homocitrulline has been researched along with Innate Inflammatory Response in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bergum, B; Bielecka, E; Binder, V; Blom, AM; Brun, JG; Dahl-Knudsen, A; Enghild, JJ; Hellvard, A; Jonsson, R; Koro, C; Mydel, P; Potempa, J; Scavenius, C | 1 |
| Barnard, J; DiDonato, JA; Hazen, SL; Hörkkö, S; Kummu, O; Nicholls, SJ; Reynolds, WF; Rodriguez, ER; Topol, EJ; Wang, Z | 1 |
| Ischiropoulos, H; Rader, DJ | 1 |
3 other study(ies) available for homocitrulline and Innate Inflammatory Response
| Article | Year |
|---|---|
Carbamylation of immunoglobulin abrogates activation of the classical complement pathway.
Topics: Adhesins, Bacterial; Antibodies, Monoclonal, Murine-Derived; Antibody-Dependent Cell Cytotoxicity; Arthritis, Rheumatoid; Citrulline; Complement Activation; Complement C1q; Complement Pathway, Classical; Cyanates; Cysteine Endopeptidases; Gingipain Cysteine Endopeptidases; Humans; Immunoglobulin G; Immunologic Factors; Inflammation; Lysine; Mass Spectrometry; Protein Binding; Protein Processing, Post-Translational; Rituximab | 2014 |
Protein carbamylation links inflammation, smoking, uremia and atherogenesis.
Topics: Animals; Atherosclerosis; Blood Proteins; Carbamates; Cholesterol; Citrulline; Clinical Trials as Topic; Coronary Artery Disease; Cyanates; Dose-Response Relationship, Drug; Female; Foam Cells; Humans; Inflammation; Jurkat Cells; Lipoproteins, LDL; Macrophages; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Models, Biological; Oxidation-Reduction; Peroxidase; Predictive Value of Tests; Protein Processing, Post-Translational; Receptors, LDL; Scavenger Receptors, Class A; Smoking; Uremia | 2007 |
'Multipurpose oxidase' in atherogenesis.
Topics: Animals; Apoptosis; Atherosclerosis; Carbamates; Cardiovascular Diseases; Cell Proliferation; Citrulline; Endothelial Cells; Humans; Inflammation; Lipoproteins, HDL; Lipoproteins, LDL; Lysine; Macrophages; Mice; Models, Biological; Muscle, Smooth; Oxidation-Reduction; Peroxidase; Scavenger Receptors, Class A; Smoking; Substrate Specificity; Thiocyanates | 2007 |