gq1b-ganglioside has been researched along with Mydriasis* in 6 studies
6 other study(ies) available for gq1b-ganglioside and Mydriasis
| Article | Year |
|---|---|
A case presenting electrophysiological and immunological characteristics of Fisher syndrome and Lambert-Eaton myasthenic syndrome.
Topics: Action Potentials; Aged; Asymptomatic Diseases; Ataxia; Autoantibodies; Blepharoptosis; Calcium Channels, P-Type; Calcium Channels, Q-Type; Diplopia; Electrodiagnosis; Electromyography; Female; Gangliosides; Humans; Hypesthesia; Lambert-Eaton Myasthenic Syndrome; Median Nerve; Miller Fisher Syndrome; Muscle Weakness; Mydriasis; Neural Conduction; Oculomotor Muscles; Tibial Nerve; Ulnar Nerve | 2021 |
Overlap of acute mydriasis and acute pharyngeal weakness associated with anti-GQ1b antibodies.
Topics: Gangliosides; Humans; Immunoglobulin G; Male; Mydriasis; Neural Conduction; Pharyngeal Diseases; Young Adult | 2018 |
Unique presentation of anti-GQ1b antibody syndrome.
Topics: Adolescent; Autoantibodies; Electromyography; Gangliosides; Humans; Magnetic Resonance Angiography; Male; Miller Fisher Syndrome; Mydriasis; Ophthalmoplegia; Spinal Puncture | 2016 |
[Mydriasis and velopharyngeal palsy as clinical signs of Miller-Fisher syndrome].
Topics: Gangliosides; Humans; Male; Middle Aged; Miller Fisher Syndrome; Mydriasis; Neurologic Examination; Paralysis; Pharyngeal Diseases | 2012 |
Acute bilateral mydriasis associated with anti-GQ1b antibody.
Miller Fisher syndrome (MFS) is an autoimmune neuropathy characterized by external ophthalmoplegia, ataxia and areflexia. Mydriasis is present in 35% of typical MFS. We report five patients with acute bilateral mydriasis, either isolated or associated with external ophthalmoplegia for which the presumed diagnosis of "atypical MFS" was confirmed by the positivity of anti-GQ1b antibodies. Acute bilateral mydriasis raises important differential diagnoses in clinical practice. This report demonstrates that acute mydriasis can be autoimmune mediated and that anti-GQ1b antibodies are useful to confirm the diagnosis in unexplained cases. Topics: Adolescent; Adult; Autoantibodies; Autoantigens; Female; Gangliosides; Humans; Male; Middle Aged; Miller Fisher Syndrome; Mydriasis; Ophthalmoplegia | 2010 |
Acute onset of a bilateral areflexical mydriasis in Miller-Fisher syndrome: a rare neuro-ophthalmologic disease.
Miller-Fisher syndrome (MFS) is characterized by variable ophthalmoplegia, ataxia, and tendon areflexia. It seems to be a variant of Guillain-Barré syndrome (GBS), but unlike in GBS, there is a primitive involvement of the ocular motor nerves, and in some cases there is brainstem or cerebellum direct damage. The unusual case of MFS in the current study started with a bilateral areflexical mydriasis and a slight failure of accommodative-convergence. Ocular-movement abnormalities developed progressively with a palsy of the upward gaze and a bilateral internuclear ophthalmoplegia to a complete ophthalmoplegia. In the serum of this patient, high titers of an IgG anti-GQ1b ganglioside and IgG anti-cerebellum. anti-Purkinje cells in particular, were found. The former autoantibody has been connected to cases of MFS, of GBS with associated ophthalmoplegia, and with other acute ocular nerve palsies. The anti-cerebellum autoantibody could explain central nervous system involvement in MFS. The role of these findings and clinical implications in MFS and in other neuro-ophthalmologic diseases are discussed. Topics: Accommodation, Ocular; Acute Disease; Autoantibodies; Cerebellum; Child; Convergence, Ocular; Dexamethasone; Gangliosides; Humans; Immunoglobulin G; Immunoglobulins, Intravenous; Male; Miller Fisher Syndrome; Mydriasis; Ophthalmoplegia; Plasmapheresis; Purkinje Cells | 2000 |