halofuginone has been researched along with Hyperplasia in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (50.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| DiRenzo, D; Goel, SA; Guo, LW; Kent, KC; Little, C; Roenneburg, D; Shi, XD; Takayama, T; Wang, B | 1 |
| Brown, DM; Callow, AD; Choi, ET; Ryan, US; Sehgal, NL | 1 |
| Aingorn, H; Genina, O; Miao, HQ; Nagler, A; Pines, M; Vlodavsky, I | 1 |
| Bona, C; McGaha, TL; Phelps, RG; Spiera, H | 1 |
4 other study(ies) available for halofuginone and Hyperplasia
| Article | Year |
|---|---|
Halofuginone stimulates adaptive remodeling and preserves re-endothelialization in balloon-injured rat carotid arteries.
Topics: Adaptation, Biological; Angiogenesis Inhibitors; Angioplasty, Balloon; Animals; Carotid Arteries; Carotid Artery Injuries; Cell Proliferation; Cells, Cultured; Collagen Type I; Endothelium, Vascular; Fibroblasts; Humans; Hyperplasia; Male; Models, Animal; Myocytes, Smooth Muscle; Organ Specificity; Piperidines; Postoperative Complications; Quinazolinones; Rats; Rats, Sprague-Dawley; Smad3 Protein; Vascular Remodeling | 2014 |
Halofuginone, a specific collagen type I inhibitor, reduces anastomotic intimal hyperplasia.
Topics: Anastomosis, Surgical; Animals; Blotting, Northern; Carotid Artery, Common; Cell Division; Cells, Cultured; Collagen; Dose-Response Relationship, Drug; Hyperplasia; Muscle, Smooth, Vascular; Piperidines; Procollagen; Quinazolines; Quinazolinones; Rabbits; RNA, Messenger; Tropoelastin; Tunica Intima | 1995 |
Inhibition of collagen synthesis, smooth muscle cell proliferation, and injury-induced intimal hyperplasia by halofuginone.
Topics: Animals; Arteries; Cattle; Cell Division; Cells, Cultured; Collagen; Humans; Hyperplasia; Muscle, Smooth, Vascular; Piperidines; Quinazolines; Quinazolinones; Rabbits; Tunica Intima | 1997 |
Halofuginone, an inhibitor of type-I collagen synthesis and skin sclerosis, blocks transforming-growth-factor-beta-mediated Smad3 activation in fibroblasts.
Topics: Animals; Cells, Cultured; Collagen; Collagen Type I; DNA-Binding Proteins; Fibroblasts; Gene Expression; Hyperplasia; Male; Mice; Mice, Inbred C57BL; Mice, Inbred Strains; Mice, Mutant Strains; Piperidines; Quinazolines; Quinazolinones; Receptors, Transforming Growth Factor beta; Sclerosis; Signal Transduction; Skin; Skin Diseases; Smad3 Protein; Trans-Activators; Transforming Growth Factor beta | 2002 |