ha 1004 has been researched along with Lymphoma in 2 studies
N-(2-guanidinoethyl)-5-isoquinolinesulfonamide: structure given in first source
Lymphoma: A general term for various neoplastic diseases of the lymphoid tissue.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Further, we show that treatment of the lymphomas with HA1004, a serine/threonine protein kinase inhibitor, restores their susceptibility to ionomycin-induced apoptosis." | 1.31 | HA1004, an inhibitor of serine/threonine protein kinases, restores the sensitivity of thymic lymphomas to Ca2+-mediated apoptosis through a protein kinase A-independent mechanism. ( Cebrat, M; Kalas, W; Matuszyk, J; Strzadala, L, 2002) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kochel, I | 1 |
| Rapak, A | 1 |
| Ziolo, E | 1 |
| Strzadala, L | 2 |
| Matuszyk, J | 1 |
| Cebrat, M | 1 |
| Kalas, W | 1 |
2 other studies available for ha 1004 and Lymphoma
| Article | Year |
|---|---|
Nur77 nuclear import and its NBRE-binding activity in thymic lymphoma cells are regulated by different mechanisms sensitive to FK506 or HA1004.
Topics: Active Transport, Cell Nucleus; Animals; Cell Line, Tumor; Cell Nucleus; DNA-Binding Proteins; Dose- | 2005 |
HA1004, an inhibitor of serine/threonine protein kinases, restores the sensitivity of thymic lymphomas to Ca2+-mediated apoptosis through a protein kinase A-independent mechanism.
Topics: Animals; Apoptosis; Calcium; Cyclic AMP; Cyclic AMP-Dependent Protein Kinases; Cyclic GMP; Enzyme Ac | 2002 |