hydrogen sulfide has been researched along with Lung Injury, Acute in 27 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The aim of this work was to test the effect of treatment with hydrogen sulfide (H2S) on hyperoxia-induced acute lung injury in mice." | 7.79 | Treatment with exogenous hydrogen sulfide attenuates hyperoxia-induced acute lung injury in mice. ( Chen, JS; He, DM; Li, HD; Qin, ZC; Zhang, QX; Zhang, ZR, 2013) |
| "To investigate the influence of hydrogen sulfide (H₂S) on p38 MAPK signaling pathway during acute lung injury (ALI) caused by lipopolysaccharide (LPS), the rats were randomly divided into six groups: control group, LPS group, LPS + NaHS group, LPS + PPG (cystathionine-γ-lyase inhibitor) group, NaHS group and PPG group." | 7.78 | [Hydrogen sulfide reduces lipopolysaccharide-induced acute lung injury and inhibits expression of phosphorylated p38 MAPK in rats]. ( Dong, ZF; Fan, YM; Huang, XL; Ling, YL, 2012) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 7.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "To observe the changes of pulmonary surfactant (PS) in rats with acute lung injury(ALI) induced by lipopolysaccharide (LPS) and to explore the effects of hydrogen sulfide (H2S) on PS." | 7.77 | [Effects of hydrogen sulfide on pulmonary surfactant in rats with acute lung injury induced by lipopolysccharide]. ( Ding, CM; Gong, JP; Jin, PL; Li, LF; Wang, P; Zhang, JX, 2011) |
| "To explore the potential effects of hydrogen sulfide (H(2)S) on nuclear factor kappa B (NF-κB) and intercellular adhesion molecule 1 (ICAM-1) in lung tissue with oleic acid (OA)-induced acute lung injury (ALI) in rats." | 7.77 | [Effects of hydrogen sulfide on the expressions of nuclear factor-κB and intercellular adhesion molecule 1 in pulmonary tissue of rats with acute lung injury]. ( DU, JB; Guan, L; Jin, HF; Li, M; Li, TS; Liu, ZW; Zhao, B, 2011) |
| "Endogenous H(2)S could inhibit the oxidative stress in lung tissue in oleic acid-induced acute lung injury in rats." | 7.77 | Effect of endogenous hydrogen sulfide on oxidative stress in oleic acid-induced acute lung injury in rats. ( Fu, Y; Liu, ZW; Wang, C; Wang, HY; Zhao, B, 2011) |
| "To explore the role of hydrogen sulfide/cystathionine-gamma-lyase (H(2)S/CSE) system in lipopolysaccharide (LPS)- induced acute lung injury (ALI) in rats and the underlying mechanisms." | 7.75 | [Role of hydrogen sulfide/cystathionine-gamma-lyase system in acute lung injury induced by lipopolysaccharide in rats]. ( Huang, XL; Ling, YL; Tian, FJ; Wei, P; Zhou, XH, 2009) |
| "The animal model of acute lung injury (ALI) caused by intravenous injection of lipopolysaccharides (LPS) and cultured human peripheral blood polymorphonuclear neutrophil (PMN) were used to study the effects of sodium hydrosulfide (NaHS), hydrogen sulfide (H2S) donor, on LPS-induced PMN accumulation, microvascular permeability and PMN apoptosis." | 7.75 | [Role of polymorphonuclear neutrophil in exogenous hydrogen sulfide attenuating endotoxin-induced acute lung injury]. ( Ding, CH; Huang, XL; Xian, XH; Zhou, JL; Zhou, XH, 2009) |
| "To explore the role of endogenous and exogenous hydrogen sulfide (H2S) in lipopolysaccharide (LPS)-induced acute lung injury (ALI) in rats and the underlying mechanisms." | 7.75 | [Role of endogenous and exogenous hydrogen sulfide in acute lung injury induced by LPS in rats]. ( Huang, XL; Ling, YL; Wei, P; Zhou, XH, 2009) |
| "To explore the role of hydrogen sulfide (H2S) in acute lung injury (ALI) during endotoxic shock (ES) and its relationship with nitric oxide (NO) and carbon monoxide (CO)." | 7.74 | [The role of hydrogen sulfide in acute lung injury during endotoxic shock and its relationship with nitric oxide and carbon monoxide]. ( Huang, XL; Ling, YL; Wei, P; Xian, XH; Zhou, XH, 2008) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 5.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H(2)S) plays a protective role during acute lung injury." | 5.39 | Hydrogen sulfide donor regulates alveolar epithelial cell apoptosis in rats with acute lung injury. ( Li, TS; Liu, WL; Liu, ZW; Wang, C; Zhao, B, 2013) |
| "Acute lung injury (ALI) is one of the fatal outcomes after exposure to high levels of hydrogen sulfide (H2S), and the matrix metalloproteinases (MMPs) especially MMP-2 and MMP-9 are believed to be involved in the development of ALI by degrading the extracellular matrix (ECM) of blood-air barrier." | 3.80 | Dexamethasone ameliorates H₂S-induced acute lung injury by alleviating matrix metalloproteinase-2 and -9 expression. ( Chen, J; Su, C; Wang, J; Xiao, H; Zhang, H; Zhang, J; Zhu, B, 2014) |
| "The aim of this work was to test the effect of treatment with hydrogen sulfide (H2S) on hyperoxia-induced acute lung injury in mice." | 3.79 | Treatment with exogenous hydrogen sulfide attenuates hyperoxia-induced acute lung injury in mice. ( Chen, JS; He, DM; Li, HD; Qin, ZC; Zhang, QX; Zhang, ZR, 2013) |
| "To investigate the influence of hydrogen sulfide (H₂S) on p38 MAPK signaling pathway during acute lung injury (ALI) caused by lipopolysaccharide (LPS), the rats were randomly divided into six groups: control group, LPS group, LPS + NaHS group, LPS + PPG (cystathionine-γ-lyase inhibitor) group, NaHS group and PPG group." | 3.78 | [Hydrogen sulfide reduces lipopolysaccharide-induced acute lung injury and inhibits expression of phosphorylated p38 MAPK in rats]. ( Dong, ZF; Fan, YM; Huang, XL; Ling, YL, 2012) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 3.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "To observe the changes of pulmonary surfactant (PS) in rats with acute lung injury(ALI) induced by lipopolysaccharide (LPS) and to explore the effects of hydrogen sulfide (H2S) on PS." | 3.77 | [Effects of hydrogen sulfide on pulmonary surfactant in rats with acute lung injury induced by lipopolysccharide]. ( Ding, CM; Gong, JP; Jin, PL; Li, LF; Wang, P; Zhang, JX, 2011) |
| "To explore the potential effects of hydrogen sulfide (H(2)S) on nuclear factor kappa B (NF-κB) and intercellular adhesion molecule 1 (ICAM-1) in lung tissue with oleic acid (OA)-induced acute lung injury (ALI) in rats." | 3.77 | [Effects of hydrogen sulfide on the expressions of nuclear factor-κB and intercellular adhesion molecule 1 in pulmonary tissue of rats with acute lung injury]. ( DU, JB; Guan, L; Jin, HF; Li, M; Li, TS; Liu, ZW; Zhao, B, 2011) |
| "Endogenous H(2)S could inhibit the oxidative stress in lung tissue in oleic acid-induced acute lung injury in rats." | 3.77 | Effect of endogenous hydrogen sulfide on oxidative stress in oleic acid-induced acute lung injury in rats. ( Fu, Y; Liu, ZW; Wang, C; Wang, HY; Zhao, B, 2011) |
| "To explore the role of hydrogen sulfide/cystathionine-gamma-lyase (H(2)S/CSE) system in lipopolysaccharide (LPS)- induced acute lung injury (ALI) in rats and the underlying mechanisms." | 3.75 | [Role of hydrogen sulfide/cystathionine-gamma-lyase system in acute lung injury induced by lipopolysaccharide in rats]. ( Huang, XL; Ling, YL; Tian, FJ; Wei, P; Zhou, XH, 2009) |
| "The animal model of acute lung injury (ALI) caused by intravenous injection of lipopolysaccharides (LPS) and cultured human peripheral blood polymorphonuclear neutrophil (PMN) were used to study the effects of sodium hydrosulfide (NaHS), hydrogen sulfide (H2S) donor, on LPS-induced PMN accumulation, microvascular permeability and PMN apoptosis." | 3.75 | [Role of polymorphonuclear neutrophil in exogenous hydrogen sulfide attenuating endotoxin-induced acute lung injury]. ( Ding, CH; Huang, XL; Xian, XH; Zhou, JL; Zhou, XH, 2009) |
| "To explore the role of endogenous and exogenous hydrogen sulfide (H2S) in lipopolysaccharide (LPS)-induced acute lung injury (ALI) in rats and the underlying mechanisms." | 3.75 | [Role of endogenous and exogenous hydrogen sulfide in acute lung injury induced by LPS in rats]. ( Huang, XL; Ling, YL; Wei, P; Zhou, XH, 2009) |
| "To explore the role of hydrogen sulfide (H2S) in acute lung injury (ALI) during endotoxic shock (ES) and its relationship with nitric oxide (NO) and carbon monoxide (CO)." | 3.74 | [The role of hydrogen sulfide in acute lung injury during endotoxic shock and its relationship with nitric oxide and carbon monoxide]. ( Huang, XL; Ling, YL; Wei, P; Xian, XH; Zhou, XH, 2008) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 1.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H2S) is a highly toxic gaseous molecule that causes death to humans exposed to high concentrations." | 1.43 | Hydrogen sulfide donor NaHS induces death of alveolar epithelial L2 cells that is associated with cellular shrinkage, transgelin expression and myosin phosphorylation. ( Aki, T; Fujii, Y; Funakoshi, T; Noritake, K; Uemura, K; Unuma, K, 2016) |
| "Hydrogen sulfide (H(2)S) plays a protective role during acute lung injury." | 1.39 | Hydrogen sulfide donor regulates alveolar epithelial cell apoptosis in rats with acute lung injury. ( Li, TS; Liu, WL; Liu, ZW; Wang, C; Zhao, B, 2013) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 5 (18.52) | 29.6817 |
| 2010's | 20 (74.07) | 24.3611 |
| 2020's | 2 (7.41) | 2.80 |
| Authors | Studies |
|---|---|
| Li, J | 1 |
| Li, M | 2 |
| Li, L | 1 |
| Ma, J | 1 |
| Yao, C | 1 |
| Yao, S | 1 |
| Liang, Y | 1 |
| Zhang, JJ | 1 |
| Gao, Y | 1 |
| Zhang, H | 3 |
| Du, Q | 1 |
| Zhang, M | 1 |
| Li, G | 1 |
| Wang, C | 3 |
| Zhang, N | 1 |
| Zhang, J | 2 |
| Yang, ZH | 1 |
| Xu, YM | 1 |
| Jiang, XY | 1 |
| Lin, JJ | 1 |
| Wang, DS | 1 |
| Chen, YL | 1 |
| Zhao, HL | 1 |
| Liu, WL | 1 |
| Liu, ZW | 3 |
| Li, TS | 2 |
| Zhao, B | 3 |
| Ryter, SW | 1 |
| Choi, AM | 1 |
| Wang, J | 1 |
| Su, C | 1 |
| Chen, J | 2 |
| Zhu, B | 1 |
| Xiao, H | 1 |
| Qi, QY | 1 |
| Chen, W | 1 |
| Li, XL | 1 |
| Wang, YW | 1 |
| Xie, XH | 1 |
| Tang, B | 1 |
| Ma, L | 1 |
| Yao, X | 1 |
| Tan, G | 1 |
| Han, P | 1 |
| Yu, T | 1 |
| Liu, B | 1 |
| Sun, X | 1 |
| Fujii, Y | 1 |
| Funakoshi, T | 1 |
| Unuma, K | 1 |
| Noritake, K | 1 |
| Aki, T | 1 |
| Uemura, K | 1 |
| Tian, F | 1 |
| Ling, Y | 1 |
| Chen, Y | 1 |
| Wang, Z | 1 |
| Zhang, HX | 1 |
| Liu, SJ | 1 |
| Tang, XL | 1 |
| Duan, GL | 1 |
| Ni, X | 1 |
| Zhu, XY | 1 |
| Liu, YJ | 1 |
| Wang, CN | 1 |
| Huang, XL | 6 |
| Zhou, XH | 5 |
| Wei, P | 3 |
| Xian, XH | 3 |
| Ling, YL | 4 |
| Tian, FJ | 1 |
| Zhou, JL | 1 |
| Ding, CH | 1 |
| Esechie, A | 1 |
| Enkhbaatar, P | 1 |
| Traber, DL | 1 |
| Jonkam, C | 1 |
| Lange, M | 1 |
| Hamahata, A | 1 |
| Djukom, C | 1 |
| Whorton, EB | 1 |
| Hawkins, HK | 1 |
| Traber, LD | 1 |
| Szabo, C | 1 |
| ma, HJ | 1 |
| Fan, YM | 2 |
| Cao, H | 1 |
| Wu, N | 1 |
| Du, X | 1 |
| Wang, D | 1 |
| Hao, F | 1 |
| Ang, SF | 1 |
| Sio, SW | 1 |
| Moochhala, SM | 1 |
| MacAry, PA | 1 |
| Bhatia, M | 1 |
| Yu, G | 1 |
| Chi, X | 1 |
| Hei, Z | 1 |
| Shen, N | 1 |
| Zhang, W | 1 |
| Li, S | 1 |
| Wang, P | 1 |
| Zhang, JX | 1 |
| Gong, JP | 1 |
| Li, LF | 1 |
| Jin, PL | 1 |
| Ding, CM | 1 |
| Guan, L | 1 |
| Jin, HF | 1 |
| DU, JB | 1 |
| Wang, HY | 1 |
| Fu, Y | 1 |
| Ge, Y | 1 |
| Sun, W | 1 |
| Wu, ZS | 1 |
| Jiang, XZ | 1 |
| Qiu, QM | 1 |
| Hong, GL | 1 |
| Liang, H | 1 |
| Li, MF | 1 |
| Lu, ZQ | 1 |
| Dong, ZF | 1 |
| Li, HD | 1 |
| Zhang, ZR | 1 |
| Zhang, QX | 1 |
| Qin, ZC | 1 |
| He, DM | 1 |
| Chen, JS | 1 |
1 review available for hydrogen sulfide and Lung Injury, Acute
| Article | Year |
|---|---|
Gaseous therapeutics in acute lung injury.
Topics: Acute Lung Injury; Administration, Inhalation; Animals; Carbon Monoxide; Disease Models, Animal; Hum | 2011 |
26 other studies available for hydrogen sulfide and Lung Injury, Acute
| Article | Year |
|---|---|
Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury.
Topics: Acute Lung Injury; Animals; Autophagy; Cecum; Cell Line; Disease Models, Animal; Ferroptosis; Hydrog | 2022 |
[Three cases of acute lung injury caused by occupational hydrogen sulfide poisoning].
Topics: Accidents, Occupational; Acute Lung Injury; Humans; Hydrogen Sulfide; Poisoning | 2020 |
[Effects of hydrogen sulfide on mitochondria of lung in rats with ALI induced by lipopolysaccharide].
Topics: Acute Lung Injury; Adenosine Triphosphatases; Animals; Hydrogen Sulfide; Lipid Peroxidation; Lipopol | 2017 |
[The correlation between CT score of lung injury and oxygenation index in patients with acute hydrogen sulphide poisoning].
Topics: Acute Lung Injury; Blood Gas Analysis; Humans; Hydrogen Sulfide; Lung; Lung Injury; Respiratory Dist | 2017 |
Hydrogen sulfide donor regulates alveolar epithelial cell apoptosis in rats with acute lung injury.
Topics: Acute Lung Injury; Animals; Apoptosis; Epithelial Cells; Hydrogen Sulfide; In Situ Nick-End Labeling | 2013 |
Dexamethasone ameliorates H₂S-induced acute lung injury by alleviating matrix metalloproteinase-2 and -9 expression.
Topics: Acute Lung Injury; Animals; Cell Line; Dexamethasone; Disease Models, Animal; Glucocorticoids; Hydro | 2014 |
H₂S protecting against lung injury following limb ischemia-reperfusion by alleviating inflammation and water transport abnormality in rats.
Topics: Acute Lung Injury; Animals; Aquaporins; Drug Evaluation, Preclinical; Edema; Hydrogen Sulfide; Infla | 2014 |
Hydrogen sulfide ameliorates acute lung injury induced by infrarenal aortic cross-clamping by inhibiting inflammation and angiopoietin 2 release.
Topics: Acute Lung Injury; Alkynes; Angiopoietin-2; Animals; Anti-Inflammatory Agents; Aorta, Abdominal; Con | 2017 |
Hydrogen sulfide donor NaHS induces death of alveolar epithelial L2 cells that is associated with cellular shrinkage, transgelin expression and myosin phosphorylation.
Topics: Acute Lung Injury; Animals; Cell Death; Cell Line; Cell Size; Chromatin Assembly and Disassembly; Do | 2016 |
Effects of CCK-8 and Cystathionine γ-Lyase/Hydrogen Sulfide System on Acute Lung Injury in Rats.
Topics: Acute Lung Injury; Animals; Anti-Inflammatory Agents; Cystathionine gamma-Lyase; Humans; Hydrogen Su | 2017 |
H2S Attenuates LPS-Induced Acute Lung Injury by Reducing Oxidative/Nitrative Stress and Inflammation.
Topics: Acute Lung Injury; Animals; Antioxidants; Endotoxemia; Hydrogen Peroxide; Hydrogen Sulfide; Inflamma | 2016 |
[The role of hydrogen sulfide in acute lung injury during endotoxic shock and its relationship with nitric oxide and carbon monoxide].
Topics: Acute Lung Injury; Animals; Blood Pressure; Carbon Monoxide; Cystathionine gamma-Lyase; Heme Oxygena | 2008 |
[Role of hydrogen sulfide/cystathionine-gamma-lyase system in acute lung injury induced by lipopolysaccharide in rats].
Topics: Acute Lung Injury; Animals; Carbon Monoxide; Cystathionine gamma-Lyase; Disease Models, Animal; Heme | 2009 |
[Role of polymorphonuclear neutrophil in exogenous hydrogen sulfide attenuating endotoxin-induced acute lung injury].
Topics: Acute Lung Injury; Animals; Apoptosis; Disease Models, Animal; Humans; Hydrogen Sulfide; Lipopolysac | 2009 |
Beneficial effect of a hydrogen sulphide donor (sodium sulphide) in an ovine model of burn- and smoke-induced acute lung injury.
Topics: Acute Lung Injury; Animals; Blotting, Western; Burns; Cytochromes c; Disease Models, Animal; Enzyme- | 2009 |
[Role of endogenous and exogenous hydrogen sulfide in acute lung injury induced by LPS in rats].
Topics: Acute Lung Injury; Animals; Antioxidants; Bronchoalveolar Lavage Fluid; Carbon Monoxide; Heme Oxygen | 2009 |
[Effect of exogenous hydrogen sulfide on polymorphonuclear neutrophil accumulation in acute lung injury rat induced by lipopolysaccharides and its mechanism].
Topics: Acute Lung Injury; Animals; Apoptosis; Hydrogen Sulfide; Intercellular Adhesion Molecule-1; Lipopoly | 2010 |
Myocardial and lung injuries induced by hydrogen sulfide and the effectiveness of oxygen therapy in rats.
Topics: Acute Lung Injury; Air Pollutants; Alveolar Epithelial Cells; Animals; Blood Gas Analysis; Cardiomyo | 2011 |
Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation.
Topics: Acute Lung Injury; Animals; Cecum; Cyclooxygenase 2; Dinoprostone; Hydrogen Sulfide; Ligation; Lung; | 2011 |
Small volume resuscitation with 7.5% hypertonic saline, hydroxyethyl starch 130/0.4 solution and hypertonic sodium chloride hydroxyethyl starch 40 injection reduced lung injury in endotoxin shock rats: comparison with saline.
Topics: Acute Lung Injury; Animals; Blood Pressure; Bronchoalveolar Lavage Fluid; Carotid Arteries; Endotoxi | 2012 |
[Effects of hydrogen sulfide on pulmonary surfactant in rats with acute lung injury induced by lipopolysccharide].
Topics: Acute Lung Injury; Animals; Hydrogen Sulfide; Lipopolysaccharides; Male; Pulmonary Surfactants; Rats | 2011 |
[Effects of hydrogen sulfide on the expressions of nuclear factor-κB and intercellular adhesion molecule 1 in pulmonary tissue of rats with acute lung injury].
Topics: Acute Lung Injury; Animals; Hydrogen Sulfide; Intercellular Adhesion Molecule-1; Lung; Male; NF-kapp | 2011 |
Effect of endogenous hydrogen sulfide on oxidative stress in oleic acid-induced acute lung injury in rats.
Topics: Acute Lung Injury; Animals; Glutathione; Hydrogen Sulfide; Lung; Male; Malondialdehyde; Oleic Acid; | 2011 |
[Effect of ulinastatin on oxidative stress and nuclear factor E2-related factor 2 expression in the lung tissues of acute hydrogen sulfide intoxicated rats].
Topics: Acute Lung Injury; Animals; Glycoproteins; Hydrogen Sulfide; Lung; Male; NF-E2-Related Factor 2; Oxi | 2012 |
[Hydrogen sulfide reduces lipopolysaccharide-induced acute lung injury and inhibits expression of phosphorylated p38 MAPK in rats].
Topics: Acute Lung Injury; Animals; Hydrogen Sulfide; Intercellular Adhesion Molecule-1; Lipopolysaccharides | 2012 |
Treatment with exogenous hydrogen sulfide attenuates hyperoxia-induced acute lung injury in mice.
Topics: Acute Lung Injury; Animals; Hydrogen Sulfide; Hyperoxia; Interleukins; Malondialdehyde; Mice; Mice, | 2013 |