hydrogen sulfide has been researched along with Necrosis in 15 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Necrosis: The death of cells in an organ or tissue due to disease, injury or failure of the blood supply.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Acute hydrogen sulfide (H2S) poisoning produces a coma, the outcome of which ranges from full recovery to severe neurological deficits." | 7.81 | Immediate and Long-Term Outcome of Acute H2S Intoxication Induced Coma in Unanesthetized Rats: Effects of Methylene Blue. ( Chenuel, B; Cooper, TK; Haouzi, P; Sonobe, T, 2015) |
| "Acute hydrogen sulfide (H2S) poisoning produces a coma, the outcome of which ranges from full recovery to severe neurological deficits." | 3.81 | Immediate and Long-Term Outcome of Acute H2S Intoxication Induced Coma in Unanesthetized Rats: Effects of Methylene Blue. ( Chenuel, B; Cooper, TK; Haouzi, P; Sonobe, T, 2015) |
| " The aim of this study was to determine effects of inhibiting hydrogen sulphide biosynthesis by DL-propargyl glycine (an irreversible inhibitor of cystathionine gamma-lyase) on inflammation, necrosis and renal function, following treatment with gentamicin in rats." | 3.78 | Inhibition of cystathionine gamma-lyase and the biosynthesis of endogenous hydrogen sulphide ameliorates gentamicin-induced nephrotoxicity. ( Dam, VP; Kinobe, RT; Ross, A; Scott, JL, 2012) |
| "After 72 h of incubation with 100 ng/mL of hydrogen sulfide, necrosis was found in less than 10% of human gingival fibroblasts, whereas apoptosis was significantly increased (p < 0." | 3.74 | Oral malodorous compound causes apoptosis and genomic DNA damage in human gingival fibroblasts. ( Imai, T; Kamoda, T; Murata, T; Qian, W; Sato, T; Tanaka, T; Yaegaki, K, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (13.33) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (26.67) | 29.6817 |
| 2010's | 9 (60.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Imamura, F | 1 |
| Cooper, TK | 2 |
| Hasegawa-Ishii, S | 1 |
| Sonobe, T | 2 |
| Haouzi, P | 2 |
| Lin, J | 1 |
| Chen, M | 1 |
| Liu, D | 1 |
| Guo, R | 1 |
| Lin, K | 1 |
| Deng, H | 1 |
| Zhi, X | 1 |
| Zhang, W | 1 |
| Feng, J | 1 |
| Wu, W | 1 |
| Grambow, E | 1 |
| Augustin, VA | 1 |
| Strüder, D | 1 |
| Kundt, G | 1 |
| Klar, E | 1 |
| Vollmar, B | 1 |
| Chenuel, B | 1 |
| Ge, N | 1 |
| Liu, C | 1 |
| Li, G | 1 |
| Xie, L | 1 |
| Zhang, Q | 1 |
| Li, L | 1 |
| Hao, N | 1 |
| Zhang, J | 1 |
| Yaegaki, K | 1 |
| Qian, W | 1 |
| Murata, T | 1 |
| Imai, T | 1 |
| Sato, T | 1 |
| Tanaka, T | 1 |
| Kamoda, T | 1 |
| Li, HH | 1 |
| Xu, J | 1 |
| Wasserloos, KJ | 1 |
| Li, J | 1 |
| Tyurina, YY | 1 |
| Kagan, VE | 1 |
| Wang, X | 1 |
| Chen, AF | 1 |
| Liu, ZQ | 1 |
| Stoyanovsky, D | 1 |
| Pitt, BR | 1 |
| Zhang, LM | 1 |
| Salloum, FN | 1 |
| Das, A | 1 |
| Samidurai, A | 1 |
| Hoke, NN | 1 |
| Chau, VQ | 1 |
| Ockaili, RA | 1 |
| Stasch, JP | 1 |
| Kukreja, RC | 1 |
| Dam, VP | 1 |
| Scott, JL | 1 |
| Ross, A | 1 |
| Kinobe, RT | 1 |
| Zuka, M | 1 |
| Chinaka, S | 1 |
| Matsumoto, Y | 1 |
| Takayama, N | 1 |
| Hitomi, Y | 1 |
| Nakamura, H | 1 |
| Ohshima, T | 1 |
| Christia-Lotter, A | 1 |
| Bartoli, C | 1 |
| Piercecchi-Marti, MD | 1 |
| Demory, D | 1 |
| Pelissier-Alicot, AL | 1 |
| Sanvoisin, A | 1 |
| Leonetti, G | 1 |
| Mirandola, P | 1 |
| Gobbi, G | 1 |
| Sponzilli, I | 1 |
| Pambianco, M | 1 |
| Malinverno, C | 1 |
| Cacchioli, A | 1 |
| De Panfilis, G | 1 |
| Vitale, M | 1 |
| Ago, M | 1 |
| Ago, K | 1 |
| Ogata, M | 1 |
| Lan, WH | 1 |
| Lopez, A | 1 |
| Prior, M | 1 |
| Yong, S | 1 |
| Lillie, L | 1 |
| Lefebvre, M | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Effect Off Full Mouth Disinfection And Scaling Root Planing Per Quadrant In Halitosis in Patients With Advanced Chronic Periodontitis: Randomized Controlled Clinical Trial[NCT02368678] | 30 participants (Actual) | Interventional | 2013-04-30 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
15 other studies available for hydrogen sulfide and Necrosis
| Article | Year |
|---|---|
Hydrogen Sulfide Specifically Alters NAD(P)H Quinone Dehydrogenase 1 (NQO1) Olfactory Neurons in the Rat.
Topics: Animals; Hydrogen Sulfide; Male; NAD(P)H Dehydrogenase (Quinone); Necrosis; Neurons; Olfactory Mucos | 2017 |
Exogenous hydrogen sulfide protects human umbilical vein endothelial cells against high glucose‑induced injury by inhibiting the necroptosis pathway.
Topics: Amino Acid Chloromethyl Ketones; Apoptosis; Caspase 3; Cytoprotection; Glucose; Human Umbilical Vein | 2018 |
The effects of hydrogen sulfide on microvascular circulation in the axial pattern flap ear model in hairless mice.
Topics: Animals; Capillaries; Ear; Edema; Hydrogen Sulfide; Mice, Hairless; Microcirculation; Morpholines; N | 2018 |
Immediate and Long-Term Outcome of Acute H2S Intoxication Induced Coma in Unanesthetized Rats: Effects of Methylene Blue.
Topics: Animals; Brain; Coma; Hydrogen Sulfide; Male; Methylene Blue; Necrosis; Neurons; Rats; Rats, Sprague | 2015 |
Hydrosulfide attenuates acute myocardial ischemic injury through the glycogen synthase kinase-3β/β-catenin signaling pathway.
Topics: Animals; Apoptosis; beta Catenin; Biomarkers; Blood Pressure; Disease Models, Animal; Glycogen Synth | 2016 |
Oral malodorous compound causes apoptosis and genomic DNA damage in human gingival fibroblasts.
Topics: Annexin A5; Apoptosis; Caspase 3; Cell Line; Cell Survival; Coloring Agents; DNA; DNA Damage; DNA Fr | 2008 |
Cytoprotective effects of albumin, nitrosated or reduced, in cultured rat pulmonary vascular cells.
Topics: Animals; Apoptosis; Arteries; Cells, Cultured; Cytoprotection; Endocytosis; Endothelial Cells; Human | 2011 |
Cinaciguat, a novel activator of soluble guanylate cyclase, protects against ischemia/reperfusion injury: role of hydrogen sulfide.
Topics: Animals; Apoptosis; Benzoates; Cell Survival; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Cyst | 2012 |
Inhibition of cystathionine gamma-lyase and the biosynthesis of endogenous hydrogen sulphide ameliorates gentamicin-induced nephrotoxicity.
Topics: Alkynes; Animals; Anti-Bacterial Agents; Cystathionine gamma-Lyase; Female; Gentamicins; Glycine; Hy | 2012 |
Fatal and non-fatal cases of lime sulfide exposure and pathogenetic mechanisms underlying pancreatic injury: Case reports with an animal experiment.
Topics: Adult; Aged; Air Pollutants; Amylases; Animals; Calcium Compounds; Esophagus; Female; Forensic Patho | 2012 |
Fatal occupational inhalation of hydrogen sulfide.
Topics: Adult; Air Pollutants; Creatine Kinase; Fatal Outcome; Humans; Hydrogen Sulfide; Male; Myocardium; N | 2007 |
Exogenous hydrogen sulfide induces functional inhibition and cell death of cytotoxic lymphocytes subsets.
Topics: Annexin A5; CD8-Positive T-Lymphocytes; Cell Culture Techniques; Cell Death; Cell Proliferation; Cel | 2007 |
Two fatalities by hydrogen sulfide poisoning: variation of pathological and toxicological findings.
Topics: Adult; Air Pollutants; Biomarkers; Bronchi; Forensic Pathology; Forensic Toxicology; Humans; Hydroge | 2008 |
Neutralization effect of some agents on the antimicrobial activity of ammoniacal silver nitrate.
Topics: Animals; Anti-Bacterial Agents; Blood; Blood Coagulation; Dental Pulp Cavity; Dentin; Enterococcus f | 1978 |
Nasal lesions in rats exposed to hydrogen sulfide for four hours.
Topics: Animals; Epithelium; Hydrogen Sulfide; Male; Nasal Cavity; Nasal Mucosa; Necrosis; Rats; Rats, Inbre | 1988 |