heme has been researched along with Atypical Hemolytic-Uremic Syndrome in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Allain, F; Boulanger, E; Denys, A; Frimat, M; Gibier, JB; Glowacki, F; Gnemmi, V; Grolaux, G; Grunenwald, A; Howsam, M; Laboux, T; Lancel, S; Lebas, C; Lopez, B; Maanaoui, M; Provôt, F; Roumenina, L | 1 |
| Boulanger, E; Delguste, F; Dimitrov, JD; Fremeaux-Bacchi, V; Frimat, M; Gnemmi, V; Grunenwald, A; Hazzan, M; Leon, J; Mathieson, PW; May, O; Merle, NS; Paule, R; Payet, C; Robe-Rybkine, T; Roumenina, LT; Satchell, SC | 1 |
| Dimitrov, JD; Fremeaux-Bacchi, V; Frimat, M; Halbwachs-Mecarelli, L; Poitou, C; Roumenina, LT; Tabarin, F | 1 |
3 other study(ies) available for heme and Atypical Hemolytic-Uremic Syndrome
| Article | Year |
|---|---|
Hemolysis is associated with altered heparan sulfate of the endothelial glycocalyx and with local complement activation in thrombotic microangiopathies.
Topics: Atypical Hemolytic Uremic Syndrome; Complement Activation; Complement System Proteins; Endothelial Cells; Glycocalyx; Heme; Hemolysis; Heparitin Sulfate; Humans; Kidney Diseases; Retrospective Studies; Thrombotic Microangiopathies | 2023 |
Heme Drives Susceptibility of Glomerular Endothelium to Complement Overactivation Due to Inefficient Upregulation of Heme Oxygenase-1.
Topics: Animals; Atypical Hemolytic Uremic Syndrome; Biopsy; Complement Activation; Complement C3; Disease Models, Animal; Disease Susceptibility; Endothelial Cells; Endothelium, Vascular; Female; Heme; Heme Oxygenase-1; Hemolysis; Human Umbilical Vein Endothelial Cells; Humans; Kidney Glomerulus; Kruppel-Like Factor 4; Membrane Proteins; Mice; Mice, Inbred C57BL; Microvessels; Primary Cell Culture; Thrombomodulin; Up-Regulation | 2018 |
Complement activation by heme as a secondary hit for atypical hemolytic uremic syndrome.
Topics: Atypical Hemolytic Uremic Syndrome; Cell Line; Cell Membrane; Complement Activation; Complement C3; Complement C3-C5 Convertases; Complement C3b; Complement Pathway, Alternative; Endothelial Cells; Heme; Hemolytic-Uremic Syndrome; Humans; Mutation; P-Selectin; Protein Binding | 2013 |