Page last updated: 2024-10-28

gyki 52466 and Benign Psychomotor Epilepsy, Childhood

gyki 52466 has been researched along with Benign Psychomotor Epilepsy, Childhood in 2 studies

GYKI 52466: an AMPA (non-NMDA) receptor antagonist; structure given in first source

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Suzuki, F1
Heinrich, C1
Boehrer, A1
Mitsuya, K1
Kurokawa, K1
Matsuda, M1
Depaulis, A1
Domínguez, MI1
Blasco-Ibáñez, JM1
Crespo, C1
Nacher, J1
Marqués-Marí, AI1
Martínez-Guijarro, FJ1

Other Studies

2 other studies available for gyki 52466 and Benign Psychomotor Epilepsy, Childhood

ArticleYear
Glutamate receptor antagonists and benzodiazepine inhibit the progression of granule cell dispersion in a mouse model of mesial temporal lobe epilepsy.
    Epilepsia, 2005, Volume: 46, Issue:2

    Topics: Animals; Benzodiazepines; Cell Count; Dentate Gyrus; Disease Models, Animal; Dizocilpine Maleate; Ep

2005
Neural overexcitation and implication of NMDA and AMPA receptors in a mouse model of temporal lobe epilepsy implying zinc chelation.
    Epilepsia, 2006, Volume: 47, Issue:5

    Topics: Animals; Benzodiazepines; Cell Death; Chelating Agents; Disease Models, Animal; Ditiocarb; Dizocilpi

2006