halofuginone has been researched along with Becker Muscular Dystrophy in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (28.57) | 29.6817 |
2010's | 4 (57.14) | 24.3611 |
2020's | 1 (14.29) | 2.80 |
Authors | Studies |
---|---|
Evans, P; Genin, O; Halevy, O; Mordechay, S; Pines, M; Smullen, S | 1 |
Evans, P; Genin, O; Halevy, O; Mordechay, S; Pines, M; Wellner, G | 1 |
Barzilai-Tutsch, H; Bodanovsky, A; Genin, O; Guttman, N; Halevy, O; Levy, O; Pines, M | 1 |
Angelini, C; Genin, O; Halevy, O; Levi, O; Pines, M | 1 |
Barzilai-Tutsch, H; Bodanovsky, A; Halevy, O; Maimon, H; Pines, M | 1 |
Anderson, JE; Halevy, O; Huebner, KD; Jassal, DS; Pines, M | 1 |
McLoon, LK | 1 |
7 other study(ies) available for halofuginone and Becker Muscular Dystrophy
Article | Year |
---|---|
Differential Effects of Halofuginone Enantiomers on Muscle Fibrosis and Histopathology in Duchenne Muscular Dystrophy.
Topics: Animals; Disease Models, Animal; Fibrosis; Male; Mice; Mice, Inbred mdx; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Piperidines; Quinazolinones | 2021 |
Hydroxy group requirement for halofuginone-dependent inhibition of muscle fibrosis and improvement of histopathology in the mdx mouse model for Duchenne muscular dystrophy.
Topics: Animals; Disease Models, Animal; Fibrillar Collagens; Fibrosis; Humans; Male; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Muscle Fibers, Skeletal; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Phosphorylation; Piperidines; Quinazolinones; Signal Transduction; Smad3 Protein; Utrophin | 2019 |
Halofuginone improves muscle-cell survival in muscular dystrophies.
Topics: Actins; Animals; Apoptosis; bcl-2 Homologous Antagonist-Killer Protein; bcl-2-Associated X Protein; Cell Survival; Diaphragm; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Gene Expression Regulation; Humans; Macrophages; Mice; Mice, Inbred mdx; Mixed Function Oxygenases; Muscular Dystrophy, Duchenne; Myoblasts; Myofibrils; PAX7 Transcription Factor; Phosphatidylinositol 3-Kinases; Piperidines; Primary Cell Culture; Quinazolinones; Signal Transduction | 2014 |
Inhibition of muscle fibrosis results in increases in both utrophin levels and the number of revertant myofibers in Duchenne muscular dystrophy.
Topics: Animals; Biopsy; Child; Child, Preschool; Collagen; Collagen Type I; Gene Expression Regulation; Humans; Immunohistochemistry; Male; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Muscle Fibers, Skeletal; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Piperidines; Quadriceps Muscle; Quinazolinones; Utrophin | 2015 |
Halofuginone promotes satellite cell activation and survival in muscular dystrophies.
Topics: Animals; Apoptosis; Cell Cycle; Cell Survival; Male; MAP Kinase Signaling System; Mice; Mice, Inbred C57BL; Muscle Fibers, Skeletal; Muscular Dystrophies, Limb-Girdle; Muscular Dystrophy, Duchenne; Phosphatidylinositol 3-Kinases; Piperidines; Proto-Oncogene Proteins c-akt; Quinazolinones; Satellite Cells, Skeletal Muscle; Signal Transduction | 2016 |
Functional resolution of fibrosis in mdx mouse dystrophic heart and skeletal muscle by halofuginone.
Topics: Actins; Age Factors; Animals; Cell Proliferation; Collagen Type I; Collagen Type III; Diaphragm; Disease Models, Animal; Fibrosis; Heart; Hepatocyte Growth Factor; Mice; Mice, Inbred mdx; Muscle Strength; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Myocardium; Physical Endurance; Piperidines; Protein Synthesis Inhibitors; Quadriceps Muscle; Quinazolinones; Respiratory Mechanics; Running; Time Factors; Ventricular Function, Left | 2008 |
Focusing on fibrosis: halofuginone-induced functional improvement in the mdx mouse model of Duchenne muscular dystrophy.
Topics: Age Factors; Animals; Collagen Type I; Collagen Type III; Disease Models, Animal; Fibrosis; Heart; Mice; Mice, Inbred mdx; Muscle Strength; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Myocardium; Physical Endurance; Piperidines; Protein Synthesis Inhibitors; Quinazolinones; Respiratory Mechanics; Time Factors; Ventricular Function, Left | 2008 |