hydrogen sulfide has been researched along with Visceral Pain in 7 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Visceral Pain: Pain originating from internal organs (VISCERA) associated with autonomic phenomena (PALLOR; SWEATING; NAUSEA; and VOMITING). It often becomes a REFERRED PAIN.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Intracolonic administration of NaHS, an H(2)S donor, evoked visceral pain-like nociceptive behavior and referred hyperalgesia in mice, an effect abolished by NNC 55-0396, a selective T-type Ca(2+)-channel blocker, or by knockdown of Ca(v)3." | 7.78 | Colonic hydrogen sulfide-induced visceral pain and referred hyperalgesia involve activation of both Ca(v)3.2 and TRPA1 channels in mice. ( Kawabata, A; Noguchi, Y; Okawa, Y; Sekiguchi, F; Tsubota-Matsunami, M, 2012) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 5.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| " In the present study, we tested the effects (in rats) of a simple extract of medicinal cannabis (called "MFF") for its ability to promote resolution of colitis, to prevent gastric damage induced by naproxen, and to reduce gastric distention-induced visceral pain." | 3.79 | Pro-resolution, protective and anti-nociceptive effects of a cannabis extract in the rat gastrointestinal tract. ( Ferraz, JG; Flannigan, KL; McKnight, W; Tuitt, D; Wallace, JL; Wang, L, 2013) |
| " Intracolonic administration of NaHS, an H(2)S donor, evoked visceral pain-like nociceptive behavior and referred hyperalgesia in mice, an effect abolished by NNC 55-0396, a selective T-type Ca(2+)-channel blocker, or by knockdown of Ca(v)3." | 3.78 | Colonic hydrogen sulfide-induced visceral pain and referred hyperalgesia involve activation of both Ca(v)3.2 and TRPA1 channels in mice. ( Kawabata, A; Noguchi, Y; Okawa, Y; Sekiguchi, F; Tsubota-Matsunami, M, 2012) |
| "Hydrogen sulfide (H₂S) functions as a neuromodulator, but whether it modulates visceral pain is not well known." | 3.78 | Upregulation of cystathionine β-synthetase expression contributes to visceral hyperalgesia induced by heterotypic intermittent stress in rats. ( Hu, S; Jiang, X; Qu, R; Wang, Y; Xiao, Y; Xu, GY, 2012) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 1.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 6 (85.71) | 24.3611 |
| 2020's | 1 (14.29) | 2.80 |
| Authors | Studies |
|---|---|
| Sekiguchi, F | 3 |
| Koike, N | 1 |
| Shimada, Y | 1 |
| Sugimoto, K | 1 |
| Masuda, H | 1 |
| Nakamura, T | 1 |
| Yamaguchi, H | 1 |
| Tanabe, G | 1 |
| Marumoto, S | 1 |
| Kasanami, Y | 1 |
| Tsubota, M | 2 |
| Ohkubo, T | 1 |
| Yoshida, S | 1 |
| Kawabata, A | 3 |
| Wallace, JL | 1 |
| Flannigan, KL | 1 |
| McKnight, W | 1 |
| Wang, L | 1 |
| Ferraz, JG | 1 |
| Tuitt, D | 1 |
| Terada, Y | 1 |
| Fujimura, M | 1 |
| Nishimura, S | 1 |
| Cenac, N | 1 |
| Castro, M | 1 |
| Desormeaux, C | 1 |
| Colin, P | 1 |
| Sie, M | 1 |
| Ranger, M | 1 |
| Vergnolle, N | 1 |
| Tsubota-Matsunami, M | 1 |
| Noguchi, Y | 1 |
| Okawa, Y | 1 |
| Li, L | 1 |
| Xie, R | 1 |
| Hu, S | 2 |
| Wang, Y | 2 |
| Yu, T | 1 |
| Xiao, Y | 2 |
| Jiang, X | 2 |
| Gu, J | 1 |
| Hu, CY | 1 |
| Xu, GY | 2 |
| Qu, R | 1 |
7 other studies available for hydrogen sulfide and Visceral Pain
| Article | Year |
|---|---|
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Ca
Topics: Animals; Calcium Channels, T-Type; Cystitis; HEK293 Cells; Humans; Hydrogen Sulfide; Mice; Pancreati | 2023 |
Pro-resolution, protective and anti-nociceptive effects of a cannabis extract in the rat gastrointestinal tract.
Topics: Analgesics; Animals; Anti-Inflammatory Agents, Non-Steroidal; Cannabis; Colitis; Gastrointestinal Tr | 2013 |
Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis.
Topics: Analysis of Variance; Animals; Benzimidazoles; Calcium Channel Blockers; Calcium Channels, T-Type; C | 2015 |
A novel orally administered trimebutine compound (GIC-1001) is anti-nociceptive and features peripheral opioid agonistic activity and Hydrogen Sulphide-releasing capacity in mice.
Topics: Analgesics; Analgesics, Opioid; Animals; Benzenesulfonates; Colon; Disease Models, Animal; Hydrogen | 2016 |
Colonic hydrogen sulfide-induced visceral pain and referred hyperalgesia involve activation of both Ca(v)3.2 and TRPA1 channels in mice.
Topics: Animals; Benzimidazoles; Calcium Channels, T-Type; Cyclopropanes; Female; Hydrogen Sulfide; Hyperalg | 2012 |
Upregulation of cystathionine beta-synthetase expression by nuclear factor-kappa B activation contributes to visceral hypersensitivity in adult rats with neonatal maternal deprivation.
Topics: Animals; Cystathionine beta-Synthase; Female; Ganglia, Spinal; Hydrogen Sulfide; Hyperalgesia; Irrit | 2012 |
Upregulation of cystathionine β-synthetase expression contributes to visceral hyperalgesia induced by heterotypic intermittent stress in rats.
Topics: Animals; Cystathionine beta-Synthase; Enzyme Inhibitors; Hydrogen Sulfide; Hyperalgesia; Intestinal | 2012 |