goralatide has been researched along with Left Ventricular Hypertrophy in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Carretero, OA; Cingolani, OH; Liu, YH; Rhaleb, NE; Villanueva, M; Yang, XP | 1 |
| Brigstock, DR; Carretero, OA; Cavasin, MA; Peng, H; Rasoul, S; Rhaleb, NE; Sanchez-Mendoza, A; Zhuo, J | 1 |
| Bohnemeier, H; Cleutjens, JP; Crijns, HJ; Paul, M; Pinto, YM; Pokharel, S; Schunkert, H; Sharma, UC; Tian, XL; van Geel, PP | 1 |
| Carretero, OA; Ezan, E; Liu, YH; Mehta, D; Peng, H; Rhaleb, NE; Yang, XP | 1 |
4 other study(ies) available for goralatide and Left Ventricular Hypertrophy
| Article | Year |
|---|---|
Reduction of cardiac fibrosis decreases systolic performance without affecting diastolic function in hypertensive rats.
Topics: Animals; Capillaries; Cell Size; Collagen; Diastole; Fibrosis; Heart Ventricles; Hydroxyproline; Hypertension; Hypertrophy, Left Ventricular; Myocardium; Oligopeptides; Pressure; Rats; Rats, Inbred SHR; Rats, Inbred WKY; Systole | 2004 |
Antifibrotic effect of Ac-SDKP and angiotensin-converting enzyme inhibition in hypertension.
Topics: Angiotensin-Converting Enzyme Inhibitors; Animals; Blood Pressure; Captopril; Cell Division; Collagen; Connective Tissue Growth Factor; Drug Therapy, Combination; Fibrosis; Growth Inhibitors; Heart Rate; Hypertension; Hypertrophy, Left Ventricular; Immediate-Early Proteins; Intercellular Signaling Peptides and Proteins; Kidney; Macrophages; Male; Mast Cells; Monocytes; Myocardium; Myocytes, Cardiac; Oligopeptides; Rats; Rats, Sprague-Dawley; Transforming Growth Factor beta | 2004 |
Increased myocardial collagen content in transgenic rats overexpressing cardiac angiotensin-converting enzyme is related to enhanced breakdown of N-acetyl-Ser-Asp-Lys-Pro and increased phosphorylation of Smad2/3.
Topics: Activin Receptors, Type I; Angiotensin II; Animals; Animals, Genetically Modified; Binding, Competitive; Collagen; DNA-Binding Proteins; DNA, Complementary; Fibrosis; Humans; Hypertrophy, Left Ventricular; Myocardium; Oligopeptides; Peptidyl-Dipeptidase A; Phosphorylation; Protein Processing, Post-Translational; Protein Serine-Threonine Kinases; Rats; Rats, Sprague-Dawley; Receptor, Transforming Growth Factor-beta Type I; Receptors, Transforming Growth Factor beta; Smad2 Protein; Smad3 Protein; Substrate Specificity; Trans-Activators; Transforming Growth Factor beta; Transforming Growth Factor beta1 | 2004 |
Long-term effect of N-acetyl-seryl-aspartyl-lysyl-proline on left ventricular collagen deposition in rats with 2-kidney, 1-clip hypertension.
Topics: Animals; Blood Pressure; Cell Division; Collagen; Dose-Response Relationship, Drug; Heart; Heart Rate; Heart Ventricles; Hypertension, Renovascular; Hypertrophy, Left Ventricular; Macrophages; Male; Monocytes; Myocardium; Oligopeptides; Rats; Rats, Sprague-Dawley; Time Factors | 2001 |