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ha 1004 and Cell Transformation, Neoplastic

ha 1004 has been researched along with Cell Transformation, Neoplastic in 1 studies

N-(2-guanidinoethyl)-5-isoquinolinesulfonamide: structure given in first source

Cell Transformation, Neoplastic: Cell changes manifested by escape from control mechanisms, increased growth potential, alterations in the cell surface, karyotypic abnormalities, morphological and biochemical deviations from the norm, and other attributes conferring the ability to invade, metastasize, and kill.

Research Excerpts

ExcerptRelevanceReference
" Protein kinase inhibitors block v-Src- and v-Fps-induced 9E3 gene expression with the same dose-response curves seen for PKC agonist-induced 9E3 gene expression."1.28Evidence that v-src and v-fps gene products use a protein kinase C-mediated pathway to induce expression of a transformation-related gene. ( Berg, KL; Foster, DA; Joseph, C; Qureshi, SA; Spangler, R, 1989)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19901 (100.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Spangler, R1
Joseph, C1
Qureshi, SA1
Berg, KL1
Foster, DA1

Other Studies

1 other study available for ha 1004 and Cell Transformation, Neoplastic

ArticleYear
Evidence that v-src and v-fps gene products use a protein kinase C-mediated pathway to induce expression of a transformation-related gene.
    Proceedings of the National Academy of Sciences of the United States of America, 1989, Volume: 86, Issue:18

    Topics: 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine; 8-Bromo Cyclic Adenosine Monophosphate; Animals; Bucl

1989