hydrogen sulfide has been researched along with Acute Edematous Pancreatitis in 23 studies
Hydrogen Sulfide: A flammable, poisonous gas with a characteristic odor of rotten eggs. It is used in the manufacture of chemicals, in metallurgy, and as an analytical reagent. (From Merck Index, 11th ed)
hydrogen sulfide : A sulfur hydride consisting of a single sulfur atom bonded to two hydrogen atoms. A highly poisonous, flammable gas with a characteristic odour of rotten eggs, it is often produced by bacterial decomposition of organic matter in the absence of oxygen.
thiol : An organosulfur compound in which a thiol group, -SH, is attached to a carbon atom of any aliphatic or aromatic moiety.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Previous studies have shown that during severe acute pancreatitis (SAP) attacks, hydrogen sulfide (H2S) is released in the colon." | 7.96 | Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis. ( Liao, R; Liu, Y; Qiang, Z; Xue, L; Zhang, C, 2020) |
| "To study the change in endogenous hydrogen sulfide (H2S) in patients with acute pancreatitis and its relationship to coagulation function." | 7.83 | [Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function]. ( Lu, G; Wang, H; Wu, A, 2016) |
| "The present study investigated whether chemokines are involved in hydrogen sulfide (H2S)-associated pathogenesis of acute pancreatitis and associated lung injury." | 7.74 | Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury. ( Bhatia, M; Moore, PK; Tamizhselvi, R, 2008) |
| "Inflammation is a natural response to tissue injury." | 6.72 | Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis. ( Bhatia, M; Kumar, A, 2021) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 6.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 5.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| "Acute pancreatitis was induced in mice by 10 hourly intraperitoneal injections of caerulein (50 (g/kg)." | 5.35 | Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis. ( Bhatia, M; Moochhala, SM; Ng, SW; Sidhapuriwala, JN; Tamizhselvi, R, 2008) |
| "Hydrogen sulfide (H2S) is a naturally occurring gas with potent vasodilator activity." | 5.33 | Role of hydrogen sulfide in acute pancreatitis and associated lung injury. ( Bhatia, M; Fu, D; Lau, HY; Moochhala, SM; Moore, PK; Wong, FL, 2005) |
| "Previous studies have shown that during severe acute pancreatitis (SAP) attacks, hydrogen sulfide (H2S) is released in the colon." | 3.96 | Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis. ( Liao, R; Liu, Y; Qiang, Z; Xue, L; Zhang, C, 2020) |
| "To study the change in endogenous hydrogen sulfide (H2S) in patients with acute pancreatitis and its relationship to coagulation function." | 3.83 | [Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function]. ( Lu, G; Wang, H; Wu, A, 2016) |
| "This study aimed to determine the effect of hydrogen sulfide (H2S) on Toll-like receptor 4 (TLR4)-mediated innate immune signaling in acute pancreatitis (AP) via substance P." | 3.77 | Preprotachykinin-A gene deletion regulates hydrogen sulfide-induced toll-like receptor 4 signaling pathway in cerulein-treated pancreatic acinar cells. ( Bhatia, M; Koh, YH; Shrivastava, P; Tamizhselvi, R; Zhang, H, 2011) |
| "The present study investigated whether chemokines are involved in hydrogen sulfide (H2S)-associated pathogenesis of acute pancreatitis and associated lung injury." | 3.74 | Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury. ( Bhatia, M; Moore, PK; Tamizhselvi, R, 2008) |
| "Inflammation is a natural response to tissue injury." | 2.72 | Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis. ( Bhatia, M; Kumar, A, 2021) |
| "Inflammation is a response to traumatic, infectious, post-ischemic, toxic, or autoimmune injury." | 2.52 | H2S and Inflammation: An Overview. ( Bhatia, M, 2015) |
| "Hydrogen sulfide displays a dual role in the mechanism of AP according to its concentration in the system." | 2.50 | From nitric oxide to hyperbaric oxygen: invisible and subtle but nonnegligible gaseous signaling molecules in acute pancreatitis. ( Dong, DL; Iv, JC; Li, L; Sun, B; Wang, G; Wu, LF, 2014) |
| "Hydrogen sulfide (H(2)S) plays an important role in cardiovascular, central nervous, and gastrointestinal systems." | 2.46 | Hydrogen sulfide and substance P in inflammation. ( Bhatia, M, 2010) |
| "In the mice with cerulein-induced acute pancreatitis, the referred hyperalgesia was suppressed by NNC 55-0396 (NNC), a selective T-channel inhibitor; zinc chloride; or ascorbic acid, known to inhibit Ca(v)3." | 1.42 | Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis. ( Fujimura, M; Kawabata, A; Nishimura, S; Sekiguchi, F; Terada, Y; Tsubota, M, 2015) |
| "Hydrogen sulfide (H2S) has been reported to be involved in the signaling of the inflammatory response; however, there are differing views as to whether it is pro- or anti-inflammatory." | 1.39 | The effect of CSE gene deletion in caerulein-induced acute pancreatitis in the mouse. ( Ang, AD; Bhatia, M; Hegde, A; Ishii, I; Rivers-Auty, J, 2013) |
| "Acute pancreatitis was induced in mice by 10 hourly intraperitoneal injections of caerulein (50 (g/kg)." | 1.35 | Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis. ( Bhatia, M; Moochhala, SM; Ng, SW; Sidhapuriwala, JN; Tamizhselvi, R, 2008) |
| "Hydrogen sulfide (H2S) is a naturally occurring gas with potent vasodilator activity." | 1.33 | Role of hydrogen sulfide in acute pancreatitis and associated lung injury. ( Bhatia, M; Fu, D; Lau, HY; Moochhala, SM; Moore, PK; Wong, FL, 2005) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (13.04) | 29.6817 |
| 2010's | 16 (69.57) | 24.3611 |
| 2020's | 4 (17.39) | 2.80 |
| Authors | Studies |
|---|---|
| Kumar, A | 1 |
| Bhatia, M | 10 |
| Sekiguchi, F | 2 |
| Koike, N | 1 |
| Shimada, Y | 1 |
| Sugimoto, K | 1 |
| Masuda, H | 1 |
| Nakamura, T | 1 |
| Yamaguchi, H | 1 |
| Tanabe, G | 1 |
| Marumoto, S | 1 |
| Kasanami, Y | 1 |
| Tsubota, M | 2 |
| Ohkubo, T | 1 |
| Yoshida, S | 1 |
| Kawabata, A | 2 |
| Liu, Y | 2 |
| Wei, Z | 1 |
| Zhou, J | 1 |
| Ma, Z | 1 |
| Liao, R | 1 |
| Xue, L | 1 |
| Qiang, Z | 1 |
| Zhang, C | 1 |
| Bronowicka-Adamska, P | 1 |
| Hutsch, T | 1 |
| Gawryś-Kopczyńska, M | 1 |
| Maksymiuk, K | 1 |
| Wróbel, M | 1 |
| Sundar, V | 1 |
| Tamizhselvi, R | 5 |
| Lu, G | 1 |
| Wu, A | 1 |
| Wang, H | 1 |
| Manickam, V | 1 |
| Ang, AD | 2 |
| Rivers-Auty, J | 1 |
| Hegde, A | 2 |
| Ishii, I | 1 |
| Wang, G | 2 |
| Iv, JC | 1 |
| Wu, LF | 1 |
| Li, L | 2 |
| Dong, DL | 1 |
| Sun, B | 2 |
| Terada, Y | 1 |
| Fujimura, M | 1 |
| Nishimura, S | 1 |
| Thangaraj, KR | 2 |
| Priyadarshini, SJ | 1 |
| Qureshi, IN | 2 |
| Joseph, AJ | 2 |
| Balasubramanian, KA | 2 |
| Ramachandran, A | 2 |
| Ji, L | 1 |
| Qu, F | 1 |
| Zhang, G | 1 |
| Wang, Y | 1 |
| Bai, X | 1 |
| Pan, S | 1 |
| Xue, D | 1 |
| David, D | 1 |
| Kurien, RT | 1 |
| Chowdhury, SD | 1 |
| Goel, A | 1 |
| Dutta, AK | 1 |
| Simon, EG | 1 |
| Shrivastava, P | 1 |
| Koh, YH | 1 |
| Zhang, H | 1 |
| Sidhapuriwala, JN | 2 |
| Zhu, YZ | 1 |
| Zuka, M | 1 |
| Chinaka, S | 1 |
| Matsumoto, Y | 1 |
| Takayama, N | 1 |
| Hitomi, Y | 1 |
| Nakamura, H | 1 |
| Ohshima, T | 1 |
| Wong, FL | 1 |
| Fu, D | 1 |
| Lau, HY | 1 |
| Moochhala, SM | 2 |
| Moore, PK | 2 |
| Ng, SW | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Intermittent Fasting as a Primary Means for Improving Quality of Life for Acute and Chronic Pancreatitis[NCT04760847] | 64 participants (Anticipated) | Interventional | 2023-12-07 | Not yet recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
5 reviews available for hydrogen sulfide and Acute Edematous Pancreatitis
| Article | Year |
|---|---|
Role of Hydrogen Sulfide, Substance P and Adhesion Molecules in Acute Pancreatitis.
Topics: Cell Adhesion Molecules; Endothelial Cells; Humans; Hydrogen Sulfide; Inflammation; Integrins; Leuko | 2021 |
From nitric oxide to hyperbaric oxygen: invisible and subtle but nonnegligible gaseous signaling molecules in acute pancreatitis.
Topics: Acute Disease; Animals; Carbon Monoxide; Gases; Humans; Hydrogen; Hydrogen Sulfide; Hyperbaric Oxyge | 2014 |
H₂S and substance P in inflammation.
Topics: Acute Disease; Animals; Burns; Carrageenan; Ceruletide; Disease Models, Animal; Drug Synergism; Edem | 2015 |
H2S and Inflammation: An Overview.
Topics: Animals; Arthritis; Humans; Hydrogen Sulfide; Inflammation; Pancreatitis; Pulmonary Disease, Chronic | 2015 |
Hydrogen sulfide and substance P in inflammation.
Topics: Air Pollutants; Amino Acid Sequence; Animals; Humans; Hydrogen Sulfide; Inflammation; Inflammation M | 2010 |
18 other studies available for hydrogen sulfide and Acute Edematous Pancreatitis
| Article | Year |
|---|---|
A hydrolysate of poly-trans-[(2-carboxyethyl)germasesquioxane] (Ge-132) suppresses Ca
Topics: Animals; Calcium Channels, T-Type; Cystitis; HEK293 Cells; Humans; Hydrogen Sulfide; Mice; Pancreati | 2023 |
Simultaneous multi-signal quantification for highly precise serodiagnosis utilizing a rationally constructed platform.
Topics: Acute Disease; Animals; Electrochemical Techniques; Hydrogen Sulfide; Luminescent Measurements; Mice | 2019 |
Release of endogenous hydrogen sulfide in enteric nerve cells suppresses intestinal motility during severe acute pancreatitis.
Topics: Animals; Cell Movement; Chromones; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; Cytokines | 2020 |
Hydrogen sulfide formation in experimental model of acute pancreatitis.
Topics: Animals; Bile Ducts, Extrahepatic; Cystathionine; Cystathionine beta-Synthase; Cystathionine gamma-L | 2019 |
Inhibition of Rb phosphorylation leads to H
Topics: Animals; Antineoplastic Agents; Ceruletide; DNA; Gene Expression Regulation; Hydrogen Sulfide; Lung | 2020 |
[Change in endogeous hydrogen sulfide in patients with acute pancreatitis and its relationship to coagulation function].
Topics: Acute Disease; Antithrombin III; Blood Coagulation; Case-Control Studies; Factor VIII; Humans; Hydro | 2016 |
Menadione (vitamin K3) inhibits hydrogen sulfide and substance P via NF-кB pathway in caerulein-induced acute pancreatitis and associated lung injury in mice.
Topics: Amylases; Animals; Ceruletide; Gene Expression Regulation; Hydrogen Sulfide; Interleukin-1beta; Lung | 2019 |
The effect of CSE gene deletion in caerulein-induced acute pancreatitis in the mouse.
Topics: Animals; Ceruletide; Cystathionine gamma-Lyase; Gene Expression Regulation; Hydrogen Sulfide; Lung D | 2013 |
Roles of Cav3.2 and TRPA1 channels targeted by hydrogen sulfide in pancreatic nociceptive processing in mice with or without acute pancreatitis.
Topics: Analysis of Variance; Animals; Benzimidazoles; Calcium Channel Blockers; Calcium Channels, T-Type; C | 2015 |
Plasma Citrulline, Glycans, and Hydrogen Sulfide in Patients With Acute Pancreatitis: Possible Markers of Intestinal Damage.
Topics: Acute Disease; Adult; Biomarkers; Case-Control Studies; Citrulline; Female; Humans; Hydrogen Sulfide | 2016 |
Hydrogen sulphide exacerbates acute pancreatitis by over-activating autophagy via AMPK/mTOR pathway.
Topics: Alkynes; AMP-Activated Protein Kinases; Animals; Autophagy; Cell Line; Disease Progression; Glycine; | 2016 |
Plasma hydrogen sulphide does not predict severity of acute pancreatitis in humans.
Topics: Acute Disease; Biomarkers; Female; Humans; Hydrogen Sulfide; Male; Pancreatitis; Severity of Illness | 2016 |
Preprotachykinin-A gene deletion regulates hydrogen sulfide-induced toll-like receptor 4 signaling pathway in cerulein-treated pancreatic acinar cells.
Topics: Acute Disease; Animals; Base Sequence; Ceruletide; DNA Primers; Gene Deletion; Hydrogen Sulfide; Imm | 2011 |
Effects of S-propargyl-cysteine (SPRC) in caerulein-induced acute pancreatitis in mice.
Topics: Acute Disease; Amylases; Animals; Anti-Inflammatory Agents, Non-Steroidal; Ceruletide; Cysteine; Cyt | 2012 |
Fatal and non-fatal cases of lime sulfide exposure and pathogenetic mechanisms underlying pancreatic injury: Case reports with an animal experiment.
Topics: Adult; Aged; Air Pollutants; Amylases; Animals; Calcium Compounds; Esophagus; Female; Forensic Patho | 2012 |
Role of hydrogen sulfide in acute pancreatitis and associated lung injury.
Topics: Acute Disease; Alkynes; Amylases; Animals; Cystathionine beta-Synthase; Cystathionine gamma-Lyase; C | 2005 |
Pro-inflammatory effects of hydrogen sulphide on substance P in caerulein-induced acute pancreatitis.
Topics: Acute Disease; Alkynes; Animals; Ceruletide; Cystathionine beta-Synthase; Glycine; Hydrogen Sulfide; | 2008 |
Inhibition of hydrogen sulfide synthesis attenuates chemokine production and protects mice against acute pancreatitis and associated lung injury.
Topics: Acute Disease; Alkynes; Animals; Ceruletide; Chemokine CCL5; Chemokines; Cystathionine gamma-Lyase; | 2008 |