hydrogen sulfide has been researched along with Brain Ischemia in 33 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
Brain Ischemia: Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Hydrogen sulfide (H2S) plays multiple roles in the function of the central nervous system in physiological and pathological conditions, such as cerebral ischemia." | 7.81 | Effect of the Inhibition of Hydrogen Sulfide Synthesis on Ischemic Injury and Oxidative Stress Biomarkers in a Transient Model of Focal Cerebral Ischemia in Rats. ( Bandegi, AR; Hadadha, M; Vakili, A, 2015) |
| " Infarction, neurological scores, brain edema, Evans blue (EB) extravasation, and tight junction protein expression were examined at 48 h after MCAO." | 7.80 | Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia. ( Alkayed, NJ; Ao, G; Cheng, J; Du, H; Hu, L; Jia, J; Liu, CF; Liu, H; Wang, Y; Xiao, Y, 2014) |
| "To explore the neuroprotective mechanism of exogenous hydrogen sulfide after cerebral ischemia-reperfusion (I/R) in rats." | 7.79 | [Neuroprotective mechanism of hydrogen sulfide after cerebral ischemia-reperfusion in rats]. ( Shen, Q; Yang, XS; Yin, J; Zeng, QH, 2013) |
| "The present study was undertaken to study the effects of exogenous hydrogen sulfide (H(2)S) on global cerebral ischemia-reperfusion(I/R) and the underlying mechanisms." | 7.79 | Exogenous hydrogen sulfide protects against global cerebral ischemia/reperfusion injury via its anti-oxidative, anti-inflammatory and anti-apoptotic effects in rats. ( Chen, G; Liu, Y; Ou, D; Tu, C; Xiao, X; Yin, J; Zhao, J, 2013) |
| "We investigated whether mild hypothermia combined with sodium hydrosulfide treatment during resuscitation improves neuron survival following cerebral ischemia-reperfusion injury beyond that observed for the individual treatments." | 5.43 | Mild Hypothermia Combined with Hydrogen Sulfide Treatment During Resuscitation Reduces Hippocampal Neuron Apoptosis Via NR2A, NR2B, and PI3K-Akt Signaling in a Rat Model of Cerebral Ischemia-Reperfusion Injury. ( Dai, HB; Duan, ML; Ji, XJ; Lv, J; Ma, RM; Miao, XL; Xu, MM; Zhu, SH, 2016) |
| "Hydrogen sulfide (H2 S) is a potent vasodilator and regulates cardiovascular homeostasis." | 5.40 | Hydrogen sulfide treatment induces angiogenesis after cerebral ischemia. ( Choi, IY; Jang, H; Kim, YJ; Lee, SH; Oh, MY; Ryu, WS; Yang, HS; Yoon, BW, 2014) |
| "Hydrogen sulfide (H(2)S) is a gaseous messenger and serves as an important neuromodulator in central nervous system." | 5.36 | Dynamic change of hydrogen sulfide during global cerebral ischemia-reperfusion and its effect in rats. ( Du, A; Li, D; Mayhan, WG; Ren, C; Sui, J; Zhao, H, 2010) |
| "Emerging evidence has suggested that hydrogen sulfide (H2S) may alleviate the cellular damage associated with cerebral ischemia/reperfusion (I/R) injury." | 3.85 | The administration of hydrogen sulphide prior to ischemic reperfusion has neuroprotective effects in an acute stroke model. ( Choi, CG; Choi, Y; Ha, HK; Ham, SJ; Jeon, SB; Jung, SC; Kim, J; Kim, JK; Kim, KW; Kim, ST; Kwon, JI; Shim, WH; Sung, YS; Woo, CW; Woo, DC, 2017) |
| "Hydrogen sulfide (H2S) plays multiple roles in the function of the central nervous system in physiological and pathological conditions, such as cerebral ischemia." | 3.81 | Effect of the Inhibition of Hydrogen Sulfide Synthesis on Ischemic Injury and Oxidative Stress Biomarkers in a Transient Model of Focal Cerebral Ischemia in Rats. ( Bandegi, AR; Hadadha, M; Vakili, A, 2015) |
| " Infarction, neurological scores, brain edema, Evans blue (EB) extravasation, and tight junction protein expression were examined at 48 h after MCAO." | 3.80 | Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia. ( Alkayed, NJ; Ao, G; Cheng, J; Du, H; Hu, L; Jia, J; Liu, CF; Liu, H; Wang, Y; Xiao, Y, 2014) |
| "To explore the neuroprotective mechanism of exogenous hydrogen sulfide after cerebral ischemia-reperfusion (I/R) in rats." | 3.79 | [Neuroprotective mechanism of hydrogen sulfide after cerebral ischemia-reperfusion in rats]. ( Shen, Q; Yang, XS; Yin, J; Zeng, QH, 2013) |
| "The present study was undertaken to study the effects of exogenous hydrogen sulfide (H(2)S) on global cerebral ischemia-reperfusion(I/R) and the underlying mechanisms." | 3.79 | Exogenous hydrogen sulfide protects against global cerebral ischemia/reperfusion injury via its anti-oxidative, anti-inflammatory and anti-apoptotic effects in rats. ( Chen, G; Liu, Y; Ou, D; Tu, C; Xiao, X; Yin, J; Zhao, J, 2013) |
| "We observed recently that elevated plasma cysteine levels are associated with poor clinical outcome in acute stroke patients." | 3.73 | Hydrogen sulfide is a mediator of cerebral ischemic damage. ( Chen, CP; Halliwell, B; Moore, PK; Qu, K; Wong, PT, 2006) |
| "We investigated whether mild hypothermia combined with sodium hydrosulfide treatment during resuscitation improves neuron survival following cerebral ischemia-reperfusion injury beyond that observed for the individual treatments." | 1.43 | Mild Hypothermia Combined with Hydrogen Sulfide Treatment During Resuscitation Reduces Hippocampal Neuron Apoptosis Via NR2A, NR2B, and PI3K-Akt Signaling in a Rat Model of Cerebral Ischemia-Reperfusion Injury. ( Dai, HB; Duan, ML; Ji, XJ; Lv, J; Ma, RM; Miao, XL; Xu, MM; Zhu, SH, 2016) |
| "Transient cerebral ischemia was induced using the Pulsinelli four-vessel occlusion method." | 1.42 | Hydrogen sulphide and mild hypothermia activate the CREB signaling pathway and prevent ischemia-reperfusion injury. ( Dai, HB; Duan, ML; Hu, YM; Ji, X; Li, WY; Ma, RM; Miao, XL; Zhang, LD; Zhu, SH, 2015) |
| "Hydrogen sulfide (H2S) is a new type of neurotransmitter that regulates the nervous, circulatory and immune systems as well as various adverse factors that can reduce cerebral vascular or brain parenchyma injury." | 1.40 | H₂S attenuates cognitive deficits through Akt1/JNK3 signaling pathway in ischemic stroke. ( Dong, H; Du, Y; Fu, Y; Huang, X; Liu, H; Liu, Y; Ma, K; Qi, D; Song, Y; Sun, Y; Wen, X; Wu, J; Zhang, F, 2014) |
| "Hydrogen sulfide (H2 S) is a potent vasodilator and regulates cardiovascular homeostasis." | 1.40 | Hydrogen sulfide treatment induces angiogenesis after cerebral ischemia. ( Choi, IY; Jang, H; Kim, YJ; Lee, SH; Oh, MY; Ryu, WS; Yang, HS; Yoon, BW, 2014) |
| "The rat model of global cerebral ischemia/reperfusion injury was established by bilateral common carotid arteries occlusion combined with hemorrhagic hypotension." | 1.39 | [Protective effect of H2S pretreatment on cerebral ischemia-reperfusion injury and its mechanisms in rats]. ( Gao, L; Gu, LZ; Guo, J; Qin, H, 2013) |
| "S-memantine was synthesized by chemically combining a slow releasing H(2)S donor 4-(3-thioxo-3H-1,2-dithiol-4-yl)-benzoic acid (ACS48) with a NMDAR antagonist memantine." | 1.38 | A novel hydrogen sulfide-releasing N-methyl-D-aspartate receptor antagonist prevents ischemic neuronal death. ( Arai, K; Atochin, DN; Ichinose, F; Khatri, A; Kida, K; Kosugi, S; Marutani, E; Nguyen, R; Tokuda, K; Van Leyen, K, 2012) |
| "Hydrogen sulfide functions as a vasodilator as well as a regulator of neuronal ion channels and multiple intracellular signaling pathways." | 1.37 | Gain-of-function polymorphisms of cystathionine β-synthase and delayed cerebral ischemia following aneurysmal subarachnoid hemorrhage. ( Badjatia, N; Claassen, J; Connolly, ES; DeRosa, PA; Ducruet, AF; Fernandez, L; Grobelny, BT; Hickman, ZL; Kotchetkov, IS; Lee, K; Mayer, SA; Narula, R; Zacharia, BE, 2011) |
| "Hydrogen sulfide (H(2)S) is a gaseous messenger and serves as an important neuromodulator in central nervous system." | 1.36 | Dynamic change of hydrogen sulfide during global cerebral ischemia-reperfusion and its effect in rats. ( Du, A; Li, D; Mayhan, WG; Ren, C; Sui, J; Zhao, H, 2010) |
| "In aged humans, stroke is a major cause of disability for which no neuroprotective measures are available." | 1.35 | Strategies to improve post-stroke behavioral recovery in aged subjects. ( Bălşeanu, A; Buga, AM; Mogoantă, L; Popa-Wagner, A, 2009) |
| "Focal cerebral ischemia was produced by reversible occlusion of the right middle cerebral artery in 17-month-old male Sprague-Dawley rats." | 1.35 | Long-term hypothermia reduces infarct volume in aged rats after focal ischemia. ( Balseanu, AT; Buga, AM; Florian, B; Grisk, O; Kessler, C; Popa-Wagner, A; Vintilescu, R; Walker, LC, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (9.09) | 29.6817 |
| 2010's | 26 (78.79) | 24.3611 |
| 2020's | 4 (12.12) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, F | 3 |
| Zhou, H | 1 |
| Zhang, X | 1 |
| Ni, SJ | 1 |
| Yao, ZY | 1 |
| Wei, X | 2 |
| Heng, X | 1 |
| Qu, SY | 1 |
| Zhao, X | 1 |
| Qi, YY | 1 |
| Ge, PY | 1 |
| Xu, CP | 1 |
| Yang, NY | 1 |
| Cao, Y | 1 |
| Zhu, HX | 1 |
| Guo, R | 1 |
| Zhang, QC | 1 |
| Huang, Y | 1 |
| Omorou, M | 1 |
| Gao, M | 1 |
| Mu, C | 1 |
| Xu, W | 1 |
| Xu, H | 1 |
| Sun, X | 2 |
| Wang, Y | 3 |
| Wen, S | 1 |
| Huang, K | 1 |
| Huang, J | 1 |
| Chu, X | 1 |
| Pang, L | 1 |
| Zhang, M | 1 |
| Wu, X | 1 |
| Xu, Y | 1 |
| He, M | 1 |
| Yang, J | 1 |
| Li, J | 1 |
| Li, Y | 2 |
| Ao, G | 3 |
| Cheng, J | 3 |
| Jia, J | 2 |
| Woo, CW | 1 |
| Kwon, JI | 1 |
| Kim, KW | 1 |
| Kim, JK | 1 |
| Jeon, SB | 1 |
| Jung, SC | 1 |
| Choi, CG | 1 |
| Kim, ST | 1 |
| Kim, J | 1 |
| Ham, SJ | 1 |
| Shim, WH | 1 |
| Sung, YS | 1 |
| Ha, HK | 1 |
| Choi, Y | 1 |
| Woo, DC | 1 |
| Sandu, RE | 1 |
| Dumbrava, D | 1 |
| Surugiu, R | 1 |
| Glavan, DG | 1 |
| Gresita, A | 1 |
| Petcu, EB | 1 |
| Qin, H | 1 |
| Gu, LZ | 1 |
| Gao, L | 1 |
| Guo, J | 1 |
| Yin, J | 2 |
| Zeng, QH | 1 |
| Shen, Q | 1 |
| Yang, XS | 1 |
| Hu, L | 1 |
| Liu, H | 2 |
| Xiao, Y | 2 |
| Du, H | 1 |
| Alkayed, NJ | 1 |
| Liu, CF | 1 |
| Wen, X | 1 |
| Qi, D | 1 |
| Sun, Y | 2 |
| Huang, X | 1 |
| Zhang, F | 1 |
| Wu, J | 1 |
| Fu, Y | 1 |
| Ma, K | 1 |
| Du, Y | 1 |
| Dong, H | 1 |
| Liu, Y | 2 |
| Song, Y | 1 |
| Jang, H | 1 |
| Oh, MY | 1 |
| Kim, YJ | 1 |
| Choi, IY | 1 |
| Yang, HS | 1 |
| Ryu, WS | 1 |
| Lee, SH | 1 |
| Yoon, BW | 1 |
| Hu, Y | 1 |
| Li, R | 1 |
| Yang, H | 1 |
| Luo, H | 1 |
| Chen, Z | 1 |
| Yu, Q | 1 |
| Lu, Z | 1 |
| Tao, L | 1 |
| Yang, L | 1 |
| Guo, Y | 1 |
| Yang, Y | 1 |
| Ding, Q | 1 |
| Dai, HB | 2 |
| Ji, X | 1 |
| Zhu, SH | 2 |
| Hu, YM | 1 |
| Zhang, LD | 1 |
| Miao, XL | 2 |
| Ma, RM | 2 |
| Duan, ML | 2 |
| Li, WY | 1 |
| Xu, MM | 1 |
| Lv, J | 1 |
| Ji, XJ | 1 |
| Hadadha, M | 1 |
| Vakili, A | 1 |
| Bandegi, AR | 1 |
| Zhang, Y | 1 |
| Chen, S | 1 |
| Buga, AM | 3 |
| Bălşeanu, A | 1 |
| Popa-Wagner, A | 3 |
| Mogoantă, L | 1 |
| Ren, C | 1 |
| Du, A | 1 |
| Li, D | 1 |
| Sui, J | 1 |
| Mayhan, WG | 1 |
| Zhao, H | 1 |
| Grobelny, BT | 1 |
| Ducruet, AF | 1 |
| DeRosa, PA | 1 |
| Kotchetkov, IS | 1 |
| Zacharia, BE | 1 |
| Hickman, ZL | 1 |
| Fernandez, L | 1 |
| Narula, R | 1 |
| Claassen, J | 1 |
| Lee, K | 1 |
| Badjatia, N | 1 |
| Mayer, SA | 1 |
| Connolly, ES | 1 |
| Qi, M | 1 |
| Hang, C | 1 |
| Zhu, L | 1 |
| Shi, J | 1 |
| Li, Z | 1 |
| Xie, Y | 1 |
| Yang, Z | 1 |
| Zhang, T | 1 |
| Minamishima, S | 1 |
| Ichinose, F | 2 |
| Zhou, J | 1 |
| Wu, PF | 1 |
| Chen, JG | 1 |
| Joseph, C | 1 |
| Vintilescu, R | 2 |
| Balseanu, AT | 2 |
| Moldovan, M | 1 |
| Junker, H | 1 |
| Walker, L | 1 |
| Lotze, M | 1 |
| Marutani, E | 1 |
| Kosugi, S | 1 |
| Tokuda, K | 1 |
| Khatri, A | 1 |
| Nguyen, R | 1 |
| Atochin, DN | 1 |
| Kida, K | 1 |
| Van Leyen, K | 1 |
| Arai, K | 1 |
| Li, GF | 1 |
| Luo, HK | 1 |
| Li, LF | 1 |
| Zhang, QZ | 1 |
| Xie, LJ | 1 |
| Jiang, H | 1 |
| Li, LP | 1 |
| Hao, N | 1 |
| Wang, WW | 1 |
| Zhang, JX | 1 |
| Duan, L | 1 |
| Bai, L | 1 |
| Tian, M | 1 |
| Li, W | 1 |
| Zhang, B | 1 |
| Tu, C | 1 |
| Zhao, J | 1 |
| Ou, D | 1 |
| Chen, G | 1 |
| Xiao, X | 1 |
| Sutherland, BA | 1 |
| Harrison, JC | 1 |
| Nair, SM | 1 |
| Sammut, IA | 1 |
| Qu, K | 1 |
| Chen, CP | 1 |
| Halliwell, B | 1 |
| Moore, PK | 1 |
| Wong, PT | 1 |
| Florian, B | 1 |
| Grisk, O | 1 |
| Walker, LC | 1 |
| Kessler, C | 1 |
6 reviews available for hydrogen sulfide and Brain Ischemia
| Article | Year |
|---|---|
Hydrogen sulfide and its donors for the treatment of cerebral ischaemia-reperfusion injury: A comprehensive review.
Topics: Animals; Brain Injuries; Brain Ischemia; Cerebral Infarction; Hydrogen Sulfide; Mammals; Oxidative S | 2023 |
Cellular and Molecular Mechanisms Underlying Non-Pharmaceutical Ischemic Stroke Therapy in Aged Subjects.
Topics: Animals; Brain Ischemia; Caloric Restriction; Comorbidity; Diabetes Mellitus; Diet, High-Fat; Diseas | 2017 |
Involvement of endothelial-derived relaxing factors in the regulation of cerebral blood flow.
Topics: Animals; Brain Ischemia; Cerebrovascular Circulation; Endothelium-Dependent Relaxing Factors; Endoth | 2011 |
[Emerging role of hydrogen sulfide in biology].
Topics: Animals; Anti-Inflammatory Agents; Apoptosis; Brain Ischemia; Cardiopulmonary Resuscitation; Heart A | 2010 |
Targeting gaseous molecules to protect against cerebral ischaemic injury: mechanisms and prospects.
Topics: Animals; Brain Ischemia; Carbon Monoxide; Cytoprotection; Drug Delivery Systems; Gases; Humans; Hydr | 2012 |
Inhalation gases or gaseous mediators as neuroprotectants for cerebral ischaemia.
Topics: Administration, Inhalation; Animals; Brain Ischemia; Carbon Monoxide; Clinical Trials as Topic; Drug | 2013 |
27 other studies available for hydrogen sulfide and Brain Ischemia
| Article | Year |
|---|---|
SAM, a cystathionine beta-synthase activator, promotes hydrogen sulfide to promote neural repair resulting from massive cerebral infarction induced by middle cerebral artery occlusion.
Topics: Animals; Apoptosis; Brain Ischemia; Cystathionine beta-Synthase; Glucose; Glutamic Acid; Humans; Hyd | 2022 |
Vagus nerve stimulated by microbiota-derived hydrogen sulfide mediates the regulation of berberine on microglia in transient middle cerebral artery occlusion rats.
Topics: Animals; Berberine; Brain Ischemia; Hydrogen Sulfide; Infarction, Middle Cerebral Artery; Microbiota | 2022 |
Novel controlled and targeted releasing hydrogen sulfide system exerts combinational cerebral and myocardial protection after cardiac arrest.
Topics: Allyl Compounds; Animals; Antioxidants; Brain Ischemia; Cells, Cultured; Delayed-Action Preparations | 2021 |
The cystathionine β-synthase/hydrogen sulfide pathway contributes to microglia-mediated neuroinflammation following cerebral ischemia.
Topics: AMP-Activated Protein Kinases; Animals; Brain Ischemia; Cells, Cultured; Cerebral Cortex; Cystathion | 2017 |
The administration of hydrogen sulphide prior to ischemic reperfusion has neuroprotective effects in an acute stroke model.
Topics: Animals; Apoptosis; Aspartic Acid; Brain Ischemia; Disease Models, Animal; Glutamic Acid; Humans; Hy | 2017 |
[Protective effect of H2S pretreatment on cerebral ischemia-reperfusion injury and its mechanisms in rats].
Topics: Animals; Brain Ischemia; Free Radicals; Hippocampus; HSP70 Heat-Shock Proteins; Hydrogen Sulfide; Ma | 2013 |
[Neuroprotective mechanism of hydrogen sulfide after cerebral ischemia-reperfusion in rats].
Topics: Animals; Brain Ischemia; HSP20 Heat-Shock Proteins; Hydrogen Sulfide; Male; Neurons; Neuroprotective | 2013 |
Hydrogen sulfide protects blood-brain barrier integrity following cerebral ischemia.
Topics: Animals; Behavior, Animal; Blood-Brain Barrier; Blotting, Western; Brain Edema; Brain Ischemia; Colo | 2014 |
H₂S attenuates cognitive deficits through Akt1/JNK3 signaling pathway in ischemic stroke.
Topics: Animals; Brain Ischemia; CA1 Region, Hippocampal; Cell Survival; Chromones; Cognition Disorders; Enz | 2014 |
Hydrogen sulfide treatment induces angiogenesis after cerebral ischemia.
Topics: Analysis of Variance; Angiogenesis Inhibitors; Animals; Brain Infarction; Brain Ischemia; Cells, Cul | 2014 |
Sirtuin 6 is essential for sodium sulfide-mediated cytoprotective effect in ischemia/reperfusion-stimulated brain endothelial cells.
Topics: Animals; Antioxidants; Brain Ischemia; Catalase; Cell Death; Cell Hypoxia; Cell Line; Cystathionine | 2015 |
ROS-Dependent Neuroprotective Effects of NaHS in Ischemia Brain Injury Involves the PARP/AIF Pathway.
Topics: Animals; Antioxidants; Brain; Brain Injuries; Brain Ischemia; Cell Line; Hydrogen Sulfide; Male; Neu | 2015 |
Hydrogen sulphide and mild hypothermia activate the CREB signaling pathway and prevent ischemia-reperfusion injury.
Topics: Analysis of Variance; Animals; Blotting, Western; Brain Injuries; Brain Ischemia; Brain-Derived Neur | 2015 |
Mild Hypothermia Combined with Hydrogen Sulfide Treatment During Resuscitation Reduces Hippocampal Neuron Apoptosis Via NR2A, NR2B, and PI3K-Akt Signaling in a Rat Model of Cerebral Ischemia-Reperfusion Injury.
Topics: Animals; Apoptosis; Brain Ischemia; Combined Modality Therapy; Disease Models, Animal; Hippocampus; | 2016 |
Effect of the Inhibition of Hydrogen Sulfide Synthesis on Ischemic Injury and Oxidative Stress Biomarkers in a Transient Model of Focal Cerebral Ischemia in Rats.
Topics: Aminooxyacetic Acid; Animals; Biomarkers; Brain; Brain Ischemia; Disease Models, Animal; Enzyme Inhi | 2015 |
Synthesis and biological evaluation of novel hydrogen sulfide releasing nicotinic acid derivatives.
Topics: Animals; Brain Ischemia; Cell Line; Cell Survival; Disease Models, Animal; Dose-Response Relationshi | 2016 |
Strategies to improve post-stroke behavioral recovery in aged subjects.
Topics: Aging; Animals; Behavior; Brain; Brain Ischemia; Cerebral Infarction; Disease Models, Animal; Feedba | 2009 |
Dynamic change of hydrogen sulfide during global cerebral ischemia-reperfusion and its effect in rats.
Topics: Animals; Blotting, Western; Brain Ischemia; Central Nervous System Agents; Cerebral Cortex; Cystathi | 2010 |
Gain-of-function polymorphisms of cystathionine β-synthase and delayed cerebral ischemia following aneurysmal subarachnoid hemorrhage.
Topics: Aged; Brain Ischemia; Cystathionine beta-Synthase; Female; Genetic Predisposition to Disease; Homocy | 2011 |
Protective effects of exogenous hydrogen sulfide on neurons of hippocampus in a rat model of brain ischemia.
Topics: Animals; Behavior, Animal; Brain Ischemia; Hippocampus; Hydrogen Sulfide; Long-Term Potentiation; Ma | 2011 |
Prolonged gaseous hypothermia prevents the upregulation of phagocytosis-specific protein annexin 1 and causes low-amplitude EEG activity in the aged rat brain after cerebral ischemia.
Topics: Aging; Animals; Annexin A1; Blotting, Western; Brain; Brain Ischemia; Disease Models, Animal; Electr | 2012 |
A novel hydrogen sulfide-releasing N-methyl-D-aspartate receptor antagonist prevents ischemic neuronal death.
Topics: Animals; Brain Injuries; Brain Ischemia; Cell Death; Cell Line, Tumor; Cells, Cultured; Drug Design; | 2012 |
Dual effects of hydrogen sulphide on focal cerebral ischaemic injury via modulation of oxidative stress-induced apoptosis.
Topics: Animals; Apoptosis; Apoptosis Regulatory Proteins; Brain Infarction; Brain Ischemia; Cerebral Cortex | 2012 |
Effects of exogenous hydrogen sulfide on brain metabolism and early neurological function in rabbits after cardiac arrest.
Topics: Administration, Inhalation; Animals; Body Temperature; Brain; Brain Ischemia; Cardiopulmonary Resusc | 2012 |
Exogenous hydrogen sulfide protects against global cerebral ischemia/reperfusion injury via its anti-oxidative, anti-inflammatory and anti-apoptotic effects in rats.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Antioxidants; Apoptosis; Biomarkers; Blotting, Wes | 2013 |
Hydrogen sulfide is a mediator of cerebral ischemic damage.
Topics: Air Pollutants; Animals; Brain Injuries; Brain Ischemia; Cerebral Cortex; Cysteine; Dizocilpine Male | 2006 |
Long-term hypothermia reduces infarct volume in aged rats after focal ischemia.
Topics: Aging; Animals; Apoptosis; Apoptosis Regulatory Proteins; Body Temperature; Brain; Brain Infarction; | 2008 |