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gyki 52466 and Epilepsy, Temporal Lobe

gyki 52466 has been researched along with Epilepsy, Temporal Lobe in 2 studies

GYKI 52466: an AMPA (non-NMDA) receptor antagonist; structure given in first source

Epilepsy, Temporal Lobe: A localization-related (focal) form of epilepsy characterized by recurrent seizures that arise from foci within the TEMPORAL LOBE, most commonly from its mesial aspect. A wide variety of psychic phenomena may be associated, including illusions, hallucinations, dyscognitive states, and affective experiences. The majority of complex partial seizures (see EPILEPSY, COMPLEX PARTIAL) originate from the temporal lobes. Temporal lobe seizures may be classified by etiology as cryptogenic, familial, or symptomatic. (From Adams et al., Principles of Neurology, 6th ed, p321).

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Suzuki, F1
Heinrich, C1
Boehrer, A1
Mitsuya, K1
Kurokawa, K1
Matsuda, M1
Depaulis, A1
Domínguez, MI1
Blasco-Ibáñez, JM1
Crespo, C1
Nacher, J1
Marqués-Marí, AI1
Martínez-Guijarro, FJ1

Other Studies

2 other studies available for gyki 52466 and Epilepsy, Temporal Lobe

ArticleYear
Glutamate receptor antagonists and benzodiazepine inhibit the progression of granule cell dispersion in a mouse model of mesial temporal lobe epilepsy.
    Epilepsia, 2005, Volume: 46, Issue:2

    Topics: Animals; Benzodiazepines; Cell Count; Dentate Gyrus; Disease Models, Animal; Dizocilpine Maleate; Ep

2005
Neural overexcitation and implication of NMDA and AMPA receptors in a mouse model of temporal lobe epilepsy implying zinc chelation.
    Epilepsia, 2006, Volume: 47, Issue:5

    Topics: Animals; Benzodiazepines; Cell Death; Chelating Agents; Disease Models, Animal; Ditiocarb; Dizocilpi

2006