hydrogen sulfide has been researched along with Sepsis in 37 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Sepsis: Systemic inflammatory response syndrome with a proven or suspected infectious etiology. When sepsis is associated with organ dysfunction distant from the site of infection, it is called severe sepsis. When sepsis is accompanied by HYPOTENSION despite adequate fluid infusion, it is called SEPTIC SHOCK.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The view of hydrogen sulfide has changed from a toxic by-product to a crucial signaling molecule, with enormous potential as a pharmacological target for diseases ranging from heart disease to sepsis." | 8.88 | Hydrogen sulfide as a therapeutic target for inflammation. ( Badiei, A; Bhatia, M; Rivers, JR, 2012) |
| "There is ample evidence that nitric oxide, carbon monoxide, and H2S may exert cytoprotective effects in shock states due to their vasomotor, antioxidant, and anti-inflammatory properties as well as their potential to induce a hibernation-like metabolic state called 'suspended animation' resulting from inhibition of cytochrome-c-oxidase." | 8.85 | Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide. ( Baumgart, K; Radermacher, P; Wagner, F, 2009) |
| "To determine alterations of circulating levels of hydrogen sulfide and substance P in patients with sepsis compared to non-sepsis patients with similar disease severity and organ dysfunction." | 7.85 | Circulating levels of hydrogen sulfide and substance P in patients with sepsis. ( Bhatia, M; Chambers, S; Gaddam, RR; Murdoch, D; Shaw, G, 2017) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 7.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "Hydrogen sulfide (H(2)S) has been shown to induce transient receptor potential vanilloid 1 (TRPV1)-mediated neurogenic inflammation in polymicrobial sepsis." | 7.77 | Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM, 2011) |
| "Capsazepine treatment attenuates significantly systemic inflammation and multiple organ damage caused by sepsis, and protects against sepsis-induced mortality." | 7.76 | Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis. ( Ang, SF; Bhatia, M; Moochhala, SM, 2010) |
| " However, whether endogenous H2S would regulate sepsis-associated lung inflammation via substance P (SP) and its receptors remains unknown." | 7.74 | Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury. ( Adhikari, S; Bhatia, M; Hegde, A; Moochhala, SM; Ng, SW; Zhang, H, 2007) |
| "Sepsis is is anabnormalhost immune responsecausedbyinfection." | 7.01 | Application and value of hydrogen sulfide modulated autophagy in sepsis. ( Liu, C; Sun, Y, 2023) |
| "Sepsis is one of the main causes of death in critically ill patients." | 6.66 | [Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis]. ( Chai, C; He, M; Kang, F; Nie, J; Zhang, C, 2020) |
| "Hydrogen sulfide (H2S) is a novel signaling molecule most recently found to be of fundamental importance in cellular function as a regulator of apoptosis, inflammation, and perfusion." | 6.55 | Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia. ( DeLeon, ER; Drucker, NA; Ferkowicz, MJ; Jensen, AR; Khaneki, S; Markel, TA; Olson, KR; Yoder, MC, 2017) |
| "Hydrogen sulfide is a novel gasotransmitter that has been shown to play a major role in regulating vascular tone." | 6.53 | The role of hydrogen sulfide in burns. ( Akter, F, 2016) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 6.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 5.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H2S) has recently been recognized as a novel gaseous transmitter with several anti-inflammatory properties." | 5.62 | Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis. ( Antonakos, N; Bauer, M; Damoraki, G; Droggiti, DE; Giamarellos-Bourboulis, EJ; Gkavogianni, T; Jentho, E; Karageorgos, A; Karakike, E; Katrini, K; Katsouda, A; Koufargyris, P; Kouloulias, V; Papapetropoulos, A; Platoni, K; Renieris, G; Sabracos, L; Szabo, C; Wang, R; Weis, S, 2021) |
| "Sepsis is a major cause of mortality, and dysregulation of the immune response plays a central role in this syndrome." | 5.40 | Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition. ( Colombani, PM; Ferlito, M; Fox-Talbot, K; Fulton, WB; Marchionni, L; Paolocci, N; Steenbergen, C; Wang, Q, 2014) |
| "In septic rats pretreated with AOAA, sepsis-associated hippocampus inflammation was reduced." | 5.37 | Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy. ( Chen, D; Lan, X; Li, C; Liu, B; Pan, H; Yang, G, 2011) |
| "Sepsis was induced by cecal ligation and puncture (CLP)." | 5.36 | Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation. ( Alves-Filho, JC; Assreuy, J; Carlos, D; Cunha, FQ; Czaikoski, PG; Ferreira, SH; Freitas, A; Hothersall, JS; Montenegro, MF; Nascimento, DC; Neto, AF; Orrico, MI; Rossi, MA; Souto, FO; Spiller, F, 2010) |
| "The view of hydrogen sulfide has changed from a toxic by-product to a crucial signaling molecule, with enormous potential as a pharmacological target for diseases ranging from heart disease to sepsis." | 4.88 | Hydrogen sulfide as a therapeutic target for inflammation. ( Badiei, A; Bhatia, M; Rivers, JR, 2012) |
| "There is ample evidence that nitric oxide, carbon monoxide, and H2S may exert cytoprotective effects in shock states due to their vasomotor, antioxidant, and anti-inflammatory properties as well as their potential to induce a hibernation-like metabolic state called 'suspended animation' resulting from inhibition of cytochrome-c-oxidase." | 4.85 | Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide. ( Baumgart, K; Radermacher, P; Wagner, F, 2009) |
| "Age, APACHE score at ICU admission, neurological disease, sepsis and duration of mechanical ventilation were all independent risk factors for the development of delirium in ICU patients." | 4.40 | Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19. ( , 2023) |
| "To investigate the effect of hydrogen sulfide (H2S) on myocardial injury in sepsis-induced myocardial dysfunction (SIMD), male C57BL/6 mice were intraperitoneally injected with lipopolysaccharide (LPS) (10 mg/kg, i." | 4.31 | Hydrogen sulfide alleviates lipopolysaccharide-induced myocardial injury through TLR4-NLRP3 pathway. ( Bai, L; He, K; Jin, S; Liu, B; Miao, Y; Wu, Y; Xia, Y; Zhang, W; Zhang, X, 2023) |
| "To determine alterations of circulating levels of hydrogen sulfide and substance P in patients with sepsis compared to non-sepsis patients with similar disease severity and organ dysfunction." | 3.85 | Circulating levels of hydrogen sulfide and substance P in patients with sepsis. ( Bhatia, M; Chambers, S; Gaddam, RR; Murdoch, D; Shaw, G, 2017) |
| "Hydrogen sulfide (H(2)S) has been shown to induce transient receptor potential vanilloid 1 (TRPV1)-mediated neurogenic inflammation in polymicrobial sepsis." | 3.77 | Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM, 2011) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 3.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "Capsazepine treatment attenuates significantly systemic inflammation and multiple organ damage caused by sepsis, and protects against sepsis-induced mortality." | 3.76 | Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis. ( Ang, SF; Bhatia, M; Moochhala, SM, 2010) |
| " However, whether endogenous H2S would regulate sepsis-associated lung inflammation via substance P (SP) and its receptors remains unknown." | 3.74 | Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury. ( Adhikari, S; Bhatia, M; Hegde, A; Moochhala, SM; Ng, SW; Zhang, H, 2007) |
| "Sepsis is is anabnormalhost immune responsecausedbyinfection." | 3.01 | Application and value of hydrogen sulfide modulated autophagy in sepsis. ( Liu, C; Sun, Y, 2023) |
| "Sepsis is one of the main causes of death in critically ill patients." | 2.66 | [Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis]. ( Chai, C; He, M; Kang, F; Nie, J; Zhang, C, 2020) |
| "Hydrogen sulfide (H2S) is a novel signaling molecule most recently found to be of fundamental importance in cellular function as a regulator of apoptosis, inflammation, and perfusion." | 2.55 | Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia. ( DeLeon, ER; Drucker, NA; Ferkowicz, MJ; Jensen, AR; Khaneki, S; Markel, TA; Olson, KR; Yoder, MC, 2017) |
| "Hydrogen sulfide is a novel gasotransmitter that has been shown to play a major role in regulating vascular tone." | 2.53 | The role of hydrogen sulfide in burns. ( Akter, F, 2016) |
| "Inflammation is a response to traumatic, infectious, post-ischemic, toxic, or autoimmune injury." | 2.52 | H2S and Inflammation: An Overview. ( Bhatia, M, 2015) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 2.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 1.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H2S) has recently been recognized as a novel gaseous transmitter with several anti-inflammatory properties." | 1.62 | Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis. ( Antonakos, N; Bauer, M; Damoraki, G; Droggiti, DE; Giamarellos-Bourboulis, EJ; Gkavogianni, T; Jentho, E; Karageorgos, A; Karakike, E; Katrini, K; Katsouda, A; Koufargyris, P; Kouloulias, V; Papapetropoulos, A; Platoni, K; Renieris, G; Sabracos, L; Szabo, C; Wang, R; Weis, S, 2021) |
| "Sepsis is a major cause of mortality, and dysregulation of the immune response plays a central role in this syndrome." | 1.40 | Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition. ( Colombani, PM; Ferlito, M; Fox-Talbot, K; Fulton, WB; Marchionni, L; Paolocci, N; Steenbergen, C; Wang, Q, 2014) |
| "Hydrogen sulfide metabolism was associated with an increase in O2 consumption from a baseline 96." | 1.37 | The liver as a central regulator of hydrogen sulfide. ( Clemens, MG; Culberson, CR; Narasimhan, S; Norris, EJ, 2011) |
| "In septic rats pretreated with AOAA, sepsis-associated hippocampus inflammation was reduced." | 1.37 | Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy. ( Chen, D; Lan, X; Li, C; Liu, B; Pan, H; Yang, G, 2011) |
| "Sepsis was induced by cecal ligation and puncture (CLP)." | 1.36 | Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation. ( Alves-Filho, JC; Assreuy, J; Carlos, D; Cunha, FQ; Czaikoski, PG; Ferreira, SH; Freitas, A; Hothersall, JS; Montenegro, MF; Nascimento, DC; Neto, AF; Orrico, MI; Rossi, MA; Souto, FO; Spiller, F, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (2.70) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 7 (18.92) | 29.6817 |
| 2010's | 20 (54.05) | 24.3611 |
| 2020's | 9 (24.32) | 2.80 |
| Authors | Studies |
|---|---|
| Li, J | 1 |
| Li, M | 1 |
| Li, L | 1 |
| Ma, J | 1 |
| Yao, C | 1 |
| Yao, S | 1 |
| Zhou, T | 1 |
| Qian, H | 1 |
| Zheng, N | 1 |
| Lu, Q | 1 |
| Han, Y | 1 |
| Zhao, YH | 1 |
| Cao, GD | 1 |
| Guo, LC | 1 |
| Cheng, QH | 1 |
| Xia, Y | 1 |
| Zhang, W | 1 |
| He, K | 1 |
| Bai, L | 1 |
| Miao, Y | 1 |
| Liu, B | 2 |
| Zhang, X | 1 |
| Jin, S | 1 |
| Wu, Y | 1 |
| Sun, Y | 1 |
| Liu, C | 1 |
| Nie, J | 1 |
| Kang, F | 1 |
| Zhang, C | 1 |
| Chai, C | 1 |
| He, M | 2 |
| Renieris, G | 1 |
| Droggiti, DE | 1 |
| Katrini, K | 1 |
| Koufargyris, P | 1 |
| Gkavogianni, T | 1 |
| Karakike, E | 1 |
| Antonakos, N | 1 |
| Damoraki, G | 1 |
| Karageorgos, A | 1 |
| Sabracos, L | 1 |
| Katsouda, A | 1 |
| Jentho, E | 1 |
| Weis, S | 1 |
| Wang, R | 1 |
| Bauer, M | 1 |
| Szabo, C | 2 |
| Platoni, K | 1 |
| Kouloulias, V | 1 |
| Papapetropoulos, A | 1 |
| Giamarellos-Bourboulis, EJ | 1 |
| Cui, W | 2 |
| Chen, J | 1 |
| Yu, F | 1 |
| Liu, W | 1 |
| Jensen, AR | 1 |
| Drucker, NA | 1 |
| Khaneki, S | 1 |
| Ferkowicz, MJ | 1 |
| Yoder, MC | 1 |
| DeLeon, ER | 1 |
| Olson, KR | 1 |
| Markel, TA | 1 |
| Bee, N | 1 |
| White, R | 1 |
| Petros, AJ | 1 |
| Gaddam, RR | 2 |
| Chambers, S | 1 |
| Murdoch, D | 1 |
| Shaw, G | 1 |
| Bhatia, M | 14 |
| Toliver-Kinsky, T | 1 |
| Törö, G | 1 |
| Lee, SJ | 1 |
| Shatalin, K | 1 |
| Nudler, E | 1 |
| Xu, C | 1 |
| Zhao, HY | 1 |
| Reitsema, VA | 1 |
| Star, BS | 1 |
| de Jager, VD | 1 |
| van Meurs, M | 1 |
| Henning, RH | 1 |
| Bouma, HR | 1 |
| Ferlito, M | 1 |
| Wang, Q | 1 |
| Fulton, WB | 1 |
| Colombani, PM | 1 |
| Marchionni, L | 1 |
| Fox-Talbot, K | 1 |
| Paolocci, N | 1 |
| Steenbergen, C | 1 |
| Akter, F | 1 |
| Badiei, A | 2 |
| Chambers, ST | 1 |
| Baumgart, K | 2 |
| Georgieff, M | 1 |
| Radermacher, P | 2 |
| Calzia, E | 1 |
| Zhang, H | 5 |
| Moochhala, SM | 6 |
| Wagner, F | 1 |
| Shanmugam, MK | 1 |
| Ang, SF | 3 |
| Kapoor, A | 1 |
| Thiemermann, C | 1 |
| Spiller, F | 1 |
| Orrico, MI | 1 |
| Nascimento, DC | 1 |
| Czaikoski, PG | 1 |
| Souto, FO | 1 |
| Alves-Filho, JC | 1 |
| Freitas, A | 1 |
| Carlos, D | 1 |
| Montenegro, MF | 1 |
| Neto, AF | 1 |
| Ferreira, SH | 1 |
| Rossi, MA | 1 |
| Hothersall, JS | 1 |
| Assreuy, J | 1 |
| Cunha, FQ | 1 |
| Norris, EJ | 1 |
| Culberson, CR | 1 |
| Narasimhan, S | 1 |
| Clemens, MG | 1 |
| MacAry, PA | 2 |
| Sio, SW | 1 |
| Chen, D | 1 |
| Pan, H | 1 |
| Li, C | 1 |
| Lan, X | 1 |
| Yang, G | 1 |
| Rivers, JR | 1 |
| Zhi, L | 3 |
| Moore, PK | 3 |
| Moochhala, S | 1 |
| Hegde, A | 1 |
| Ng, SW | 1 |
| Adhikari, S | 1 |
| Smith, RF | 1 |
| Dayton, SL | 1 |
| Chipps, DD | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| A Phase II Double Blind Randomized Controlled Trial of High Dose Vitamin B12 in Septic Shock[NCT03783091] | Phase 2 | 20 participants (Anticipated) | Interventional | 2019-08-05 | Recruiting | ||
| Hydrogen Sulfide as Prognostic Factor[NCT01088490] | 50 participants (Actual) | Observational | 2010-01-31 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
11 reviews available for hydrogen sulfide and Sepsis
| Article | Year |
|---|---|
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Application and value of hydrogen sulfide modulated autophagy in sepsis.
Topics: Autophagy; Carbon Monoxide; Humans; Hydrogen Sulfide; Nitric Oxide; Sepsis | 2023 |
[Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis].
Topics: Autophagy; Humans; Hydrogen Sulfide; Mitochondria; Sepsis; Signal Transduction | 2020 |
Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia.
Topics: Apoptosis; Cell Death; Humans; Hydrogen Sulfide; Ischemia; Sepsis; Signal Transduction | 2017 |
Metabolic Resuscitation Strategies to Prevent Organ Dysfunction in Sepsis.
Topics: Caloric Restriction; Combined Modality Therapy; Energy Metabolism; Humans; Hydrogen Sulfide; Hypothe | 2019 |
H2S and Inflammation: An Overview.
Topics: Animals; Arthritis; Humans; Hydrogen Sulfide; Inflammation; Pancreatitis; Pulmonary Disease, Chronic | 2015 |
The role of hydrogen sulfide in burns.
Topics: Animals; Burns; Humans; Hydrogen Sulfide; Inflammation; Sepsis; Wound Healing | 2016 |
Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide.
Topics: Animals; Carbon Monoxide; Hydrogen Sulfide; Microcirculation; Nitric Oxide; Nitric Oxide Synthase Ty | 2009 |
The role of pro-inflammatory molecules and pharmacological agents in acute pancreatitis and sepsis.
Topics: Animals; Apoptosis; Ceruletide; Chemokines; Female; Humans; Hydrogen Sulfide; Inflammation Mediators | 2010 |
Hydrogen sulfide and substance P in inflammation.
Topics: Air Pollutants; Amino Acid Sequence; Animals; Humans; Hydrogen Sulfide; Inflammation; Inflammation M | 2010 |
Hydrogen sulfide as a therapeutic target for inflammation.
Topics: Animals; Anti-Inflammatory Agents; Humans; Hydrogen Sulfide; Inflammation; Molecular Targeted Therap | 2012 |
1 trial available for hydrogen sulfide and Sepsis
26 other studies available for hydrogen sulfide and Sepsis
| Article | Year |
|---|---|
Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury.
Topics: Acute Lung Injury; Animals; Autophagy; Cecum; Cell Line; Disease Models, Animal; Ferroptosis; Hydrog | 2022 |
GYY4137 ameliorates sepsis-induced cardiomyopathy via NLRP3 pathway.
Topics: Animals; Cardiomyopathies; Cytokines; Hydrogen Sulfide; Inflammasomes; Lipopolysaccharides; Mice; Mo | 2022 |
[Hydrogen Sulfide Ameliorates Myocardial Injury Caused by Sepsis Through Suppressing ROS-Mediated Endoplasmic Reticulum Stress].
Topics: Activating Transcription Factor 4; Animals; Apoptosis; Cystathionine gamma-Lyase; Endoplasmic Reticu | 2022 |
Hydrogen sulfide alleviates lipopolysaccharide-induced myocardial injury through TLR4-NLRP3 pathway.
Topics: Animals; Cardiomyopathies; Heart Injuries; Hydrogen Sulfide; Inflammation; Lipopolysaccharides; Male | 2023 |
Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis.
Topics: Animals; Cystathionine gamma-Lyase; Humans; Hydrogen Sulfide; Mice; Mice, Knockout; Pseudomonas aeru | 2021 |
GYY4137 protected the integrity of the blood-brain barrier via activation of the Nrf2/ARE pathway in mice with sepsis.
Topics: Animals; Apoptosis; Biological Transport; Blood-Brain Barrier; Brain; Disease Models, Animal; Hydrog | 2021 |
Hydrogen Sulfide in Exhaled Gases From Ventilated Septic Neonates and Children: A Preliminary Report.
Topics: Adolescent; Biomarkers; Breath Tests; Case-Control Studies; Child; Child, Preschool; Exhalation; Fem | 2017 |
Circulating levels of hydrogen sulfide and substance P in patients with sepsis.
Topics: Aged; C-Reactive Protein; Calcitonin; Female; Humans; Hydrogen Sulfide; Intensive Care Units; Interl | 2017 |
H
Topics: Animals; Escherichia coli; Escherichia coli Infections; Host-Pathogen Interactions; Hydrogen Sulfide | 2019 |
[Effect and Mechanism of Hydrogen Sulfide on Septic Rats with Myocardial Injury].
Topics: Animals; Calcitonin; Hydrogen Sulfide; Interleukin-10; Myocardium; NF-kappa B; Protective Agents; Ra | 2018 |
Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition.
Topics: Animals; Apoptosis; Bacteria; Caspase 3; Cecum; Cytokines; Endoplasmic Reticulum Stress; Enzyme Acti | 2014 |
Cystathionine-γ-lyase gene silencing with siRNA in monocytes/ macrophages attenuates inflammation in cecal ligation and puncture-induced sepsis in the mouse.
Topics: Animals; Cystathionine gamma-Lyase; Disease Models, Animal; Gene Silencing; Humans; Hydrogen Sulfide | 2016 |
Cardioprotection by hydrogen sulfide: suspended animation, inflammation, and apoptosis.
Topics: Animals; Apoptosis; Gene Deletion; Humans; Hydrogen Sulfide; Hypoxia; Inflammation; Models, Biologic | 2009 |
Endogenous hydrogen sulfide regulates inflammatory response by activating the ERK pathway in polymicrobial sepsis.
Topics: Animals; Bacteremia; Cecum; Enzyme Activation; Hydrogen Sulfide; Inflammation Mediators; Ligation; M | 2008 |
Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis.
Topics: Alkynes; Animals; Capsaicin; Cystathionine gamma-Lyase; Glycine; Hydrogen Sulfide; Liver; Lung; Male | 2010 |
Hydrogen sulfide, neurogenic inflammation, and cardioprotection: a tale of rotten eggs and vanilloid receptors.
Topics: Animals; Cardiotonic Agents; Cystathionine gamma-Lyase; Humans; Hydrogen Sulfide; Liver; Lung; Mice; | 2010 |
Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation.
Topics: Animals; CD11b Antigen; Cell Movement; Down-Regulation; Endothelium, Vascular; Hydrogen Sulfide; Int | 2010 |
The liver as a central regulator of hydrogen sulfide.
Topics: Animals; Cecum; Hydrogen Sulfide; In Vitro Techniques; Liver; Male; Oxygen Consumption; Phenylephrin | 2011 |
Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling.
Topics: Animals; Capsaicin; Cell Nucleus; Chemokines; Cytokines; Extracellular Signal-Regulated MAP Kinases; | 2011 |
Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation.
Topics: Acute Lung Injury; Animals; Cecum; Cyclooxygenase 2; Dinoprostone; Hydrogen Sulfide; Ligation; Lung; | 2011 |
Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy.
Topics: Aminooxyacetic Acid; Animals; Brain Diseases; Cystathionine beta-Synthase; Cytokines; Disease Models | 2011 |
Role of hydrogen sulfide in cecal ligation and puncture-induced sepsis in the mouse.
Topics: Alkynes; Animals; Cecum; Disease Models, Animal; Enzyme Inhibitors; Glycine; Hydrogen Sulfide; Liver | 2006 |
Hydrogen sulfide acts as an inflammatory mediator in cecal ligation and puncture-induced sepsis in mice by upregulating the production of cytokines and chemokines via NF-kappaB.
Topics: Alkynes; Animals; Cecum; Chemokines; Cystathionine gamma-Lyase; Cytokines; Disease Models, Animal; G | 2007 |
Endogenous hydrogen sulfide regulates leukocyte trafficking in cecal ligation and puncture-induced sepsis.
Topics: Alkynes; Animals; Cell Adhesion; Cell Adhesion Molecules; Chemokine CXCL2; Enzyme Inhibitors; Glycin | 2007 |
Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury.
Topics: Animals; Cecum; Gene Deletion; Hydrogen Sulfide; Inflammation Mediators; Ligation; Lung; Male; Mice; | 2007 |
Recognition of Citrobacter diversus in the clinical laboratory.
Topics: Alcohols; Anti-Bacterial Agents; Bacteriological Techniques; Bacteriuria; Burns; Child; Citrates; Cu | 1973 |