hydrogen sulfide has been researched along with Infarction, Middle Cerebral Artery in 12 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Infarction, Middle Cerebral Artery: NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Infarction, neurological scores, brain edema, Evans blue (EB) extravasation, and tight junction protein expression were examined at 48 h after MCAO." | 7.80 | Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia. ( Alkayed, NJ; Ao, G; Cheng, J; Du, H; Hu, L; Jia, J; Liu, CF; Liu, H; Wang, Y; Xiao, Y, 2014) |
| "Cysteine is known to cause neuronal cell death and has been reported to be elevated in brain ischemia, but it has not been studied in clinical stroke." | 7.73 | High plasma cyst(e)ine level may indicate poor clinical outcome in patients with acute stroke: possible involvement of hydrogen sulfide. ( Chang, HM; Chen, CP; Chimon, GN; Halliwell, B; Ng, YK; Qu, K; Rumpel, H; Seah, AB; Wong, MC; Wong, PT, 2006) |
| "Rats underwent 2-h middle cerebral artery occlusion (MCAO) and received 40 ppm or 80 ppm H2S inhalation for 3h at the beginning of reperfusion." | 5.42 | Hydrogen sulfide induces neuroprotection against experimental stroke in rats by down-regulation of AQP4 via activating PKC. ( Cheng, L; Chi, M; Deng, L; Pan, H; Wang, G; Wei, X; Yao, X; Zhang, B, 2015) |
| "Focal ischemia was induced by 60-min middle cerebral artery occlusion (MCAO), followed by 23-h reperfusion." | 5.40 | Hydrogen sulfide protects the brain against ischemic reperfusion injury in a transient model of focal cerebral ischemia. ( Aboutaleb, N; Asadi, Y; Gheibi, A; Gheibi, S; Kalalian-Moghaddam, H; Khaksari, M; Mehrjerdi, FZ; Vakili, A, 2014) |
| " Infarction, neurological scores, brain edema, Evans blue (EB) extravasation, and tight junction protein expression were examined at 48 h after MCAO." | 3.80 | Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia. ( Alkayed, NJ; Ao, G; Cheng, J; Du, H; Hu, L; Jia, J; Liu, CF; Liu, H; Wang, Y; Xiao, Y, 2014) |
| "We observed recently that elevated plasma cysteine levels are associated with poor clinical outcome in acute stroke patients." | 3.73 | Hydrogen sulfide is a mediator of cerebral ischemic damage. ( Chen, CP; Halliwell, B; Moore, PK; Qu, K; Wong, PT, 2006) |
| "Cysteine is known to cause neuronal cell death and has been reported to be elevated in brain ischemia, but it has not been studied in clinical stroke." | 3.73 | High plasma cyst(e)ine level may indicate poor clinical outcome in patients with acute stroke: possible involvement of hydrogen sulfide. ( Chang, HM; Chen, CP; Chimon, GN; Halliwell, B; Ng, YK; Qu, K; Rumpel, H; Seah, AB; Wong, MC; Wong, PT, 2006) |
| "Rats underwent 2-h middle cerebral artery occlusion (MCAO) and received 40 ppm or 80 ppm H2S inhalation for 3h at the beginning of reperfusion." | 1.42 | Hydrogen sulfide induces neuroprotection against experimental stroke in rats by down-regulation of AQP4 via activating PKC. ( Cheng, L; Chi, M; Deng, L; Pan, H; Wang, G; Wei, X; Yao, X; Zhang, B, 2015) |
| "Focal ischemia was induced by 60-min middle cerebral artery occlusion (MCAO), followed by 23-h reperfusion." | 1.40 | Hydrogen sulfide protects the brain against ischemic reperfusion injury in a transient model of focal cerebral ischemia. ( Aboutaleb, N; Asadi, Y; Gheibi, A; Gheibi, S; Kalalian-Moghaddam, H; Khaksari, M; Mehrjerdi, FZ; Vakili, A, 2014) |
| "Focal cerebral ischemia was produced by reversible occlusion of the right middle cerebral artery in 17-month-old male Sprague-Dawley rats." | 1.35 | Long-term hypothermia reduces infarct volume in aged rats after focal ischemia. ( Balseanu, AT; Buga, AM; Florian, B; Grisk, O; Kessler, C; Popa-Wagner, A; Vintilescu, R; Walker, LC, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (25.00) | 29.6817 |
| 2010's | 6 (50.00) | 24.3611 |
| 2020's | 3 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhang, Q | 2 |
| Shen, Z | 2 |
| Shen, Y | 2 |
| Ma, M | 1 |
| Jue, H | 1 |
| Zhu, Y | 2 |
| Guo, W | 2 |
| Wang, F | 1 |
| Zhou, H | 1 |
| Zhang, X | 1 |
| Ni, SJ | 1 |
| Yao, ZY | 1 |
| Wei, X | 2 |
| Heng, X | 1 |
| Qu, SY | 1 |
| Zhao, X | 1 |
| Qi, YY | 1 |
| Ge, PY | 1 |
| Xu, CP | 1 |
| Yang, NY | 1 |
| Cao, Y | 1 |
| Zhu, HX | 1 |
| Guo, R | 1 |
| Zhang, QC | 1 |
| Guo, L | 1 |
| Wang, Z | 1 |
| Miao, L | 1 |
| Wang, M | 1 |
| Wu, J | 1 |
| Gheibi, S | 1 |
| Aboutaleb, N | 1 |
| Khaksari, M | 1 |
| Kalalian-Moghaddam, H | 1 |
| Vakili, A | 1 |
| Asadi, Y | 1 |
| Mehrjerdi, FZ | 1 |
| Gheibi, A | 1 |
| Wang, Y | 1 |
| Jia, J | 1 |
| Ao, G | 1 |
| Hu, L | 1 |
| Liu, H | 1 |
| Xiao, Y | 1 |
| Du, H | 1 |
| Alkayed, NJ | 1 |
| Liu, CF | 1 |
| Cheng, J | 1 |
| Zhang, B | 1 |
| Cheng, L | 1 |
| Chi, M | 1 |
| Deng, L | 1 |
| Pan, H | 1 |
| Yao, X | 1 |
| Wang, G | 1 |
| Sandu, RE | 1 |
| Uzoni, A | 1 |
| Ciobanu, O | 1 |
| Moldovan, M | 1 |
| Anghel, A | 1 |
| Radu, E | 1 |
| Coogan, AN | 1 |
| Popa-Wagner, A | 2 |
| Li, GF | 1 |
| Luo, HK | 1 |
| Li, LF | 1 |
| Zhang, QZ | 1 |
| Xie, LJ | 1 |
| Jiang, H | 1 |
| Li, LP | 1 |
| Hao, N | 1 |
| Wang, WW | 1 |
| Zhang, JX | 1 |
| Qu, K | 2 |
| Chen, CP | 2 |
| Halliwell, B | 2 |
| Moore, PK | 1 |
| Wong, PT | 2 |
| Chimon, GN | 1 |
| Seah, AB | 1 |
| Chang, HM | 1 |
| Wong, MC | 1 |
| Ng, YK | 1 |
| Rumpel, H | 1 |
| Florian, B | 1 |
| Vintilescu, R | 1 |
| Balseanu, AT | 1 |
| Buga, AM | 1 |
| Grisk, O | 1 |
| Walker, LC | 1 |
| Kessler, C | 1 |
12 other studies available for hydrogen sulfide and Infarction, Middle Cerebral Artery
| Article | Year |
|---|---|
The regulatory role of MiR-203 in oxidative stress induced cell injury through the CBS/H
Topics: Animals; Antagomirs; Apoptosis; Cell Line, Tumor; Cystathionine beta-Synthase; Humans; Hydrogen Sulf | 2022 |
SAM, a cystathionine beta-synthase activator, promotes hydrogen sulfide to promote neural repair resulting from massive cerebral infarction induced by middle cerebral artery occlusion.
Topics: Animals; Apoptosis; Brain Ischemia; Cystathionine beta-Synthase; Glucose; Glutamic Acid; Humans; Hyd | 2022 |
Vagus nerve stimulated by microbiota-derived hydrogen sulfide mediates the regulation of berberine on microglia in transient middle cerebral artery occlusion rats.
Topics: Animals; Berberine; Brain Ischemia; Hydrogen Sulfide; Infarction, Middle Cerebral Artery; Microbiota | 2022 |
MiR-125b-5p is involved in oxygen and glucose deprivation injury in PC-12 cells via CBS/H
Topics: Animals; Apoptosis; Cell Hypoxia; Cystathionine beta-Synthase; Gene Knockdown Techniques; Hydrogen S | 2018 |
Hydrogen sulfide protects the brain against ischemic reperfusion injury in a transient model of focal cerebral ischemia.
Topics: Animals; Apoptosis; Brain; Edema; Hydrogen Sulfide; Infarction, Middle Cerebral Artery; Male; Neurop | 2014 |
Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia.
Topics: Animals; Behavior, Animal; Blood-Brain Barrier; Blotting, Western; Brain Edema; Brain Ischemia; Colo | 2014 |
Hydrogen sulfide induces neuroprotection against experimental stroke in rats by down-regulation of AQP4 via activating PKC.
Topics: Administration, Inhalation; Animals; Aquaporin 4; Brain; Brain Edema; Disease Models, Animal; Down-R | 2015 |
Post-stroke gaseous hypothermia increases vascular density but not neurogenesis in the ischemic penumbra of aged rats.
Topics: Aging; Animals; Blood Pressure; Body Temperature; Disease Models, Animal; Doublecortin Domain Protei | 2016 |
Dual effects of hydrogen sulphide on focal cerebral ischaemic injury via modulation of oxidative stress-induced apoptosis.
Topics: Animals; Apoptosis; Apoptosis Regulatory Proteins; Brain Infarction; Brain Ischemia; Cerebral Cortex | 2012 |
Hydrogen sulfide is a mediator of cerebral ischemic damage.
Topics: Air Pollutants; Animals; Brain Injuries; Brain Ischemia; Cerebral Cortex; Cysteine; Dizocilpine Male | 2006 |
High plasma cyst(e)ine level may indicate poor clinical outcome in patients with acute stroke: possible involvement of hydrogen sulfide.
Topics: Adult; Aged; Aged, 80 and over; Amino Acids; Animals; Brain; Cysteine; Cystine; Disease Models, Anim | 2006 |
Long-term hypothermia reduces infarct volume in aged rats after focal ischemia.
Topics: Aging; Animals; Apoptosis; Apoptosis Regulatory Proteins; Body Temperature; Brain; Brain Infarction; | 2008 |