histamine has been researched along with Atherosclerosis in 21 studies
Atherosclerosis: A thickening and loss of elasticity of the walls of ARTERIES that occurs with formation of ATHEROSCLEROTIC PLAQUES within the ARTERIAL INTIMA.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Previous studies have reported a relationship between CCL22 and atherosclerosis and the role of histamine in this pathway." | 7.81 | CCL22/Macrophage-derived Chemokine Expression in Apolipoprotein E-deficient Mice and Effects of Histamine in the Setting of Atherosclerosis. ( Guo, X; Harada, M; Kimura, S; Nabeshima, A; Noguchi, H; Sasaguri, Y; Wang, KY; Watanabe, T; Yamada, S, 2015) |
| " In this study, we investigate the relationship between monocytes/macrophages and histamine in atherosclerosis and discover that histamine levels regulate various immunologically important molecules and influences atherosclerotic progression." | 7.78 | Expression of macrophage-derived chemokine (CCL22) in atherosclerosis and regulation by histamine via the H2 receptor. ( Guo, X; Kimura, S; Sasaguri, Y; Shimajiri, S; Tanimoto, A; Tasaki, T; Wang, KY; Yamada, S, 2012) |
| "Histamine and histamine receptors are found in atherosclerotic lesions, and their signaling and subsequent proatherogenic or proinflammatory gene expression are involved in atherogenesis." | 7.77 | Histamine deficiency decreases atherosclerosis and inflammatory response in apolipoprotein E knockout mice independently of serum cholesterol level. ( Ding, Y; Guo, X; Hirano, K; Kato, S; Kohno, K; Murata, Y; Ohtsu, H; Sasaguri, Y; Shimajiri, S; Tanimoto, A; Tsutsui, M; Wang, KY; Watanabe, T; Yamada, S, 2011) |
| "Histamine is a low-molecular-weight amine, synthesized from l-histidine by histidine decarboxylase." | 6.43 | Histamine network in atherosclerosis. ( Ohtsu, H; Sasaguri, Y; Tanimoto, A, 2006) |
| "We found that pronounced hyperlipidemia-induced atherosclerotic progression occurred in HH1R/apoE-DKO mice, but in HH2R/apoE-DKO mice less atherosclerosis, despite pro-atherogenic serum cholesterol levels compared with apoE-KO mice." | 5.42 | Novel function of histamine signaling in hyperlipidemia-induced atherosclerosis: Histamine H1 receptors protect and H2 receptors accelerate atherosclerosis. ( Sasaguri, Y; Tanimoto, A; Wang, KY; Yamada, S, 2015) |
| "Modulation of histamine and Stat6-signaling may represent an attractive therapeutic strategy for the prevention or treatment of atherosclerosis." | 3.85 | Histamine promotes the differentiation of macrophages from CD11b ( Chen, R; Cheng, D; Ding, S; Ge, J; Huang, Z; Shi, H; Tan, H; Wang, TC; Xu, L; Yang, X; Zhang, W; Zou, Y, 2017) |
| " Previous studies have reported a relationship between CCL22 and atherosclerosis and the role of histamine in this pathway." | 3.81 | CCL22/Macrophage-derived Chemokine Expression in Apolipoprotein E-deficient Mice and Effects of Histamine in the Setting of Atherosclerosis. ( Guo, X; Harada, M; Kimura, S; Nabeshima, A; Noguchi, H; Sasaguri, Y; Wang, KY; Watanabe, T; Yamada, S, 2015) |
| "Histamine, a potent inflammatory mediator, has been known to cause the pathogenesis of atherosclerosis." | 3.79 | Down-regulation of histamine-induced endothelial cell activation as potential anti-atherosclerotic activity of peptides from Spirulina maxima. ( Kim, SK; Vo, TS, 2013) |
| " Then the rabbit atherosclerotic plaque rupture and thrombosis were triggered by injection of viper venom and histamine." | 3.79 | Pravastatin inhibits plaque rupture and subsequent thrombus formation in atherosclerotic rabbits with hyperlipidemia. ( Huang, C; Huang, Z; Jiang, H; Wu, G; Xie, Q; Xu, L, 2013) |
| " In this study, we investigate the relationship between monocytes/macrophages and histamine in atherosclerosis and discover that histamine levels regulate various immunologically important molecules and influences atherosclerotic progression." | 3.78 | Expression of macrophage-derived chemokine (CCL22) in atherosclerosis and regulation by histamine via the H2 receptor. ( Guo, X; Kimura, S; Sasaguri, Y; Shimajiri, S; Tanimoto, A; Tasaki, T; Wang, KY; Yamada, S, 2012) |
| "Histamine and histamine receptors are found in atherosclerotic lesions, and their signaling and subsequent proatherogenic or proinflammatory gene expression are involved in atherogenesis." | 3.77 | Histamine deficiency decreases atherosclerosis and inflammatory response in apolipoprotein E knockout mice independently of serum cholesterol level. ( Ding, Y; Guo, X; Hirano, K; Kato, S; Kohno, K; Murata, Y; Ohtsu, H; Sasaguri, Y; Shimajiri, S; Tanimoto, A; Tsutsui, M; Wang, KY; Watanabe, T; Yamada, S, 2011) |
| "Histamine, a potent inflammatory mediator, has multiple effects on the pathogenesis of atherosclerosis." | 3.74 | Histamine induces Egr-1 expression in human aortic endothelial cells via the H1 receptor-mediated protein kinase Cdelta-dependent ERK activation pathway. ( Cui, MZ; Hao, F; Tan, M; Xu, X, 2008) |
| "In the allergic bronchial asthma model using HDC-KO mice it was found that histamine positively controls eosinophilia, but not bronchial hypersensitivity." | 2.46 | Histamine synthesis and lessons learned from histidine decarboxylase deficient mice. ( Ohtsu, H, 2010) |
| "Histamine also plays a role in inducing atherosclerosis in the mouse model." | 2.44 | Progress in allergy signal research on mast cells: the role of histamine in immunological and cardiovascular disease and the transporting system of histamine in the cell. ( Ohtsu, H, 2008) |
| "Histamine is a low-molecular-weight amine, synthesized from l-histidine by histidine decarboxylase." | 2.43 | Histamine network in atherosclerosis. ( Ohtsu, H; Sasaguri, Y; Tanimoto, A, 2006) |
| "We found that pronounced hyperlipidemia-induced atherosclerotic progression occurred in HH1R/apoE-DKO mice, but in HH2R/apoE-DKO mice less atherosclerosis, despite pro-atherogenic serum cholesterol levels compared with apoE-KO mice." | 1.42 | Novel function of histamine signaling in hyperlipidemia-induced atherosclerosis: Histamine H1 receptors protect and H2 receptors accelerate atherosclerosis. ( Sasaguri, Y; Tanimoto, A; Wang, KY; Yamada, S, 2015) |
| "No thrombus was found in the control group." | 1.38 | In vivo MR imaging of plaque disruption and thrombus formation in an atherosclerotic rabbit model. ( Feng, T; Ma, X; Shang, J; Zeng, C; Zhang, Z; Zhao, L; Zhao, Q, 2012) |
| "Histamine is known to increase vascular permeability." | 1.36 | Histamine H1 receptor promotes atherosclerotic lesion formation by increasing vascular permeability for low-density lipoproteins. ( Akhmedov, A; Becher, B; Borén, J; Hofmann, J; Johansen, P; Lüscher, TF; Mocharla, P; Rohrer, L; Rozenberg, I; Sluka, SH; Soliz, J; Steffel, J; Tanner, FC; Watanabe, T, 2010) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (9.52) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (19.05) | 29.6817 |
| 2010's | 14 (66.67) | 24.3611 |
| 2020's | 1 (4.76) | 2.80 |
| Authors | Studies |
|---|---|
| Ma, Y | 2 |
| Li, S | 3 |
| Yang, H | 1 |
| Zhang, Y | 8 |
| Li, H | 6 |
| Zhou, L | 3 |
| Lin, J | 1 |
| Chen, Y | 1 |
| Hou, Y | 1 |
| Zhang, X | 7 |
| Liu, T | 3 |
| Zhou, X | 2 |
| Wang, Y | 8 |
| Halim, AA | 1 |
| Alsayed, B | 1 |
| Embarak, S | 1 |
| Yaseen, T | 1 |
| Dabbous, S | 1 |
| Fontaine, O | 1 |
| Dueluzeau, R | 1 |
| Raibaud, P | 1 |
| Chabanet, C | 1 |
| Popoff, MR | 1 |
| Badoual, J | 1 |
| Gabilan, JC | 1 |
| Andremont, A | 1 |
| Gómez, L | 1 |
| Andrés, S | 1 |
| Sánchez, J | 1 |
| Alonso, JM | 1 |
| Rey, J | 1 |
| López, F | 1 |
| Jiménez, A | 1 |
| Yan, Z | 1 |
| Zhao, Y | 4 |
| Wang, J | 6 |
| Huang, L | 2 |
| Hu, K | 1 |
| Liu, H | 4 |
| Wang, H | 3 |
| Guo, Z | 1 |
| Song, Y | 1 |
| Huang, H | 4 |
| Yang, R | 1 |
| Owen, TW | 1 |
| Al-Kaysi, RO | 1 |
| Bardeen, CJ | 1 |
| Cheng, Q | 1 |
| Wu, S | 1 |
| Cheng, T | 1 |
| Wang, B | 4 |
| Zhang, Q | 4 |
| Wu, X | 2 |
| Yao, Y | 3 |
| Ochiai, T | 1 |
| Ishiguro, H | 2 |
| Nakano, R | 2 |
| Kubota, Y | 2 |
| Hara, M | 1 |
| Sunada, K | 1 |
| Hashimoto, K | 1 |
| Kajioka, J | 1 |
| Fujishima, A | 1 |
| Jiao, J | 3 |
| Gai, QY | 3 |
| Wang, W | 2 |
| Zang, YP | 2 |
| Niu, LL | 2 |
| Fu, YJ | 3 |
| Wang, X | 4 |
| Yao, LP | 1 |
| Qin, QP | 1 |
| Wang, ZY | 1 |
| Liu, J | 4 |
| Aleksic Sabo, V | 1 |
| Knezevic, P | 1 |
| Borges-Argáez, R | 1 |
| Chan-Balan, R | 1 |
| Cetina-Montejo, L | 1 |
| Ayora-Talavera, G | 1 |
| Sansores-Peraza, P | 1 |
| Gómez-Carballo, J | 1 |
| Cáceres-Farfán, M | 1 |
| Jang, J | 1 |
| Akin, D | 1 |
| Bashir, R | 1 |
| Yu, Z | 1 |
| Zhu, J | 2 |
| Jiang, H | 2 |
| He, C | 2 |
| Xiao, Z | 1 |
| Xu, J | 2 |
| Sun, Q | 1 |
| Han, D | 1 |
| Lei, H | 1 |
| Zhao, K | 2 |
| Zhu, L | 1 |
| Li, X | 4 |
| Fu, H | 2 |
| Wilson, BK | 1 |
| Step, DL | 1 |
| Maxwell, CL | 1 |
| Gifford, CA | 1 |
| Richards, CJ | 1 |
| Krehbiel, CR | 1 |
| Warner, JM | 1 |
| Doerr, AJ | 1 |
| Erickson, GE | 1 |
| Guretzky, JA | 1 |
| Rasby, RJ | 1 |
| Watson, AK | 1 |
| Klopfenstein, TJ | 1 |
| Sun, Y | 4 |
| Liu, Z | 3 |
| Pham, TD | 1 |
| Lee, BK | 1 |
| Yang, FC | 1 |
| Wu, KH | 1 |
| Lin, WP | 1 |
| Hu, MK | 1 |
| Lin, L | 3 |
| Shao, J | 1 |
| Sun, M | 1 |
| Xu, G | 1 |
| Xu, N | 1 |
| Wang, R | 1 |
| Liu, S | 1 |
| He, H | 1 |
| Dong, X | 2 |
| Yang, M | 2 |
| Yang, Q | 1 |
| Duan, S | 1 |
| Yu, Y | 2 |
| Han, J | 2 |
| Zhang, C | 4 |
| Chen, L | 2 |
| Yang, X | 2 |
| Li, W | 3 |
| Wang, T | 2 |
| Campbell, DA | 1 |
| Gao, K | 1 |
| Zager, RA | 1 |
| Johnson, ACM | 1 |
| Guillem, A | 1 |
| Keyser, J | 1 |
| Singh, B | 1 |
| Steubl, D | 1 |
| Schneider, MP | 1 |
| Meiselbach, H | 1 |
| Nadal, J | 1 |
| Schmid, MC | 1 |
| Saritas, T | 1 |
| Krane, V | 1 |
| Sommerer, C | 1 |
| Baid-Agrawal, S | 1 |
| Voelkl, J | 1 |
| Kotsis, F | 1 |
| Köttgen, A | 1 |
| Eckardt, KU | 1 |
| Scherberich, JE | 1 |
| Yao, L | 2 |
| Sun, L | 3 |
| Zhu, Z | 1 |
| Naren, N | 1 |
| Zhang, XX | 2 |
| Gentile, GL | 1 |
| Rupert, AS | 1 |
| Carrasco, LI | 1 |
| Garcia, EM | 1 |
| Kumar, NG | 1 |
| Walsh, SW | 1 |
| Jefferson, KK | 1 |
| Guest, RL | 1 |
| Samé Guerra, D | 1 |
| Wissler, M | 1 |
| Grimm, J | 1 |
| Silhavy, TJ | 1 |
| Lee, JH | 2 |
| Yoo, JS | 1 |
| Kim, Y | 1 |
| Kim, JS | 2 |
| Lee, EJ | 1 |
| Roe, JH | 1 |
| Delorme, M | 1 |
| Bouchard, PA | 1 |
| Simon, M | 1 |
| Simard, S | 1 |
| Lellouche, F | 1 |
| D'Urzo, KA | 1 |
| Mok, F | 1 |
| D'Urzo, AD | 1 |
| Koneru, B | 1 |
| Lopez, G | 1 |
| Farooqi, A | 1 |
| Conkrite, KL | 1 |
| Nguyen, TH | 1 |
| Macha, SJ | 1 |
| Modi, A | 1 |
| Rokita, JL | 1 |
| Urias, E | 1 |
| Hindle, A | 1 |
| Davidson, H | 1 |
| Mccoy, K | 1 |
| Nance, J | 1 |
| Yazdani, V | 1 |
| Irwin, MS | 1 |
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| Wheeler, DA | 1 |
| Maris, JM | 1 |
| Diskin, SJ | 1 |
| Reynolds, CP | 1 |
| Abhilash, L | 1 |
| Kalliyil, A | 1 |
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| Hartley, AM | 2 |
| Meunier, B | 2 |
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| Xu, JY | 1 |
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| Haraux, F | 1 |
| Rich, PR | 1 |
| Kamalanathan, M | 1 |
| Doyle, SM | 1 |
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| Achberger, AM | 1 |
| Wade, TL | 1 |
| Schwehr, K | 1 |
| Santschi, PH | 1 |
| Sylvan, JB | 1 |
| Quigg, A | 1 |
| Leong, W | 1 |
| Xu, W | 2 |
| Gao, S | 1 |
| Zhai, X | 1 |
| Wang, C | 2 |
| Gilson, E | 1 |
| Ye, J | 1 |
| Lu, Y | 1 |
| Yan, R | 1 |
| Hu, Z | 1 |
| You, Q | 1 |
| Cai, Q | 1 |
| Yang, D | 1 |
| Gu, S | 1 |
| Dai, H | 1 |
| Zhao, X | 1 |
| Gui, C | 1 |
| Gui, J | 1 |
| Wu, PK | 1 |
| Hong, SK | 1 |
| Starenki, D | 1 |
| Oshima, K | 1 |
| Shao, H | 1 |
| Gestwicki, JE | 1 |
| Tsai, S | 1 |
| Park, JI | 1 |
| Zhao, R | 1 |
| Gu, Z | 1 |
| Dong, C | 2 |
| Guo, G | 1 |
| Li, L | 4 |
| Barrett, HE | 1 |
| Meester, EJ | 1 |
| van Gaalen, K | 1 |
| van der Heiden, K | 1 |
| Krenning, BJ | 1 |
| Beekman, FJ | 1 |
| de Blois, E | 1 |
| de Swart, J | 1 |
| Verhagen, HJ | 1 |
| Maina, T | 1 |
| Nock, BA | 1 |
| Norenberg, JP | 1 |
| de Jong, M | 1 |
| Gijsen, FJH | 1 |
| Bernsen, MR | 1 |
| Martínez-Milla, J | 1 |
| Galán-Arriola, C | 1 |
| Carnero, M | 1 |
| Cobiella, J | 1 |
| Pérez-Camargo, D | 1 |
| Bautista-Hernández, V | 1 |
| Rigol, M | 1 |
| Solanes, N | 1 |
| Villena-Gutierrez, R | 1 |
| Lobo, M | 1 |
| Mateo, J | 1 |
| Vilchez-Tschischke, JP | 1 |
| Salinas, B | 1 |
| Cussó, L | 1 |
| López, GJ | 1 |
| Fuster, V | 1 |
| Desco, M | 1 |
| Sanchez-González, J | 1 |
| Ibanez, B | 1 |
| van den Berg, P | 1 |
| Schweitzer, DH | 1 |
| van Haard, PMM | 1 |
| Geusens, PP | 1 |
| van den Bergh, JP | 1 |
| Zhu, X | 1 |
| Huang, X | 2 |
| Xu, H | 2 |
| Yang, G | 2 |
| Lin, Z | 1 |
| Salem, HF | 1 |
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| Hayton, TW | 1 |
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| Shi, J | 2 |
| Carreno, J | 1 |
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| Hautajärvi, H | 1 |
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| Känsäkoski, P | 1 |
| Rysä, J | 1 |
| Hakkola, J | 1 |
| Hukkanen, J | 1 |
| Zhu, N | 1 |
| Li, Y | 4 |
| Du, Q | 1 |
| Hao, P | 1 |
| Cao, X | 1 |
| Li, CX | 1 |
| Zhao, S | 1 |
| Luo, XM | 1 |
| Feng, JX | 1 |
| Gonzalez-Cotto, M | 1 |
| Guo, L | 1 |
| Karwan, M | 1 |
| Sen, SK | 1 |
| Barb, J | 1 |
| Collado, CJ | 1 |
| Elloumi, F | 1 |
| Palmieri, EM | 1 |
| Boelte, K | 1 |
| Kolodgie, FD | 1 |
| Finn, AV | 1 |
| Biesecker, LG | 1 |
| McVicar, DW | 1 |
| Qu, F | 1 |
| Deng, Z | 1 |
| Xie, Y | 2 |
| Tang, J | 3 |
| Chen, Z | 2 |
| Luo, W | 1 |
| Xiong, D | 1 |
| Zhao, D | 1 |
| Fang, J | 1 |
| Zhou, Z | 1 |
| Niu, PP | 1 |
| Song, B | 1 |
| Xu, YM | 1 |
| Zhang, Z | 3 |
| Qiu, N | 1 |
| Yin, J | 1 |
| Zhang, J | 3 |
| Guo, W | 1 |
| Liu, M | 2 |
| Chen, D | 5 |
| Luo, K | 1 |
| He, Z | 2 |
| Zheng, G | 1 |
| Xu, F | 1 |
| Sun, W | 1 |
| Yin, F | 1 |
| van Hest, JCM | 1 |
| Du, L | 2 |
| Shi, X | 1 |
| Kang, S | 1 |
| Duan, W | 1 |
| Zhang, S | 2 |
| Feng, J | 2 |
| Qi, N | 1 |
| Shen, G | 1 |
| Ren, H | 1 |
| Shang, Q | 1 |
| Zhao, W | 2 |
| Yang, Z | 2 |
| Jiang, X | 2 |
| Alame, M | 1 |
| Cornillot, E | 1 |
| Cacheux, V | 1 |
| Tosato, G | 1 |
| Four, M | 1 |
| De Oliveira, L | 1 |
| Gofflot, S | 1 |
| Delvenne, P | 1 |
| Turtoi, E | 1 |
| Cabello-Aguilar, S | 1 |
| Nishiyama, M | 1 |
| Turtoi, A | 1 |
| Costes-Martineau, V | 1 |
| Colinge, J | 1 |
| Guo, Q | 1 |
| Quan, M | 1 |
| Dong, J | 1 |
| Bai, J | 1 |
| Han, R | 1 |
| Cai, Y | 1 |
| Lv, YQ | 1 |
| Chen, Q | 1 |
| Lyu, HD | 1 |
| Deng, L | 1 |
| Zhou, D | 1 |
| Xiao, X | 1 |
| De Langhe, S | 1 |
| Billadeau, DD | 1 |
| Lou, Z | 1 |
| Zhang, JS | 1 |
| Xue, Z | 1 |
| Shen, XD | 1 |
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| Kohno, K | 1 |
| Zhao, Q | 1 |
| Shang, J | 1 |
| Feng, T | 1 |
| Zeng, C | 1 |
| Bai, W | 1 |
| Zheng, X | 1 |
| Tasaki, T | 1 |
| Xie, Q | 1 |
| Huang, C | 1 |
| JANIAKOWA, A | 1 |
| ORZECHOWSKA, K | 1 |
| WAHL, M | 1 |
| Yan, F | 1 |
5 reviews available for histamine and Atherosclerosis
| Article | Year |
|---|---|
Topics: A549 Cells; Acetylmuramyl-Alanyl-Isoglutamine; Acinetobacter baumannii; Acute Lung Injury; Adaptor P | 2016 |
Topics: A549 Cells; Acetylmuramyl-Alanyl-Isoglutamine; Acinetobacter baumannii; Acute Lung Injury; Adaptor P | 2016 |
Topics: A549 Cells; Acetylmuramyl-Alanyl-Isoglutamine; Acinetobacter baumannii; Acute Lung Injury; Adaptor P | 2016 |
Topics: A549 Cells; Acetylmuramyl-Alanyl-Isoglutamine; Acinetobacter baumannii; Acute Lung Injury; Adaptor P | 2016 |
Histamine synthesis and lessons learned from histidine decarboxylase deficient mice.
Topics: Anaphylaxis; Animals; Asthma; Atherosclerosis; Crohn Disease; Epigenesis, Genetic; Gene Expression R | 2010 |
Pathophysiologic role of histamine: evidence clarified by histidine decarboxylase gene knockout mice.
Topics: Anaphylaxis; Animals; Asthma; Atherosclerosis; Dermatitis, Allergic Contact; Histamine; Histidine De | 2012 |
Histamine network in atherosclerosis.
Topics: Animals; Atherosclerosis; Histamine; Histamine Antagonists; Histidine Decarboxylase; Humans; Mice; R | 2006 |
Progress in allergy signal research on mast cells: the role of histamine in immunological and cardiovascular disease and the transporting system of histamine in the cell.
Topics: Anaphylaxis; Animals; Asthma; Atherosclerosis; Biological Transport; Cardiovascular Diseases; Enceph | 2008 |
1 trial available for histamine and Atherosclerosis
16 other studies available for histamine and Atherosclerosis
| Article | Year |
|---|---|
Acid suppressants use and risk of atherosclerotic cardiovascular disease in middle-aged and older adults.
Topics: Aged; Antihypertensive Agents; Atherosclerosis; Cardiovascular Diseases; Histamine; Humans; Lansopra | 2022 |
Histamine promotes the differentiation of macrophages from CD11b
Topics: Animals; Atherosclerosis; CD11b Antigen; Cell Differentiation; Dendritic Cells; Female; Flow Cytomet | 2017 |
Down-regulation of histamine-induced endothelial cell activation as potential anti-atherosclerotic activity of peptides from Spirulina maxima.
Topics: Atherosclerosis; Cell Adhesion; Cell Line; Chemokine CCL2; Down-Regulation; Early Growth Response Pr | 2013 |
CCL22/Macrophage-derived Chemokine Expression in Apolipoprotein E-deficient Mice and Effects of Histamine in the Setting of Atherosclerosis.
Topics: Animals; Apolipoproteins E; Atherosclerosis; Cells, Cultured; Chemokine CCL22; Disease Models, Anima | 2015 |
Novel function of histamine signaling in hyperlipidemia-induced atherosclerosis: Histamine H1 receptors protect and H2 receptors accelerate atherosclerosis.
Topics: Animals; Atherosclerosis; Blotting, Western; Chromatography, High Pressure Liquid; Disease Models, A | 2015 |
Oxidized low-density lipoprotein contributes to atherogenesis via co-activation of macrophages and mast cells.
Topics: Atherosclerosis; Cell Adhesion; Cell Adhesion Molecules; Cell Line; Cell Membrane; Cells, Cultured; | 2015 |
Histamine induces Egr-1 expression in human aortic endothelial cells via the H1 receptor-mediated protein kinase Cdelta-dependent ERK activation pathway.
Topics: Aorta; Atherosclerosis; Cells, Cultured; CREB-Binding Protein; Early Growth Response Protein 1; Endo | 2008 |
Histamine H1 receptor promotes atherosclerotic lesion formation by increasing vascular permeability for low-density lipoproteins.
Topics: Animals; Aortic Diseases; Aortitis; Apolipoproteins E; Atherosclerosis; Bone Marrow Transplantation; | 2010 |
Histamine deficiency decreases atherosclerosis and inflammatory response in apolipoprotein E knockout mice independently of serum cholesterol level.
Topics: Animals; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Blotting, Western; Cholesterol; Choles | 2011 |
In vivo MR imaging of plaque disruption and thrombus formation in an atherosclerotic rabbit model.
Topics: Animals; Aorta, Abdominal; Aortic Diseases; Atherosclerosis; Catheterization; Cholesterol, Dietary; | 2012 |
Prostaglandin E1 dose-dependently promotes stability of atherosclerotic plaque in a rabbit model.
Topics: Alprostadil; Angioplasty, Balloon; Animals; Anti-Inflammatory Agents; Aorta, Abdominal; Aortic Disea | 2012 |
Expression of macrophage-derived chemokine (CCL22) in atherosclerosis and regulation by histamine via the H2 receptor.
Topics: Aorta; Atherosclerosis; Cell Line, Tumor; Cell Movement; Chemokine CCL22; Chemokines; Dendritic Cell | 2012 |
Pravastatin inhibits plaque rupture and subsequent thrombus formation in atherosclerotic rabbits with hyperlipidemia.
Topics: 6-Ketoprostaglandin F1 alpha; Animals; Anticholesteremic Agents; Aorta; Aorta, Abdominal; Atheroscle | 2013 |
[Histamine level in atherosclerosis].
Topics: Arteriosclerosis; Atherosclerosis; Histamine; Humans | 1960 |
[Anomalies of amine metabolism and atherosclerosis (possibilities of their prevention)].
Topics: Arteriosclerosis; Atherosclerosis; Cholesterol; Histamine; Humans; Lipid Metabolism; Proteins; Serot | 1963 |
Aqueous extract of rhubarb stabilizes vulnerable atherosclerotic plaques due to depression of inflammation and lipid accumulation.
Topics: Animals; Anticholesteremic Agents; Aorta, Abdominal; Aortic Rupture; Atherosclerosis; Cholesterol, H | 2008 |