hydrogen sulfide has been researched along with Pancreatitis in 23 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Pancreatitis: INFLAMMATION of the PANCREAS. Pancreatitis is classified as acute unless there are computed tomographic or endoscopic retrograde cholangiopancreatographic findings of CHRONIC PANCREATITIS (International Symposium on Acute Pancreatitis, Atlanta, 1992). The two most common forms of acute pancreatitis are ALCOHOLIC PANCREATITIS and gallstone pancreatitis.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Previous studies have shown that during severe acute pancreatitis (SAP) attacks, hydrogen sulfide (H2S) is released in the colon." | 7.96 | Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis. ( Liao, R; Liu, Y; Qiang, Z; Xue, L; Zhang, C, 2020) |
| "To study the change in endogenous hydrogen sulfide (H2S) in patients with acute pancreatitis and its relationship to coagulation function." | 7.83 | [Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function]. ( Lu, G; Wang, H; Wu, A, 2016) |
| "The present study investigated whether chemokines are involved in hydrogen sulfide (H2S)-associated pathogenesis of acute pancreatitis and associated lung injury." | 7.74 | Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury. ( Bhatia, M; Moore, PK; Tamizhselvi, R, 2008) |
| "Inflammation is a natural response to tissue injury." | 6.72 | Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis. ( Bhatia, M; Kumar, A, 2021) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 6.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 5.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| "Acute pancreatitis was induced in mice by 10 hourly intraperitoneal injections of caerulein (50 (g/kg)." | 5.35 | Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis. ( Bhatia, M; Moochhala, SM; Ng, SW; Sidhapuriwala, JN; Tamizhselvi, R, 2008) |
| "Hydrogen sulfide (H2S) is a naturally occurring gas with potent vasodilator activity." | 5.33 | Role of hydrogen sulfide in acute pancreatitis and associated lung injury. ( Bhatia, M; Fu, D; Lau, HY; Moochhala, SM; Moore, PK; Wong, FL, 2005) |
| "Previous studies have shown that during severe acute pancreatitis (SAP) attacks, hydrogen sulfide (H2S) is released in the colon." | 3.96 | Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis. ( Liao, R; Liu, Y; Qiang, Z; Xue, L; Zhang, C, 2020) |
| "To study the change in endogenous hydrogen sulfide (H2S) in patients with acute pancreatitis and its relationship to coagulation function." | 3.83 | [Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function]. ( Lu, G; Wang, H; Wu, A, 2016) |
| "This study aimed to determine the effect of hydrogen sulfide (H2S) on Toll-like receptor 4 (TLR4)-mediated innate immune signaling in acute pancreatitis (AP) via substance P." | 3.77 | Preprotachykinin-A gene deletion regulates hydrogen sulfide-induced toll-like receptor 4 signaling pathway in cerulein-treated pancreatic acinar cells. ( Bhatia, M; Koh, YH; Shrivastava, P; Tamizhselvi, R; Zhang, H, 2011) |
| "The present study investigated whether chemokines are involved in hydrogen sulfide (H2S)-associated pathogenesis of acute pancreatitis and associated lung injury." | 3.74 | Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury. ( Bhatia, M; Moore, PK; Tamizhselvi, R, 2008) |
| "Inflammation is a natural response to tissue injury." | 2.72 | Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis. ( Bhatia, M; Kumar, A, 2021) |
| "Inflammation is a response to traumatic, infectious, post-ischemic, toxic, or autoimmune injury." | 2.52 | H2S and Inflammation: An Overview. ( Bhatia, M, 2015) |
| "Hydrogen sulfide displays a dual role in the mechanism of AP according to its concentration in the system." | 2.50 | From nitric oxide to hyperbaric oxygen: invisible and subtle but nonnegligible gaseous signaling molecules in acute pancreatitis. ( Dong, DL; Iv, JC; Li, L; Sun, B; Wang, G; Wu, LF, 2014) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 2.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 1.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| "Hydrogen sulfide (H2S) has been reported to be involved in the signaling of the inflammatory response; however, there are differing views as to whether it is pro- or anti-inflammatory." | 1.39 | The effect of CSE gene deletion in caerulein-induced acute pancreatitis in the mouse. ( Ang, AD; Bhatia, M; Hegde, A; Ishii, I; Rivers-Auty, J, 2013) |
| "Acute pancreatitis was induced in mice by 10 hourly intraperitoneal injections of caerulein (50 (g/kg)." | 1.35 | Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis. ( Bhatia, M; Moochhala, SM; Ng, SW; Sidhapuriwala, JN; Tamizhselvi, R, 2008) |
| "Hydrogen sulfide (H2S) is a naturally occurring gas with potent vasodilator activity." | 1.33 | Role of hydrogen sulfide in acute pancreatitis and associated lung injury. ( Bhatia, M; Fu, D; Lau, HY; Moochhala, SM; Moore, PK; Wong, FL, 2005) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (13.04) | 29.6817 |
| 2010's | 16 (69.57) | 24.3611 |
| 2020's | 4 (17.39) | 2.80 |
| Authors | Studies |
|---|---|
| Kumar, A | 1 |
| Bhatia, M | 10 |
| Sekiguchi, F | 2 |
| Koike, N | 1 |
| Shimada, Y | 1 |
| Sugimoto, K | 1 |
| Masuda, H | 1 |
| Nakamura, T | 1 |
| Yamaguchi, H | 1 |
| Tanabe, G | 1 |
| Marumoto, S | 1 |
| Kasanami, Y | 1 |
| Tsubota, M | 2 |
| Ohkubo, T | 1 |
| Yoshida, S | 1 |
| Kawabata, A | 2 |
| Liu, Y | 2 |
| Wei, Z | 1 |
| Zhou, J | 1 |
| Ma, Z | 1 |
| Liao, R | 1 |
| Xue, L | 1 |
| Qiang, Z | 1 |
| Zhang, C | 1 |
| Bronowicka-Adamska, P | 1 |
| Hutsch, T | 1 |
| Gawryś-Kopczyńska, M | 1 |
| Maksymiuk, K | 1 |
| Wróbel, M | 1 |
| Sundar, V | 1 |
| Tamizhselvi, R | 5 |
| Lu, G | 1 |
| Wu, A | 1 |
| Wang, H | 1 |
| Manickam, V | 1 |
| Ang, AD | 2 |
| Rivers-Auty, J | 1 |
| Hegde, A | 2 |
| Ishii, I | 1 |
| Wang, G | 2 |
| Iv, JC | 1 |
| Wu, LF | 1 |
| Li, L | 2 |
| Dong, DL | 1 |
| Sun, B | 2 |
| Terada, Y | 1 |
| Fujimura, M | 1 |
| Nishimura, S | 1 |
| Thangaraj, KR | 2 |
| Priyadarshini, SJ | 1 |
| Qureshi, IN | 2 |
| Joseph, AJ | 2 |
| Balasubramanian, KA | 2 |
| Ramachandran, A | 2 |
| Ji, L | 1 |
| Qu, F | 1 |
| Zhang, G | 1 |
| Wang, Y | 1 |
| Bai, X | 1 |
| Pan, S | 1 |
| Xue, D | 1 |
| David, D | 1 |
| Kurien, RT | 1 |
| Chowdhury, SD | 1 |
| Goel, A | 1 |
| Dutta, AK | 1 |
| Simon, EG | 1 |
| Shrivastava, P | 1 |
| Koh, YH | 1 |
| Zhang, H | 1 |
| Sidhapuriwala, JN | 2 |
| Zhu, YZ | 1 |
| Zuka, M | 1 |
| Chinaka, S | 1 |
| Matsumoto, Y | 1 |
| Takayama, N | 1 |
| Hitomi, Y | 1 |
| Nakamura, H | 1 |
| Ohshima, T | 1 |
| Wong, FL | 1 |
| Fu, D | 1 |
| Lau, HY | 1 |
| Moochhala, SM | 2 |
| Moore, PK | 2 |
| Ng, SW | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Intermittent Fasting as a Primary Means for Improving Quality of Life for Acute and Chronic Pancreatitis[NCT04760847] | 64 participants (Anticipated) | Interventional | 2023-12-07 | Not yet recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
5 reviews available for hydrogen sulfide and Pancreatitis
| Article | Year |
|---|---|
Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis.
Topics: Cell Adhesion Molecules; Endothelial Cells; Humans; Hydrogen Sulfide; Inflammation; Integrins; Leuko | 2021 |
From nitric oxide to hyperbaric oxygen: invisible and subtle but nonnegligible gaseous signaling molecules in acute pancreatitis.
Topics: Acute Disease; Animals; Carbon Monoxide; Gases; Humans; Hydrogen; Hydrogen Sulfide; Hyperbaric Oxyge | 2014 |
H₂S and substance P in inflammation.
Topics: Acute Disease; Animals; Burns; Carrageenan; Ceruletide; Disease Models, Animal; Drug Synergism; Edem | 2015 |
H2S and Inflammation: An Overview.
Topics: Animals; Arthritis; Humans; Hydrogen Sulfide; Inflammation; Pancreatitis; Pulmonary Disease, Chronic | 2015 |
Hydrogen sulfide and substance P in inflammation.
Topics: Air Pollutants; Amino Acid Sequence; Animals; Humans; Hydrogen Sulfide; Inflammation; Inflammation M | 2010 |
18 other studies available for hydrogen sulfide and Pancreatitis
| Article | Year |
|---|---|
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Ca
Topics: Animals; Calcium Channels, T-Type; Cystitis; HEK293 Cells; Humans; Hydrogen Sulfide; Mice; Pancreati | 2023 |
Simultaneous multi-signal quantification for highly precise serodiagnosis utilizing a rationally constructed platform.
Topics: Acute Disease; Animals; Electrochemical Techniques; Hydrogen Sulfide; Luminescent Measurements; Mice | 2019 |
Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis.
Topics: Animals; Cell Movement; Chromones; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; Cytokines | 2020 |
Hydrogen sulfide formation in experimental model of acute pancreatitis.
Topics: Animals; Bile Ducts, Extrahepatic; Cystathionine; Cystathionine beta-Synthase; Cystathionine gamma-L | 2019 |
Inhibition of Rb phosphorylation leads to H
Topics: Animals; Antineoplastic Agents; Ceruletide; DNA; Gene Expression Regulation; Hydrogen Sulfide; Lung | 2020 |
[Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function].
Topics: Acute Disease; Antithrombin III; Blood Coagulation; Case-Control Studies; Factor VIII; Humans; Hydro | 2016 |
Menadione (vitamin K3) inhibits hydrogen sulfide and substance P via NF-кB pathway in caerulein-induced acute pancreatitis and associated lung injury in mice.
Topics: Amylases; Animals; Ceruletide; Gene Expression Regulation; Hydrogen Sulfide; Interleukin-1beta; Lung | 2019 |
The effect of CSE gene deletion in caerulein-induced acute pancreatitis in the mouse.
Topics: Animals; Ceruletide; Cystathionine gamma-Lyase; Gene Expression Regulation; Hydrogen Sulfide; Lung D | 2013 |
Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis.
Topics: Analysis of Variance; Animals; Benzimidazoles; Calcium Channel Blockers; Calcium Channels, T-Type; C | 2015 |
Plasma Citrulline, Glycans, and Hydrogen Sulfide in Patients With Acute Pancreatitis: Possible Markers of Intestinal Damage.
Topics: Acute Disease; Adult; Biomarkers; Case-Control Studies; Citrulline; Female; Humans; Hydrogen Sulfide | 2016 |
Hydrogen sulphide exacerbates acute pancreatitis by over-activating autophagy via AMPK/mTOR pathway.
Topics: Alkynes; AMP-Activated Protein Kinases; Animals; Autophagy; Cell Line; Disease Progression; Glycine; | 2016 |
Plasma hydrogen sulphide does not predict severity of acute pancreatitis in humans.
Topics: Acute Disease; Biomarkers; Female; Humans; Hydrogen Sulfide; Male; Pancreatitis; Severity of Illness | 2016 |
Preprotachykinin-A gene deletion regulates hydrogen sulfide-induced toll-like receptor 4 signaling pathway in cerulein-treated pancreatic acinar cells.
Topics: Acute Disease; Animals; Base Sequence; Ceruletide; DNA Primers; Gene Deletion; Hydrogen Sulfide; Imm | 2011 |
Effects of S-propargyl-cysteine (SPRC) in caerulein-induced acute pancreatitis in mice.
Topics: Acute Disease; Amylases; Animals; Anti-Inflammatory Agents, Non-Steroidal; Ceruletide; Cysteine; Cyt | 2012 |
Fatal and non-fatal cases of lime sulfide exposure and pathogenetic mechanisms underlying pancreatic injury: Case reports with an animal experiment.
Topics: Adult; Aged; Air Pollutants; Amylases; Animals; Calcium Compounds; Esophagus; Female; Forensic Patho | 2012 |
Role of hydrogen sulfide in acute pancreatitis and associated lung injury.
Topics: Acute Disease; Alkynes; Amylases; Animals; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; C | 2005 |
Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis.
Topics: Acute Disease; Alkynes; Animals; Ceruletide; Cystathionine beta-Synthase; Glycine; Hydrogen Sulfide; | 2008 |
Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury.
Topics: Acute Disease; Alkynes; Animals; Ceruletide; Chemokine CCL5; Chemokines; Cystathionine gamma-Lyase; | 2008 |