halothane has been researched along with Cerebral Infarction in 14 studies
Cerebral Infarction: The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
| Excerpt | Relevance | Reference |
|---|---|---|
| " Although anesthesia by halothane via face-mask allowed better control of depth of anesthesia than chloral hydrate, we have found this method to be unsatisfactory due to insufficient control of ventilation and waste of anesthetic gases." | 3.71 | Anesthetic methods in rats determine outcome after experimental focal cerebral ischemia: mechanical ventilation is required to obtain controlled experimental conditions. ( Baethmann, A; Schmid-Elsaesser, R; Zausinger, S, 2002) |
| "In the short-duration groups, cerebral infarct volumes were not significantly different between anaesthetics (short-duration halothane: 288 (61) mm(3), mean (SD); short-duration desflurane: 269 (71) mm(3), P>0." | 1.32 | Desflurane affords greater protection than halothane against focal cerebral ischaemia in the rat. ( Ducouret, P; Gérard, JL; Haelewyn, B; Hanouz, JL; MacKenzie, ET; Roussel, S; Yvon, A, 2003) |
| "Halothane-pretreated animals were subjected to MCAO 24 hours after AP." | 1.31 | Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent. ( Dirnagl, U; Fütterer, C; Isaev, NK; Kapinya, KJ; Löwl, D; Maurer, M; Waschke, KF, 2002) |
| "Four groups received sham head trauma (surgical incision of the scalp but no trauma) and were treated with (A) nothing, (B) chronic ethanol (6% ethanol in drinking water for 40 days), (C) acute ethanol 1." | 1.30 | The influence of acute and chronic alcohol treatment on brain edema, cerebral infarct volume and neurological outcome following experimental head trauma in rats. ( Artru, AA; Donato, T; Lam, AM; Laohaprasit, V; Paez, A; Shapira, Y, 1997) |
| "Halothane is a strong inhibitor of potassium evoked spreading depression (SD) in cats." | 1.30 | Reduction of infarct volume by halothane: effect on cerebral blood flow or perifocal spreading depression-like depolarizations. ( Fujita, T; Graf, R; Heiss, WD; Hübel, K; Rosner, G; Saito, R, 1997) |
| "9%) was administered intravenously at a dosage of 1% of body weight immediately after suture removal following a 2-hour period of MCA occlusion." | 1.30 | Effect of delayed albumin hemodilution on infarction volume and brain edema after transient middle cerebral artery occlusion in rats. ( Belayev, L; Busto, R; Clemens, JA; Ginsberg, MD; Zhao, W, 1997) |
| "These results indicate that cerebral infarction induces bladder hyperactivity in awake rats and that urethan or MK-801 inhibits the development of this hyperactivity, most likely by blocking glutamatergic transmission in the brain." | 1.30 | Influence of anesthesia on bladder hyperactivity induced by middle cerebral artery occlusion in the rat. ( de Groat, WC; Namiki, M; Yokoyama, O; Yoshiyama, M, 1997) |
| "Ketamine was administered as a 50 mg/kg i." | 1.28 | Effects of ketamine on outcome from temporary middle cerebral artery occlusion in the spontaneously hypertensive rat. ( Baker, MT; Ridenour, TR; Todd, MM; Warner, DS, 1991) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (14.29) | 18.7374 |
| 1990's | 7 (50.00) | 18.2507 |
| 2000's | 4 (28.57) | 29.6817 |
| 2010's | 1 (7.14) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Lückl, J | 1 |
| Dreier, JP | 1 |
| Szabados, T | 1 |
| Wiesenthal, D | 1 |
| Bari, F | 1 |
| Greenberg, JH | 1 |
| Haelewyn, B | 1 |
| Yvon, A | 1 |
| Hanouz, JL | 1 |
| MacKenzie, ET | 1 |
| Ducouret, P | 1 |
| Gérard, JL | 1 |
| Roussel, S | 1 |
| JACOBSON, I | 1 |
| LAWSON, DD | 1 |
| Baldauf, K | 1 |
| Henrich-Noack, P | 1 |
| Reymann, KG | 1 |
| Shapira, Y | 1 |
| Lam, AM | 1 |
| Paez, A | 1 |
| Artru, AA | 1 |
| Laohaprasit, V | 1 |
| Donato, T | 1 |
| Saito, R | 1 |
| Graf, R | 1 |
| Hübel, K | 1 |
| Fujita, T | 1 |
| Rosner, G | 1 |
| Heiss, WD | 1 |
| Belayev, L | 1 |
| Busto, R | 1 |
| Zhao, W | 1 |
| Clemens, JA | 1 |
| Ginsberg, MD | 1 |
| Yokoyama, O | 1 |
| Yoshiyama, M | 1 |
| Namiki, M | 1 |
| de Groat, WC | 1 |
| Harukuni, I | 1 |
| Traystman, RJ | 1 |
| Bhardwaj, A | 1 |
| Koehler, RC | 1 |
| Kirsch, JR | 1 |
| Sarraf-Yazdi, S | 1 |
| Sheng, H | 1 |
| Brinkhous, AD | 1 |
| Pearlstein, RD | 1 |
| Warner, DS | 2 |
| Zausinger, S | 1 |
| Baethmann, A | 1 |
| Schmid-Elsaesser, R | 1 |
| Kapinya, KJ | 1 |
| Löwl, D | 1 |
| Fütterer, C | 1 |
| Maurer, M | 1 |
| Waschke, KF | 1 |
| Isaev, NK | 1 |
| Dirnagl, U | 1 |
| Ridenour, TR | 1 |
| Todd, MM | 1 |
| Baker, MT | 1 |
| Gelb, AW | 1 |
| Floyd, P | 1 |
| Lok, P | 1 |
| Peerless, SJ | 1 |
| Farrell, M | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Inhalatorial Sedation in Patient With SAH Versus Conventional Intravenous Sedation (GAS-SAH)[NCT00830843] | Phase 4 | 13 participants (Actual) | Interventional | 2009-01-31 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
14 other studies available for halothane and Cerebral Infarction
| Article | Year |
|---|---|
Peri-infarct flow transients predict outcome in rat focal brain ischemia.
Topics: Anesthesia; Anesthesia, Inhalation; Animals; Basal Ganglia; Blood Pressure; Brain Ischemia; Carbon D | 2012 |
Desflurane affords greater protection than halothane against focal cerebral ischaemia in the rat.
Topics: Analysis of Variance; Anesthetics, Inhalation; Animals; Blood Pressure; Brain Ischemia; Carbon Dioxi | 2003 |
THE EFFECT OF HYPERBARIC OXYGEN ON EXPERIMENTAL CEREBRAL INFARCTION IN THE DOG. WITH PRELIMINARY CORRELATIONS OF CEREBRAL BLOOD FLOW AT 2 ATMOSPHERES OF OXYGEN.
Topics: Anesthesia; Anesthesia, Inhalation; Atmosphere; Bis-Trimethylammonium Compounds; Blood Flow Velocity | 1963 |
Detrimental effects of halothane narcosis on damage after endothelin-1-induced MCAO.
Topics: Anesthetics, Inhalation; Animals; Brain Ischemia; Cerebral Infarction; Endothelin-1; Endothelium, Va | 2007 |
The influence of acute and chronic alcohol treatment on brain edema, cerebral infarct volume and neurological outcome following experimental head trauma in rats.
Topics: Administration, Oral; Alcoholism; Anesthesia, General; Animals; Brain; Brain Edema; Cerebral Hemorrh | 1997 |
Reduction of infarct volume by halothane: effect on cerebral blood flow or perifocal spreading depression-like depolarizations.
Topics: Anesthetics, Inhalation; Animals; Brain Ischemia; Cats; Cerebral Infarction; Cerebrovascular Circula | 1997 |
Effect of delayed albumin hemodilution on infarction volume and brain edema after transient middle cerebral artery occlusion in rats.
Topics: Anesthetics, Inhalation; Animals; Behavior, Animal; Brain Edema; Cerebral Arteries; Cerebral Infarct | 1997 |
Influence of anesthesia on bladder hyperactivity induced by middle cerebral artery occlusion in the rat.
Topics: Anesthesia, Inhalation; Anesthesia, Intravenous; Animals; Cerebral Arteries; Cerebral Infarction; Di | 1997 |
Intravenous basic fibroblast growth factor does not ameliorate brain injury resulting from transient focal ischemia in cats.
Topics: Analysis of Variance; Anesthetics, Inhalation; Animals; Brain; Brain Edema; Cats; Cause of Death; Ce | 1998 |
Effects of postischemic halothane administration on outcome from transient focal cerebral ischemia in the rat.
Topics: Anesthesia, Inhalation; Anesthetics, Inhalation; Animals; Body Temperature; Brain; Cerebral Cortex; | 1999 |
Anesthetic methods in rats determine outcome after experimental focal cerebral ischemia: mechanical ventilation is required to obtain controlled experimental conditions.
Topics: Acidosis, Respiratory; Anesthesia; Anesthetics; Animals; Brain; Brain Ischemia; Cerebral Infarction; | 2002 |
Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent.
Topics: Anesthetics, Inhalation; Animals; Body Temperature; Brain; Brain Infarction; Brain Ischemia; Cells, | 2002 |
Effects of ketamine on outcome from temporary middle cerebral artery occlusion in the spontaneously hypertensive rat.
Topics: Animals; Arterial Occlusive Diseases; Blood Pressure; Cerebral Arterial Diseases; Cerebral Arteries; | 1991 |
A prophylactic bolus of thiopentone does not protect against prolonged focal cerebral ischaemia.
Topics: Animals; Brain; Cats; Cerebral Infarction; Drug Therapy, Combination; Electroencephalography; Haloth | 1986 |