Page last updated: 2024-09-02

higenamine and Injury, Myocardial Reperfusion

higenamine has been researched along with Injury, Myocardial Reperfusion in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (66.67)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dong, YR; Wu, MP; Xiong, J; Yan, C; Zhang, YS; Zhou, QM1
Cao, J; Min, S; Zhang, GX; Zhang, YJ; Zheng, CD1
Chang, KC; Kang, YJ; Kim, HJ; Lee, JH; Lee, YS; Park, MK; Seo, HG; Yun-Choi, HS1

Other Studies

3 other study(ies) available for higenamine and Injury, Myocardial Reperfusion

ArticleYear
Higenamine protects ischemia/reperfusion induced cardiac injury and myocyte apoptosis through activation of β2-AR/PI3K/AKT signaling pathway.
    Pharmacological research, 2016, Volume: 104

    Topics: Alkaloids; Animals; Apoptosis; Cardiotonic Agents; Caspase 3; Cells, Cultured; Hydrogen Peroxide; Male; Mice, Inbred C57BL; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Rats, Sprague-Dawley; Receptors, Adrenergic, beta-2; Signal Transduction; Tetrahydroisoquinolines

2016
Protective effect of Shenfu injection on myocardial mitochondria injured by ischemia-reperfusion in rabbits.
    Chinese medical journal, 2005, Mar-20, Volume: 118, Issue:6

    Topics: Alkaloids; Animals; Calcium; Creatine Kinase; Creatine Kinase, MB Form; Drugs, Chinese Herbal; Female; Ginsenosides; Injections; Isoenzymes; Male; Malondialdehyde; Mitochondria, Heart; Myocardial Reperfusion Injury; Myocardium; Protective Agents; Rabbits; Tetrahydroisoquinolines

2005
Higenamine reduces apoptotic cell death by induction of heme oxygenase-1 in rat myocardial ischemia-reperfusion injury.
    Apoptosis : an international journal on programmed cell death, 2006, Volume: 11, Issue:7

    Topics: Alkaloids; Animals; Antioxidants; Apoptosis; bcl-2-Associated X Protein; Blotting, Western; Cardiotonic Agents; Caspase 3; Caspases; Cytochromes c; Cytochromes c2; Enzyme Inhibitors; Glutathione; Heme Oxygenase (Decyclizing); In Situ Nick-End Labeling; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Oxidation-Reduction; Peroxynitrous Acid; Proto-Oncogene Proteins c-bcl-2; Protoporphyrins; Rats; Rats, Sprague-Dawley; Tetrahydroisoquinolines

2006