effect. ( Bonetti, PO; Herrmann, J; Lavi, R; Lerman, A; Lerman, LO; Mannheim, D, 2011) |
| " LVH was characterized by increases in the ratios of heart and left ventricular weights to body weight, increased myocyte cross-sectional areas, myocardial and perivascular fibrosis, and elevated cardiac hydroxyproline." | 3.75 | K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function. ( Gao, S; Long, CL; Wang, H; Wang, RH, 2009) |
| "Canrenone attenuates LV interstitial remodeling and reduces filling pressures in rats with postinfarction heart failure." | 3.71 | Effects of canrenone on myocardial reactive fibrosis in a rat model of postinfarction heart failure. ( Casaburi, C; Cittadini, A; Di Gianni, A; Monti, MG; Saccà, L; Saldamarco, L; Scherillo, G; Serpico, R; Vanasia, M, 2002) |
| "The combination of ACE inhibitor and ARB, independently of the hypotensive effect, improved LV phenotypic change and increased LV endothelin-1 production and collagen accumulation, diastolic dysfunction, and survival in a rat heart failure model more effectively than either agent alone, thereby providing solid experimental evidence that the combination of these 2 agents is more beneficial than monotherapy for treatment of heart failure." | 3.71 | Effects of combination of ACE inhibitor and angiotensin receptor blocker on cardiac remodeling, cardiac function, and survival in rat heart failure. ( Iwao, H; Izumi, Y; Kawano, H; Kim, S; Kimoto, M; Yoshiyama, M; Zhan, Y, 2001) |
| "Endothelial dysfunction can lead to congestive heart failure and the activation of endothelial ATP-sensitive potassium (K(ATP)) channels may contribute to endothelial protection." | 1.40 | Natakalim improves post-infarction left ventricular remodeling by restoring the coordinated balance between endothelial function and cardiac hypertrophy. ( Cui, WY; Long, CL; Wang, H; Zhang, YF; Zhong, ML; Zhou, HM, 2014) |
| "Chronic heart failure is a multifactorial, progressive disease of many causes and is associated with complex ventricular remodeling." | 1.32 | The structural examination of myocardial samples from patients with end-stage heart failure supported by ventricular assist devices using electron microscopy and amino acid analysis reveals low degree of reverse remodeling. ( Arusoglu, L; Becker, G; Böthig, D; El Banayosy, A; Heimann, P; Jacob, M; Kassner, A; Körfer, R; Mannherz, HG; Meyer, HE; Milting, H; Morshuis, M, 2004) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 5 (22.73) | 18.7374 |
| 1990's | 6 (27.27) | 18.2507 |
| 2000's | 9 (40.91) | 29.6817 |
| 2010's | 2 (9.09) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhou, HM | 1 |
| Zhong, ML | 1 |
| Zhang, YF | 1 |
| Cui, WY | 1 |
| Long, CL | 2 |
| Wang, H | 2 |
| Gao, S | 1 |
| Wang, RH | 1 |
| Mannheim, D | 1 |
| Herrmann, J | 1 |
| Bonetti, PO | 1 |
| Lavi, R | 1 |
| Lerman, LO | 1 |
| Lerman, A | 1 |
| Cittadini, A | 1 |
| Casaburi, C | 1 |
| Monti, MG | 1 |
| Di Gianni, A | 1 |
| Serpico, R | 1 |
| Scherillo, G | 1 |
| Saldamarco, L | 1 |
| Vanasia, M | 1 |
| Saccà, L | 1 |
| DE DEUXCHAISNES, CN | 1 |
| KRANE, SM | 1 |
| Ahn, J | 1 |
| Varagic, J | 1 |
| Slama, M | 1 |
| Susic, D | 1 |
| Frohlich, ED | 1 |
| Milting, H | 1 |
| Jacob, M | 1 |
| Kassner, A | 1 |
| Heimann, P | 1 |
| Mannherz, HG | 1 |
| Becker, G | 1 |
| Meyer, HE | 1 |
| Böthig, D | 1 |
| Arusoglu, L | 1 |
| Morshuis, M | 1 |
| Körfer, R | 1 |
| El Banayosy, A | 1 |
| Itter, G | 1 |
| Jung, W | 1 |
| Juretschke, P | 1 |
| Schoelkens, BA | 1 |
| Linz, W | 1 |
| Du, XJ | 1 |
| Gao, XM | 1 |
| Kiriazis, H | 1 |
| Moore, XL | 1 |
| Ming, Z | 1 |
| Su, Y | 1 |
| Finch, AM | 1 |
| Hannan, RA | 1 |
| Dart, AM | 1 |
| Graham, RM | 1 |
| Bristow, MR | 1 |
| Ginsburg, R | 1 |
| Minobe, W | 1 |
| Cubicciotti, RS | 1 |
| Sageman, WS | 1 |
| Lurie, K | 1 |
| Billingham, ME | 1 |
| Harrison, DC | 1 |
| Stinson, EB | 1 |
| Conrad, CH | 1 |
| Brooks, WW | 1 |
| Hayes, JA | 1 |
| Sen, S | 2 |
| Robinson, KG | 1 |
| Bing, OH | 1 |
| Komamura, K | 1 |
| Vatner, SF | 1 |
| Yang, CM | 1 |
| Kandaswamy, V | 1 |
| Young, D | 1 |
| Turcani, M | 1 |
| Jacob, R | 1 |
| Clair, MJ | 1 |
| Krombach, RS | 1 |
| Coker, ML | 1 |
| Heslin, TL | 1 |
| Kribbs, SB | 1 |
| de Gasparo, M | 1 |
| Spinale, FG | 1 |
| Kim, HE | 1 |
| Dalal, SS | 1 |
| Young, E | 1 |
| Legato, MJ | 1 |
| Weisfeldt, ML | 1 |
| D'Armiento, J | 1 |
| O'Brien, DW | 1 |
| Fu, Y | 1 |
| Parker, HR | 1 |
| Chan, SY | 1 |
| Idikio, H | 1 |
| Scott, PG | 1 |
| Jugdutt, BI | 1 |
| Kim, S | 1 |
| Yoshiyama, M | 1 |
| Izumi, Y | 1 |
| Kawano, H | 1 |
| Kimoto, M | 1 |
| Zhan, Y | 1 |
| Iwao, H | 1 |
| Randhawa, AK | 1 |
| Singal, PK | 1 |
| Siri, FM | 1 |
| Smith, RM | 1 |
| Jansen, HH | 1 |
| Leclercq, JF | 1 |
| Berjal, G | 1 |
| Durand, M | 1 |
| Hatt, PY | 1 |
| Swynghedauw, B | 1 |
1 review available for hydroxyproline and Heart Failure
| Article | Year |
|---|---|
PAGET'S DISEASE OF BONE: CLINICAL AND METABOLIC OBSERVATIONS.
Topics: Alkaline Phosphatase; Aspirin; Blood; Bone and Bones; Calcium; Citrates; Fluorides; Geriatrics; Hear | 1964 |
21 other studies available for hydroxyproline and Heart Failure
| Article | Year |
|---|---|
Natakalim improves post-infarction left ventricular remodeling by restoring the coordinated balance between endothelial function and cardiac hypertrophy.
Topics: Administration, Oral; Allyl Compounds; Animals; Blood Pressure; Cardiomegaly; Dose-Response Relation | 2014 |
K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function.
Topics: Animals; Aorta, Abdominal; Atrial Natriuretic Factor; Blood Pressure; Cardiovascular Agents; Disease | 2009 |
Simvastatin preserves diastolic function in experimental hypercholesterolemia independently of its lipid lowering effect.
Topics: Animals; Diastole; Disease Models, Animal; Female; Heart Failure; Hydroxymethylglutaryl-CoA Reductas | 2011 |
Effects of canrenone on myocardial reactive fibrosis in a rat model of postinfarction heart failure.
Topics: Administration, Oral; Aldosterone; Animals; Canrenone; Collagen; Cyclodextrins; Disease Models, Anim | 2002 |
Cardiac structural and functional responses to salt loading in SHR.
Topics: Aging; Animals; Blood Pressure; Cardiomegaly; Echocardiography; Fibrosis; Heart; Heart Failure; Hydr | 2004 |
The structural examination of myocardial samples from patients with end-stage heart failure supported by ventricular assist devices using electron microscopy and amino acid analysis reveals low degree of reverse remodeling.
Topics: Adolescent; Adult; Aged; Child; Collagen; Heart Failure; Heart Transplantation; Heart Ventricles; He | 2004 |
A model of chronic heart failure in spontaneous hypertensive rats (SHR).
Topics: Animals; Aorta; Blood Pressure; Disease Models, Animal; Heart Failure; Heart Rate; Hydroxyproline; H | 2004 |
Transgenic alpha1A-adrenergic activation limits post-infarct ventricular remodeling and dysfunction and improves survival.
Topics: Actins; Aging; Animals; Atrial Natriuretic Factor; Collagen; Echocardiography; Female; Fibronectins; | 2006 |
Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts.
Topics: Adenylyl Cyclases; Adolescent; Adult; Catecholamines; Contractile Proteins; Creatine Kinase; Dihydro | 1982 |
Myocardial fibrosis and stiffness with hypertrophy and heart failure in the spontaneously hypertensive rat.
Topics: Aging; Animals; Cardiomegaly; Endomyocardial Fibrosis; Heart Failure; Hydroxyproline; Hypertension; | 1995 |
[Alterations in left ventricular systolic and diastolic function in conscious dogs with pacing-induced heart failure].
Topics: Animals; Cardiac Pacing, Artificial; Collagen; Diastole; Dogs; Heart Failure; Hydroxyproline; Myocar | 1993 |
Changes in collagen phenotypes during progression and regression of cardiac hypertrophy.
Topics: Aging; Angiotensin-Converting Enzyme Inhibitors; Animals; Blotting, Northern; Captopril; Cardiomegal | 1997 |
Minoxidil accelerates heart failure development in rats with ascending aortic constriction.
Topics: Animals; Aorta; Collagen; Constriction, Pathologic; Heart; Heart Failure; Hydroxyproline; Hypertroph | 1998 |
Angiotensin AT1 receptor inhibition in pacing induced heart failure: effects on left ventricular myocardial collagen content and composition.
Topics: Animals; Cardiac Pacing, Artificial; Collagen; Echocardiography; Heart Failure; Humans; Hydroxyproli | 1998 |
Disruption of the myocardial extracellular matrix leads to cardiac dysfunction.
Topics: Age Factors; Animals; Cardiomegaly; Collagen; Diastole; Extracellular Matrix; Gene Expression; Heart | 2000 |
Differential morphometric and ultrastructural remodelling in the left atrium and left ventricle in rapid ventricular pacing-induced heart failure.
Topics: Animals; Cardiac Pacing, Artificial; Collagen; Dogs; Heart Atria; Heart Failure; Heart Ventricles; H | 2000 |
Effects of combination of ACE inhibitor and angiotensin receptor blocker on cardiac remodeling, cardiac function, and survival in rat heart failure.
Topics: Angiotensin Receptor Antagonists; Angiotensin-Converting Enzyme Inhibitors; Animals; Antihypertensiv | 2001 |
Pressure overload-induced cardiac hypertrophy with and without dilation.
Topics: Animals; Aorta; Biopsy; Blood Pressure; Body Weight; Cardiomegaly; Collagen; Disease Models, Animal; | 1992 |
Effects of plasma norepinephrine elevation on the heart's adaptation to chronic aortic constriction in rats.
Topics: Animals; Aortic Diseases; Blood Pressure; Constriction, Pathologic; DNA; Heart; Heart Failure; Hemod | 1986 |
[Quantitative connective tissue conditions in ventricle walls of insufficient hearts, demonstrated by the example of hydroxyproline determination].
Topics: Cardiomegaly; Connective Tissue; Heart; Heart Failure; Heart Ventricles; Humans; Hydroxyproline; Myo | 1967 |
[Primary cardiac failure in aged subjects].
Topics: Adenosine Triphosphatases; Adult; Age Factors; Aged; Atrial Fibrillation; Autopsy; Cardiomegaly; Car | 1974 |