hydrogen sulfide has been researched along with Heart Diseases in 16 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Heart Diseases: Pathological conditions involving the HEART including its structural and functional abnormalities.
Excerpt | Relevance | Reference |
---|---|---|
"Cirrhosis was induced by surgical ligation of bile duct in rats." | 5.38 | The role of endogenous hydrogen sulfide in pathogenesis of chronotropic dysfunction in rats with cirrhosis. ( Babaei-Karamshahlou, M; Hajizadeh, S; Hooshmand, B; Mani, AR, 2012) |
"Fibrosis is defined as the pathological progress of excessive extracellular matrix (ECM), such as collagen, fibronectin, and elastin deposition, as the regenerative capacity of cells cannot satisfy the dynamic repair of chronic damage." | 2.72 | Gasotransmitters: Potential Therapeutic Molecules of Fibrotic Diseases. ( Cao, Y; Chen, Y; Jiang, F; Kong, G; Li, Y; Liu, L; Tang, M; Wang, Q; Yuan, S; Zhang, Q, 2021) |
"Hydrogen sulfide (H2S) has been long recognized as a highly poisonous gas that is rapidly lethal in intoxicating dosage." | 2.52 | Hydrogen sulfide and cardioprotection--Mechanistic insights and clinical translatability. ( Salloum, FN, 2015) |
"Hydrogen sulfide (H2S) has antifibrotic activity in the kidneys, heart, lungs, and other organs." | 1.51 | Exogenous H2S mitigates myocardial fibrosis in diabetic rats through suppression of the canonical Wnt pathway. ( Chen, Y; Jia, Q; Ma, SF; Mehmood, S; Wang, Y; Yang, R, 2019) |
"Cirrhosis was induced by surgical ligation of bile duct in rats." | 1.38 | The role of endogenous hydrogen sulfide in pathogenesis of chronotropic dysfunction in rats with cirrhosis. ( Babaei-Karamshahlou, M; Hajizadeh, S; Hooshmand, B; Mani, AR, 2012) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (25.00) | 29.6817 |
2010's | 9 (56.25) | 24.3611 |
2020's | 3 (18.75) | 2.80 |
Authors | Studies |
---|---|
Chen, Y | 2 |
Yuan, S | 1 |
Cao, Y | 1 |
Kong, G | 1 |
Jiang, F | 1 |
Li, Y | 1 |
Wang, Q | 2 |
Tang, M | 1 |
Zhang, Q | 1 |
Liu, L | 1 |
Bai, L | 1 |
Dai, J | 1 |
Xia, Y | 1 |
He, K | 1 |
Xue, H | 1 |
Guo, Q | 1 |
Tian, D | 1 |
Xiao, L | 1 |
Zhang, X | 1 |
Teng, X | 1 |
Wu, Y | 1 |
Jin, S | 1 |
Merz, T | 1 |
Denoix, N | 1 |
Wigger, D | 1 |
Waller, C | 1 |
Wepler, M | 1 |
Vettorazzi, S | 1 |
Tuckermann, J | 1 |
Radermacher, P | 1 |
McCook, O | 1 |
Kuang, Q | 1 |
Xue, N | 1 |
Chen, J | 1 |
Shen, Z | 2 |
Cui, X | 1 |
Fang, Y | 1 |
Ding, X | 1 |
Yang, R | 1 |
Jia, Q | 1 |
Ma, SF | 1 |
Wang, Y | 1 |
Mehmood, S | 1 |
Salloum, FN | 1 |
Shen, Y | 1 |
Luo, S | 1 |
Guo, W | 1 |
Zhu, YZ | 1 |
Liu, MH | 1 |
Lin, XL | 1 |
Zhang, Y | 1 |
He, J | 1 |
Tan, TP | 1 |
Wu, SJ | 1 |
Liu, J | 1 |
Tian, W | 1 |
Chen, L | 1 |
Yu, S | 1 |
Li, J | 1 |
Yuan, C | 1 |
Finnbjornsdottir, RG | 1 |
Carlsen, HK | 1 |
Thorsteinsson, T | 1 |
Oudin, A | 1 |
Lund, SH | 1 |
Gislason, T | 1 |
Rafnsson, V | 1 |
Yong, QC | 2 |
Lee, SW | 1 |
Foo, CS | 1 |
Neo, KL | 2 |
Chen, X | 1 |
Bian, JS | 2 |
Liao, F | 1 |
Zheng, Y | 1 |
Geng, B | 2 |
Louis, XL | 1 |
Murphy, R | 1 |
Thandapilly, SJ | 1 |
Yu, L | 1 |
Netticadan, T | 1 |
Babaei-Karamshahlou, M | 1 |
Hooshmand, B | 1 |
Hajizadeh, S | 1 |
Mani, AR | 1 |
Llorca, J | 1 |
Salas, A | 1 |
Prieto-Salceda, D | 1 |
Chinchon-Bengoechea, V | 1 |
Delgado-RodrÃguez, M | 1 |
Hu, LF | 1 |
Pan, TT | 1 |
Chang, L | 1 |
Yu, F | 1 |
Zhao, J | 1 |
Jiang, H | 1 |
Du, J | 1 |
Tang, C | 1 |
Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
---|---|---|---|---|---|---|---|
Plasma Measurement of Hydrogen Sulfide, Nitric Oxide and Stress Hyperglycemia in Acute Myocardial Infarction[NCT03829605] | 50 participants (Anticipated) | Observational | 2019-02-15 | Not yet recruiting | |||
[information is prepared from clinicaltrials.gov, extracted Sep-2024] |
4 reviews available for hydrogen sulfide and Heart Diseases
Article | Year |
---|---|
Gasotransmitters: Potential Therapeutic Molecules of Fibrotic Diseases.
Topics: Antioxidants; Fibrosis; Gasotransmitters; Heart Diseases; Humans; Hydrogen Sulfide; Liver Cirrhosis; | 2021 |
Hydrogen sulfide and cardioprotection--Mechanistic insights and clinical translatability.
Topics: Animals; Cardiotonic Agents; Heart; Heart Diseases; Humans; Hydrogen Sulfide; Oxidative Stress; Sign | 2015 |
The Cardioprotective Effects of Hydrogen Sulfide in Heart Diseases: From Molecular Mechanisms to Therapeutic Potential.
Topics: Animals; Heart Diseases; Heart Failure; Hydrogen Sulfide; Mitochondria; Nitric Oxide; Protective Age | 2015 |
[Hydrogen sulfide--a novel cardiac disease protector].
Topics: Animals; Calcium Channel Blockers; Calcium Channels, L-Type; Heart Diseases; Humans; Hydrogen Sulfid | 2012 |
12 other studies available for hydrogen sulfide and Heart Diseases
Article | Year |
---|---|
Hydrogen Sulfide Ameliorated High Choline-Induced Cardiac Dysfunction by Inhibiting cGAS-STING-NLRP3 Inflammasome Pathway.
Topics: Animals; Caspase 1; Choline; Cystathionine gamma-Lyase; Heart Diseases; Hydrogen Sulfide; Inflammaso | 2022 |
The Role of Glucocorticoid Receptor and Oxytocin Receptor in the Septic Heart in a Clinically Relevant, Resuscitated Porcine Model With Underlying Atherosclerosis.
Topics: Animals; Atherosclerosis; Cystathionine gamma-Lyase; Disease Models, Animal; Gene Expression Regulat | 2020 |
Low Plasma Hydrogen Sulfide Is Associated with Impaired Renal Function and Cardiac Dysfunction.
Topics: Adult; Aged; Biomarkers; Disease Progression; Echocardiography; Female; Glomerular Filtration Rate; | 2018 |
Exogenous H2S mitigates myocardial fibrosis in diabetic rats through suppression of the canonical Wnt pathway.
Topics: Animals; Collagen; Diabetes Mellitus, Experimental; Fibrosis; Heart; Heart Diseases; Hydrogen Sulfid | 2019 |
Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting reactive oxygen species-activated extracellular signal-regulated kinase 1/2 in H9c2 cardiac myocytes.
Topics: Acetylcysteine; Animals; Antibiotics, Antineoplastic; Cardiotonic Agents; Cell Line; Cystathionine g | 2015 |
Association between Daily Hydrogen Sulfide Exposure and Incidence of Emergency Hospital Visits: A Population-Based Study.
Topics: Adult; Aged; Aged, 80 and over; Emergency Service, Hospital; Environmental Exposure; Female; Heart D | 2016 |
Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.
Topics: Alkynes; Animals; Antibodies, Blocking; Blood Pressure; Chromones; Dimethyl Sulfoxide; Electrocardio | 2008 |
Garlic extracts prevent oxidative stress, hypertrophy and apoptosis in cardiomyocytes: a role for nitric oxide and hydrogen sulfide.
Topics: Animals; Antioxidants; Apoptosis; Cysteine; Disulfides; Garlic; Heart; Heart Diseases; Hydrogen Sulf | 2012 |
The role of endogenous hydrogen sulfide in pathogenesis of chronotropic dysfunction in rats with cirrhosis.
Topics: Alkynes; Aminooxyacetic Acid; Animals; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; Fibro | 2012 |
Nitrogen dioxide increases cardiorespiratory admissions in Torrelavega (Spain).
Topics: Air Pollutants; Heart Diseases; Hospitalization; Humans; Hydrogen Sulfide; Nitrogen Dioxide; Nitroge | 2005 |
Cyclooxygenase-2 mediates the delayed cardioprotection induced by hydrogen sulfide preconditioning in isolated rat cardiomyocytes.
Topics: Air Pollutants; Animals; Blotting, Western; Cell Separation; Cell Survival; Cyclooxygenase 2; Cycloo | 2008 |
Hydrogen sulfide inhibits myocardial injury induced by homocysteine in rats.
Topics: Animals; Cystathionine gamma-Lyase; Disease Models, Animal; Electron Transport Complex IV; Endoplasm | 2008 |