halothane has been researched along with Hepatolenticular Degeneration in 2 studies
Hepatolenticular Degeneration: A rare autosomal recessive disease characterized by the deposition of copper in the BRAIN; LIVER; CORNEA; and other organs. It is caused by defects in the ATP7B gene encoding copper-transporting ATPase 2 (EC 3.6.3.4), also known as the Wilson disease protein. The overload of copper inevitably leads to progressive liver and neurological dysfunction such as LIVER CIRRHOSIS; TREMOR; ATAXIA and intellectual deterioration. Hepatic dysfunction may precede neurologic dysfunction by several years.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 2 (100.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Stevens, WC | 1 |
Eger, EI | 1 |
White, A | 1 |
Halsey, MJ | 1 |
Munger, W | 1 |
Gibbons, RD | 1 |
Dolan, W | 1 |
Shargel, R | 1 |
Read, AE | 1 |
1 review available for halothane and Hepatolenticular Degeneration
Article | Year |
---|---|
Immunological liver disease.
Topics: Acidosis, Renal Tubular; Autoimmune Diseases; Chemical and Drug Induced Liver Injury; Cholangiograph | 1973 |
1 other study available for halothane and Hepatolenticular Degeneration
Article | Year |
---|---|
Comparative toxicities of halothane, isoflurane, and diethyl ether at subanesthetic concentrations in laboratory animals.
Topics: Anesthesia, Inhalation; Anesthetics; Animals; Body Weight; Chemical and Drug Induced Liver Injury; D | 1975 |