Page last updated: 2024-08-24

halofuginone and Hypertrophy, Left Ventricular

halofuginone has been researched along with Hypertrophy, Left Ventricular in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Arabacilar, P; Bantscheff, M; Bao, W; Bergamini, G; Bernard, RE; Del Rosario, J; Eisennagel, SH; Faelth Savitski, M; Force, TL; Gatto, GJ; Guo, Y; Halsey, WS; Hu, E; Hughes, AM; Joberty, G; Kirkpatrick, RB; Lal, H; Livi, GP; Lu, Q; Nayal, M; Olzinski, AR; Platchek, MC; Qin, P; Qu, XA; Quaile, MP; Rajpal, DK; Roethke, T; Sathe, GM; Schnackenberg, CG; Willette, RN; Wright, F; Xie, W	1

Other Studies

1 other study(ies) available for halofuginone and Hypertrophy, Left Ventricular

Article	Year
Activation of the Amino Acid Response Pathway Blunts the Effects of Cardiac Stress. Journal of the American Heart Association, 2017, May-09, Volume: 6, Issue:5 Topics: Amino Acids; Amino Acyl-tRNA Synthetases; Animals; Autophagy; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Enzyme Inhibitors; Fibroblasts; Fibrosis; Heart Failure; Humans; Hypertrophy, Left Ventricular; Induced Pluripotent Stem Cells; Male; Mice, Inbred C57BL; Myocytes, Cardiac; Piperidines; Protein Serine-Threonine Kinases; Protein Synthesis Inhibitors; Quinazolinones; Stress, Physiological; Time Factors; Ventricular Function, Left; Ventricular Remodeling	2017

Article

Year

Activation of the Amino Acid Response Pathway Blunts the Effects of Cardiac Stress.

Journal of the American Heart Association, 2017, May-09, Volume: 6, Issue:5

Topics: Amino Acids; Amino Acyl-tRNA Synthetases; Animals; Autophagy; Cells, Cultured; Disease Models, Animal; Dose-Response Relationship, Drug; Enzyme Inhibitors; Fibroblasts; Fibrosis; Heart Failure; Humans; Hypertrophy, Left Ventricular; Induced Pluripotent Stem Cells; Male; Mice, Inbred C57BL; Myocytes, Cardiac; Piperidines; Protein Serine-Threonine Kinases; Protein Synthesis Inhibitors; Quinazolinones; Stress, Physiological; Time Factors; Ventricular Function, Left; Ventricular Remodeling

2017