hydrogen sulfide has been researched along with Blood Poisoning in 37 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The view of hydrogen sulfide has changed from a toxic by-product to a crucial signaling molecule, with enormous potential as a pharmacological target for diseases ranging from heart disease to sepsis." | 8.88 | Hydrogen sulfide as a therapeutic target for inflammation. ( Badiei, A; Bhatia, M; Rivers, JR, 2012) |
| "There is ample evidence that nitric oxide, carbon monoxide, and H2S may exert cytoprotective effects in shock states due to their vasomotor, antioxidant, and anti-inflammatory properties as well as their potential to induce a hibernation-like metabolic state called 'suspended animation' resulting from inhibition of cytochrome-c-oxidase." | 8.85 | Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide. ( Baumgart, K; Radermacher, P; Wagner, F, 2009) |
| "To determine alterations of circulating levels of hydrogen sulfide and substance P in patients with sepsis compared to non-sepsis patients with similar disease severity and organ dysfunction." | 7.85 | Circulating levels of hydrogen sulfide and substance P in patients with sepsis. ( Bhatia, M; Chambers, S; Gaddam, RR; Murdoch, D; Shaw, G, 2017) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 7.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "Hydrogen sulfide (H(2)S) has been shown to induce transient receptor potential vanilloid 1 (TRPV1)-mediated neurogenic inflammation in polymicrobial sepsis." | 7.77 | Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM, 2011) |
| "Capsazepine treatment attenuates significantly systemic inflammation and multiple organ damage caused by sepsis, and protects against sepsis-induced mortality." | 7.76 | Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis. ( Ang, SF; Bhatia, M; Moochhala, SM, 2010) |
| " However, whether endogenous H2S would regulate sepsis-associated lung inflammation via substance P (SP) and its receptors remains unknown." | 7.74 | Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury. ( Adhikari, S; Bhatia, M; Hegde, A; Moochhala, SM; Ng, SW; Zhang, H, 2007) |
| "Sepsis is is anabnormalhost immune responsecausedbyinfection." | 7.01 | Application and value of hydrogen sulfide modulated autophagy in sepsis. ( Liu, C; Sun, Y, 2023) |
| "Sepsis is one of the main causes of death in critically ill patients." | 6.66 | [Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis]. ( Chai, C; He, M; Kang, F; Nie, J; Zhang, C, 2020) |
| "Hydrogen sulfide (H2S) is a novel signaling molecule most recently found to be of fundamental importance in cellular function as a regulator of apoptosis, inflammation, and perfusion." | 6.55 | Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia. ( DeLeon, ER; Drucker, NA; Ferkowicz, MJ; Jensen, AR; Khaneki, S; Markel, TA; Olson, KR; Yoder, MC, 2017) |
| "Hydrogen sulfide is a novel gasotransmitter that has been shown to play a major role in regulating vascular tone." | 6.53 | The role of hydrogen sulfide in burns. ( Akter, F, 2016) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 6.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 5.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H2S) has recently been recognized as a novel gaseous transmitter with several anti-inflammatory properties." | 5.62 | Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis. ( Antonakos, N; Bauer, M; Damoraki, G; Droggiti, DE; Giamarellos-Bourboulis, EJ; Gkavogianni, T; Jentho, E; Karageorgos, A; Karakike, E; Katrini, K; Katsouda, A; Koufargyris, P; Kouloulias, V; Papapetropoulos, A; Platoni, K; Renieris, G; Sabracos, L; Szabo, C; Wang, R; Weis, S, 2021) |
| "Sepsis is a major cause of mortality, and dysregulation of the immune response plays a central role in this syndrome." | 5.40 | Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition. ( Colombani, PM; Ferlito, M; Fox-Talbot, K; Fulton, WB; Marchionni, L; Paolocci, N; Steenbergen, C; Wang, Q, 2014) |
| "In septic rats pretreated with AOAA, sepsis-associated hippocampus inflammation was reduced." | 5.37 | Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy. ( Chen, D; Lan, X; Li, C; Liu, B; Pan, H; Yang, G, 2011) |
| "Sepsis was induced by cecal ligation and puncture (CLP)." | 5.36 | Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation. ( Alves-Filho, JC; Assreuy, J; Carlos, D; Cunha, FQ; Czaikoski, PG; Ferreira, SH; Freitas, A; Hothersall, JS; Montenegro, MF; Nascimento, DC; Neto, AF; Orrico, MI; Rossi, MA; Souto, FO; Spiller, F, 2010) |
| "The view of hydrogen sulfide has changed from a toxic by-product to a crucial signaling molecule, with enormous potential as a pharmacological target for diseases ranging from heart disease to sepsis." | 4.88 | Hydrogen sulfide as a therapeutic target for inflammation. ( Badiei, A; Bhatia, M; Rivers, JR, 2012) |
| "There is ample evidence that nitric oxide, carbon monoxide, and H2S may exert cytoprotective effects in shock states due to their vasomotor, antioxidant, and anti-inflammatory properties as well as their potential to induce a hibernation-like metabolic state called 'suspended animation' resulting from inhibition of cytochrome-c-oxidase." | 4.85 | Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide. ( Baumgart, K; Radermacher, P; Wagner, F, 2009) |
| "Age, APACHE score at ICU admission, neurological disease, sepsis and duration of mechanical ventilation were all independent risk factors for the development of delirium in ICU patients." | 4.40 | Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19. ( , 2023) |
| "To investigate the effect of hydrogen sulfide (H2S) on myocardial injury in sepsis-induced myocardial dysfunction (SIMD), male C57BL/6 mice were intraperitoneally injected with lipopolysaccharide (LPS) (10 mg/kg, i." | 4.31 | Hydrogen sulfide alleviates lipopolysaccharide-induced myocardial injury through TLR4-NLRP3 pathway. ( Bai, L; He, K; Jin, S; Liu, B; Miao, Y; Wu, Y; Xia, Y; Zhang, W; Zhang, X, 2023) |
| "To determine alterations of circulating levels of hydrogen sulfide and substance P in patients with sepsis compared to non-sepsis patients with similar disease severity and organ dysfunction." | 3.85 | Circulating levels of hydrogen sulfide and substance P in patients with sepsis. ( Bhatia, M; Chambers, S; Gaddam, RR; Murdoch, D; Shaw, G, 2017) |
| "Hydrogen sulfide (H(2)S) has been shown to induce transient receptor potential vanilloid 1 (TRPV1)-mediated neurogenic inflammation in polymicrobial sepsis." | 3.77 | Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM, 2011) |
| "Hydrogen sulfide (H(2)S) has been shown to promote transient receptor potential vanilloid type 1 (TRPV1)-mediated neurogenic inflammation in sepsis and its associated multiple organ failure, including acute lung injury (ALI)." | 3.77 | Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation. ( Ang, SF; Bhatia, M; MacAry, PA; Moochhala, SM; Sio, SW, 2011) |
| "Capsazepine treatment attenuates significantly systemic inflammation and multiple organ damage caused by sepsis, and protects against sepsis-induced mortality." | 3.76 | Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis. ( Ang, SF; Bhatia, M; Moochhala, SM, 2010) |
| " However, whether endogenous H2S would regulate sepsis-associated lung inflammation via substance P (SP) and its receptors remains unknown." | 3.74 | Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury. ( Adhikari, S; Bhatia, M; Hegde, A; Moochhala, SM; Ng, SW; Zhang, H, 2007) |
| "Sepsis is is anabnormalhost immune responsecausedbyinfection." | 3.01 | Application and value of hydrogen sulfide modulated autophagy in sepsis. ( Liu, C; Sun, Y, 2023) |
| "Sepsis is one of the main causes of death in critically ill patients." | 2.66 | [Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis]. ( Chai, C; He, M; Kang, F; Nie, J; Zhang, C, 2020) |
| "Hydrogen sulfide (H2S) is a novel signaling molecule most recently found to be of fundamental importance in cellular function as a regulator of apoptosis, inflammation, and perfusion." | 2.55 | Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia. ( DeLeon, ER; Drucker, NA; Ferkowicz, MJ; Jensen, AR; Khaneki, S; Markel, TA; Olson, KR; Yoder, MC, 2017) |
| "Hydrogen sulfide is a novel gasotransmitter that has been shown to play a major role in regulating vascular tone." | 2.53 | The role of hydrogen sulfide in burns. ( Akter, F, 2016) |
| "Inflammation is a response to traumatic, infectious, post-ischemic, toxic, or autoimmune injury." | 2.52 | H2S and Inflammation: An Overview. ( Bhatia, M, 2015) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 2.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "Sepsis often leads to multiple organ failure or even death and is a significant health problem that contributes to a heavy economic burden." | 1.72 | Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury. ( Li, J; Li, L; Li, M; Ma, J; Yao, C; Yao, S, 2022) |
| "Hydrogen sulfide (H2S) has recently been recognized as a novel gaseous transmitter with several anti-inflammatory properties." | 1.62 | Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis. ( Antonakos, N; Bauer, M; Damoraki, G; Droggiti, DE; Giamarellos-Bourboulis, EJ; Gkavogianni, T; Jentho, E; Karageorgos, A; Karakike, E; Katrini, K; Katsouda, A; Koufargyris, P; Kouloulias, V; Papapetropoulos, A; Platoni, K; Renieris, G; Sabracos, L; Szabo, C; Wang, R; Weis, S, 2021) |
| "Sepsis is a major cause of mortality, and dysregulation of the immune response plays a central role in this syndrome." | 1.40 | Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition. ( Colombani, PM; Ferlito, M; Fox-Talbot, K; Fulton, WB; Marchionni, L; Paolocci, N; Steenbergen, C; Wang, Q, 2014) |
| "Hydrogen sulfide metabolism was associated with an increase in O2 consumption from a baseline 96." | 1.37 | The liver as a central regulator of hydrogen sulfide. ( Clemens, MG; Culberson, CR; Narasimhan, S; Norris, EJ, 2011) |
| "In septic rats pretreated with AOAA, sepsis-associated hippocampus inflammation was reduced." | 1.37 | Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy. ( Chen, D; Lan, X; Li, C; Liu, B; Pan, H; Yang, G, 2011) |
| "Sepsis was induced by cecal ligation and puncture (CLP)." | 1.36 | Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation. ( Alves-Filho, JC; Assreuy, J; Carlos, D; Cunha, FQ; Czaikoski, PG; Ferreira, SH; Freitas, A; Hothersall, JS; Montenegro, MF; Nascimento, DC; Neto, AF; Orrico, MI; Rossi, MA; Souto, FO; Spiller, F, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (2.70) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 7 (18.92) | 29.6817 |
| 2010's | 20 (54.05) | 24.3611 |
| 2020's | 9 (24.32) | 2.80 |
| Authors | Studies |
|---|---|
| Li, J | 1 |
| Li, M | 1 |
| Li, L | 1 |
| Ma, J | 1 |
| Yao, C | 1 |
| Yao, S | 1 |
| Zhou, T | 1 |
| Qian, H | 1 |
| Zheng, N | 1 |
| Lu, Q | 1 |
| Han, Y | 1 |
| Zhao, YH | 1 |
| Cao, GD | 1 |
| Guo, LC | 1 |
| Cheng, QH | 1 |
| Xia, Y | 1 |
| Zhang, W | 1 |
| He, K | 1 |
| Bai, L | 1 |
| Miao, Y | 1 |
| Liu, B | 2 |
| Zhang, X | 1 |
| Jin, S | 1 |
| Wu, Y | 1 |
| Sun, Y | 1 |
| Liu, C | 1 |
| Nie, J | 1 |
| Kang, F | 1 |
| Zhang, C | 1 |
| Chai, C | 1 |
| He, M | 2 |
| Renieris, G | 1 |
| Droggiti, DE | 1 |
| Katrini, K | 1 |
| Koufargyris, P | 1 |
| Gkavogianni, T | 1 |
| Karakike, E | 1 |
| Antonakos, N | 1 |
| Damoraki, G | 1 |
| Karageorgos, A | 1 |
| Sabracos, L | 1 |
| Katsouda, A | 1 |
| Jentho, E | 1 |
| Weis, S | 1 |
| Wang, R | 1 |
| Bauer, M | 1 |
| Szabo, C | 2 |
| Platoni, K | 1 |
| Kouloulias, V | 1 |
| Papapetropoulos, A | 1 |
| Giamarellos-Bourboulis, EJ | 1 |
| Cui, W | 2 |
| Chen, J | 1 |
| Yu, F | 1 |
| Liu, W | 1 |
| Jensen, AR | 1 |
| Drucker, NA | 1 |
| Khaneki, S | 1 |
| Ferkowicz, MJ | 1 |
| Yoder, MC | 1 |
| DeLeon, ER | 1 |
| Olson, KR | 1 |
| Markel, TA | 1 |
| Bee, N | 1 |
| White, R | 1 |
| Petros, AJ | 1 |
| Gaddam, RR | 2 |
| Chambers, S | 1 |
| Murdoch, D | 1 |
| Shaw, G | 1 |
| Bhatia, M | 14 |
| Toliver-Kinsky, T | 1 |
| Törö, G | 1 |
| Lee, SJ | 1 |
| Shatalin, K | 1 |
| Nudler, E | 1 |
| Xu, C | 1 |
| Zhao, HY | 1 |
| Reitsema, VA | 1 |
| Star, BS | 1 |
| de Jager, VD | 1 |
| van Meurs, M | 1 |
| Henning, RH | 1 |
| Bouma, HR | 1 |
| Ferlito, M | 1 |
| Wang, Q | 1 |
| Fulton, WB | 1 |
| Colombani, PM | 1 |
| Marchionni, L | 1 |
| Fox-Talbot, K | 1 |
| Paolocci, N | 1 |
| Steenbergen, C | 1 |
| Akter, F | 1 |
| Badiei, A | 2 |
| Chambers, ST | 1 |
| Baumgart, K | 2 |
| Georgieff, M | 1 |
| Radermacher, P | 2 |
| Calzia, E | 1 |
| Zhang, H | 5 |
| Moochhala, SM | 6 |
| Wagner, F | 1 |
| Shanmugam, MK | 1 |
| Ang, SF | 3 |
| Kapoor, A | 1 |
| Thiemermann, C | 1 |
| Spiller, F | 1 |
| Orrico, MI | 1 |
| Nascimento, DC | 1 |
| Czaikoski, PG | 1 |
| Souto, FO | 1 |
| Alves-Filho, JC | 1 |
| Freitas, A | 1 |
| Carlos, D | 1 |
| Montenegro, MF | 1 |
| Neto, AF | 1 |
| Ferreira, SH | 1 |
| Rossi, MA | 1 |
| Hothersall, JS | 1 |
| Assreuy, J | 1 |
| Cunha, FQ | 1 |
| Norris, EJ | 1 |
| Culberson, CR | 1 |
| Narasimhan, S | 1 |
| Clemens, MG | 1 |
| MacAry, PA | 2 |
| Sio, SW | 1 |
| Chen, D | 1 |
| Pan, H | 1 |
| Li, C | 1 |
| Lan, X | 1 |
| Yang, G | 1 |
| Rivers, JR | 1 |
| Zhi, L | 3 |
| Moore, PK | 3 |
| Moochhala, S | 1 |
| Hegde, A | 1 |
| Ng, SW | 1 |
| Adhikari, S | 1 |
| Smith, RF | 1 |
| Dayton, SL | 1 |
| Chipps, DD | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| A Phase II Double Blind Randomized Controlled Trial of High Dose Vitamin B12 in Septic Shock[NCT03783091] | Phase 2 | 20 participants (Anticipated) | Interventional | 2019-08-05 | Recruiting | ||
| Hydrogen Sulfide as Prognostic Factor[NCT01088490] | 50 participants (Actual) | Observational | 2010-01-31 | Completed | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
11 reviews available for hydrogen sulfide and Blood Poisoning
| Article | Year |
|---|---|
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Effect of 12-week of aerobic exercise on hormones and lipid profile status in adolescent girls with polycystic ovary syndrome: A study during COVID-19.
Topics: Actin Cytoskeleton; Actins; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenosine Triphosp | 2023 |
Application and value of hydrogen sulfide modulated autophagy in sepsis.
Topics: Autophagy; Carbon Monoxide; Humans; Hydrogen Sulfide; Nitric Oxide; Sepsis | 2023 |
[Research on the signal pathway of hydrogen sulfide regulating autophagy to protect intestinal injury in sepsis].
Topics: Autophagy; Humans; Hydrogen Sulfide; Mitochondria; Sepsis; Signal Transduction | 2020 |
Hydrogen Sulfide: A Potential Novel Therapy for the Treatment of Ischemia.
Topics: Apoptosis; Cell Death; Humans; Hydrogen Sulfide; Ischemia; Sepsis; Signal Transduction | 2017 |
Metabolic Resuscitation Strategies to Prevent Organ Dysfunction in Sepsis.
Topics: Caloric Restriction; Combined Modality Therapy; Energy Metabolism; Humans; Hydrogen Sulfide; Hypothe | 2019 |
H2S and Inflammation: An Overview.
Topics: Animals; Arthritis; Humans; Hydrogen Sulfide; Inflammation; Pancreatitis; Pulmonary Disease, Chronic | 2015 |
The role of hydrogen sulfide in burns.
Topics: Animals; Burns; Humans; Hydrogen Sulfide; Inflammation; Sepsis; Wound Healing | 2016 |
Applying gases for microcirculatory and cellular oxygenation in sepsis: effects of nitric oxide, carbon monoxide, and hydrogen sulfide.
Topics: Animals; Carbon Monoxide; Hydrogen Sulfide; Microcirculation; Nitric Oxide; Nitric Oxide Synthase Ty | 2009 |
The role of pro-inflammatory molecules and pharmacological agents in acute pancreatitis and sepsis.
Topics: Animals; Apoptosis; Ceruletide; Chemokines; Female; Humans; Hydrogen Sulfide; Inflammation Mediators | 2010 |
Hydrogen sulfide and substance P in inflammation.
Topics: Air Pollutants; Amino Acid Sequence; Animals; Humans; Hydrogen Sulfide; Inflammation; Inflammation M | 2010 |
Hydrogen sulfide as a therapeutic target for inflammation.
Topics: Animals; Anti-Inflammatory Agents; Humans; Hydrogen Sulfide; Inflammation; Molecular Targeted Therap | 2012 |
1 trial available for hydrogen sulfide and Blood Poisoning
26 other studies available for hydrogen sulfide and Blood Poisoning
| Article | Year |
|---|---|
Hydrogen sulfide attenuates ferroptosis and stimulates autophagy by blocking mTOR signaling in sepsis-induced acute lung injury.
Topics: Acute Lung Injury; Animals; Autophagy; Cecum; Cell Line; Disease Models, Animal; Ferroptosis; Hydrog | 2022 |
GYY4137 ameliorates sepsis-induced cardiomyopathy via NLRP3 pathway.
Topics: Animals; Cardiomyopathies; Cytokines; Hydrogen Sulfide; Inflammasomes; Lipopolysaccharides; Mice; Mo | 2022 |
[Hydrogen Sulfide Ameliorates Myocardial Injury Caused by Sepsis Through Suppressing ROS-Mediated Endoplasmic Reticulum Stress].
Topics: Activating Transcription Factor 4; Animals; Apoptosis; Cystathionine gamma-Lyase; Endoplasmic Reticu | 2022 |
Hydrogen sulfide alleviates lipopolysaccharide-induced myocardial injury through TLR4-NLRP3 pathway.
Topics: Animals; Cardiomyopathies; Heart Injuries; Hydrogen Sulfide; Inflammation; Lipopolysaccharides; Male | 2023 |
Host cystathionine-γ lyase derived hydrogen sulfide protects against Pseudomonas aeruginosa sepsis.
Topics: Animals; Cystathionine gamma-Lyase; Humans; Hydrogen Sulfide; Mice; Mice, Knockout; Pseudomonas aeru | 2021 |
GYY4137 protected the integrity of the blood-brain barrier via activation of the Nrf2/ARE pathway in mice with sepsis.
Topics: Animals; Apoptosis; Biological Transport; Blood-Brain Barrier; Brain; Disease Models, Animal; Hydrog | 2021 |
Hydrogen Sulfide in Exhaled Gases From Ventilated Septic Neonates and Children: A Preliminary Report.
Topics: Adolescent; Biomarkers; Breath Tests; Case-Control Studies; Child; Child, Preschool; Exhalation; Fem | 2017 |
Circulating levels of hydrogen sulfide and substance P in patients with sepsis.
Topics: Aged; C-Reactive Protein; Calcitonin; Female; Humans; Hydrogen Sulfide; Intensive Care Units; Interl | 2017 |
H
Topics: Animals; Escherichia coli; Escherichia coli Infections; Host-Pathogen Interactions; Hydrogen Sulfide | 2019 |
[Effect and Mechanism of Hydrogen Sulfide on Septic Rats with Myocardial Injury].
Topics: Animals; Calcitonin; Hydrogen Sulfide; Interleukin-10; Myocardium; NF-kappa B; Protective Agents; Ra | 2018 |
Hydrogen sulfide [corrected] increases survival during sepsis: protective effect of CHOP inhibition.
Topics: Animals; Apoptosis; Bacteria; Caspase 3; Cecum; Cytokines; Endoplasmic Reticulum Stress; Enzyme Acti | 2014 |
Cystathionine-γ-lyase gene silencing with siRNA in monocytes/ macrophages attenuates inflammation in cecal ligation and puncture-induced sepsis in the mouse.
Topics: Animals; Cystathionine gamma-Lyase; Disease Models, Animal; Gene Silencing; Humans; Hydrogen Sulfide | 2016 |
Cardioprotection by hydrogen sulfide: suspended animation, inflammation, and apoptosis.
Topics: Animals; Apoptosis; Gene Deletion; Humans; Hydrogen Sulfide; Hypoxia; Inflammation; Models, Biologic | 2009 |
Endogenous hydrogen sulfide regulates inflammatory response by activating the ERK pathway in polymicrobial sepsis.
Topics: Animals; Bacteremia; Cecum; Enzyme Activation; Hydrogen Sulfide; Inflammation Mediators; Ligation; M | 2008 |
Hydrogen sulfide promotes transient receptor potential vanilloid 1-mediated neurogenic inflammation in polymicrobial sepsis.
Topics: Alkynes; Animals; Capsaicin; Cystathionine gamma-Lyase; Glycine; Hydrogen Sulfide; Liver; Lung; Male | 2010 |
Hydrogen sulfide, neurogenic inflammation, and cardioprotection: a tale of rotten eggs and vanilloid receptors.
Topics: Animals; Cardiotonic Agents; Cystathionine gamma-Lyase; Humans; Hydrogen Sulfide; Liver; Lung; Mice; | 2010 |
Hydrogen sulfide improves neutrophil migration and survival in sepsis via K+ATP channel activation.
Topics: Animals; CD11b Antigen; Cell Movement; Down-Regulation; Endothelium, Vascular; Hydrogen Sulfide; Int | 2010 |
The liver as a central regulator of hydrogen sulfide.
Topics: Animals; Cecum; Hydrogen Sulfide; In Vitro Techniques; Liver; Male; Oxygen Consumption; Phenylephrin | 2011 |
Hydrogen sulfide and neurogenic inflammation in polymicrobial sepsis: involvement of substance P and ERK-NF-κB signaling.
Topics: Animals; Capsaicin; Cell Nucleus; Chemokines; Cytokines; Extracellular Signal-Regulated MAP Kinases; | 2011 |
Hydrogen sulfide upregulates cyclooxygenase-2 and prostaglandin E metabolite in sepsis-evoked acute lung injury via transient receptor potential vanilloid type 1 channel activation.
Topics: Acute Lung Injury; Animals; Cecum; Cyclooxygenase 2; Dinoprostone; Hydrogen Sulfide; Ligation; Lung; | 2011 |
Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy.
Topics: Aminooxyacetic Acid; Animals; Brain Diseases; Cystathionine beta-Synthase; Cytokines; Disease Models | 2011 |
Role of hydrogen sulfide in cecal ligation and puncture-induced sepsis in the mouse.
Topics: Alkynes; Animals; Cecum; Disease Models, Animal; Enzyme Inhibitors; Glycine; Hydrogen Sulfide; Liver | 2006 |
Hydrogen sulfide acts as an inflammatory mediator in cecal ligation and puncture-induced sepsis in mice by upregulating the production of cytokines and chemokines via NF-kappaB.
Topics: Alkynes; Animals; Cecum; Chemokines; Cystathionine gamma-Lyase; Cytokines; Disease Models, Animal; G | 2007 |
Endogenous hydrogen sulfide regulates leukocyte trafficking in cecal ligation and puncture-induced sepsis.
Topics: Alkynes; Animals; Cell Adhesion; Cell Adhesion Molecules; Chemokine CXCL2; Enzyme Inhibitors; Glycin | 2007 |
Hydrogen sulfide up-regulates substance P in polymicrobial sepsis-associated lung injury.
Topics: Animals; Cecum; Gene Deletion; Hydrogen Sulfide; Inflammation Mediators; Ligation; Lung; Male; Mice; | 2007 |
Recognition of Citrobacter diversus in the clinical laboratory.
Topics: Alcohols; Anti-Bacterial Agents; Bacteriological Techniques; Bacteriuria; Burns; Child; Citrates; Cu | 1973 |