hydrogen sulfide has been researched along with Myocarditis in 7 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Myocarditis: Inflammatory processes of the muscular walls of the heart (MYOCARDIUM) which result in injury to the cardiac muscle cells (MYOCYTES, CARDIAC). Manifestations range from subclinical to sudden death (DEATH, SUDDEN). Myocarditis in association with cardiac dysfunction is classified as inflammatory CARDIOMYOPATHY usually caused by INFECTION, autoimmune diseases, or responses to toxic substances. Myocarditis is also a common cause of DILATED CARDIOMYOPATHY and other cardiomyopathies.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To explore the impact of hydrogen sulfide (H2S) on the heme oxygenase—1 (HO—1)/carbon monoxide (CO) system in coxsackie virus B3 (CVB3)—induced myocarditis." | 7.81 | Regulation of the Heme Oxygenase-1/carbon monoxide system by hydrogen sulfide in murine coxsackievirus B3-induced myocarditis. ( Chu, M; Rong, X; Wu, R; Wu, T; Xia, T; Zhang, S, 2015) |
| "This study is to determine the therapeutic effects of Panax notoginseng saponins (PNSs) on coxsackievirus B3 (CVB3)-induced myocarditis, and whether cystathionine-γ-lyase (CSE)/hydrogen sulfide (H2S) pathway is involved." | 7.81 | Panax Notoginseng Saponins Ameliorates Coxsackievirus B3-Induced Myocarditis by Activating the Cystathionine-γ-Lyase/Hydrogen Sulfide Pathway. ( Chu, M; Geng, Z; Jia, L; Lu, J; Pan, L; Rong, X; Wang, Z; Wu, R; Zhang, C; Zhang, Y; Zhao, Q, 2015) |
| "To explore the protective effect of hydrogen sulfide (H2S) on viral myocarditis (VMC) mice and its mechanism." | 7.80 | [Protective effect of hydrogen sulfide on mice with experimental viral myocarditis and its mechanism]. ( Liu, L; Qian, H, 2014) |
| "Previous studies have shown that hydrogen sulfide (H2S) plays key roles in a number of biological processes, including vasorelaxation, inflammation, apoptosis, ischemia/reperfusion and oxidative stress, which are involved in the pathogenesis of myocarditis." | 7.76 | [Expression of cystathionine-γ-lyase/hydrogen sulfide pathway in CVB3-induced myocarditis in mice]. ( Chen, Q; Hua, W; Jiang, JB; Qiu, HX; Rong, X; Wu, RZ; Zhang, YH, 2010) |
| "To explore the impact of hydrogen sulfide (H2S) on the heme oxygenase—1 (HO—1)/carbon monoxide (CO) system in coxsackie virus B3 (CVB3)—induced myocarditis." | 3.81 | Regulation of the Heme Oxygenase-1/carbon monoxide system by hydrogen sulfide in murine coxsackievirus B3-induced myocarditis. ( Chu, M; Rong, X; Wu, R; Wu, T; Xia, T; Zhang, S, 2015) |
| "This study is to determine the therapeutic effects of Panax notoginseng saponins (PNSs) on coxsackievirus B3 (CVB3)-induced myocarditis, and whether cystathionine-γ-lyase (CSE)/hydrogen sulfide (H2S) pathway is involved." | 3.81 | Panax Notoginseng Saponins Ameliorates Coxsackievirus B3-Induced Myocarditis by Activating the Cystathionine-γ-Lyase/Hydrogen Sulfide Pathway. ( Chu, M; Geng, Z; Jia, L; Lu, J; Pan, L; Rong, X; Wang, Z; Wu, R; Zhang, C; Zhang, Y; Zhao, Q, 2015) |
| "To explore the protective effect of hydrogen sulfide (H2S) on viral myocarditis (VMC) mice and its mechanism." | 3.80 | [Protective effect of hydrogen sulfide on mice with experimental viral myocarditis and its mechanism]. ( Liu, L; Qian, H, 2014) |
| "To explore the effects and potential mechanisms of hydrogen sulfide (H2S) in CVB3-induced mice with myocarditis." | 3.79 | Cardioprotection of H2S by downregulating iNOS and upregulating HO-1 expression in mice with CVB3-induced myocarditis. ( Chen, Q; Gao, L; Gong, F; Hua, W; Xie, C; Zhou, S, 2013) |
| "Previous studies have shown that hydrogen sulfide (H2S) plays key roles in a number of biological processes, including vasorelaxation, inflammation, apoptosis, ischemia/reperfusion and oxidative stress, which are involved in the pathogenesis of myocarditis." | 3.76 | [Expression of cystathionine-γ-lyase/hydrogen sulfide pathway in CVB3-induced myocarditis in mice]. ( Chen, Q; Hua, W; Jiang, JB; Qiu, HX; Rong, X; Wu, RZ; Zhang, YH, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (14.29) | 29.6817 |
| 2010's | 6 (85.71) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hua, W | 3 |
| Chen, Q | 3 |
| Gong, F | 1 |
| Xie, C | 1 |
| Zhou, S | 1 |
| Gao, L | 1 |
| Qian, H | 1 |
| Liu, L | 1 |
| Wu, Z | 1 |
| Peng, H | 1 |
| Du, Q | 1 |
| Lin, W | 1 |
| Liu, Y | 1 |
| Zhang, S | 1 |
| Wu, T | 2 |
| Xia, T | 1 |
| Rong, X | 4 |
| Chu, M | 2 |
| Wu, R | 3 |
| Pan, L | 1 |
| Zhang, Y | 2 |
| Lu, J | 1 |
| Geng, Z | 1 |
| Jia, L | 1 |
| Wang, Z | 1 |
| Zhao, Q | 1 |
| Zhang, C | 1 |
| Jiang, J | 1 |
| Qiu, H | 1 |
| Jiang, JB | 1 |
| Wu, RZ | 1 |
| Qiu, HX | 1 |
| Zhang, YH | 1 |
7 other studies available for hydrogen sulfide and Myocarditis
| Article | Year |
|---|---|
Cardioprotection of H2S by downregulating iNOS and upregulating HO-1 expression in mice with CVB3-induced myocarditis.
Topics: Animals; Blotting, Western; Coxsackievirus Infections; Enterovirus B, Human; Heme Oxygenase-1; Hydro | 2013 |
[Protective effect of hydrogen sulfide on mice with experimental viral myocarditis and its mechanism].
Topics: Animals; Blotting, Western; Catalase; Coxsackievirus Infections; Enterovirus; Enzyme-Linked Immunoso | 2014 |
GYY4137, a hydrogen sulfide‑releasing molecule, inhibits the inflammatory response by suppressing the activation of nuclear factor‑kappa B and mitogen‑activated protein kinases in Coxsackie virus B3‑infected rat cardiomyocytes.
Topics: Active Transport, Cell Nucleus; Animals; Anti-Inflammatory Agents; Cells, Cultured; Coxsackievirus I | 2015 |
Regulation of the Heme Oxygenase-1/carbon monoxide system by hydrogen sulfide in murine coxsackievirus B3-induced myocarditis.
Topics: Alkynes; Animals; Carbon Monoxide; Carboxyhemoglobin; Enterovirus B, Human; Glycine; HeLa Cells; Hem | 2015 |
Panax Notoginseng Saponins Ameliorates Coxsackievirus B3-Induced Myocarditis by Activating the Cystathionine-γ-Lyase/Hydrogen Sulfide Pathway.
Topics: Analysis of Variance; Animals; Biomarkers; Blotting, Western; Coxsackievirus Infections; Cystathioni | 2015 |
The dual role of the cystathionine gamma-lyase/hydrogen sulfide pathway in CVB3-induced myocarditis in mice.
Topics: Animals; Coxsackievirus Infections; Cystathionine gamma-Lyase; Disease Models, Animal; HeLa Cells; H | 2009 |
[Expression of cystathionine-γ-lyase/hydrogen sulfide pathway in CVB3-induced myocarditis in mice].
Topics: Animals; Coxsackievirus Infections; Cystathionine gamma-Lyase; Enterovirus B, Human; Extracellular S | 2010 |