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hydroxyindoleacetic acid and Chronic Pain

hydroxyindoleacetic acid has been researched along with Chronic Pain in 2 studies

(5-hydroxyindol-3-yl)acetic acid : A member of the class of indole-3-acetic acids that is indole-3-acetic acid substituted by a hydroxy group at C-5.

Chronic Pain: Aching sensation that persists for more than a few months. It may or may not be associated with trauma or disease, and may persist after the initial injury has healed. Its localization, character, and timing are more vague than with acute pain.

Research Excerpts

ExcerptRelevanceReference
"Chronic pain was evaluated by thermal hyperalgesia in Hargreaves test and mechanical allodynia in von Frey test, depressive-like behaviors were evaluated by immobility time in forced swim test and tail suspension test."1.48Chronic trans-astaxanthin treatment exerts antihyperalgesic effect and corrects co-morbid depressive like behaviors in mice with chronic pain. ( Ding, L; Jiang, X; Jing, C; Liu, F; Pang, C; Yan, Q; Zhang, L, 2018)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Jiang, X1
Yan, Q1
Liu, F1
Jing, C1
Ding, L1
Zhang, L1
Pang, C1
Shiiba, SJ1
Yamamoto, S1
Sasaki, H1
Nishi, M1
Ishikawa, K1
Yasuda, S1
Tokuda, N1
Nakanishi, O1
Ishikawa, T1

Other Studies

2 other studies available for hydroxyindoleacetic acid and Chronic Pain

ArticleYear
Chronic trans-astaxanthin treatment exerts antihyperalgesic effect and corrects co-morbid depressive like behaviors in mice with chronic pain.
    Neuroscience letters, 2018, Jan-01, Volume: 662

    Topics: Analgesics; Animals; Behavior, Animal; Chronic Pain; Cytokines; Depression; Hydroxyindoleacetic Acid

2018
Cutaneous magnetic stimulation reduces rat chronic pain via activation of the supra-spinal descending pathway.
    Cellular and molecular neurobiology, 2012, Volume: 32, Issue:2

    Topics: Analgesia; Animals; Astrocytes; Chronic Pain; Constriction, Pathologic; Efferent Pathways; Glial Fib

2012