hydrogen sulfide has been researched along with Cardiovascular Stroke in 58 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The aim of the present study was to explore whether hydrogen sulfide (H2S) protects against ischemic heart failure (HF) by inhibiting the necroptosis pathway." | 8.12 | Hydrogen sulfide protects against ischemic heart failure by inhibiting RIP1/RIP3/MLKL-mediated necroptosis. ( Chang, L; Dai, J; Gong, J; Lu, H; Luo, Y; Ma, F; Zhu, Y, 2022) |
| "Exogenous hydrogen sulfide (H2S) leads to down-regulation of inflammatory responses and provides myocardial protection during acute ischemia/reperfusion injury; however its role during chronic heart failure (CHF) due to myocardial infarction (MI) is yet to be unveiled." | 7.78 | Hydrogen sulfide mitigates cardiac remodeling during myocardial infarction via improvement of angiogenesis. ( Lominadze, D; Metreveli, N; Mishra, PK; Qipshidze, N; Tyagi, SC, 2012) |
| "The role of hydrogen sulfide (H(2)S) in myocardial infarction (MI) has not been previously studied." | 7.74 | Hydrogen sulfide and its possible roles in myocardial ischemia in experimental rats. ( Ho, P; Huang, SH; Loke, YY; Lu, J; Moore, PK; Tan, CS; Wang, ZJ; Whiteman, M; Zhu, YC; Zhu, YZ, 2007) |
| "We investigated whether (endogenous) hydrogen sulfide (H2S) protects the heart against myocardial ischemia and reperfusion injury." | 7.73 | The production of hydrogen sulfide limits myocardial ischemia and reperfusion injury and contributes to the cardioprotective effects of preconditioning with endotoxin, but not ischemia in the rat. ( McDonald, MC; Sivarajah, A; Thiemermann, C, 2006) |
| "Hydrogen sulfide (H2S) has been demonstrated to possess cardioprotective effects post MI and during the progress of cardiac remodeling." | 5.43 | Hydrogen Sulfide Recruits Macrophage Migration by Integrin β1-Src-FAK/Pyk2-Rac Pathway in Myocardial Infarction. ( Miao, L; Shen, X; Xin, H; Xin, X; Zhu, YZ, 2016) |
| "Hydrogen sulfide (H2S) has been shown to exert cardioprotective effects in MI." | 5.43 | Hydrogen Sulfide Mitigates Myocardial Infarction via Promotion of Mitochondrial Biogenesis-Dependent M2 Polarization of Macrophages. ( Miao, L; Moore, PK; Shen, X; Shen, Y; Whiteman, M; Xin, H; Xin, X; Zhu, YZ, 2016) |
| " In the present study, we investigated the cardioprotective effects and pharmacokinetic properties of a controlled release formulation of SPRC (CR-SPRC) in an in vivo rat model of myocardial infarction (MI)." | 5.42 | Cardioprotective effects and pharmacokinetic properties of a controlled release formulation of a novel hydrogen sulfide donor in rats with acute myocardial infarction. ( Huang, C; Lin, S; Liu, H; Liu, X; Tran, BH; Wang, S; Zhang, Q; Zhu, YZ, 2015) |
| "The aim of the present study was to explore whether hydrogen sulfide (H2S) protects against ischemic heart failure (HF) by inhibiting the necroptosis pathway." | 4.12 | Hydrogen sulfide protects against ischemic heart failure by inhibiting RIP1/RIP3/MLKL-mediated necroptosis. ( Chang, L; Dai, J; Gong, J; Lu, H; Luo, Y; Ma, F; Zhu, Y, 2022) |
| "The present study aimed to elucidate the mechanisms by which hydrogen sulfide (H2S) attenuates left ventricular remodeling after myocardial infarction (MI)." | 3.85 | Hydrogen Sulfide Reduces Recruitment of CD11b ( Li, H; Wang, JN; Wu, B; Wu, SW; Wu, T; Zhang, L; Zhang, YE, 2017) |
| "Exogenous hydrogen sulfide (H2S) leads to down-regulation of inflammatory responses and provides myocardial protection during acute ischemia/reperfusion injury; however its role during chronic heart failure (CHF) due to myocardial infarction (MI) is yet to be unveiled." | 3.78 | Hydrogen sulfide mitigates cardiac remodeling during myocardial infarction via improvement of angiogenesis. ( Lominadze, D; Metreveli, N; Mishra, PK; Qipshidze, N; Tyagi, SC, 2012) |
| "In this study, we determined the cardioprotective effects of S-propargyl-cysteine (SPRC), a structural analog of S-allylcysteine (SAC), using in vivo models of acute myocardial infarction (MI) and in vitro hypoxic cardiomyocytes models." | 3.75 | S-propargyl-cysteine protects both adult rat hearts and neonatal cardiomyocytes from ischemia/hypoxia injury: the contribution of the hydrogen sulfide-mediated pathway. ( Liu, HR; Mu, Q; Rose, P; Wang, Q; Zhu, YZ, 2009) |
| "PKG activation with tadalafil limits myocardial infarction and preserves LV function through H(2)S signaling." | 3.75 | Phosphodiesterase-5 inhibitor, tadalafil, protects against myocardial ischemia/reperfusion through protein-kinase g-dependent generation of hydrogen sulfide. ( Abbate, A; Chau, VQ; Hoke, NN; Kukreja, RC; Ockaili, RA; Salloum, FN; Toldo, S; Varma, A, 2009) |
| "The role of hydrogen sulfide (H(2)S) in myocardial infarction (MI) has not been previously studied." | 3.74 | Hydrogen sulfide and its possible roles in myocardial ischemia in experimental rats. ( Ho, P; Huang, SH; Loke, YY; Lu, J; Moore, PK; Tan, CS; Wang, ZJ; Whiteman, M; Zhu, YC; Zhu, YZ, 2007) |
| "To investigate the changes of plasma hydrogen sulfide (H(2)S) in patients with coronary heart disease (CHD)." | 3.73 | [Changes of the new gaseous transmitter H2S in patients with coronary heart disease]. ( Geng, B; Jiang, HL; Li, ZL; Tang, CS; Wu, HC, 2005) |
| "We investigated whether (endogenous) hydrogen sulfide (H2S) protects the heart against myocardial ischemia and reperfusion injury." | 3.73 | The production of hydrogen sulfide limits myocardial ischemia and reperfusion injury and contributes to the cardioprotective effects of preconditioning with endotoxin, but not ischemia in the rat. ( McDonald, MC; Sivarajah, A; Thiemermann, C, 2006) |
| "Hydrogen sulfide (H2S) was identified as the third gasotransmitter in 1996 following the discoveries of the biological importance of nitric oxide and carbon monoxide." | 2.55 | Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure. ( Donnarumma, E; Lefer, DJ; Trivedi, RK, 2017) |
| "Hydrogen sulfide (H2S) is an important signaling molecule with promising protective effects in many physiological and pathological processes." | 1.51 | Controllable thioester-based hydrogen sulfide slow-releasing donors as cardioprotective agents. ( Liu, J; Liu, Y; Luo, S; Xie, S; Xu, J; Xu, S; Yao, H; Zhu, Z, 2019) |
| "Hydrogen sulfide (H2S) has been demonstrated to possess cardioprotective effects post MI and during the progress of cardiac remodeling." | 1.43 | Hydrogen Sulfide Recruits Macrophage Migration by Integrin β1-Src-FAK/Pyk2-Rac Pathway in Myocardial Infarction. ( Miao, L; Shen, X; Xin, H; Xin, X; Zhu, YZ, 2016) |
| "Hydrogen sulfide (H2S) has been shown to exert cardioprotective effects in MI." | 1.43 | Hydrogen Sulfide Mitigates Myocardial Infarction via Promotion of Mitochondrial Biogenesis-Dependent M2 Polarization of Macrophages. ( Miao, L; Moore, PK; Shen, X; Shen, Y; Whiteman, M; Xin, H; Xin, X; Zhu, YZ, 2016) |
| "Pretreatment with zofenopril significantly reduced myocardial infarct size and cardiac troponin I levels after I/R injury in both mice and swine." | 1.43 | Zofenopril Protects Against Myocardial Ischemia-Reperfusion Injury by Increasing Nitric Oxide and Hydrogen Sulfide Bioavailability. ( Ali, MJ; Bradley, JM; Cirino, G; Donnarumma, E; Evangelista, S; Goodchild, TT; Islam, KN; Jenkins, JS; Lefer, DJ; Organ, CL; Patel, RA; Polhemus, DJ; Rushing, AM; Scarborough, AL, 2016) |
| "Hydrogen sulfide (H2S), which is a member of the gasotransmitter family, plays an important physiological and pathological role in cardiovascular system." | 1.43 | Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts. ( Bai, S; Chen, J; Gao, J; Li, H; Li, L; Li, X; Sun, W; Wang, R; Wang, Y; Wu, L; Xu, C, 2016) |
| " In the present study, we investigated the cardioprotective effects and pharmacokinetic properties of a controlled release formulation of SPRC (CR-SPRC) in an in vivo rat model of myocardial infarction (MI)." | 1.42 | Cardioprotective effects and pharmacokinetic properties of a controlled release formulation of a novel hydrogen sulfide donor in rats with acute myocardial infarction. ( Huang, C; Lin, S; Liu, H; Liu, X; Tran, BH; Wang, S; Zhang, Q; Zhu, YZ, 2015) |
| "Hydrogen sulfide (H2S) is an important endogenous modulator of diverse physiological and pathophysiological processes." | 1.42 | GYY4137 attenuates remodeling, preserves cardiac function and modulates the natriuretic peptide response to ischemia. ( Liew, OW; Lilyanna, S; Martinez, EC; Moore, PK; Peh, MT; Richards, AM; Wang, P, 2015) |
| "Hydrogen sulfide (H(2)S) displays anti-inflammatory and cytoprotective activities to attenuate myocardial ischemia-reperfusion (MIR)-induced injury, but its role in MIR in diabetics is not known." | 1.37 | The protective role of hydrogen sulfide in myocardial ischemia-reperfusion-induced injury in diabetic rats. ( Gao, Y; Kang, K; Li, W; Sun, L; Sun, X; Yao, X; Zhang, Y, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 5 (8.62) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 17 (29.31) | 29.6817 |
| 2010's | 26 (44.83) | 24.3611 |
| 2020's | 10 (17.24) | 2.80 |
| Authors | Studies |
|---|---|
| El-Sayed, SS | 1 |
| Shahin, RM | 1 |
| Fahmy, A | 1 |
| Elshazly, SM | 1 |
| Liu, Q | 1 |
| Ji, G | 1 |
| Chu, Y | 1 |
| Hao, T | 1 |
| Qian, M | 1 |
| Zhao, Q | 1 |
| Kolluru, GK | 1 |
| Shackelford, RE | 1 |
| Shen, X | 3 |
| Dominic, P | 1 |
| Kevil, CG | 2 |
| Ma, F | 2 |
| Zhu, Y | 1 |
| Chang, L | 2 |
| Gong, J | 1 |
| Luo, Y | 2 |
| Dai, J | 1 |
| Lu, H | 2 |
| Bechelli, C | 1 |
| Macabrey, D | 1 |
| Deglise, S | 1 |
| Allagnat, F | 1 |
| Kang, SC | 1 |
| Sohn, EH | 1 |
| Lee, SR | 1 |
| Ravindran, S | 1 |
| Gopalakrishnan, S | 1 |
| Kurian, GA | 1 |
| Li, Y | 1 |
| Liu, M | 1 |
| Yi, J | 1 |
| Song, X | 1 |
| Zheng, X | 1 |
| Liu, D | 1 |
| Wang, S | 2 |
| Chu, C | 1 |
| Yang, J | 1 |
| Zhang, J | 1 |
| Cai, X | 1 |
| Zhang, Q | 2 |
| Li, X | 2 |
| Li, S | 1 |
| Ma, J | 1 |
| Zhu, W | 2 |
| Liu, X | 2 |
| Wei, M | 1 |
| Tu, W | 1 |
| Shen, Y | 2 |
| Liu, J | 2 |
| Lai, X | 1 |
| Yu, P | 1 |
| Wang, L | 2 |
| Tang, Y | 1 |
| He, H | 1 |
| Wei, W | 1 |
| Donnarumma, E | 2 |
| Trivedi, RK | 1 |
| Lefer, DJ | 3 |
| Liang, W | 1 |
| Chen, J | 2 |
| Li, L | 2 |
| Li, M | 1 |
| Wei, X | 1 |
| Tan, B | 1 |
| Shang, Y | 1 |
| Fan, G | 1 |
| Wang, W | 1 |
| Liu, W | 1 |
| Yao, H | 1 |
| Luo, S | 1 |
| Xie, S | 1 |
| Liu, Y | 3 |
| Xu, J | 1 |
| Zhu, Z | 1 |
| Xu, S | 1 |
| Wang, Z | 1 |
| Tran, B | 1 |
| Zhong, R | 1 |
| Xiong, Y | 1 |
| Dai, T | 1 |
| Wu, J | 1 |
| Xin, X | 3 |
| Guo, W | 2 |
| Xie, Y | 1 |
| Mao, Y | 1 |
| Zhu, YZ | 9 |
| Zhang, Y | 2 |
| Li, H | 3 |
| Zhao, G | 1 |
| Sun, A | 2 |
| Zong, NC | 1 |
| Li, Z | 1 |
| Zhu, H | 1 |
| Zou, Y | 2 |
| Yang, X | 1 |
| Ge, J | 2 |
| Salloum, FN | 3 |
| Sturz, GR | 1 |
| Yin, C | 1 |
| Rehman, S | 1 |
| Hoke, NN | 3 |
| Kukreja, RC | 3 |
| Xi, L | 1 |
| Ma, SF | 1 |
| Ding, YJ | 1 |
| Chen, Y | 1 |
| Pu, SX | 1 |
| Wu, HJ | 1 |
| Wang, ZF | 1 |
| Tao, BB | 1 |
| Wang, WW | 1 |
| Zhu, YC | 3 |
| Bibli, SI | 1 |
| Andreadou, I | 1 |
| Chatzianastasiou, A | 1 |
| Tzimas, C | 1 |
| Sanoudou, D | 1 |
| Kranias, E | 1 |
| Brouckaert, P | 1 |
| Coletta, C | 1 |
| Szabo, C | 3 |
| Kremastinos, DT | 1 |
| Iliodromitis, EK | 1 |
| Papapetropoulos, A | 1 |
| Tran, BH | 1 |
| Huang, C | 1 |
| Lin, S | 1 |
| Liu, H | 1 |
| Lilyanna, S | 1 |
| Peh, MT | 1 |
| Liew, OW | 1 |
| Wang, P | 1 |
| Moore, PK | 5 |
| Richards, AM | 1 |
| Martinez, EC | 1 |
| Li, C | 1 |
| Hu, M | 1 |
| Wang, Y | 2 |
| Deng, J | 1 |
| Yan, X | 1 |
| Miao, L | 2 |
| Xin, H | 2 |
| Li, N | 1 |
| Wang, MJ | 1 |
| Jin, S | 1 |
| Bai, YD | 1 |
| Hou, CL | 1 |
| Ma, FF | 1 |
| Li, XH | 1 |
| Whiteman, M | 3 |
| Karwi, QG | 1 |
| Wood, ME | 1 |
| Torregrossa, R | 1 |
| Baxter, GF | 1 |
| Ali, MJ | 1 |
| Rushing, AM | 1 |
| Scarborough, AL | 1 |
| Bradley, JM | 1 |
| Organ, CL | 1 |
| Islam, KN | 1 |
| Polhemus, DJ | 1 |
| Evangelista, S | 1 |
| Cirino, G | 1 |
| Jenkins, JS | 1 |
| Patel, RA | 1 |
| Goodchild, TT | 1 |
| Gao, J | 1 |
| Sun, W | 1 |
| Bai, S | 1 |
| Wang, R | 2 |
| Wu, L | 1 |
| Xu, C | 1 |
| Ueda, K | 1 |
| Fukuma, N | 1 |
| Takimoto, E | 1 |
| Wu, T | 1 |
| Wu, B | 1 |
| Zhang, L | 1 |
| Wu, SW | 1 |
| Wang, JN | 1 |
| Zhang, YE | 1 |
| Yong, QC | 1 |
| Lee, SW | 2 |
| Foo, CS | 1 |
| Neo, KL | 2 |
| Chen, X | 2 |
| Bian, JS | 3 |
| Pan, TT | 2 |
| Chen, YQ | 1 |
| Wang, Q | 2 |
| Liu, HR | 1 |
| Mu, Q | 1 |
| Rose, P | 1 |
| Calvert, JW | 1 |
| Jha, S | 1 |
| Gundewar, S | 1 |
| Elrod, JW | 1 |
| Ramachandran, A | 1 |
| Pattillo, CB | 1 |
| Osipov, RM | 1 |
| Robich, MP | 1 |
| Feng, J | 2 |
| Clements, RT | 2 |
| Glazer, HP | 1 |
| Sodha, NR | 2 |
| Bianchi, C | 2 |
| Sellke, FW | 2 |
| Zhuo, Y | 1 |
| Chen, PF | 1 |
| Zhang, AZ | 1 |
| Zhong, H | 1 |
| Chen, CQ | 1 |
| Horvath, EM | 1 |
| Stahl, GL | 1 |
| Chau, VQ | 2 |
| Abbate, A | 1 |
| Varma, A | 1 |
| Ockaili, RA | 2 |
| Toldo, S | 1 |
| Wang, X | 1 |
| Gao, Y | 1 |
| Yao, X | 1 |
| Li, W | 1 |
| Kang, K | 1 |
| Sun, L | 1 |
| Sun, X | 1 |
| Minamishima, S | 1 |
| Ichinose, F | 1 |
| Xie, X | 1 |
| Huang, Z | 1 |
| Hu, X | 1 |
| Jia, J | 1 |
| Das, A | 1 |
| Samidurai, A | 1 |
| Stasch, JP | 1 |
| Qipshidze, N | 1 |
| Metreveli, N | 1 |
| Mishra, PK | 1 |
| Lominadze, D | 1 |
| Tyagi, SC | 1 |
| KAIPAINEN, WJ | 1 |
| KOPTEVA, EG | 1 |
| SHIKHOVA, NM | 1 |
| KAPLUN, SIa | 1 |
| SHIKHOV, MM | 1 |
| Jiang, HL | 1 |
| Wu, HC | 1 |
| Li, ZL | 1 |
| Geng, B | 1 |
| Tang, CS | 1 |
| Sivarajah, A | 1 |
| McDonald, MC | 1 |
| Thiemermann, C | 1 |
| Wang, ZJ | 1 |
| Ho, P | 1 |
| Loke, YY | 1 |
| Huang, SH | 1 |
| Tan, CS | 1 |
| Lu, J | 1 |
| Hu, Y | 1 |
| Khin, ES | 1 |
| Chuah, SC | 1 |
| Das, DK | 1 |
| Zhu, XY | 1 |
| Yan, XH | 1 |
| Chen, SJ | 1 |
| Bliksøen, M | 1 |
| Kaljusto, ML | 1 |
| Vaage, J | 1 |
| Stensløkken, KO | 1 |
| Ji, Y | 1 |
| Pang, QF | 1 |
| Xu, G | 1 |
| Wang, JK | 1 |
| Zeng, YM | 1 |
| Tolonen, M | 1 |
| Hernberg, S | 1 |
| Nurminen, M | 1 |
| Tiitola, K | 1 |
| Vathenen, AS | 1 |
| Emberton, P | 1 |
| Wales, JM | 1 |
| Sorokina, EI | 1 |
| Nazarova, IN | 1 |
| Krasnikov, VE | 1 |
5 reviews available for hydrogen sulfide and Cardiovascular Stroke
| Article | Year |
|---|---|
Sulfide regulation of cardiovascular function in health and disease.
Topics: Heart; Heart Failure; Humans; Hydrogen Sulfide; Myocardial Infarction; Sulfides | 2023 |
Clinical Potential of Hydrogen Sulfide in Peripheral Arterial Disease.
Topics: Atherosclerosis; Humans; Hydrogen Sulfide; Myocardial Infarction; Peripheral Arterial Disease; Quali | 2023 |
Hydrogen Sulfide as a Potential Alternative for the Treatment of Myocardial Fibrosis.
Topics: Animals; Cardiomyopathies; Diabetes Mellitus; Fibrosis; Humans; Hydrogen Sulfide; Hypertension; Myoc | 2020 |
Protective Actions of H2S in Acute Myocardial Infarction and Heart Failure.
Topics: Acute Disease; Animals; Cardiotonic Agents; Heart Failure; Humans; Hydrogen Sulfide; Mitochondria; M | 2017 |
[Emerging role of hydrogen sulfide in biology].
Topics: Animals; Anti-Inflammatory Agents; Apoptosis; Brain Ischemia; Cardiopulmonary Resuscitation; Heart A | 2010 |
53 other studies available for hydrogen sulfide and Cardiovascular Stroke
| Article | Year |
|---|---|
Quercetin ameliorated remote myocardial injury induced by renal ischemia/reperfusion in rats: Role of Rho-kinase and hydrogen sulfide.
Topics: Animals; Antioxidants; Hydrogen Sulfide; Kidney; Male; Myocardial Infarction; Quercetin; Rats; Rats, | 2021 |
Enzyme-responsive hybrid prodrug of nitric oxide and hydrogen sulfide for heart failure therapy.
Topics: Cardiotonic Agents; Diuretics; Enzyme Inhibitors; Heart Failure; Humans; Hydrogen Sulfide; Myocardia | 2022 |
Hydrogen sulfide protects against ischemic heart failure by inhibiting RIP1/RIP3/MLKL-mediated necroptosis.
Topics: Animals; Heart Failure; Hydrogen Sulfide; Mice; Myocardial Infarction; Necroptosis; Protein Kinases | 2022 |
Beneficial effect of sodium thiosulfate extends beyond myocardial tissue in isoproterenol model of infarction: Implication for nootropic effects.
Topics: Animals; Behavior, Animal; Disease Models, Animal; Heart; Hydrogen Sulfide; Isoproterenol; Male; Mit | 2020 |
Exogenous hydrogen sulfide inhibits apoptosis by regulating endoplasmic reticulum stress-autophagy axis and improves myocardial reconstruction after acute myocardial infarction.
Topics: Animals; Apoptosis; Autophagy; Cell Line; Cystathionine gamma-Lyase; Disease Models, Animal; Endopla | 2020 |
Hydrogen sulfide restores sevoflurane postconditioning mediated cardioprotection in diabetic rats: Role of SIRT1/Nrf2 signaling-modulated mitochondrial dysfunction and oxidative stress.
Topics: Animals; Cardiotonic Agents; Diabetes Mellitus, Experimental; Heme Oxygenase (Decyclizing); Hydrogen | 2021 |
Liraglutide restores late cardioprotective effects of remote preconditioning in diabetic rats via activation of hydrogen sulfide and nuclear factor erythroid 2-related factor 2 signaling pathway.
Topics: Animals; Diabetes Mellitus, Experimental; Hydrogen Sulfide; Ischemic Preconditioning; Ischemic Preco | 2021 |
Conductive Hydrogen Sulfide-Releasing Hydrogel Encapsulating ADSCs for Myocardial Infarction Treatment.
Topics: Adipose Tissue; Allografts; Animals; Hydrogels; Hydrogen Sulfide; Male; Myocardial Infarction; Myoca | 2019 |
Controllable thioester-based hydrogen sulfide slow-releasing donors as cardioprotective agents.
Topics: Animals; Cardiotonic Agents; Cell Line; Disease Models, Animal; Esters; Hydrogen Peroxide; Hydrogen | 2019 |
ZYZ-803 Mitigates Endoplasmic Reticulum Stress-Related Necroptosis after Acute Myocardial Infarction through Downregulating the RIP3-CaMKII Signaling Pathway.
Topics: Acute Disease; Animals; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Cardiotonic Agents; Down | 2019 |
Hydrogen sulfide attenuates the recruitment of CD11b⁺Gr-1⁺ myeloid cells and regulates Bax/Bcl-2 signaling in myocardial ischemia injury.
Topics: Animals; Anti-Inflammatory Agents; Apoptosis; bcl-2-Associated X Protein; CD11b Antigen; Cell Moveme | 2014 |
Beetroot juice reduces infarct size and improves cardiac function following ischemia-reperfusion injury: Possible involvement of endogenous H2S.
Topics: Animals; Beta vulgaris; Blotting, Western; Hydrogen Sulfide; Male; Mice; Myocardial Infarction; Poly | 2015 |
Hydrogen Sulfide Targets the Cys320/Cys529 Motif in Kv4.2 to Inhibit the Ito Potassium Channels in Cardiomyocytes and Regularizes Fatal Arrhythmia in Myocardial Infarction.
Topics: Amino Acid Motifs; Animals; Arrhythmias, Cardiac; Cysteine; Disulfides; HEK293 Cells; Humans; Hydrog | 2015 |
Cardioprotection by H2S engages a cGMP-dependent protein kinase G/phospholamban pathway.
Topics: Animals; Calcium-Binding Proteins; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Disease Models, | 2015 |
Cardioprotective effects and pharmacokinetic properties of a controlled release formulation of a novel hydrogen sulfide donor in rats with acute myocardial infarction.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Cardiotonic Agents; Creatine Kinase; Cysteine; Del | 2015 |
GYY4137 attenuates remodeling, preserves cardiac function and modulates the natriuretic peptide response to ischemia.
Topics: Animals; Atrial Natriuretic Factor; Cardiotonic Agents; Humans; Hydrogen Sulfide; Ischemia; Morpholi | 2015 |
Hydrogen sulfide preconditioning protects against myocardial ischemia/reperfusion injury in rats through inhibition of endo/sarcoplasmic reticulum stress.
Topics: Animals; Apoptosis; Gene Expression Regulation; HSP70 Heat-Shock Proteins; Humans; Hydrogen Sulfide; | 2015 |
Hydrogen Sulfide Recruits Macrophage Migration by Integrin β1-Src-FAK/Pyk2-Rac Pathway in Myocardial Infarction.
Topics: Animals; Cell Hypoxia; Cell Movement; Cystathionine gamma-Lyase; Endocytosis; Focal Adhesion Kinase | 2016 |
The H2S Donor NaHS Changes the Expression Pattern of H2S-Producing Enzymes after Myocardial Infarction.
Topics: Animals; Cell Hypoxia; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; Cytoprotection; Hydro | 2016 |
Hydrogen Sulfide Mitigates Myocardial Infarction via Promotion of Mitochondrial Biogenesis-Dependent M2 Polarization of Macrophages.
Topics: Animals; Disease Models, Animal; Fatty Acids; Heart Function Tests; Hydrogen Sulfide; Lipolysis; Mac | 2016 |
Pharmacological postconditioning against myocardial infarction with a slow-releasing hydrogen sulfide donor, GYY4137.
Topics: Animals; Cytoprotection; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Glycoge | 2016 |
Zofenopril Protects Against Myocardial Ischemia-Reperfusion Injury by Increasing Nitric Oxide and Hydrogen Sulfide Bioavailability.
Topics: Animals; Antihypertensive Agents; Biological Availability; Blotting, Western; Captopril; Cystathioni | 2016 |
Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts.
Topics: Aging; Animals; Autophagy; Cardiotonic Agents; Cell Survival; Hydrogen Sulfide; Ischemic Postconditi | 2016 |
Hydrogen Sulfide, a Potential Cardioprotective Gas Activating a Life Span Regulator.
Topics: Animals; Biomarkers; Cardiotonic Agents; Disease Models, Animal; Evidence-Based Medicine; Humans; Hy | 2016 |
Hydrogen Sulfide Reduces Recruitment of CD11b
Topics: Animals; Apoptosis; CD11b Antigen; Flow Cytometry; Hydrogen Sulfide; Male; Mice; Mice, Inbred C57BL; | 2017 |
Endogenous hydrogen sulphide mediates the cardioprotection induced by ischemic postconditioning.
Topics: Alkynes; Animals; Antibodies, Blocking; Blood Pressure; Chromones; Dimethyl Sulfoxide; Electrocardio | 2008 |
All in the timing: a comparison between the cardioprotection induced by H2S preconditioning and post-infarction treatment.
Topics: Animals; Dose-Response Relationship, Drug; Heart; Hydrogen Sulfide; Ischemic Preconditioning, Myocar | 2009 |
S-propargyl-cysteine protects both adult rat hearts and neonatal cardiomyocytes from ischemia/hypoxia injury: the contribution of the hydrogen sulfide-mediated pathway.
Topics: Animals; Animals, Newborn; Cell Hypoxia; Cell Survival; Cystathionine gamma-Lyase; Cysteine; Disease | 2009 |
Hydrogen sulfide mediates cardioprotection through Nrf2 signaling.
Topics: Active Transport, Cell Nucleus; Air Pollutants; Animals; bcl-Associated Death Protein; bcl-X Protein | 2009 |
Effect of hydrogen sulfide in a porcine model of myocardial ischemia-reperfusion: comparison of different administration regimens and characterization of the cellular mechanisms of protection.
Topics: Animals; Apoptosis; Cardiotonic Agents; Cell Survival; Coronary Circulation; Disease Models, Animal; | 2009 |
Cardioprotective effect of hydrogen sulfide in ischemic reperfusion experimental rats and its influence on expression of survivin gene.
Topics: Alkynes; Animals; Apoptosis; Blood Pressure; Blotting, Western; Cardiotonic Agents; Cystathionine ga | 2009 |
Hydrogen sulfide therapy attenuates the inflammatory response in a porcine model of myocardial ischemia/reperfusion injury.
Topics: Animals; Anti-Inflammatory Agents; Coronary Vessels; Cytokines; Free Radicals; Hydrogen Sulfide; Imm | 2009 |
Phosphodiesterase-5 inhibitor, tadalafil, protects against myocardial ischemia/reperfusion through protein-kinase g-dependent generation of hydrogen sulfide.
Topics: Animals; Carbolines; Cyclic GMP-Dependent Protein Kinases; Cystathionine beta-Synthase; Female; Hemo | 2009 |
Toxic gas, lifesaver.
Topics: Animals; Blood Pressure; Cardiovascular Physiological Phenomena; Humans; Hydrogen Sulfide; Metabolis | 2010 |
Hydrogen sulfide attenuates cardiac dysfunction in a rat model of heart failure: a mechanism through cardiac mitochondrial protection.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; Blood Pressure; Cardiotonic Agents; Caspase 3; Cytoc | 2011 |
The protective role of hydrogen sulfide in myocardial ischemia-reperfusion-induced injury in diabetic rats.
Topics: Alkynes; Animals; Apoptosis; Caspase 3; Diabetes Mellitus, Experimental; Down-Regulation; Enzyme Inh | 2011 |
Transplantation of mesenchymal stem cells preconditioned with hydrogen sulfide enhances repair of myocardial infarction in rats.
Topics: Analysis of Variance; Animals; Apoptosis; Blotting, Western; Cell Culture Techniques; Cell Survival; | 2012 |
Cinaciguat, a novel activator of soluble guanylate cyclase, protects against ischemia/reperfusion injury: role of hydrogen sulfide.
Topics: Animals; Apoptosis; Benzoates; Cell Survival; Cyclic GMP; Cyclic GMP-Dependent Protein Kinases; Cyst | 2012 |
Hydrogen sulfide mitigates cardiac remodeling during myocardial infarction via improvement of angiogenesis.
Topics: Angiostatins; Animals; Blotting, Western; Echocardiography; Endostatins; Hydrogen Sulfide; Immunohis | 2012 |
Hydrogen sulfide in toxication; rapidly transient changes in the electrocardiogram suggestive of myocardial infarction.
Topics: Cardiovascular Diseases; Electrocardiography; Humans; Hydrogen Sulfide; Myocardial Infarction; Poiso | 1954 |
[Experimental myocardial infarct and hydrogen sulfide baths].
Topics: Balneology; Baths; Hydrogen Sulfide; Myocardial Infarction | 1957 |
[Changes of the new gaseous transmitter H2S in patients with coronary heart disease].
Topics: Adult; Aged; Angina, Unstable; Coronary Disease; Female; Humans; Hydrogen Sulfide; Male; Middle Aged | 2005 |
The production of hydrogen sulfide limits myocardial ischemia and reperfusion injury and contributes to the cardioprotective effects of preconditioning with endotoxin, but not ischemia in the rat.
Topics: Alkynes; Animals; Cardiotonic Agents; Cystathionine gamma-Lyase; Decanoic Acids; Endotoxins; Enzyme | 2006 |
Hydrogen sulfide and its possible roles in myocardial ischemia in experimental rats.
Topics: Alkynes; Animals; Cell Death; Cells, Cultured; Culture Media; Cystathionine gamma-Lyase; Disease Mod | 2007 |
Cardioprotection induced by hydrogen sulfide preconditioning involves activation of ERK and PI3K/Akt pathways.
Topics: Animals; Benzophenanthridines; Cardiotonic Agents; Cell Survival; Chromones; Disease Models, Animal; | 2008 |
S-allylcysteine mediates cardioprotection in an acute myocardial infarction rat model via a hydrogen sulfide-mediated pathway.
Topics: Animals; Cardiotonic Agents; Cystathionine gamma-Lyase; Cysteine; Garlic; Humans; Hydrogen Sulfide; | 2007 |
Hydrogen sulfide preconditioning by garlic when it starts to smell.
Topics: Animals; Cardiotonic Agents; Cysteine; Garlic; Humans; Hydrogen Sulfide; Myocardial Infarction; Plan | 2007 |
H(2)S protects myocardium against ischemia/reperfusion injury and its effect on c-Fos protein expression in rats.
Topics: Animals; Cardiotonic Agents; Coronary Vessels; Down-Regulation; Hemodynamics; Hydrogen Sulfide; Male | 2008 |
Effects of hydrogen sulphide on ischaemia-reperfusion injury and ischaemic preconditioning in the isolated, perfused rat heart.
Topics: Animals; Blotting, Western; Cardiotonic Agents; Drug Evaluation, Preclinical; HSP72 Heat-Shock Prote | 2008 |
Exogenous hydrogen sulfide postconditioning protects isolated rat hearts against ischemia-reperfusion injury.
Topics: Animals; Blood Pressure; Buffers; Coronary Circulation; Creatine Kinase; Decanoic Acids; Dose-Respon | 2008 |
A follow-up study of coronary heart disease in viscose rayon workers exposed to carbon disulphide.
Topics: Air; Angina Pectoris; Blood Pressure; Carbon Disulfide; Cardiac Volume; Coronary Disease; Dose-Respo | 1975 |
Hydrogen sulphide poisoning in factory worker.
Topics: Adult; Emotions; Humans; Hydrogen Sulfide; Male; Myocardial Infarction; Occupational Diseases | 1988 |
[Change in the functional state of the myocardium under the influence of chamber hydrogen sulfide baths at an early stage of posthospital rehabilitation of myocardial infarct patients].
Topics: Adult; Baths; Electrocardiography; Heart; Humans; Hydrogen Sulfide; Middle Aged; Myocardial Infarcti | 1973 |