gq1b-ganglioside and Cognitive-Dysfunction

gq1b-ganglioside has been researched along with Cognitive-Dysfunction* in 2 studies

Other Studies

2 other study(ies) available for gq1b-ganglioside and Cognitive-Dysfunction

Article	Year
Ganglioside GQ1b ameliorates cognitive impairments in an Alzheimer's disease mouse model, and causes reduction of amyloid precursor protein. Scientific reports, 2019, 06-11, Volume: 9, Issue:1 Brain-derived neurotrophic factor (BDNF) plays crucial roles in memory impairments including Alzheimer's disease (AD). Previous studies have reported that tetrasialoganglioside GQ1b is involved in long-term potentiation and cognitive functions as well as BDNF expression. However, in vitro and in vivo functions of GQ1b against AD has not investigated yet. Consequently, treatment of oligomeric Aβ followed by GQ1b significantly restores Aβ Topics: Alzheimer Disease; Amyloid beta-Protein Precursor; Animals; Brain-Derived Neurotrophic Factor; Cells, Cultured; Cognitive Dysfunction; Disease Models, Animal; Gangliosides; Hippocampus; Mice, Transgenic; Neurons; Rats; tau Proteins; Up-Regulation	2019
Clinical Reasoning: A 35-year-old woman with diplopia, ataxia, and altered mental status. Neurology, 2018, 11-13, Volume: 91, Issue:20 Topics: Adult; Ataxia; Autoantibodies; Autoantigens; Autoimmune Diseases; Brain Stem; Cognitive Dysfunction; Diplopia; Encephalitis; Female; Gangliosides; Humans	2018

Article

Year

Ganglioside GQ1b ameliorates cognitive impairments in an Alzheimer's disease mouse model, and causes reduction of amyloid precursor protein.

Scientific reports, 2019, 06-11, Volume: 9, Issue:1

Brain-derived neurotrophic factor (BDNF) plays crucial roles in memory impairments including Alzheimer's disease (AD). Previous studies have reported that tetrasialoganglioside GQ1b is involved in long-term potentiation and cognitive functions as well as BDNF expression. However, in vitro and in vivo functions of GQ1b against AD has not investigated yet. Consequently, treatment of oligomeric Aβ followed by GQ1b significantly restores Aβ

Topics: Alzheimer Disease; Amyloid beta-Protein Precursor; Animals; Brain-Derived Neurotrophic Factor; Cells, Cultured; Cognitive Dysfunction; Disease Models, Animal; Gangliosides; Hippocampus; Mice, Transgenic; Neurons; Rats; tau Proteins; Up-Regulation

2019

Clinical Reasoning: A 35-year-old woman with diplopia, ataxia, and altered mental status.

Neurology, 2018, 11-13, Volume: 91, Issue:20

Topics: Adult; Ataxia; Autoantibodies; Autoantigens; Autoimmune Diseases; Brain Stem; Cognitive Dysfunction; Diplopia; Encephalitis; Female; Gangliosides; Humans

2018