hydrazine has been researched along with Anterior Cerebral Circulation Infarction in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Liu, T; Shen, H; Xia, W; Yu, H; Zhang, T | 1 |
| Ahmad, AS; Candelario-Jalil, E; DeMars, KM; Doré, S; Frankowski, JC; Hawkins, KE; Leclerc, JL; Yang, C | 1 |
| Cheng, CY; Liu, T; Mruk, DD; Shao, J; Silvestrini, B; Wang, B; Xia, W; Xie, QR; Ying, W; Yu, H; Zhang, T | 1 |
| Hara, H; Morimoto, N; Nagai, H; Shimazawa, M; Yamashima, T | 1 |
4 other study(ies) available for hydrazine and Anterior Cerebral Circulation Infarction
| Article | Year |
|---|---|
Adjudin protects against cerebral ischemia reperfusion injury by inhibition of neuroinflammation and blood-brain barrier disruption.
Topics: Animals; Blood-Brain Barrier; Brain; Brain Infarction; CD11b Antigen; Cytokines; Disease Models, Animal; Enzyme-Linked Immunosorbent Assay; Gene Expression Regulation; Hydrazines; Indazoles; Matrix Metalloproteinase 9; Mice; Microglia; Neurologic Examination; Neuroprotective Agents; Nitric Oxide Synthase Type II; Occludin; Reperfusion Injury; Zonula Occludens-1 Protein | 2014 |
Detrimental role of the EP1 prostanoid receptor in blood-brain barrier damage following experimental ischemic stroke.
Topics: Animals; Blood-Brain Barrier; Brain Infarction; Disease Models, Animal; Endothelial Cells; Gene Expression; Gene Knockout Techniques; Hydrazines; Male; Matrix Metalloproteinase 3; Matrix Metalloproteinase 9; Mice; Mice, Knockout; Neurons; Neutrophil Infiltration; Oxazepines; Permeability; Proteolysis; Rats; Receptors, Prostaglandin E, EP1 Subtype; Stroke; Tight Junction Proteins | 2015 |
Adjudin attenuates lipopolysaccharide (LPS)- and ischemia-induced microglial activation.
Topics: Analysis of Variance; Animals; Anti-Inflammatory Agents; Brain Edema; Brain Infarction; CD11b Antigen; Cell Line, Transformed; Cell Survival; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Interactions; Hydrazines; Indazoles; Infarction, Middle Cerebral Artery; Lipopolysaccharides; Male; Mice; Mice, Inbred ICR; Microglia; Nervous System Diseases; NF-kappa B; Psychomotor Performance; Signal Transduction; Time Factors | 2013 |
Minocycline inhibits oxidative stress and decreases in vitro and in vivo ischemic neuronal damage.
Topics: Animals; Antioxidants; Benzimidazoles; Benzoxazoles; Biphenyl Compounds; Brain Edema; Brain Infarction; Cell Death; Cell Survival; Cells, Cultured; Cerebral Cortex; Chromans; Dose-Response Relationship, Drug; Drug Interactions; Embryo, Mammalian; Fluorescent Dyes; Glutamic Acid; Hydrazines; Infarction, Middle Cerebral Artery; Inhibitory Concentration 50; Ischemia; Lipid Peroxidation; Male; Mice; Minocycline; Neurons; Neuroprotective Agents; Oxidative Stress; Picrates; Quinolinium Compounds; Saponins; Tetrazolium Salts; Time Factors | 2005 |