Compounds > heme
Page last updated: 2024-08-23

heme and Gasser Syndrome

heme has been researched along with Gasser Syndrome in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (25.00)24.3611
2020's3 (75.00)2.80

Authors

AuthorsStudies
Amann, K; Bauer, M; Coldewey, SM; Daniel, C; Gunzer, F; Heinemann, SH; Krieg, N; Kröller, S; Mestekemper, AN; Pirschel, W; Tolosano, E; Wissuwa, B1
van de Kar, NCAJ; van den Heuvel, LP; Wagener, FADTG1
de Wijs, S; van de Kar, NCAJ; van den Heuvel, LP; van der Velden, T; Veissi, ST; Volokhina, EB; Wagener, FADTG; Wijnsma, KL1
Dimitrov, JD; Fremeaux-Bacchi, V; Frimat, M; Halbwachs-Mecarelli, L; Poitou, C; Roumenina, LT; Tabarin, F1

Other Studies

4 other study(ies) available for heme and Gasser Syndrome

ArticleYear
Divergent roles of haptoglobin and hemopexin deficiency for disease progression of Shiga-toxin-induced hemolytic-uremic syndrome in mice.
    Kidney international, 2022, Volume: 101, Issue:6

    Topics: Animals; Disease Progression; Escherichia coli Infections; Haptoglobins; Heme; Hemoglobins; Hemolysis; Hemolytic-Uremic Syndrome; Hemopexin; Mice; Shiga Toxin; Shiga-Toxigenic Escherichia coli; Thrombotic Microangiopathies

2022
Protective mechanisms harnessing against injurious heme and preventing kidney damage in STEC-HUS: toward new therapies?
    Kidney international, 2022, Volume: 101, Issue:6

    Topics: Escherichia coli Infections; Heme; Hemolytic-Uremic Syndrome; Humans; Kidney; Shiga Toxin; Shiga-Toxigenic Escherichia coli

2022
Heme as Possible Contributing Factor in the Evolvement of Shiga-Toxin
    Frontiers in immunology, 2020, Volume: 11

    Topics: Apoptosis; Biomarkers; Child; Child, Preschool; Disease Susceptibility; Endothelial Cells; Female; Heme; Heme Oxygenase-1; Hemolytic-Uremic Syndrome; Humans; Infant; Male; Oxidation-Reduction; Phenotype; Protein Transport; Reactive Oxygen Species; Shiga-Toxigenic Escherichia coli; Stress, Physiological; Thromboplastin

2020
Complement activation by heme as a secondary hit for atypical hemolytic uremic syndrome.
    Blood, 2013, Jul-11, Volume: 122, Issue:2

    Topics: Atypical Hemolytic Uremic Syndrome; Cell Line; Cell Membrane; Complement Activation; Complement C3; Complement C3-C5 Convertases; Complement C3b; Complement Pathway, Alternative; Endothelial Cells; Heme; Hemolytic-Uremic Syndrome; Humans; Mutation; P-Selectin; Protein Binding

2013