hydrogen sulfide has been researched along with Cardiotoxicity in 9 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Cardiotoxicity: Damage to the HEART or its function secondary to exposure to toxic substances such as drugs used in CHEMOTHERAPY; IMMUNOTHERAPY; or RADIATION.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 8 (88.89) | 24.3611 |
2020's | 1 (11.11) | 2.80 |
Authors | Studies |
---|---|
Wang, Y | 2 |
Ying, X | 1 |
Zou, Z | 1 |
Yuan, A | 1 |
Xiao, Z | 1 |
Geng, N | 1 |
Qiao, Z | 1 |
Li, W | 1 |
Lu, X | 1 |
Pu, J | 1 |
Wang, S | 1 |
Chi, Q | 1 |
Hu, X | 3 |
Cong, Y | 1 |
Li, S | 2 |
Zhuang, XD | 1 |
Long, M | 1 |
Dong, XB | 1 |
Liu, DH | 1 |
Liao, XX | 1 |
Liu, MH | 3 |
Zhang, Y | 3 |
Lin, XL | 2 |
He, J | 3 |
Tan, TP | 3 |
Wu, SJ | 3 |
Yu, S | 2 |
Chen, L | 2 |
Chen, YD | 2 |
Fu, HY | 2 |
Yuan, C | 2 |
Li, J | 2 |
Liu, J | 1 |
Tian, W | 1 |
Sun, J | 1 |
Aponte, AM | 1 |
Menazza, S | 1 |
Gucek, M | 1 |
Steenbergen, C | 1 |
Murphy, E | 1 |
Guo, DM | 1 |
He, H | 1 |
Peng, J | 1 |
Tang, ZH | 1 |
Jiang, ZS | 1 |
Wu, J | 1 |
Guo, W | 1 |
Lin, SZ | 1 |
Wang, ZJ | 1 |
Kan, JT | 1 |
Chen, SY | 1 |
Zhu, YZ | 1 |
Huang, Z | 1 |
Zhuang, X | 1 |
Xie, C | 1 |
Dong, X | 1 |
Guo, Y | 1 |
Liao, X | 1 |
9 other studies available for hydrogen sulfide and Cardiotoxicity
Article | Year |
---|---|
Hydrogen sulfide alleviates mitochondrial damage and ferroptosis by regulating OPA3-NFS1 axis in doxorubicin-induced cardiotoxicity.
Topics: Carbon-Sulfur Lyases; Cardiotoxicity; Doxorubicin; Ferroptosis; Humans; Hydrogen Sulfide; Myocytes, | 2023 |
Hydrogen sulfide-induced oxidative stress leads to excessive mitochondrial fission to activate apoptosis in broiler myocardia.
Topics: Animals; Apoptosis; Cardiotoxicity; Chickens; Gene Expression Regulation; Hydrogen Sulfide; Inhalati | 2019 |
Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
Topics: Animals; Cardiotoxicity; Cell Death; Cell Line; Cell Survival; Cytoprotection; Glucose; Hydrogen Sul | 2014 |
Hydrogen sulfide attenuates doxorubicin‑induced cardiotoxicity by inhibiting calreticulin expression in H9c2 cells.
Topics: Antineoplastic Agents; Apoptosis; Calreticulin; Cardiotoxicity; Cell Line; Doxorubicin; Gene Express | 2015 |
Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibiting the expression of peroxiredoxin III in H9c2 cells.
Topics: Acetylcysteine; Animals; Cardiotonic Agents; Cardiotoxicity; Cell Death; Cystathionine gamma-Lyase; | 2016 |
Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.
Topics: Animals; Cardiotoxicity; Hydrogen Sulfide; Ischemic Postconditioning; Mice, Inbred C57BL; NADPH Oxid | 2016 |
Hydrogen sulfide protects H9c2 cardiac cells against doxorubicin-induced cytotoxicity through the PI3K/Akt/FoxO3a pathway.
Topics: Acetylcysteine; Animals; Apoptosis; Cardiotonic Agents; Cardiotoxicity; Cell Line; Chromones; Doxoru | 2016 |
Gp130-mediated STAT3 activation by S-propargyl-cysteine, an endogenous hydrogen sulfide initiator, prevents doxorubicin-induced cardiotoxicity.
Topics: Animals; Calcium; Cardiotoxicity; Cell Nucleus; Cysteine; Cytokine Receptor gp130; Doxorubicin; Hydr | 2016 |
Exogenous Hydrogen Sulfide Attenuates High Glucose-Induced Cardiotoxicity by Inhibiting NLRP3 Inflammasome Activation by Suppressing TLR4/NF-κB Pathway in H9c2 Cells.
Topics: Animals; Apoptosis; Cardiotoxicity; Cell Line; Cytoprotection; Gene Silencing; Glucose; Hydrogen Sul | 2016 |